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evolutionary psycs
study how natural selection influences behavior
heredity
how genes influence your behavior (nature)
environment
how outside situations influence your behavior (nurture)
Twin studies (genetics)
identical twin will have a higher percentage of also developing a disease.
twin studies (environment)
identical twins raised in diff environments show show differences
central nervous system
brain and spinal cord
peripheral nervous system
the rest of the NS - relays to central NS
somatic NS
voluntary movement, has sensory and motor neurons
autonomic NS
involuntary organs (heart, lungs, etc) (has sympathetic and parasympathetic NS)
sympathetic NS
fight/flight (generally activates- exception digestion)
parasympathetic NS
rest/digest (generally inhibits - exception digestion)
neuron
basic cell of the NS
dendrites
receive incoming NTs
axon
action potential travels down this
myelin sheath
speeds up action potential down axon, protects axon.
synapse
gap between neurons
sensory neurons
receive sense signals from environment - send signal to brain.
motor neurons
signals to move - send signals from brain
interneurons
calls in spinal chord/brain responsible for reflex arc.
reflex arc
important stimuli skips the brain and routes through the spinal cord for immediate reactions. (hand on hot flame)
GLIA
support cells - give nutrients and clean up around neutrons.
neurons fire with an AP
ions move across membrane sends an electrical charge down the axon
resting potential
neuron maintains a -70mv charge when not doing anything
depolarization
charge of neuron briefly switches from neg to pos - triggers the AP
threshold of depolarization
stimulus strength must reach this point to start the AP
All or nothing principle
it goes or it doesn’t. FIRE NOW OR NO FIRE.
refractory period
neuron must rest and reset before it can send another AP
neurotransmitters
chemicals released in the synaptic gap, received by neurons. Classified as excitatory or inhibitory
GABA
major inhibitory NT
glutamate
major excitatory NT (excites you)
dopamine
reward (short term) and fine movement - association with addiction (in hypothalamus)
seratonin
moods (long term) emotion, sleep (too little association with depression)
acetylcholine
memory and movement - in hippocampus (associated with alzheimers)
Norepinephrine
sympathetic NS - too little association with depression
endorphins
decrease pain
susbstance P
pain regulation (abnormality increases pain and inflammation)
oxytocin
love, bonding, childbirth, lactation
adrenaline
fight/flight
leptin
makes u full (stops hunger)
ghrelin
makes u hungry (turns you into a gremlin)
melatonin
sleep
agonist
drug that mimics NT
antagonist
drug that blocks NT
reuptake
unused NT are taken back up into sending neuron
reuptake inhibition
blocks reuptake
depressants
decrease NS activity (alcohol)
stimulants
increase NS activity (caffeine and cocaine)
hallucinogens
causes hallucinations and altered perceptions (marijuana)
opioids
relieves pain (heroin) (endorphin agonists)
cerebellum
movement, balance, coordination, procedural memory (walking a tight rope, balancing a ball)
Brainstem/Medulla
vital organs (HR, BP, breathing)
RAS
alertness, arousal, sleep, eye movement
cerebral cortex
outer portion of the brain – higher order thought processes
includes limbic system, lobes, corpus callosum
amygdala
emotions, fear
hippocampus
episodic and semantic memory (if you saw a hippo on campus you’d remember it)
hypothalamus
reward/pleasure center, eating behaviors – links endocrine system, homeostasis
thalamus
relay center for all but smell
pituitary gland
talks w/ endocrine system and hypothalamus – release hormones
occipital lobe
vision
frontal lobe
decision making, planning, judgment, movement, personality, language, executive function – includes the prefrontal cortex
prefrontal cortex
front of frontal lobe - map of motor receptors - controls skeletal movements
motor cortex
back of frontal lobe – map of our motor receptors – controls skeletal movement
parietal lobe
sensations and touch – controls association cortex – includes somatosensory cortex – map of our touch receptors
somatosensory cortex
map of our touch receptors
temporal lobe
hearing and face recognition, language
association areas
receive input from multiple areas to integrate info
broca’s area
ability to produce speech (telugu)
wernicke’s area
ability to comprehend speech (kannada)
corpus callosum
bundle of nerve fibers connective hemispheres
brain plasticity
brain changes via damage and through experiences
endocrine system
send hormones throughout the body
pituitary gland
controlled by hypothalamus. releases growth hormones.
EEG
shows brain activity (electrical output)
fMRI
show brain activity in specific regions, measure oxygen
lesion
destruction of brain tissue
multiple sclerosis
destruction of myelin sheath
myasthenia gravis
acetylcholine blocked, disrupts AP, causes paralysis and poor motor control
blindsight
lesions to primary visual cortex
prosapagnosia
face blindness - damage to occipital or temporal lobe
aphasia
cant comprehend or speak speech
phantom limb pain
pain from a limb that is no longer there
epilepsy
seizures (too much or little clutamate/GABA)
alzheimers
memory loss (destruction of acetylcholine in hippocampus)
REM rebound
after sleep disruptions or lack of REM sleep, you’ll have more intense REM sleep.
activation synthesis
brain produces random bursts of energy – stimulating lodged memories in limbic sys & brain stem. Dreams start random then develop meaning
consolidation theory
brain is combining and processing memories for storage
sensation
receive stimulus energy from environment
transduction
convert that info into APs
perception
brain interprets info
absolute threshold
detection of signal 50% (is it there)
JND
can tell the difference b/w a stronger & weaker stimulus or two similar things (coke vs pepsi, did it get darker?)
weber’s law
two stimuli must differ by a minimum proportion (the stronger the stimulus, the more you have to add to tell the difference)
synesthesia
disorder where your senses blend
lens
focuses light on retina
retina
contains photoreceptors
fovea
area of best visions (cones here)
rods
black/white, dark adaptation; way more rods than cones, located along sides of retina
cones
color, bright light (red, green, blue) – only in the fovea
ganglion cells
create optic nerve (opponent process theory happens here)
blind spot
occurs where the optic nerve leaves the eye