1/137
Looks like no tags are added yet.
Name | Mastery | Learn | Test | Matching | Spaced | Call with Kai | Chat |
|---|
No analytics yet
Send a link to your students to track their progress
plain film radiographs (X-Ray)?
ionizing electromagnetic radiation of short wavelengths to visualize osseous structures and surrounding soft tissue (limited)
common X-Ray views
- anterior-posterior
- axillary
- internal rotation
- supraspinatus outlet
digital motion X-Ray (DMX)/Fluroscopy
continuous pr pulse X-Ray imaging to capture movement in real time
computed tomography (CT)
rotates X-Ray beam 360 degrees around patient to generate multiple cross-sectional image
magnetic resonance imaging (MRI)
uses strong magnetic fields and radiofrequency pulses to produce high resolution images of soft tissue and bone marrow
arthrography
involves injection of contrast medium into joint to outline intra-articular structures
ultrasound
uses high frequency sound waves to produce dynamic, real time images of shoulder
bone scan (scintigraphy)
use IV injection of a radiotracer to highlighted areas of increased metabolic activity in bone
tendinitis
acute injury with tendon inflammation
treatment for tendinitis
- anti-inflammatory strategies
- protection, activity modification, RICE (rest, ice, compression, elevation)
tendinosis
- abnormal process or degeneration of tendon caused by repetitive microtrauma
treatment for tendinosis
- promoting collagen synthesis, tissue maturation and strength, and not reducing acute inflammation
- eccentric resistance exercise
what are the 3 stages of tendon pathology?
- reactive tendinopathy
- tendon dysrepair
- degenerative tendinopathy
reactive tendinopathy
- triggered by acute overload of tendon where tendon becomes thickened and painful but is intact
- pain with activity and mild loss of function
- common in young adults with abrupt changes in load
tendon dysrepair
- represents an attempt at healing but disorganized collagen and matrix changes
- structural integrity of tendon declines
- symptoms more persistent
- seen with ongoing mechanical overload and inadequate rest
degenerative tendinopathy
- advanced disruption of tendon structure
- symptoms: chronic pain, weakness, functional loss, high risk of tendon rupture
- common in middle to older adults from cumulative microtrauma
primary impingement tendinopathy/tendinosis?
- impingement of RTC beneath coracoacromial arch which results from decreased coracoacromial space and is mechanical in nature and occurs without GH instability
- intrinsic causes (cuff muscles) or extrinsic causes (shape of acromion)
secondary impingement tendinopathy/tendinosis
- occurs secondary to primary problem of instability with anterior or anterior/inferior instability being a key diagnostic criteria
sequence of events for secondary impingement tendinopathy/tendinosis
- initial weakness in biceps and RTC muscles overloads passive mechanisms (ligaments and labrum)
- dynamic stabilizers (RTC and biceps) try to compensate but fail from fatigue
- humeral head undergoes abnormal translation leading to mechanical impingement of RTC
posterior internal impingement
- specific type of secondary impingement that occurs secondary to contact or impingement of articular sides of supraspinatus/infraspinatus tendons
posterior internal impingement commonly occurs in athletes doing what?
OH activities in extreme ER and abduction (baseball players)
describe RTC tears
- in OH athletes and after acute overload events, a vulnerable tendon may fail
- normal aging of cuff tissue characterized by continuous repetitive micro-trauma can predispose tendon for rupture
what is the sizing for RTC tears?
- small
degrees of rotator cuff degeneration
- grade 0: no fatty streaks
- grade 1: some fatty streaks
- grade 2: muscle > fat
- grade 3: muscle = fat
- grade 4: muscle < fat
S/S on RTC tendinopathy (partial)
- painful arc
- pain/weakness in empty can or ER resistance
S/S of calcific tendinopathy?
- acute or severe pain episodes
- painful arc
- night pain
- calcific deposits
S/S of RTC full tear
- night pain
- weakness
- drop arm test
- positive hornblowers
S/S of subacromial-subdeltoid bursitis
- lateral shoulder pain
- painful arc
S/S of long head of biceps tendinopathy?
- anterior groove tendernes
- pain with Speeds or Yergasons
- pain with forward elevation
S/S of long head of biceps instability/SLAP
- deep joint pain
- clicking
- symptoms provoked with O'Briens Tests
S/S of AC Joint Pain
- localized AC tenderness
- pain with cross body adduction
S/S of GH microinstability
- instability symptoms
- humeral head migration
S/S GH osteoarthritis
- stiffness/crepitus
- > 50
- radiographic changes
S/S of cervical radiculopathy
- neck pain
- dermatomal patterns
- Spurlings/ULTT
- provocation/alleviation with compression/traction
S/S of suprascapular neuropathy
- aching posterior shoulder pain with infra-supra atrophy
- ER weakness
proximal biceps tendinosis
- inflammation and tendinosis of biceps tendon
- degenerative condition involved in repetitive activities like throwing, golf, sports with humeral rotation above horizontal
clinical presentation of proximal biceps tendinosis?
- isolated disorder of LH biceps with pain over anterior shoulder and bicipital groove
- pain to palpation over bicipital groove
- pain with contraction and passive max lengthening
- painful arc between 60-120
subacromial bursitis
- acute or chronic inflammation of the subacromial bursa
- repetitIve shoulder movement cause reactive accumulation of fluid within bursa
- often occurs secondary to repetitive activities and linked to impingement and RTC tears
clinical presentation of subacromial bursitis
- onset sudden
- tenderness to palpation over anterolateral aspect of shoulder
- pain with abduction beginning at 60 and decreasing at 100-120
- resisted testing normal
calcific tendinitis
- chronic tendinitis with formation of mineral deposits in and around tendon
- likely a result of tendon degeneration caused by chronic fibrosis and necrosis
what are the 2 phases of calcific tendinitis?
- precalcific: asymptomatic transformation of tendon (formative - calcium deposits in tendon, resorptive - calcium reabsorbed)
- postcalcific: remodeling of collagen
scapular dyskinesis
- symptomatic/asymptomatic change in scapular position of dynamic motion
- 3 subtypes
- can occur from any consequential factor leading to alterations in scapular position/motion
factors cause scapular dyskinesis
- impaired posture
- impaired muscle function
- muscle inflexibility
- proprioceptive dysfunction
3/4 subtypes of scapular dyskinesis?
- 1: at rest, prominence of the inferior medial border
- 2: at rest, prominence of entire scapular border
- 3: prominence of superior border
- 4: normal
scapular dyskinesis test
- unweighted shoulder flexion and abduction watching scapula
- 1-2 lbs shoulder flexion and abduction watching scapula
what are the 3 clinical stages of SA and RTC tendinopathy?
- stage 1: edema & inflammation
- stage 2: fibrosis & tendinitis/tendinosis
- stage 3: tendon degeneration & rupture
describe stage 1: edema and inflammation for SA and RTC tendinopathy?
- age 25 or younger
- consistent with contractile lesion
- anterior/lateral shoulder in C5 distribution
- aching pain triggered by activity (OH and lifting objects out)
- tender over GT
- painful arc between 60-120
- changes typically reversible
describe stage 2: fibrosis & tendinitis/tendinosis for SA and RTC tendinopathy
- 25-40 yrs
- continuation of stage 1 S/S
- increases pain with ADLS, at rest, and night
- increasing tenderness
- worsening pain in C5 distribution
- changes in accessory joint motion
- changes not typically clinically reversible
describe stage 3: tendon degeneration & rupture
- 40+
- increased pain with ADLs, at night
- progressive weakness in abd and ER
- atrophy of muscles
- capsular restrictions and pathological end-feels
- changes not clinically or surgically reversible
how many stages are in Neer's Stages of subacromial impingement?
3
describe stage 1 of Neer's stages of subacromial impingement
- less than 25
- intermittent pain with OH activities
- edema and hemorrhage
- reversible cuff edema
- non-surgical management
describe stage 2 of Neer's stages of subacromial impingement
- 25-40 yrs
- fibrosis and tendinosis
- irreversible tendon changes
- conservative and surgical management
describe stage 3 of Neer's stages of subacromial impingement
- 40+
- significant tendon degeneration
- RTC Tear, biceps tedon rupture and bony changes
- surgical management
what is painful arc special test?
active elevation watching when there is pain
what is Neer Impingement Test?
passive forward elevation with IR at end-range
what is Hawkins Kennedy Test?
passive IR at 90 degrees of flexion
what is empty can test?
elevation or resisted elevation in scapular plane with IR
what is the lift off test?
resisted IR from behind back
what is the drop arm test?
AROM and resisted eccentric lowering
what is the ER lag sign?
resisted hold in ER
what is the Hornblowers sign?
resisted ER in abduction
what are 3 tests that great the best cluster for subacromial impingement?
- painful arc
- empty can
- ER resistance
GH joint instability
- asymptomatic or symptomatic condition characterized by abnormal movement between humeral head and glenoid
- inability to keep humeral head center in glenoid fossa when arm is elevated or placed in certain positions
GH joint subluxation
- symptomatic translation of the humeral head on fossa without complete separation of articular surfaces
GH joint dislocation
- complete loss of articulation of joint
what directions is GH joint instability most common?
- anterior
- anterior/inferior
FEDS classification system
- frequency (solitary 1, occasional 2-5, frequent 5+)
- etiology (traumatic, atraumatic)
- primary direction (anterior, inferior, posterior)
- severity (subluxation, dislocation)
what are the frequency categories for the FEDS system?
- solitary = 1 episode
- occasional = 2-5
- frequent > 5
what are the etiology categories for the FEDS system?
- traumatic: history of injury
- atraumatic: no history of injury
what are the primary direction categories for the FEDS system?
- anterior
- inferior
- posterior
what are the severity categories for the FEDS system?
- subluxation: spontaneous reduction
- dislocation: required physical force
what does hawkins classification scheme for GH joint include?
- frequency
- etiology
- directions
- degree
- volition
what are the frequency categories for Hawkins scheme?
- acute
- chronic
- recurrent
what are the etiology categories for Hawkins scheme?
- traumatic (macrotrauma)
- atraumatic
- microtrauma
- congenital
- neuromuscular
what are the direction categories for the Hawkins Scheme?
- anterior
- posterior
- inferior
- multidirectional
what are the degree categories for Hawkins scheme?
- subluxation
- dislocation
what are the volition categories for Hawkins scheme?
- involuntary
- voluntary
what are capsular tears of GH joint?
results from direct or indirect loading over humerus
what are capsular detachments?
detachment of labrum from direct or indirect loading over humerus
what is the clinical presentation of shoulder dislocations?
- severe acute pain with an overt MOI
- arm is held across body in IR with varying degrees of abduction
what is anterior GH instability?
- most usual form of GH instability
- MOI: traumatic event that places shoulder in extreme abduction and ER
clinical presentation of anterior GH instability
- history of pain and dysfunction associated with instability
- excessive looseness
- atrophy
- humeral head more palpable anteriorly
- positive anterior apprehension, load and shift, and crank test
how do most anterior GH dislocations occur?
- result of axial loading of arm in abducted and ER position, OR abducted, extended, and ER position
what is the greatest risk of injury with an anterior dislocation?
axillary nerve
what is the management of anterior dislocations?
- young: sling for 4-6 weeks
- older: sling for 1-2 to decrease stiffness
what is the rehab progression for anterior dislocations?
- immobilization phase: progressive isometrics
- post-immobilization phase: progressive resistive exercise
what are common associated lesions with a anterior instability/dislocations?
- hill-sachs lesion
- reverse hill sachs lesion
- bankart lesion
- perthes bankart lesion
hill-sachs lesion?
- compression fracture of posterolateral aspect of humeral head
reverse hill sachs lesion?
- compression fracture of anterior-medial humeral head
bankart lesion
avulsion of capsule and glenoid labrum from anterior rim of glenoid
perthes bankart lesion
anterioinferior labrum is torn and lifted from glenoid edge but remains intact to periosteum
how do most posterior GH instability/disclocations occur?
- result from axial loading of arm in adducted and IR position OR adducted, flexed, and IR
- can also occur during electrocution or grand mal seizures
what are the clinical findings for posterior GH instability/dislocation?
- limitations ino elevation above 90
- prominence over posterior shoulder
- flattening of anterior aspect of shoulder
- positive clunk test
- GIRD
what are early interventions for an unstable shoulder?
- education
- immobilization
- rest
- pain control
what are the immobilization recommendations for an unstable shoulder?
- < 20: 6 weeks
- 20-30: 2-3 weeks
- 30-40: 10-14 days
- > 40: 3-5 days
what are intermediate interventions for an unstable shoulder?
- education
- RTC and scapular strengthening
- isometrics
- anterior instability precautions
what is atraumatic instability?
instability without significant or notable injury
what are potential causes of atraumatic instability?
- loss of negative intra-articular pressure
- failure of GH ligaments
- failure of dynamic stabilizers
- altered NM control
- anatomical variations
- disuse atrophy
- familial and systemic factors
what is a SLAP lesion?
injury to the superior glenoid labrum that extends from anterior to posterior relative to the biceps tendon
what are the 4 types of SLAP lesions?
- type 1: degenerative fraying of superior labrum with bicep intact
- type 2: avulsion of superior labrum and biceps from glenoid
- type 3: bucket handle tear with intact biceps
- type 4: bucket handle tear extending into biceps