Module 2 - Shoulder Pathology

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Last updated 11:29 AM on 9/23/26
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138 Terms

1
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plain film radiographs (X-Ray)?

ionizing electromagnetic radiation of short wavelengths to visualize osseous structures and surrounding soft tissue (limited)

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common X-Ray views

- anterior-posterior

- axillary

- internal rotation

- supraspinatus outlet

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digital motion X-Ray (DMX)/Fluroscopy

continuous pr pulse X-Ray imaging to capture movement in real time

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computed tomography (CT)

rotates X-Ray beam 360 degrees around patient to generate multiple cross-sectional image

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magnetic resonance imaging (MRI)

uses strong magnetic fields and radiofrequency pulses to produce high resolution images of soft tissue and bone marrow

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arthrography

involves injection of contrast medium into joint to outline intra-articular structures

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ultrasound

uses high frequency sound waves to produce dynamic, real time images of shoulder

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bone scan (scintigraphy)

use IV injection of a radiotracer to highlighted areas of increased metabolic activity in bone

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tendinitis

acute injury with tendon inflammation

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treatment for tendinitis

- anti-inflammatory strategies

- protection, activity modification, RICE (rest, ice, compression, elevation)

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tendinosis

- abnormal process or degeneration of tendon caused by repetitive microtrauma

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treatment for tendinosis

- promoting collagen synthesis, tissue maturation and strength, and not reducing acute inflammation

- eccentric resistance exercise

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what are the 3 stages of tendon pathology?

- reactive tendinopathy

- tendon dysrepair

- degenerative tendinopathy

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reactive tendinopathy

- triggered by acute overload of tendon where tendon becomes thickened and painful but is intact

- pain with activity and mild loss of function

- common in young adults with abrupt changes in load

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tendon dysrepair

- represents an attempt at healing but disorganized collagen and matrix changes

- structural integrity of tendon declines

- symptoms more persistent

- seen with ongoing mechanical overload and inadequate rest

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degenerative tendinopathy

- advanced disruption of tendon structure

- symptoms: chronic pain, weakness, functional loss, high risk of tendon rupture

- common in middle to older adults from cumulative microtrauma

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primary impingement tendinopathy/tendinosis?

- impingement of RTC beneath coracoacromial arch which results from decreased coracoacromial space and is mechanical in nature and occurs without GH instability

- intrinsic causes (cuff muscles) or extrinsic causes (shape of acromion)

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secondary impingement tendinopathy/tendinosis

- occurs secondary to primary problem of instability with anterior or anterior/inferior instability being a key diagnostic criteria

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sequence of events for secondary impingement tendinopathy/tendinosis

- initial weakness in biceps and RTC muscles overloads passive mechanisms (ligaments and labrum)

- dynamic stabilizers (RTC and biceps) try to compensate but fail from fatigue

- humeral head undergoes abnormal translation leading to mechanical impingement of RTC

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posterior internal impingement

- specific type of secondary impingement that occurs secondary to contact or impingement of articular sides of supraspinatus/infraspinatus tendons

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posterior internal impingement commonly occurs in athletes doing what?

OH activities in extreme ER and abduction (baseball players)

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describe RTC tears

- in OH athletes and after acute overload events, a vulnerable tendon may fail

- normal aging of cuff tissue characterized by continuous repetitive micro-trauma can predispose tendon for rupture

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what is the sizing for RTC tears?

- small

24
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degrees of rotator cuff degeneration

- grade 0: no fatty streaks

- grade 1: some fatty streaks

- grade 2: muscle > fat

- grade 3: muscle = fat

- grade 4: muscle < fat

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S/S on RTC tendinopathy (partial)

- painful arc

- pain/weakness in empty can or ER resistance

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S/S of calcific tendinopathy?

- acute or severe pain episodes

- painful arc

- night pain

- calcific deposits

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S/S of RTC full tear

- night pain

- weakness

- drop arm test

- positive hornblowers

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S/S of subacromial-subdeltoid bursitis

- lateral shoulder pain

- painful arc

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S/S of long head of biceps tendinopathy?

- anterior groove tendernes

- pain with Speeds or Yergasons

- pain with forward elevation

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S/S of long head of biceps instability/SLAP

- deep joint pain

- clicking

- symptoms provoked with O'Briens Tests

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S/S of AC Joint Pain

- localized AC tenderness

- pain with cross body adduction

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S/S of GH microinstability

- instability symptoms

- humeral head migration

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S/S GH osteoarthritis

- stiffness/crepitus

- > 50

- radiographic changes

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S/S of cervical radiculopathy

- neck pain

- dermatomal patterns

- Spurlings/ULTT

- provocation/alleviation with compression/traction

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S/S of suprascapular neuropathy

- aching posterior shoulder pain with infra-supra atrophy

- ER weakness

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proximal biceps tendinosis

- inflammation and tendinosis of biceps tendon

- degenerative condition involved in repetitive activities like throwing, golf, sports with humeral rotation above horizontal

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clinical presentation of proximal biceps tendinosis?

- isolated disorder of LH biceps with pain over anterior shoulder and bicipital groove

- pain to palpation over bicipital groove

- pain with contraction and passive max lengthening

- painful arc between 60-120

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subacromial bursitis

- acute or chronic inflammation of the subacromial bursa

- repetitIve shoulder movement cause reactive accumulation of fluid within bursa

- often occurs secondary to repetitive activities and linked to impingement and RTC tears

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clinical presentation of subacromial bursitis

- onset sudden

- tenderness to palpation over anterolateral aspect of shoulder

- pain with abduction beginning at 60 and decreasing at 100-120

- resisted testing normal

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calcific tendinitis

- chronic tendinitis with formation of mineral deposits in and around tendon

- likely a result of tendon degeneration caused by chronic fibrosis and necrosis

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what are the 2 phases of calcific tendinitis?

- precalcific: asymptomatic transformation of tendon (formative - calcium deposits in tendon, resorptive - calcium reabsorbed)

- postcalcific: remodeling of collagen

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scapular dyskinesis

- symptomatic/asymptomatic change in scapular position of dynamic motion

- 3 subtypes

- can occur from any consequential factor leading to alterations in scapular position/motion

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factors cause scapular dyskinesis

- impaired posture

- impaired muscle function

- muscle inflexibility

- proprioceptive dysfunction

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3/4 subtypes of scapular dyskinesis?

- 1: at rest, prominence of the inferior medial border

- 2: at rest, prominence of entire scapular border

- 3: prominence of superior border

- 4: normal

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scapular dyskinesis test

- unweighted shoulder flexion and abduction watching scapula

- 1-2 lbs shoulder flexion and abduction watching scapula

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what are the 3 clinical stages of SA and RTC tendinopathy?

- stage 1: edema & inflammation

- stage 2: fibrosis & tendinitis/tendinosis

- stage 3: tendon degeneration & rupture

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describe stage 1: edema and inflammation for SA and RTC tendinopathy?

- age 25 or younger

- consistent with contractile lesion

- anterior/lateral shoulder in C5 distribution

- aching pain triggered by activity (OH and lifting objects out)

- tender over GT

- painful arc between 60-120

- changes typically reversible

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describe stage 2: fibrosis & tendinitis/tendinosis for SA and RTC tendinopathy

- 25-40 yrs

- continuation of stage 1 S/S

- increases pain with ADLS, at rest, and night

- increasing tenderness

- worsening pain in C5 distribution

- changes in accessory joint motion

- changes not typically clinically reversible

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describe stage 3: tendon degeneration & rupture

- 40+

- increased pain with ADLs, at night

- progressive weakness in abd and ER

- atrophy of muscles

- capsular restrictions and pathological end-feels

- changes not clinically or surgically reversible

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how many stages are in Neer's Stages of subacromial impingement?

3

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describe stage 1 of Neer's stages of subacromial impingement

- less than 25

- intermittent pain with OH activities

- edema and hemorrhage

- reversible cuff edema

- non-surgical management

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describe stage 2 of Neer's stages of subacromial impingement

- 25-40 yrs

- fibrosis and tendinosis

- irreversible tendon changes

- conservative and surgical management

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describe stage 3 of Neer's stages of subacromial impingement

- 40+

- significant tendon degeneration

- RTC Tear, biceps tedon rupture and bony changes

- surgical management

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what is painful arc special test?

active elevation watching when there is pain

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what is Neer Impingement Test?

passive forward elevation with IR at end-range

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what is Hawkins Kennedy Test?

passive IR at 90 degrees of flexion

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what is empty can test?

elevation or resisted elevation in scapular plane with IR

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what is the lift off test?

resisted IR from behind back

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what is the drop arm test?

AROM and resisted eccentric lowering

60
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what is the ER lag sign?

resisted hold in ER

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what is the Hornblowers sign?

resisted ER in abduction

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what are 3 tests that great the best cluster for subacromial impingement?

- painful arc

- empty can

- ER resistance

63
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GH joint instability

- asymptomatic or symptomatic condition characterized by abnormal movement between humeral head and glenoid

- inability to keep humeral head center in glenoid fossa when arm is elevated or placed in certain positions

64
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GH joint subluxation

- symptomatic translation of the humeral head on fossa without complete separation of articular surfaces

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GH joint dislocation

- complete loss of articulation of joint

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what directions is GH joint instability most common?

- anterior

- anterior/inferior

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FEDS classification system

- frequency (solitary 1, occasional 2-5, frequent 5+)

- etiology (traumatic, atraumatic)

- primary direction (anterior, inferior, posterior)

- severity (subluxation, dislocation)

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what are the frequency categories for the FEDS system?

- solitary = 1 episode

- occasional = 2-5

- frequent > 5

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what are the etiology categories for the FEDS system?

- traumatic: history of injury

- atraumatic: no history of injury

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what are the primary direction categories for the FEDS system?

- anterior

- inferior

- posterior

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what are the severity categories for the FEDS system?

- subluxation: spontaneous reduction

- dislocation: required physical force

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what does hawkins classification scheme for GH joint include?

- frequency

- etiology

- directions

- degree

- volition

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what are the frequency categories for Hawkins scheme?

- acute

- chronic

- recurrent

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what are the etiology categories for Hawkins scheme?

- traumatic (macrotrauma)

- atraumatic

- microtrauma

- congenital

- neuromuscular

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what are the direction categories for the Hawkins Scheme?

- anterior

- posterior

- inferior

- multidirectional

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what are the degree categories for Hawkins scheme?

- subluxation

- dislocation

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what are the volition categories for Hawkins scheme?

- involuntary

- voluntary

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what are capsular tears of GH joint?

results from direct or indirect loading over humerus

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what are capsular detachments?

detachment of labrum from direct or indirect loading over humerus

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what is the clinical presentation of shoulder dislocations?

- severe acute pain with an overt MOI

- arm is held across body in IR with varying degrees of abduction

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what is anterior GH instability?

- most usual form of GH instability

- MOI: traumatic event that places shoulder in extreme abduction and ER

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clinical presentation of anterior GH instability

- history of pain and dysfunction associated with instability

- excessive looseness

- atrophy

- humeral head more palpable anteriorly

- positive anterior apprehension, load and shift, and crank test

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how do most anterior GH dislocations occur?

- result of axial loading of arm in abducted and ER position, OR abducted, extended, and ER position

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what is the greatest risk of injury with an anterior dislocation?

axillary nerve

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what is the management of anterior dislocations?

- young: sling for 4-6 weeks

- older: sling for 1-2 to decrease stiffness

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what is the rehab progression for anterior dislocations?

- immobilization phase: progressive isometrics

- post-immobilization phase: progressive resistive exercise

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what are common associated lesions with a anterior instability/dislocations?

- hill-sachs lesion

- reverse hill sachs lesion

- bankart lesion

- perthes bankart lesion

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hill-sachs lesion?

- compression fracture of posterolateral aspect of humeral head

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reverse hill sachs lesion?

- compression fracture of anterior-medial humeral head

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bankart lesion

avulsion of capsule and glenoid labrum from anterior rim of glenoid

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perthes bankart lesion

anterioinferior labrum is torn and lifted from glenoid edge but remains intact to periosteum

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how do most posterior GH instability/disclocations occur?

- result from axial loading of arm in adducted and IR position OR adducted, flexed, and IR

- can also occur during electrocution or grand mal seizures

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what are the clinical findings for posterior GH instability/dislocation?

- limitations ino elevation above 90

- prominence over posterior shoulder

- flattening of anterior aspect of shoulder

- positive clunk test

- GIRD

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what are early interventions for an unstable shoulder?

- education

- immobilization

- rest

- pain control

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what are the immobilization recommendations for an unstable shoulder?

- < 20: 6 weeks

- 20-30: 2-3 weeks

- 30-40: 10-14 days

- > 40: 3-5 days

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what are intermediate interventions for an unstable shoulder?

- education

- RTC and scapular strengthening

- isometrics

- anterior instability precautions

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what is atraumatic instability?

instability without significant or notable injury

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what are potential causes of atraumatic instability?

- loss of negative intra-articular pressure

- failure of GH ligaments

- failure of dynamic stabilizers

- altered NM control

- anatomical variations

- disuse atrophy

- familial and systemic factors

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what is a SLAP lesion?

injury to the superior glenoid labrum that extends from anterior to posterior relative to the biceps tendon

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what are the 4 types of SLAP lesions?

- type 1: degenerative fraying of superior labrum with bicep intact

- type 2: avulsion of superior labrum and biceps from glenoid

- type 3: bucket handle tear with intact biceps

- type 4: bucket handle tear extending into biceps