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what are the 4 definitions of abnormality
deviation from social norms
failure to function adequately
deviation from ideal mental health
statistical infrequency
deviation from social norms definition
a persons thinking or behaviour is classified as abnormal if it violates the norms of society
failure to function adequately definition
behaviour is abnormal if it prevents a person from leading a normal everyday and functional life
Rosenhan and Seligman: characteristics to define failure to function
personal distress: suffering with a disorder
maladaptive behaviour: behaviour which prevents people from achieving major life goals
unpredictable behaviour: the behaviour of abnormal people is often very variable
irrational behaviour: others cannot understand why anyone would choose to behave this way
observer discomfort: observers to the behaviour are made uncomfortable by it
deviation from ideal mental health definition
when someone does not meet the set criteria for good mental health
Jahoda’s 6 categories of ideal mental health
positive attitude towards oneself
self actualisation
autonomy
resisting stress
accurate perception of reality
environmental mastery
statistical infrequency definition
when an individual has less common characteristics and is statistically rare
define what a phobia is
an irrational fear of an object or situation
a type of anxiety disorder that produces emotional relations to a potentially dangerous stimuli
what are some behavioural symptoms of phobias
panic
avoidance
disruption of functioning
what are some emotional symptoms of phobias
persistent fear of exposure
high levels of anxiety
what are some cognitive symptoms of phobias
selective attention: if the sufferer can see the phobic stimulus it is hard to look away from it
what are the three DSM-5 categories for phobias
simple phobias: the fear of specific objects or situations
social phobias: fear of social situations
agoraphobia: fear of leaving a safe space
Describe the behaviourist explanation of acquisition of phobias
the phobic stimulus (NS) is paired with the unconditioned stimulus which creates an association between the neutral stimulus and the unconditioned stimulus
the phobic stimulus then creates the fear response that was originally created by the unconditioned stimulus
this fear response can sometimes be generalised to other stimuli
describe the behaviourist explanation of the maintenance of phobia
the avoidance of a phobia reduces anxiety
negative reinforcement
Little Albert: Watson and Rayner- aim
to see if it is possible to induce a fear of a previously neutral stimulus through classical conditioning
Little Albert: Watson and Rayner- procedure
albert was 11 months old
alberts reaction to the neutral stimuli without the unconditioned stimulus showed no fear
classical conditioning was used to see if the unconditioned stimulus (the bang) could achieve the aims of inducing fear
Little Albert: Watson and Rayner- results
after the unconditioned stimulus was introduced Albert started to show signs of fear towards the neutral stimulus and objects similar to that such as white rabbits
this meant that stimulus generalisation has occurred
Little Albert: Watson and Rayner- conclusion
a fear response had been conditioned using classical conditioning
generalisation of the fear has been made to other similar objects
Bagby 1992: research on phobias
reported on a case study of a woman who had a phobia of running water that originated from her getting her feet stuck in some rocks near a waterfall
As time went on she became increasingly panic stricken. Although she was deconditioned, the conditioned stimulus of running water was acquired
King et al 1998: research on phobias
reported that case studies showed that children thended to acquire strong phobias through a traumatic experience, which further supports the idea of phobias being aquired through classical conditioning
Behavioural therapies to treating phobias: systematic desensitisation
stage 1: functional analysis- thereapist works with a client to produce a fear hierarchy
stage 2: relaxation training- clients are taught different techniques for relaxing, e.g breating and muscle tension
stage 3: graduated exposure- over the course of 6-12 sessions, the client is gradually brought into contact with their phobic object or situations by starting at the bottom of the hierarchy and gradually moving up once no anxiety is produced in a situation
what is reciprocal inhibition
you cannot have negative thoughts about something if you are having positive thoughts about it. this is a key principle because it suggests your conditioned response cannot be fear and calm together. Therefore, if you replace the fear with calmness the treatment has worked
behavioural therapies to treating phobias: flooding
immediate exposure to fear stimulus
extinction occurs because the person realises that they are in no danger
need informed consent due to high distress
what is the difference between invitro and invivo
invitro is when you imagine exposure
invivo is when you are actually exposed
define what depression is
a mental disorder characterised by low mood and low energy levels
what are the DSM-5 criteria for depression
persistent depressive disorder: long term depression
disruptive mood dysregulation: childhood trauma
premenstrual dysphoric depression
major depressive disorder: severe/ short term depression
what are some behavioural sympotoms for unipolar depression
loss of energy
social impairment
weight changes
poor hygiene
sleep pattern disturbance
what are some emotional symptoms of unipolar depression
loss of enthusiasm
constant depressed mood
worthlessness
what are some cognitive symptoms of unipolar depression
delusions
reduced concentration
thoughts of death
poor memory
what are some behavioural symptoms of bipolar depression (mania)
high energy levels
reckless behaviour
talkative
what are some emotional sympotoms of bipolar depression (mania)
elevated mood states
irritability
lack of guilt
what are some cognitive symptoms of bipolar depression (mania)
racing thought
irrational thought processes
delusions
describe ellis’ ABC model
A: activating event
B: belief: rational/ irrational thoughts
C: consequence: desirable/ undesirable behaviours and emotions
What is Becks negative triad
this idea sees depression as a result of negative feelings about themselves, the world and the future.
How is the negative triad maintained
Arbitrary inference: conclusions drawn in the absence of sufficient evidence.
selective abstraction: conclusions drawn from just one part of a situation
overgeneralisation: conclusions drawn on the basis of a single event.
magnification/minimisation: exaggerations in evaluation of performance.
research support for becks neg triad
Boury et al (A03)
monitored students negative thoughts with the beck depression inventory, finding that depressive patients misinterpret facts and experiences in a negative fashion. They also feel hopeless about the future if asked.
What is CBT
cognitive behavioural therapy
is a combination of cognitive therapy (a way of changing maladaptive thoughts and beliefs) and behavioural therapy (a way of changing behaviour in response to these thoughts and beliefs.
what is REBT
rational emotive behavioural therapy
1-2 sessions every two week for about 15 sessions. it has 3 phases:
1- education phase: patients are taught to recognise and record events (activating) in a dysfunctional thought diary, record all negative thoughts, emotions and behaviours
2- behavioural activation: the therapist can then work to help the patients challenge their beliefs so they can be disproved.
behavioural symptoms of obsessions (OCD)
hinder everyday functioning- maladaptive/ failure to function due to reoccuring obsessions
social impairment- cannot have meaningful social interactions
behavioural symptoms of compulstions (OCD)
repetative- feel compelled to repeat behaviours
hinder everyday functioning- maladaptive, disrupts life
social impairment
emotional symptoms of obsessions (OCD)
extreme enxiety- persistant inappropriate or forbidden ideas (the urge to repeat behaviours creates anxiety)
emotional symptoms of compulsions (OCD)
distress- because they recognise the urges cannot be controlled
embarassment- sufferers know they are behaving irrationallt and creates embarasment and shame
cognitive symptoms of obsessions (OCD)
recurrent and persistant thoughts
recognised as self-generated
attentional bias
cognitive symptoms of compulsions (OCD)
uncontrolable thoughts about urges
realisation of inappropriateness
genetic explaination for OCD: the COMT gene
regulates the neurotransmitter dopamine that could be involved in OCD.
a mutation of the COMT gene produces higher levels of dopamine
genetic explaination for OCD: the SERT gene
serotonin transporter/ reuptaker
affects to transport of serotonin and a mutation in the gene produces lower levels of seratonin.
this is thought to have happened in people with OCD.
research support: genetic explaination of OCD, Samuels et al
performed gene mapping on people with OCD finding a link to a mutation on chromosome 14, implying a genetic link to the disorder
neural explaination for OCD: anatomical abnormalities
some forms of OCD have been linked to breakdowns in the imune system functioning due to infections such as lymes disease and influenza. this indicates damage to neural mechanisms due to the infections
high levels of activity been found in the orbital frontal cortex, associated with higher level thought processes- this area is thought to control impulses.
neural explaination for OCD: biochemical abnormalities
brain scans show low levels of seratonin in addition, brain scans show low levels of seratonin activity in the brain and high levels of dopamine.
research support for the neural explaination: Hu 2006
procedure- compared seratonin activity in OCD sufferers and non sufferers
results- the research showed a finding of seratonin levels to be lower in 80% of the OCD patients.
conclusion- this supports the idea of low levels of seratonin being associated with the onset of the disorder
biological therapies to treating OCD: drug therapy
Antidepressants, specifically selective serotonin reuptake inhibitors (SSRIs), are used to treat OCD by increasing serotonin levels in the synapse. They block the serotonin transporter, reducing reuptake into the presynaptic cell. This allows more serotonin to bind to receptors on the postsynaptic neuron, improving communication in serotonin pathways and helping reduce symptoms like repetitive thinking.
research support for drug therapies: koran et al
conducted research to evaluate the long-term effectiveness of drug treatments for Obsessive-Compulsive Disorder (OCD).
They found that while Selective Serotonin Reuptake Inhibitors (SSRIs) are effective in reducing OCD symptoms, many patients relapsed when the medication was discontinued. This suggests that SSRIs may manage symptoms rather than cure the disorder, and long-term use may be necessary for sustained benefits.