ECG Abnormal Findings

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Right Atrial Enlargement: Lead II
tall peaked P wave (>2.5mm)
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RAE: V1
biphasic P wave
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LAE Lead II
wide notched prolonged P wave (.12s)
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LAE V1
biphasic P wave with negative side larger than positive
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RVH V1 & V2
tall R waves
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RVH V5 & V6
deep S waves
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RVH Lead I
negative QRS
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RVH V1
Positive QRS
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RVH all leads
ST depression, T wave inversion
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RVH mean axis
right axis deviation
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LVH equation
S wave in V1/V2 + R wave in V5/V6 >35mm
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LVH all leads
St depression and T wave inversion
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LVH V1-V6
R wave amplitude greater or equal to 25mm
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LVH aVL
R wave more than or equal to 11-13mm
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1st degree AV block
Long PR segment with QRS following (>.2s)
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2nd degree AV block Type I
Pr intervals progressively increase until QRS is dropped
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2nd degree AV block Type II
PR intervals are constant with dropped QRS
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3rd degree AV block
complete dissociation between P waves and QRS complexes, unchanged PR intervals
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RBBB V1
wide positive QRS, rabbit ears
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RBBB Lead I
S wave present (shouldn't be wider than half a box)
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LBBB V1/V2
wide notched negative QRS (in most leads)
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LBBB all leads
ST-T changes opposite of the R waves proceeding them
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LBBB V5/V6
tall notched positive R waves
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LBBB I, aVL, V5, V6
absence of small normal Q waves
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LBBB mean axis
possible left axis deviation
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STEMI ST elevation
aVL, I, V2, V4, V6
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STEMI ST depression
II, III, aVF
*reciprocal findings
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STEMI Q waves
pathologic findings: 1/3 height of QRS, .4s long
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NSTEMI all leads
ST depression
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NSTEMI reciprocal findings
NONE.
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Artifact
NOT A PATIENT CONDITION
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Artifact causes
patient movement,poor skin prep, old electrodes
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Low voltage limb leads
QRS
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Low voltage precordial lead
none with a total voltage of >15mm
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Low voltage ecg calibration
must be properly calibrated to have this finding
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Types of ST segment change
ST depression and ST elevation
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ST depression due to oxygen demands
heart rate, wall stress, contractility
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ST depression due to oxygen supply
blood flow
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ST depression due to exercise ischemia
heart vessels are partially occluded which is made obvious by exercise, higher need for blood causes less blood to get through
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T wave inversion reasons
coronary heart disease, angina pectoris
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Digitalis effect
messes with baseline ST segments, cannot read for ischemia, causes sloping ST segment
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Prinzmetal Angina
ST elevation
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Ventricular Aneurysm
ST elevation, T wave inversion
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Pericarditis all leads except aVR
ST elevation
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Pericarditis all leads
PR segments are depressed
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Early repolarization
ST elevation in normal hearts
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Juvenile Pattern V1-V4
T wave inversion
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Hypocalcemia
QT interval prolongation (>.44)
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Hypercalcemia
QT interval shortened
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Hypokalemia
prominent U wave (only visible during bradycardia)
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Hyperkalemia
Tall, tented, peaked T wave, moves toward ventricular fibrillation
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Normal Sinus Rhythm
Rhythm: regular
Rate: 60-100bpm
P waves (in LII): max .12s
PR interval: .12-.2s
QRS: less than .1
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Sinus Arrhythmias
Rhythm: irregular & sinus
Rate: any rate
P waves, PR interval, QRS normal
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Sinus Arrhythmias Breathing
Decreases on expiration, increases on inspiration
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Sinus tachycardia
Rhythm: regular
Rate: higher than 100bpm
P waves: normal
PR interval: normal
QRS: normal
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Sinus bradycardia
Rhythm: regular
Rate: less than 60 bpm
P wave, PR interval, QRS: normal
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Sinus Exit Block
Dropped beat, beat comes back in line with rhythm from before
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Sinus exit block PR intervals
PR intervals are normal and regular
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Sinus Arrest
Dropped beat, rhythma before and after the pause do not line up
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Premature Atrial Contraction (PAC)
Premature beat, narrow QRS complex
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Blocked PAC
Look at T wave prior (big/different than others in same lead) & beat after
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Blocked PAC physiology
PAC happens so quickly that heart cells are in absolute refractory period, unable to conduct impulse
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Premature Junctional Contractions Three P wave options
Absent, inverted, retrograde
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PJC: Absent P wave
atria and ventricles depolarize at same time
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PJC: Inverted P wave
in correct sport, upside down, beat started at AV junction
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PJC: Retrograde P wave
Ventricles depolarize between atria
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Accelerated Junctional Rhythm: multiple leads
inverted P wave, P wave after QRS OR no P wave
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Accelerated Junctional Rhythm: BPM
60-100 bpm
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Atrial Tachycardia: all leads
three or more PAC that occur consecutively
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Atrial Tachycardia: BPM
200 +/- 50 bpm
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ST elevation
aneurysm, evolving MI, pericarditis, Prinzmetal angina, early repolarization
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ST depression
ischemia, reciprocal change of MI, NSTEMI, R/L BBB, Digitalis, R/L VH
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Left Anterior Hemiblock Lead I
small Q wave
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Left Anterior Hemiblock Lead III
small R
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Left Anterior Hemiblock QRS
QRS normal or widened to .1s
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Left Anterior Hemiblock mean electrical axis
Left shift of electrical axis: -45 to -90
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Left Posterior Hemiblock Lead I
small R wave
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Left Posterior Hemiblock Lead III
Small Q wave
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Left Posterior Hemiblock QRS
normal or widened to .1s
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Left Posterior Hemiblock electrical axis
Rightward shift of electrical axis: 120 to 180