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What is the largest organ of the body
The skin
What does the structure/ function of the skin do?
Guards the body from environmental stress
Adapts to environmental influences
What are the 2 layers of the skin
Epidermis and Dermis
What is beneath the 2 layers of the skin?
subcutaneous layer of adipose tissue
What are the functions of the skin?
Communication/ Protection
Prevents penetration, Perception, Temp. Regulation, Identification, wound repair (cools us down), Absorption/ Excretion, Production of Vitamin D
Subjective Data: Health History Questions
Past history, Change in pigmentation, change in mole (size color, Excessive dryness or moisture
Pruritus (itching), Excessive bruising, Rash or lesion, Medications, Hair loss, Change in nails
Environmental or occupational hazards (where they work or live)
Patient-Centered care: Do you perform a skin-self examination?
Objective Data: The Physical Exam
Preparation: Skin characteristics
Equipment needed: Strong direct lightening, small centimeter ruler, penlight, gloves
What are special procedures done on the Physical exam?
Wood’s light: UV light filtered through a special glass; detects fluorescing lesions; Lesions with blue-green fluorescence indicates fungal infection
Light magnifier
What is Capillary Refill (peripheral Circulation):
With the index finer or middle fingertip at heart level, depress the nail edge at least 5 seconds to blanch and then release, noting the return of color
What is does normal capillary refill show?
Normally color return is instant or at least within a few seconds (1-2) in a cold environment
What does abnormal capillary refill do?
Cyanotic nail beds or sluggish color return >3 seconds indicated clinical deadline (cardiovascular or respiratory failure, septic shock)
Objective Data: They Physical Exam
Complete Physical Examination:
Learn to consciously assess or attend to surface skin characteristics
Don’t ignore- skin’s information about circulation, nutritional status, and signs of systemic diseases and topical data (things they have been exposed to)
Know the person’s normal/baseline skin coloring and self-monitoring practices
Separate intertriginous areas (skinfolds) and inspect them (always remove socks)
What does regional examination do?
Individual seeks care because of changes
Assessment- Focused on skin alone
Inspection and Palpation
Temperature:
Hypomeia: (above-hot)
Hypermeia: (below-cool)
Moisture:
Diaphoresis (sweating profusely)
Dehydration (check skin turgor)
Texture and Thickness
Color:
General pigmentation:
Freckles, mole (nevus), birthmarks
Widespread color change
Pallor, Erythema, Cyanosis, Jaundice
Skin Color:
Pallor:
When the red-pink tones from the oxygenated hemoglobin in the blood are lost, the skin takes on the color of connective tissue (collagen) which is mostly WHITE
Skin Color: Erythema
Intense redness of the skin from excess blood (hyperemia) in the dilated superficial capillaries
Skin Color: Cyanosis
Bluish mottled color from decreased perfusion; the tissues have high levels of deoxygenated blood
Skin Color: Jaundice
A yellowish skin color indicates rising amounts of bilirubin in the blood
Inspection and Palpation
Edema:
Unilateral vs. Bilateral
Pitting Edema
Anasarca
Mobility and Turgor:
Assess skin elasticity
Poor turgor (INFANTS)
Scleroderma: autoimmune disorder-changes skin like stone
Inspection and Palpation: Lesions
Traumatic or pathologic changes
Note the color
Elevation: flat, raised, or pedunculated
Pattern or shape
Size: In centimeters (sm)
Location and distribution on the body: generalized or localized
Common Skin Lesions:
Primary Contact Dermatitis:
A rash; one area- where it was touched
Common Skin Lesions:
Allergic Drug Reaction and Tinea Versicolor
Common Skin Lesions: Tinea Corporis (Ringworm of the BODY):
fungal infection; scalp or on the body
Common Skin Lesions: Tinea Pedis: (Ringworm of the FOOT)
athletes foot
Common Skin Lesions: Psoriasis
Systemic condition in the folds
Common Skin Lesions: Herpes Zoster (Shingles):
Extremely painful- runs along pattern
Common Skin Lesion: Erythema Migrants of Lyme Disease:
Localized area of erythema
Common Skin Lesions: Labial Herpes Simplex (Cold Sores):
Usually apparel on the mouth
Lesions: Abnormal characteristics of pigmented lesions are summarized by
ABCDEF
ABCDEF: What does A stand for?
Asymmetry: Not regularly round or oval; two halves of lesion don’t look the same
ABCDEF: What does B stand for?
Border Irregularly: Notching, scallpoing, ragged edges, poorly defined margin; Important for pressure ulcers
ABCDEF: What does C stand for?
Color Variation: Areas of brown, tan, black, blue, red, white, or combination
ABCDEF: What does D stand for?
Diameter: Greater than 6 mm (size of pencil eraser)
ABCDEF: What does E stand for?
E: Elevation or Evolution
ABCDEF: What does F stand for?
Funny looking: “ugly duckling” sign- the suspicious lesion stands out as looking different compared with its neighboring nevi
Common Shapes and Configurations of Lesions:
Annular or Circular:
Beings in center and spreads to periphery
Common Shapes and Configurations of Lesions: Confluent:
Lesions run together
Common Shapes and Configurations of Lesions: Discrete
distinct and separate
Common Shapes and Configurations of Lesions: Grouped
Cluster of lesions
Common Shapes and Configurations of Lesions: Gryate
twisted, coiled, or snakelike
Common Shapes and Configurations of Lesions: Target or iris
resembles iris of eyes, concentric rings
Common Shapes and Configurations of Lesions: Linear
Scratch, streak, line (scabies), or stripe
Common Shapes and Configurations of Lesions: Polycytic
Annular lesion grow together
Common Shapes and Configurations of Lesions: Zosteriform
Linear arrangement following a unilateral nerve route
Primary Lesions
Macule:
Flat, color change; Ex: freckles
Primary Lesions: Papule
Solid, less than 1 cm; Ex: moles
Primary lesions: Patch
Macules that are larger than 1 cm; Ex: Measles
Primary lesions: Plaque
form surface elevation wider than 1 cm- Disk-shaped lesion; Ex: sclerosis
Primary lesions: Nodule
Solid, elevated, hard or soft, larger than 1 cm; xanthoma
Primary lesions: Tumor
deeper into dermis, may be benign or malignant; Lipoma, hemangioma
Primary lesions: Wheal
Superficial, raised, irregular shape; mosquito bite/ allergic reaction
Primary lesions: Urticaria (Hives)
Whereas coalesce to form extensive reaction
Primary lesions: Vesicle
Elevated cavity containing free fluid (blister); herpes simplex, chicken pox
Primary lesions: Bulla
larger than 1 cm, superficial in epidermis; Ex: friction blister, burns, contact dermatitis
Primary lesions: Cyst
Encapsulated fluid filled cavity in dermis; Ex: sebaceous cyst
Primary lesions: Pustule
Turbid fluid (pus)’ Ex: Impetigo acne
Secondary Skin Lesion
Debris on skin surface: Crust
Thickened dried out exudate
Secondary Skin Lesion
Debris on skin surface: Scale
Compact flakes on desiccated skin from shedding of dead excess keratin cells
Secondary Skin Lesion: Break in continuity of skin surface
Fissure:
Linear crack with abrupt edges extending into dermis
Secondary Skin Lesion: Break in continuity of skin surface: Erosin
Scooped Out But Shallow Depression
Secondary Skin Lesion: Break in continuity of skin surface: Ulcer
Deeper depression extending into dermis with irregular shape, may bleed, leaves scare
Secondary Skin Lesion: Break in continuity of skin surface: Excoriation
Self-inflicted abrasion that is superficial
Secondary Skin Lesion: Break in continuity of skin surface: Scar
Permanent fibrotic change after healing
Secondary Skin Lesion: Break in continuity of skin surface: Atrophic Scar
Resulting skin level is depressed with loss of tissue and thinning
Secondary Skin Lesion: Break in continuity of skin surface: Lichenification
Prolonged intense scratching leads to thickened skin producing tightly packed set of papules
Secondary Skin Lesion: Break in continuity of skin surface: Keloid
Benign excess of scar tissue beyond original injury
Pressure Injuries (Pressure Ulcer, Decubitus Ulcer)
Pressure Injuries (PIs) appear on the skin over a bony prominence when circulation is impaired
Immobilization impedes delivery of blood/oxygen/nutrients to skin and venous drainage. This results in ischemia and cell death
Risk factors:
Impaired immobility, thin fragile skin of aging, decreased sensory perception, impaired LOC, moisture from urine/ stool incontinence, shearing injury, poor nutrition and infection
What are common sites for pressure injuries?
Back- Sacrum, heel, elbow, scapula, vertebra
Side: Ankle, knee, hip, run, shoulder
What are the Pressure Injury Stages?
Stage 1: Non-Blanchable erythema (stays red)
Stage 2: Partial-thickness skin loss
Stage 3: Full-thickness skin loss (stay into dermis)
Stage 4: Full- thickness skin/tissue loss (ligaments)
Deep Tissue Pressure Injury (DTPI)
PI Caused by Medical Device
Vascular Lesions
Hemangiomas:
Caused by a benign proliferation of blood vessels in the dermis
Port-Wine Stain (Nevus Flammeus)
Strawberry Mark (Immature Hemangioma)
Cavernous Hemangioma (Mature)
Vascular Lesions: Telangiectasia
Caused by vascular dilation
Telangiectasia
Spider or Star Angioma
Venous Lake
Vascular Lesions: Purpuric Lesions:
caused by blood flowing out of breaks in the vessels. RBCs and blood pigments are deposited in the tissue (extravascular). Difficult to see in dark-skinned people
Petechiae
Ecchymosis: Bruising that came from the inside out
Purpura
Contusion (Bruise): Only one that is caused by Blunt Trauma
Malignant Skin Lesions
Basal Cell Carcinoma (flat then raised)
Squamous cell Carcinoma
Malignant Melanoma (tanned, black)
Developmental Competence: Infants and Children
Temperature regulation is not effective
Developmental Competence: Infants and Children- Normal Findings
Mongolian Spot, Cafe au lait spot (birthmark), Physiologic jaundice, Milia, port Wine stain
What is the most common skin problem in adolescents?
Acne
Severe acne includes papules, pustules, and nodules
What is turgor
Poor turgor or “tenting” indicates dehydration, especially when combined with delayed capillary refill and tachypnea; also occurs with malnutrition
Where do you test for turgor and skin mobility in infants?
Over the abdomen
Common Skin Lesions in Children
Diaper dermatitis, Intertrigo (candidiasis), Impetigo, Atpoic dermatitis (eczema), Measles (rubeola), German measles (rubella), Chickenpox (varicella)
Developmental Competence: Pregnant Women
Sweat and sebaceous glands increase secretion
Expected skin color changes due to increased hormone levels
Developmental Competence: Pregnant Women- Normal Findings:
Striae “stretch marks”, Linea nigra, Chloasma (butterfly across face), Vascular Spiders (Spider angioma)- Varicose veins
Developmental Aging Adult:
Loss of elastin, collagen, subcutaneous fat, and reduction of muscle tone
Epidermis thins and flattens > wrinkling occur
Aging skin increase risk for pressure ulcers
Developmental: Aging Adult- Normal Findings
Senile lentigines (liver spots), Seborrheic Keratosis, Actinic keratosis
Cultural and Genetics:
Genetic attributes of dark-skinned individuals afford protection against skin cancer due to melanin
Increased likelihood of skin cancer in whites than in black and Hispanic populations
Succession of genetic mutations leading to increased chromosome sensitivity to sun damage
What is hair?
Threads of Keratin
Shaft, Root, Bulb Matrix, Hair follicle
What are the two types of hair?
Vellus hair and Terminal Hair
Inspection of Hair:
Color
Hair color comes from melanin production
Vary from pale blond to total black
Graying begins as early as the 30s b/c of reduced melanin production in the follicles
Genetic factors affect the onset of graying
Inspection of Hair: Texture
Note dull, coarse, or brittle scalp of hair
Tinea Capitis: Gray, scaly, well-defined areas with broken hairs (baby's- “milk spots”)
Loss of eyebrows and scalp hair (expected) with chemotherapy or hypothyroidism
Palpation of the Hair:
Distribution:
Fine vellus hair coats the body
Coarser terminal hair grows at eyebrows, eyelashes, and scalp
Puberty- coarse curly hairs
Absent or sparse genital hair suggests endocrine abnormalities
Hirsutism
Palpation of the Hair: Lesions
Separate the hair into sections and lift to observe the scalp
Should be clean and free of any lesions or prest inhabitants
Lice- head or pubic areas
Abnormal Hair Conditions
AIDS- Related Kaposi sarcoma: patch stage
Toxic alopecia, Traction alopecia, hirsutism, Furuncle and abscess
Tinea Capitis (scalp ringworm), Seborrheic Dermatitis (cradle cap), Pediculosis Capitis (head lice), Folliculitis Barbae (“razor bumps”)
What are Nails?
Hard plates of keratin
Clear with longitudinal ridges
Pink color
Inspection of the Nails:
Shape and Contour
The profile sign
Clubbing of nails- occurs with pulmonary diseases, lung cancer, and congenital cyanotic heart disease
Consistency, Color
Abnormal Conditions of the Nails:
Scabies Paronychia, Beau Line, Splinter Hemorrhages, Onychomycosis, Late Clubbing, Pitting, Habit-tic dystrophy
Health promotion and Teaching:
Educate on the dangers of excessive UV exposure from indoor tanning equipment
Sun lamps, beds, booths
Long- term tanning can lead to something more frightening and deadly
Increased risk of melanoma
Increased risk of non-melanoma skin CA (squamous and basil cell carcinoma)
“Tan Tax”- 10% tax on use of YV indoor tanning
Teach Skin self-examination