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what two components does the hypothalumus coordinate and sitmulate
primary neural component; brain stem -SNS pathway (direct innervatino
primary hormonal components: hypothalumus- pituitary-adrenal axis (secretion of epi and NE
What is the most direct measure available for the autonomic NS
MSNA: muscle symapthetic nerve activity
what other ways can you indirectly measure ANS activiy
HRV; BEAT-BEAT VARIATION IN CARDIAC CYCLE
low variability reflects poor autonomic tone; reduced reg capacity *ability to adaptively respond to challenges)
HR recovery; PNS reactivation
what ways does ANS control during exercise
Enhance cardiorespiratory function by brachial dilation, transportation of waste product, increased HR, increased cardiac output, increased Bp
Fuel mobilization: adipose cells release fatty acids. liver cells release glucose
thermoregulation, increased sweating, vasodilation increase blood to skin
how do hormones influence metabolism?
mobilize fuel for muscle contraction
maintain blood glucose level
wha ate the two hormones involved?
glucaogon and insulin
which three sites do hormones influence
adipose tissue; fat storage is inhibited and fat mobilization is enhanced
liver: glycogen breakdown and glyconeogenesis
skeletal muscles: glycognesis and increase in free fatty acid uptake & utilization
what two areas will endocrine system affect
CV , muscle bone adipose tissues
what impact does endocrine system have during exercise on CV function
Enhanced cardiac function
blood distribution to active tissue
maintain fluid electrolyte balance
ADH and aldoesterone act on kindeys
whats the difference between NE nad Epi
NE effects quicker, and elevation of this hormone is seen at lower intensities (about 70 % VO2 MAX)
what happens in regards to NE AND epi
secreted by adrenal medullat at 80-90 % VO2 MAX
large increase after anerobic work
greater release from static vs dynamic RT
what hormones are involved in fluid balance, what do they do
ADH AND ALDOSTERONE WATER RETENTION; renin stimulates aldosterone secretion
what happnes to insulin and glucagon during exercise
I decreases and g increase during aerobic ex, no change during static becuase trying to decrease fat stores, no change during static or HIT short term because muscle tissues have fuels available for them
what is the reponse for growth hormones repsonse during exercise
slow response, increases during and post RT
what is cortisols response to exercise
slow acting, greater response to anaerobic exercise, remains elevated for hout after
what is T and PTH response to exercise (thyroid & parathyroid hormone)
inconclusive
whats the differnce in regards to hormonal adaptatinos in trained vs untrained individuals for EPI, NR, I, cortisol, GH,
trained individuals have a much lower amount being released during exercise because less is required for the same outcome. For example, increasing HR would need less epi to do so
what happes to glucagon in UT VS T
no change in TG compared to UTG,because improved efficiency and improved insulin sensitivity, trained individuals done need as much glucagon in otder to conrtol blood glucose levels