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Postpartum period
6-8 weeks after birth
Vitals indicating hemmorage
Decreased blood pressure (orthostatic hypotension is normal)
Increased HR
Saturating a pad in 15 min
Postpartum neuropathy
Loss of sensation in lower limbs usually r/t positioning of the legs after birth
Hormone changes in the postpartum period
Estrogen - drops and can cause diuresis, breast engorgement, sweating
Progesterone - drops causing increased muscle tone in body
Placental enzyme insulinase - results in reduction of blood glucose
Low estrogen and progesterone trigger anterior pituitary to produce prolactin
Oxytocin coordinates and strengthens uterine contractions to prevent hemorrhage (breastfeeding triggers release)
Thyroid levels return to normal after 3 mo, postpartum thyroiditis is a concern
Cardiovascular and hematological systems postpartum
CO declines to prelabor levels within 1-2 hours postpartum
CO returns to prepregnancy levels within a few weeks
Varicosities resolve within 6 weeks
Blood volume returns to normal 6 weeks postpartum
Hypovolemic shock usually does not occur r/t normal blood loss (300-500mL) r/t expanded blood volume during pregnancy
Coagulation factors rise after birth and put pt at risk for DVT
GI and GU systems postpartum
constipation(hemorrhoids, pain from incisions, dehydration) use stool softener especially if opioid pain management in use
Mild proteinuria on day 1 and 2 and may last up to 6 weeks
Encourage voiding right after birth (needs to be within 6-8 hours) and should measure 150mL AT LEAST
Urinary retention may occur causing displacement of the uterus but postpartum diuresis usually occurs within 12 hours after birth (>3,000 mL/day)
Fundus postpartum
Should be firm (instruct pt to empty bladder, perform fundal massage)
Located between symphysis pubis and umbilicus immediately postpartum
Rises to at or below umbilicus 12 hours pp
Decreases in size by about 1-2 cm per day and should return between symphysis pubis and umbilicus by end of first week
At around 2 weeks it should be within the pelvis and not palpable
Documentation of lochia
Scant- 2.5 cm
Light - 10 cm
Moderate - 15cm
Heavy - saturated within 2 hours (similar to a heavy period up to 2 hours after birth)
flow may increase with standing and breastfeeding
Lactational amenorrhea
Suppression of ovulation during breastfeeding r/t prolactin
If breastfeeding, approx 6 months to first ovulation but depends on breastfeeding frequency, length of feeding, and infants suck
If not breastfeeding, approx 7-9 weeks (45-94 days) s/p birth ovulation begins and menses within 12 weeks
Hyperlactation vs hypolacation
Hyperlactation: excess mammary glandular tissue growth, overstimulation of the breasts or nipples
Hypolactation: having DM, PCOS or thyroid disorders.
Manifestations of PP depression
fatigue
insomnia
flat affect
lack of appetite
headache
anxiety, anger, sadness
Frequencies of vital signs after labor
Blood pressure every 15 min for the 2 hours
Temperature every 4 hours for the first 8 hours and then at least every 8 hours
If temp is elevated after first 24 hours - infection is possible
Assessing fundus nursing interventions
position patient supine with knees slightly flexed so fundal height is not influenced by positioning
Assess q8 hours
Cup one hand above the symphysis pubis and the other hand on the fundus to support and palpate the uterus and massage in a circular motion.
Observe lochia flow as fundus is palpated
Fundus may be deviated to the right if the bladder is full. It should be firm and midline after birth.
Engorgement vs mastitis
Engorgement: fullness of breast tissue
will resolve on own, wear supportive bra, cold packs (15 on, 45 off) or cabbage leaves
empty each breast after feedings, using a pump if needed
take a warm shower or apply warm compress before breastfeeding
Mastitis: infection in a milk duct
flu like symptoms
Behaviors indicating parent-newborn bonding
holds newborn face to face
assigns meaning to newborn behavior and views positively
Names the newborn
touches and maintaines proximity
responds to cries
Behaviors that indicate impaired sense of parents bonding
apathy when newborn cries
disgust when newborn voids, stools or spits up
turns away
does not include newborn in family context
perceives newborn as incorporative
Nutrition education for postpartum patients
non lactating patients should consume 1,800-2,200 cal/day
lactating patients should add 450-500 calories onto pre pregnancy diet
Continue prenatal vitamins until 6 weeks s/p birth
Immediate care for newborn
Suction mouth, then nose after cutting umbilical cord to establish respiratory function (suction at the side of the mouth)
Obligatory nose breathing is expected
Rub back and tap feet to stimulate breathing, apply O2 as needed
Check central skin color
Dry off quickly, provide a warm surface and cover
Normal vitals for a newborn
Done in the following order:
RR: 30-60 (2x adult)
short periods of apnea <15 seconds are OK
HR: 120-160 (can be as low as 80 during sleep) use apical for 1 full min (2x adult)
B/P: 60-80/40-50 (1/2 of adult)
can be done in all four extremities
Temp: 97.6-99.6
Newborn reflexes
Rooting - stroking cheek or edge of mouth causes newborn to turn head to that side and start to suck
Moro (startle) - allow head and trunk of newborn in a sitting position to fall backward at an angle of 30 degrees. The newborn should extend and then abdult the arms, elbows and fingers to form a C
Palmar grasp - Place fingers in newborns hand, the hand should close around the fingers
Babinski - stroking outer edge of foot causes newborns toes to fan upward and out
Plantar - place finger at base of newborns toes, the newborn should curl toes downward
Lanugo vs vernix
Lanugo: excess hair
Vernix: cheesy coating on newborn's skin, providing protection from amniotic fluid and aiding in thermoregulation.
Assessment of umbilical cord
3 vessels
two arteries, one vein
Elimination assessment in the newborn
First void by 24 hours
Should void 6 or more times per day
Meconium stool 24-48 hours
Newborn medications + vaccines
Hep B vaccine
Erythromycin eye ointment (inner to outer canthus in lower conjunctival sac)
Vitamin K injection to prevent hemorrhage in vastus lateralis
Don’t give vitamin K and hep B vaccine in same thigh
Umbilical cord and circ care
Remove clamp (clamp prevents hemorrhage) when dry at about 24 hours
Stump will fall off around 2 weeks of age
Clean the cord with water
Fold the diaper down and away from the umbilical stump
Don’t bathe by submersion until cord has fallen off
For circumcised babies, apply diaper loosely and change q4 hours and apply petroleum jelly
Bathing the newborn
first bath should be delayed for 8-24 hours
Bathe every other day
Use a mild soap only PRN
Do not give baby boys baths until circumcision is healed, but you can trickle warm water over the penis
Do not wash off yellowish mucus on glans of circumcised babies
Hypoglycemia in the newborn
can be r/t cold stress
s/s: jittery, hypotonia, unstable VS, weak or high pitched cry, poor feeding, apnea, respiratory distress, low temp, seizures
Blood glucose 40-45 mg or less = hypoglycemia
Take sample on outer heel edges
Newborn screenings
PKU sample taken at 24 hours, then another in 10-14 days
Congenital heart disease screening at 24 hours and 48 hours, tests oximetry and BPs on arm and foot x3 tests an hour apart (less than 3% difference between arm and leg is passing)
Hearing screening by 1 mo
Vision: newborn should be able to focus on objects 8-12 inches from face
Types of jaundice
Assess skin and sclera
Pathological : Over 10 days or very high bilirubin
Physiological: less than 24 hours
Kernicterus is a serious condition resulting from excessive bilirubin levels that can lead to permanent neurological damage.
Cold stress in newborns
newborns have a large surface area to weight ratio and reduced metabolism
newborn keeps warm by metabolizing brown fat
becoming chilled quickly uses up brown fat and can increase oxygen demand and cause hypoglycemia
check temperature axillary
Temp stabilizes within 12 hours after birth if chilling is prevented
S/s include: mottling, skin pallor, tachypnea
Individuals with disabilities act
free public education for kids with disabilities
requires IEP
adaptations, modifications for classrooms
address behavior issues
transition to adulthood
Characteristics of ASD
Decreased communication in social situations
abnormal communicative behaviors and body languages
Repetitive motor or speech patterns (flapping hands, rocking body)
Insistence on routine
Typically appears in first 3 years at puberty
Lacks empathy/trouble showing feelings
Lacks cooperation
Language delay
Self care deficits
Head banging, hand biting
Depression - flat affect
Withdrawal
Disregards social norms
Poor eye contact
Medication tx for ASD
2nd generation antipsychotics
SSRIs
Stimulants (for hyperactivity and impulsivity)
Naltrexone (off label use)
Characteristics of ADD
Impulsivity
Inattention, hyperactivity or combined
Inattention
unable to concentrate, easily distracted, short attention span
Unable to follow instructions
Difficulty with organization
Hyperactivity:
unable to sit still
fidgeting
excessive physical movement, talking, interrupting
Impulsivity:
acting without thinking
lack of regard for consequences
no sense of danger, frequent injuries.
Medication tx for ADD
Ritalin (increase dopamine and attention)
Adderall
Atomoxetine (SSRIs)
Clonidine (alpha 2 adrenergic agonist)
Guanfacine (alpha 2 adrenergic agonist)
Neurodevelopmental disorders
Disorders that affect the development of the brain and nervous system, impacting behavior, memory, and learning. Examples include autism spectrum disorder (ASD) and attention-deficit/hyperactivity disorder (ADHD).
often have onset in developmental period
impairments of personal, social, academic or occupational functioning
Harder to dx in younger children
Often improve as the patient gets older but can persist into adulthood
More s/s of ADD
behaviors at home and at school
often found before age 12
Low frustration tolerance
temper outbursts
poor school performance
low self esteem
difficulty taking turns
poor social boundaries
intrusive behaviors
disorganized
Easily bored
Not task oriented
Intellectual developmental disorder
A type of neurodevelopmental disorder
Severe deficits in three major areas of functioning
According to DSM-5-TR:
intellectual - mental capacity, learning, reasoning, problem solving
social - work, social activities, relationships
daily functioning - ADLs
Noted during child development
Examples include
Down Syndrome
Fragile X syndrome
PKU
Etiology of neurodevelopmental disorders
Combination of genetic, and environmental factors
Before birth (genetic conditions), insufficient dietary iodine, postnatal infections (rubella, HIV)
During birth (delivery complications)
Childhood head injury or infection
Exposure to toxins such as alcohol or heavy metals
Oxidative stress: balance of free radicals and antioxidant defenses is disturbed, which in a developing brain can result in impairment
Comorbidities in neurodevelopmental disorders
Conditions that often occur alongside neurodevelopmental disorders, such as anxiety, depression, or learning disabilities.
For example, in patients with ASD:
ADHD
Epilepsy
Psychiatric/behavioral concerns
GI disorders
Are common comorbidities, especially in ASD folks with intellectual disability
In patients with ADHD:
substance use disorders
sleep disorders
anxiety disorders
Types of neurodevelopmental disorders
Learning disorders - a deficiency that involves the way someone brain receives, processes, retains and responds to information (ie dyslexia). Often comorbid with ADHD population
Motor disorders - Tic disorders (vocal and motor),repetitive movement (rocking, grinding teeth) - often comorbid with ADHD, depression and OCD
Attention-Deficit/Hyperactivity Disorder - inattention, impulsivity - often comorbid with learning disorders
Autism Spectrum Disorders - deficits in social interaction + communications, repetitive and restricted patterns of behavior, interests or activities
Communication Disorders - language difficulties, vocabulary skill decificany, speech problems (stuttering, ect) - often comorbid with hearing impairment, intellectual disabilities, developmental disabilities ASD, ADHD, TBI
Intellectual Disabilities - deficits in intellectual and adaptive functioning (down syndrome, fragile X syndrome) - often comorbid with increased aging, alzheimer’s (DS)
Identifiable risk factors for neurodevelopmental disorders
ADHD
low birth weight
tobacco and alcohol use of mom
ASD:
De novo gene mutations
Family history
advanced parental age (combined with genetic factors)
very low birth weight (combined with genetic factors)
More common in boys than girls
Nursing interventions for ASD
Refer to early intervention programs at school
Educate parents to provide structure and consistent expectations
Utilize behavior reward system
PT, OT and ST as needed
Short, concise and developmentally appropriate communication
Nursing interventions for ADHD
Refer for family therapy to improve communication and coping skills.
Use cognitive behavior therapy to improve behavior patterns and develop problem-solving skills.
Music or art therapy school program.
Pharmacological intervention with stimulant medication, such as methylphenidate, to treat unresolved signs of restlessness and distractibility
Provide short and clear explanations
Pathophysiology of gene mutations
permanent change in the DNA sequence
Can affect a single DNA base pair or large segments of a chromosome
Changes the type/amount of protein made causing it to work incorrectly or not at all
Can be neutral, harmful or beneficial (evolution vs genetic disorders)
Genogram/Pedigree
A graphical representation that displays family relationships and medical history, often used in nursing to assess genetic risks and patterns of illnesses.
uses at least 3 generations
Looks for inherited genetic disorders rather than acquired (from replication errors or damage to DNA from toxins and is more individual)
Katrotype
Diagram of all the chromosomes in a person’s cells, arranged in pairs and ordered by size, shape and number
sample taken from blood, amniotic fluid, ect
22 pairs of autosomes
1 pair of sex chromosomes (XX female, XY male)
Genetic component of cystic fibrosis
Hereditary disorder acquired from two parents having mutations in the CFTR gene
Must inherit two copies of mutated (recessive) gene
White americans more likely to have
Faulty CFTR protein changes how much salt moves in/out of cells
Causes thick and sticky mucus and increases amount of salt in sweat
Affected organs include: lungs, liver, small intestine, reproductive system
Symptoms of CF
Thick mucus - can block and damage airways of lungs
Persistent cough (dry)
Exercise intolerance
Repeat lung infection
Inflamed nasal passages
Recurrent sinusitis
Clubbing of fingers, and toes, barrel chest r/t poor oxygenation
Fever, night sweats
Salty skin and extra salty sweat, tears, saliva (high Na and Cl)
Slow growth and shorter height
Can also clog digestive system
Bulky, frothy, greasy stools full of lipids
Meconium ileus in infants
Complications of CF
Bronchiectasis
Chronic infections
Hemoptysis
Pneumothorax
Exacerbations
Nutritional deficiencies especially fat soluble vitamins (ADEK)
DM related to pancreas damage
Liver disease
Infertility - especially in men
Osteoporosis
Electrolyte imbalance
Dehydration
Mental health concerns
Diagnostic testing and criteria for CF
Heel prick on card to test for immunoreactive trypsinogen as a newborn screening
Sweat test for high levels of chloride (ensure child is well hydrated) (can be done as soon as 48 hours s/p birth)
CFTR gene analysis
Interventions for CF
Airway clearance therapy (ACT) such as manual chest physiotherapy (2x per day, not after meals)
Aerosol therapy (bronchodilators) admin before ACT
Encourage aerobic exercise
Oxygen as prescribed
High protein, high calorie diet
Admin pancreatic enzymes within 30 min of eating a meal or snack
Adding salt to foods during hot months
Antibiotics for lung infection
Stay up to date on immunizations
Good dental hygiene
Have child rest prior to meals