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what is the purpose of a cerebral angiogram
done in order to visualize the cerebral blood vessels
what are the indications for cerebral angiogram
to identify aneurysms, defects, obstructions, or vascular malformations
what must be assessed prior to cerebral angiogram
consent, shell fish allergy due to contrast dye, any anticoagulant use, BUN/creatinine to assess kidney function in order to excrete dye
how is a cerebral angiogram preformed
catheter inserted into groin or neck, use iodine, need to hold still, monitor pulses, will use fluoroscopy and x rays to take pictures after dye is injected
what are the important post procedure points with cerebral angiograms
lay flat in bed for 12-14 hours, monitor puncture site every hour, if bleeding hold pressure, neuro circulatory checks on lower extremities, whatever leg was used immobilize puncture site for 6-8 hours, force fluids after to flush out dye
what is the purpose of a CAT (cerebral CT) scan
with other without dye, done to visualize components in brain + head, painless
what are the indications for a CAT scan
see if hemorrhage, visualize an infarction or stroke abscess or tumor in cerebral cavity
what are the important pre procedure points with a CAT scan
educate patient that if contrast dye is used they may feel a flush or warm face, metallic taste in mouth, consent, shellfish or iodine allergies
what should you do post CAT scan
encourage fluids to flush out dye
what is the purpose of an electroencephalography (EEG)
painless procedure to record electrical activity of brain
what are the indications for an EEG
seizures
what must happen prior to an EEG
pt may need to stay awake night before, no caffeine or stimulants 24 hours prior, no anti seizure or stimulant meds 24-48 hours prior
how is an EEG preformed
takes about 1 hour, electrodes placed on scalp, may be asked to hyperventilate for 3-4 minutes to increase electrical activity or bright light exposure
what happens post procedure with an EEG
help remove paste from hair and get electrodes off, administer anti seizure meds and watch for seizure activity
what is the purpose of the Glasgow coma scale
determines neurological function and level of consciousness in a numeric scale
what are the indications for the Glasgow coma scale
patients with head injuries, brain tumors, or encephalitis
what are the categories of the Glasgow coma scale
best eye response (1-4), best verbal response (1-5), best motor response (1-6)
what is the purpose of a lumbar puncture
needle inserted to obtain specimen of cerebral spinal fluid, relieve pressure, or inject meds like chemo
why is informed consent important with a lumbar puncture
rare, but serious risk of brain herniation if there is increased intracranial pressure
what are the indications for lumbar puncture
MS, meningitis, infections, cancers
what happens pre procedure with a lumbar puncture
help patient void prior and get into position
how is a lumbar puncture preformed
place in lateral recumbent metal at edge of bed, position so spine is stretched, local anesthetic used, physician can use monometer to test procedure of CSF
what is important to monitor after a lumbar puncture
monitor puncture site, neuro assess every 15-30 minutes until stable, lay flat for several hours afterwords, increase fluids to 3000 mL, if headache can give oral analgesic, if persists dr can do epidural blood patch to seal hole, monitor for bleeding or drainage
what is the purpose of an MRI
use magnets to visualize cross sectional images of head
what are the indications of an MRI
need to discriminate soft tissue from tumor or bone
what is important preprocedure with an MRI
assess shellfish/iodine allergy due to contrast dye, remove all jewelry, NPO 4-8 hours if getting sedation due to claustrophobia, assess for any metal implants like pacemaker
what is the purpose of intracranial pressure monitoring
device inserted into cranium that records pressure and is connected to monitor to show pressure waveforms
what are the indications for intracranial pressure monitoring
comatose pt in ICU with head injury
what happens pre procedure with intracranial pressure monitoring
head shaved and cleansed
how is intracranial pressure monitoring preformed
ventriculostomy; intraventricular fluid filled catheter inserted into ventricles through burr holes, subarachnoid screw or bolt placed. epidural or subdural fibrotic sensor; through burr hole and less invasive
what is important post intracranial pressure monitoring
serious life threatening risk for infection, monitor site, cover with sterile dressing + always preform sterile technique, watch for signs of increased intracranial pressure on monitor (greater than 15 mmHg), vital signs every hour
damage to nerves related to long term high glucose and low insulin
diabetic neuropathy
what are the different types of diabetic neuropathy
peripheral- most common in lower extremities then upper; can lead to ulcers
autonomic- all the nerves
proximal- thigh, hips, butt, and legs, one area but bilaterally
focal- damage specific nerve in face, torso, and legs
what are the symptoms of diabetic neuropathy
burning and stabbing pain, numbness, tingling, may cause safety issues and functional impairments
what are the resulting issues from diabetic neuropathy
balance, ability to recognize pain, ability to handle ADL tasks which require strength and dexterity
what is the treatment for diabetic neuropathy
long term control of blood glucose most important to diminish damage to the nerves
what medications are used for diabetic neuropathy
anticonvulsants (gabapentin, pregabalin), antidepressants, topical treatment (capsaicin) for focal neuropathy, lidocaine injection, vitamin supplements
chronic, degenerative motor system disorder caused by the loss of dopamine producing brain cells
parkinson’s disease
what are the clinical manifestations of parkinson’s disease
tremor at rest, rigidity/stiffness, bradykinesia (slow movement), postural instability, mask like face, shuffling gait, pill rolling tremor in fingers, insomnia, depression/anxiety/Lewy body dementia, weakness, autonomic dysfunction, slow speech
what is used to diagnose parkinson’s
neuro exam, unified parkinson’s disease rating scale, and levodopa test (check UPDRS, take levodopa, check UPDRS again)
what is the treatment for parkinson’s disease
carbidopa-levidopa; take 1 hour before or 2 hours after meals, evenly spaced, cam cause dyskinesia, N/V, dizziness, hypotension, can cause on/off response causing mobile/active one moment then slow/rigid the next
what other medications supplement carbidopa-levidopa in parkinson’s treatment
anticholinergics and antihistamines for tremor/rigidity, dopamine agonists, antiviral drug (amantadine), MAO-B inhibitors, COMT inhibitors
what are the complications from parkinson’s disease
parkinsonian crisis-severe W/D from meds or emotional upset (anxiety, sweating, tachycardia, tachypnea)
aspiration pneumonia, altered cognition, falls
what are the patient education points with parkinson’s disease
prevent constipation by increasing fluids and fiber, support mobility by encouraging exercise, ROM, good posture, rest periods, extra time for ADLs, home safety, elevated toilet seat, hand rails, velcro vs. buttons, reduce stress
what are other treatment options for parkinson’s disease
deep brain stimulation (implanted electrical device stops tremor), stereotactic palliodotomy (surgeon destroys brain tissue that causes tremor, mostly replaced by DBS), controversial= fetal tissue transplant and stem cells
a spouse of a husband who has parkinson’s explains to you that her husband experiences episodes while walking where he freezes and can’t move. she asks what can be done to help with these types of episodes to prevent injury. select all that apply
a. have the husband try to change the direction of movement by moving in the opposite direction when the freeze ups occur
b, use a cane with a laster point while walking
c. have the husband try to push through the freeze ups
d. encourage the husband to consciously life the legs while walking
a., c., d.
you’re caring for a patient with parkinson’s disease who has tremors. select the option that is incorrect about tremors experienced in this disease.
a. the tremors are most likely to occur with purposeful movements
b. a common term used to describe the tremors in the hands and fingers is called pill rolling
c. tremors are one of the most common signs of parkinsons
d. tremors in this disease can occur in the hands, fingers, arms, legs, and even the lips and tongue
a.
youre providing diet education to a patient with parkinson’s disease. which statement below demonstrates the patient understands your teaching? select all that apply
a. I will limit foods high in fiber like fruits and vegetables
b. I will be sure to drink 2 L of fluid per day
c. it is very common for me to experience diarrhea with this disease
d. I will avoid taking carbidopa/levodopa with a protein rich meal
b., d.
chronic demyelinating disease producing plaques on the myelin sheath of neurons in the CNS
multiple sclerosis
who most commonly gets MS
onset occurs between 25-35 y.o women, caucasian living in colder climates of North America and Europe
what are the 4 typical courses of MS
progressive relapsing (steady decline since onset with super-impacted attacks), secondary progressive (initial relapsing that suddenly begins to have decline without periods of remission), primary progressive (steady increase in disability without attacks), relapsing remitting (most common, unpredictable attacks which may or may not leave permanent deficits followed by periods of remission)
what are the clinical manifestations of MS
fatigue, visual disturbances, dysarthria (trouble speaking), ataxia (impaired coordination), spasticity, muscle weakness, sensory changes, pain, paresthesia, bladder/bowel dysfunction, sexual dysfunction, dysphagia, mood changes, cognitive impairment
MS is diagnosed by — of other disorders
exclusion
what are the pharmacological treatments of MS
immune suppressants and modulators, interferon beta (sc every other day), corticosteroids, glatiramer (remitting relapsing MS- daily SQ), azathioprine, cyclosporine
what are the symptomatic treatments for MS
antispasmotics- baclofen, tizanidine
anticholinergics for bladder- oxybutynin
clonazepam for anxiety/tremor
what are the non pharmacologic treatments for MS
exercise and ROM to minimize contractures and spasticity; eye patches for diplopia, communication boards, refer to speech therapy for dysphagia, energy conservation, skin and falls prevention
which finding below represents a positive Romberg sign in a patient with MS?
a. the patient reports dark spots in the visual fields during the confrontation visual field test
b. when the patient closes the eyes and stands with their feet together they start to lose their balance and sway back and forth
c. the patient’s sign and symptoms increase when exposed to hot temperatures
d. the patient reports an electric shock feeling when the head and neck are moved downward
b.
a patient is receiving interferon beta for treatment of multiple sclerosis. as the nurse you will stress the importance of?
a. physical exercise to improve fatigue
b. low fat diet
c. hand hygiene and avoiding infection
d. reporting ideation of suicide
c.
what is the most common cause for generalized paralysis from virus such as mono, cold/flu, can be triggered by flu vaccine
Guillain-barre
an acute inflammatory demyelinating polyneuropathy affecting the peripheral nervous system (temporary)
Guillain-barre
what are the hallmarks of guillain-barre
characteristic ascending weakness, maximum deficit within 4 weeks, can involve paralysis of respiratory system, 3 phases (initial/acute, plateau, recovery), usual recover within 6 months and up to 2 years, excellent prognosis
how is guillain-barre diagnoses
history and clinical presentation, CSF examination, EMG
what is the medical treatment for guillain-barre
considered a medical emergency, focus of therapy is supportive care, plasmapheresis, IVIG to reduce antibodies
what are the nursing management points with guillain-barre
vital signs, respiratory and swallowing assessments, I&O’s (SIADH in 26% patients- hyponatremia + hypoosmolarity), mobility, pain, preventing complications
autoimmune disease relating to loss of acetylcholine receptors in the post-synaptic neurons of the neuromuscular junction
myasthenia-gravis
what is the hallmark sign of myasthenia-gravis
muscle weakness that increases during periods of activity and improves after periods of rest
what are the clinical manifestations of myasthenia-gravis
weakness in muscles that control eye and eyelid movement, slurred speech, swallowing, respiratory dysfunction, fatigue
how is myasthenia-gravis diagnosed
blood test (presence of immune molecules or acetylcholine receptor antibodies), edriophonium (tensilon) test (prevents ACH breakdown-have atropine sulfate on hand), nerve conduction test, EMG
what are the treatments for myasthenia-gravis
anticholinesterase agents (pyridostigmine and neostigmine, Q3h), immunosuppressive drugs (prednisone, azathioprine if prednisone ineffective), thymectomy, plasmapheriesis, intravenous immune globulin (IVIG)
what are the complications from myasthenia-gravis
myasthenia crisis (triggered by infection, fever, respiratory weakness- increase medication), cholinergic crisis (over medicated, abdominal cramps, N/V/D, hypotension-give atropine)
which meal option would be the most appropriate for a patient with myasthenia-gravis?
a. roasted potatoes and cubed steak
b. hamburger with baked fries
c. clam chowder with mashed potatoes
d. fresh veggie tray with sliced cheese cubes
c.
what is amyotropic lateral sclerosis also known as
lou gherigs disease
most common of motor neuron diseases, tends to occur in middle age (male>female), fatal disease (2-5 years), involves degeneration of anterior horn cells and corticospinal tracts
amyotropic lateral sclerosis
what are the clinical manifestations of amyotropic lateral sclerosis
progressive muscle weakness, fasciculations/spasms and cramping of muscles, impairment of use of arms/legs, thick speech and difficulty projecting voice, SOB, difficulty breathing/swallowing, drooling; usually progressives from upper extremities to shoulders, trunk and neck with lower extremities affected later
what are the treatments for amyotropic lateral sclerosis
no cure, supportive therapy only. riluzole extends life by a few months (hepatotoxic-no alcohol)
what are the medications to control symptoms in amyotropic lateral sclerosis
baclofen, diazepam for spasticity, pain control, PEG tubes with TF once they cannot swallow
in pts with amyotropic lateral sclerosis, there will eventually be a need for mechanical ventilation. what allows living months or years beyond respiratory failure
tracheostomy invasive ventilation
sudden pain in any area innervated by 5th cranial nerve branch, shooting/stabbing/burning pain, may have involuntary contraction of facial muscle or eye twitching, occurs more as people age, can coexist with MS patients
trigeminal neuralgia
what are the treatments for trigeminal neuralgia
anti-seizure meds, gabapentin and baclofen for nerve pain, surgical resection possible
unilateral inflammation of the 7th cranial nerve (facial) causing weakness or paralysis of one side of face, usually in people under 45, may also have speech or chewing difficulties/facial droop, may be mistaken for stroke, spontaneous recovery in 3-5 weeks
bell’s palsy
what is the treatment for bells palsy
corticosteroids and analgesics
loss of intellectual ability with impairment severe enough to interfere with social or occupational functioning. loss of memory, reasoning, judgment, and language
dementia
what are the possible causes of dementia
brain hypoxia, reduced blood flow due to stroke, drugs
what must be ruled out before a dementia diagnosis
delirium
what is the most common type of dementia
alzheimers
dementia risk increases with advancing —-
age
what type of dementia is due to stroke
multi-infarct or vascular
what type of dementia affects the frontal brain
pick’s disease
what type of dementia is similar to alzheimer's but is more progressive and is linked to parkinson’s
Lewy body
what type of dementia is due to age and chronic HTN
binswangers
what type of dementia is caused by plaques and tangles of tau and amyloid proteins in neurons
alzheimers
what are the essential features of Alzheimers
memory failure, personality changes, difficulty with ADL’s
what are the causes of Alzheimers
genetics (less than 5%), age, females more at risk, African American and hispanic, head injury, environmental (diabetes, high cholesterol, smoking, lack of exercise, obesity, low vitamin D, high stress and depression)
how is Alzheimers diagnosed
no definitive diagnosis except histopathological on post mortem exam. clinical diagnosis made based on patient profile and presenting behaviors and/or cognitive issues
how do you care for those with Alzheimers
no prevention or cure, treat symptoms and support caregivers and family
what are the several methods of staging Alzheimers
functional assessment staging (FAST); addresses 7 major areas of functioning
stages of deterioration; 3 major stages of degeneration (mild, moderate, severe), easier for family to understand
stages of symptom progression; three stages delineated by years up to and after diagnosis
in alzheimers, losses and symptoms vary from person to person, depending on…
extent of brain cell death and loss, location of brain cell death and loss, speed with which losses occur
what are the guidelines for reorientation in Alzheimers patients
only reorient in early stages when confused, validation used in later stages
what are the 12 core primary symptoms of Alzheimers
functional impairment, mood disorders, delusions and hallucinations, dependence in ADLS’s, inability to initiate meaningful activity, anxiety, spatial disorientation, elopement, resistance to care, food refusal, insomnia (don’t use Benadryl) and sundowning, apathy and agitation
what are some behavior interventions for patients with dementia
progressively lowered stress threshold, patients need alternating quiet and stimulating environments, behaviors have meaning, check for physical problems, pain, environment, mind stimulating activities and develop a routine