PAEA Pediatrics EOR Topics

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313 Terms

1
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what is the MC conjunctivitis seen in children? what is the cause? source?

viral conjunctivitis; Adenovirus; swimming pools

2
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Dx? preauricular lymphadenopathy, copious watery eye discharge, scanty mucoid discharge, usually unilateral with punctate staining on slit lamp examination; Tx?

dx: viral conjunctivitis

tx: supportive (cool compresses, artificial tears) +/- antihistamines for itching (Olopatadine)

3
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Dx? bilateral eye itching, tearing, redness, string discharge, chemosis (conjunctival swelling) with cobblestone appearance to inner/upper eyelids; Tx?

dx: allergic conjunctivitis

tx: topical antihistamines (H1 blockers) (Olopatadine, Pheniramine/Naphazoline, Emedastine), topical NSAID (ketorolac), topical corticosteroids (but s/e of long term use = glaucoma, cataracts, HSV keratitis)

4
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Dx? purulent eye discharge, lid crusting, no visual changes, absence of ciliary injection; Tx?

dx: bacterial conjunctivitis (MC S. aureus, Strep pneumo, H. influenzae)

tx: topical abx (erythromycin, fluoroquinolones, sulfonamides, aminoglycosides); if contact lens wearer cover for pseudomonas w/ fluoroquinolone or aminoglycoside

5
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if bacterial conjunctivitis is found to be chlamydia or gonorrhea what is the tx?

admit for IV and topical abx (ophtho emergency)

-gonoccoccal: IV ceftriaxone + topical

-chlamydia: IV azithromycin

6
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neonatal conjunctivitis is aka? if left untreated can develop what?

ophthalmia neonatorum; corneal ulceration, opacification/scarring, visual impairment/blindness

7
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standard prophylaxis given immediately after birth to prevent ophthalmia neonatorum (neonatal conjunctivitis) includes:

erythromycin ointment, tetracycline ointment, silver nitrate, or povidone-iodine

8
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if ophthalmia neonatorum (neonatal conjunctivitis) develops on day 1 after birth what is the most likely cause? day 2-5? day 5-7? day 7-11?

day 1: silver nitrate (chemical cause- prophylaxis is what can cause the condition)

day 2-5: gonococcal

day 5-7: chlamydia

day 7-11: HSV

9
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orbital (septal) cellulitis is usually secondary to _________ infection in most commonly what age group?

sinus; 7-12y; other causes include dental/facial infxns or bacteremia

10
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what is the most common sinus infection (90%) that causes secondary orbital cellulitis? what organisms are the cause?

ethmoid; S. aureus, Strep. pneumo, GABHS (Strep. pyogenes), H. influenzae

11
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work up/Dx? decreased vision, pain w/ ocular movement, proptosis (bulging eye), eyelid erythema and edema; tx?

dx: orbital cellulitis

work up: CT scan (showing infxn of fat & ocular muscles) or MRI

tx: IV antibiotics (Vanc, Clinda, Cefotaxime, Ampicillin/Sulbactam)

12
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what is the difference b/t orbital (septal) cellulitis and preseptal cellulitis?

preseptal may still have ocular pain, redness and swelling but NO visual changes or pain w/ ocular mvmt (hasn't affected the muscles)

13
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misalignment of the eyes is aka? when does stable ocular alignment present in infants?

strabismus; 2-3 mos

14
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convergent strabismus is aka? divergent strabismus is aka?

convergent: esotropia (deviated inward "cross eyed")

divergent: exotropia (deviated ouward)

15
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a + Hirschberg corneal light reflex test, diplopia, scotomas (blind spots), or amblyopia (lazy eye) are clinical manifestations of what condition? what other tests can be performed?

strabismus; cover-uncover test to determine the angle of strabismus, cover test, convergence testing

16
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how can strabismus be treated?

-patch therapy: normal eye is covered to stimulate and strengthen the affected eye

-eyeglasses

-corrective therapy: if severe or unresponsive to conservative therapy

if not treated before 2 y/o, amblyopia may occur and cause decreased visual acuity that is not correctable

17
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Dx? 1-2 days of ear pain, pruritis in the ear canal, auricular discharge, pressure/fullness, hearing usually preserved, pain with tug test and tragus pressure, auditory canal erythema/edema/debris, recent swimming pool use; MC organisms? Tx?

Dx: otitis externa

MC organisms: pseudomonas, proteus, s. aureus, s. epidermis, GABHS, anaerobes (peptostreptococcus), aspergillus

Tx: 1. protect ear against moisture (isopropyl alcohol and acetic acid) 2. ciprofloxacin/dexamethasone (ofloxacin safe if there is an associated TM perf) 3. Aminoglycoside combo (neomycin/polytrim-B/hydrocortisone -BUT not used if perf suspected bc ototoxic 4. amphotericin B if fungal

18
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malignant otitis externa is osteomyelitis at the skull base secondary to ___________ infxn; MC seen in what pt populations; Tx?

pseudomonas; MC in DM and immunocompromised pts; Tx w/ IV Ceftazidime or Piperacillin + FQ or Aminoglycoside

19
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acute otitis media is an infection of the middle ear, temporal bone and mastoid air cells that is MC preceded by

a viral URI that causes edema of eustachian tube, negative pressure, transudation of fluid and mucus in middle ear that allows for bacterial growth

20
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what are the 4 MC organisms seen in acute otitis media?

Strep pneumo, H. influenza, Moraxella catarrhalis, Strep pyogenes (same as seen in acute sinusitis)

21
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Dx: fever, otalgia, ear tugging in infants, conductive hearing loss, stuffiness, possible drainage from ear, bulging/erythematous TM w/ effusion, dec TM mobility on pneumatic otoscopy; Tx?

dx: acute otitis media

tx: 1st line- amoxicillin, 2nd line- augmentin (amoxicillin-clavulate); if PCN allergy- azithromycin, clarithromycin, erythromycin-sulfisoxazole, trimethoprim/sulfamethoxazole, if PCN adverse effect but not allergy- ceftriaxone, cefdinir, cefixine

don't forget to treat pain as well (ibuprofen or tylenol); can also perform myringotomy (surgical drainage) to relieve pain

tympanostomy if recurrent >4 times in 1 yr

22
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if bullae are seen on the TM of a pt with AOM what should you suspect?

mycoplasma pneumoniae

<p>mycoplasma pneumoniae</p>
23
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Dx? deep ear pain (worse at night), fever, mastoid tenderness and possibly fluctuance (abscess), following AOM infxn; complications?

-dx: mastoiditis (inflammation of the mastoid air cells of the temporal bone- mastoid and middle ear are connected)

-complications: hearing loss, labyrinthitis, vertigo, CN VII paralysis, brain abscess

24
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how is mastoiditis diagnosed and treated?

dx: by CT scan is 1st line test

tx: IV abx (same as w/ AOM- amoxicillin 1st line, augmentin 2nd line, azithromycin for allergy to PCN, ceftriaxone for ADR to PCN) + middle ear/mastoid drainage (myringotomy +/- tympanostomy tube placement- can obtain Cx)

if mastoiditis refractory to tx or complicated = mastoidectomy

25
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what are the 2 auditory examination tests (and what order do you perform them in)?

1st Weber (tuning fork placed on top of head)

2nd Rinne (tuning fork placed on mastoid bone by ear)

26
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if a child has conductive hearing loss in their L ear what will the Weber and Rinne tests show?

Weber: lateralizes to L ear

Rinne: BC > AC

27
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if a child has sensorineural hearing loss in the R ear what will the Weber and Rinne tests show?

Weber: lateralizes to L ear (the normal one)

Rinne: AC > BC (shows normal L ear)

28
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what are the causes of conductive vs sensorineural hearing loss?

conductive: cerumen impaction MC, damage to ossicles (otosclerosis, cholesteatoma), mastoiditis, otitis media

sensorineural: presbyacusis MC (age-related hearing loss), chronic loud noise exposure, CNS lesions (acoustic neuroma), labyrinthitis, meniere syndrome

29
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how is cerumen impaction treated?

1. cerumen softening: hydrogen peroxide 3% or carbamide peroxide (Debrox)

2. aural toilet: irrigation (as long as no TM perf- H2O must be at body temp to prevent vertigo), curette removal, suction

<p>1. cerumen softening: hydrogen peroxide 3% or carbamide peroxide (Debrox)</p><p>2. aural toilet: irrigation (as long as no TM perf- H2O must be at body temp to prevent vertigo), curette removal, suction</p>
30
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Dx? acute ear pain, hearing loss, break in the tympanic membrane, +/- conductive hearing loss, +/- bloody otorrhea, +/- tinnitus & vertigo; Tx?

dx: tympanic membrane perforation

tx: observation (most heal spontaneously) but can do surgical repair; avoid water/moisture/topical aminoglycoside (ototoxic) in ear

<p>dx: tympanic membrane perforation</p><p>tx: observation (most heal spontaneously) but can do surgical repair; avoid water/moisture/topical aminoglycoside (ototoxic) in ear</p>
31
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Dx: sneezing, nasal congestion/itching, clear rhinorrhea, worse in the morning, pale/blue turbinates, +/- nasal polyps, +/- eye, ear, throat involvement; Tx?

dx: allergic rhinitis

tx: 1st line intranasal steroids, oral antihistamines, mast cell stabilizers (cromolyn, nedocromil)

32
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what are the blood vessels involved in anterior vs posterior epistaxis? which is MC involved?

anterior: Kiesselbach's plexus MC type of epistaxis

posterior: palatine artery- this one may cause bleeding in both nares and posterior pharynx

<p>anterior: Kiesselbach's plexus MC type of epistaxis</p><p>posterior: palatine artery- this one may cause bleeding in both nares and posterior pharynx</p>
33
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tx for anterior epstaxis?

-1st line is direct pressure 10-15 min in seated position leaning forward (to reduce vessel pressure)

-topical decongestants/vasocontrictors: phenylephrine, oxymetazoline (afrin)

-cauterization: silver nitrate if cannot control bleeding and site can be seen

-nasal packing: + abx (cephalexin or clindamycin) to prevent toxic shock syndrome

-adjunct therapy: avoid exercise, spicy foods (vasodilation), moisten membranes w/ bacitracin and humidifiers

34
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acute pharyngitis/tonsillitis is MC caused by? other causes?

-viral is MC: adenovirus, rhinovirus, enterovirus, EBV, RSV, influenza A/B, herpes zoster

-bacterial: GABHS (strep. pyogenes "strep throat")

35
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tx for acute viral pharyngitis/tonsillitis

1. fever control: ibuprofen or tylenol

2. hydration

3. bed rest

36
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Dx? pt w/ sore throat, fever >100.4F/38C, pharyngotonsillar exudates, tender anterior cervical LAD, absence of cough and gram stain showing G+ cocci in chains

"strep throat" streptococcal pharyngitis (strep pyogenes)

37
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what are the Centor criteria for diagnosing strep throat?

0-1 pts: no abx or throat Cx (unless 3-14y get Cx anyway)

2-3 pts: Cx

4-5 pts: abx

<p>0-1 pts: no abx or throat Cx (unless 3-14y get Cx anyway)</p><p>2-3 pts: Cx</p><p>4-5 pts: abx</p>
38
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what is the tx for strep throat?

-1st line: PCN G or VK; others include: amoxicillin, augmentin

-if PCN allergic = macrolides (azithromycin, erythromycin, clarithromycin) or if ADR = cephalosporins or clindamycin

39
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what are some complications from strep throat?

-rheumatic fever (preventable w/ abx)

-glomerulonerphritis (not preventable w/ abx)

-peritonsillar abscess, cellulitis

40
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peritonsillar abscess is aka _______ that occurs after

quinsy; tonsillitis -> cellulitis -> abscess formation

<p>quinsy; tonsillitis -&gt; cellulitis -&gt; abscess formation</p>
41
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Dx? dysphagia, pharyngitis, muffled "hot potato voice," drooling, trismus (lock jaw), uvula deviation to contralateral side, tonsillitis, anterior cervical LAD

peritonsillar abscess but need CT scan to differentiate b/t cellulitis vs abscess

<p>peritonsillar abscess but need CT scan to differentiate b/t cellulitis vs abscess</p>
42
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how is peritonsillar abscess treated?

-abx (ampicillin/sulbactam (Unasyn), clindamycin, PCN G + metronidazole) + aspiration or I&D

-tonsillectomy if recurrent strep infxns, peritonsillar infxns, or chronic tonsillitis

43
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thrush is caused by? tx w/?

candida albicans; tx is w/ nystatin mouthwash, clotrimazole troaches or oral fluconazole

<p>candida albicans; tx is w/ nystatin mouthwash, clotrimazole troaches or oral fluconazole</p>
44
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what is the most common cause of acute epiglottitis?

H. influenzae type B infxn (incidence has gone down d/t vaccination); other rare causes are strep pneumo, s. aureus, GABHS (strep pyogenes)

45
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Dx? dysphagia, drooling, distress, fever, odynophagia, inspiratory stridor, dyspnea, hoarseness, muffled voice, tripod position, thumbprint sign on lateral XR, laryngoscopy gold standard showing cherry-red edematous epiglottis; Tx?

dx: acute epiglottitis

tx: maintain airway (comfort and keep child calm, dexamethasone to reduce airway edema, tracheal intubation for severe cases) and supportive mgmt

if bacterial cause is suspected can start on 2nd/3rd gen cephalosporin ceftriaxone or cefotaxime

46
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diaper rash is MC caused by? tx?

candida albicans

tx: topical antifungals, frequent diaper changes to reduce dampness in that area

can also be a contact dermatitis d/t prolonged exposure to urine/feces; tx w/ freq diaper changes, petroleum or zinc oxide

47
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an immediate drug reaction such as urticaria or angioedema that is Ig-E mediated is a type ___ hypersensitivity rxn

type I

48
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a cytotoxic, Ab-mediated drug reaction is a type ___ hypersensitivity rxn

type II

49
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a drug reaction caused by immune antibody-antigen complex deposition is a type ___ hypersensitivity rxn

type III

50
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a delayed (cell mediated) drug reaction like erythema multiforme is a type ___ hypersensitivity rxn

type IV

51
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what is the most common type of cutaneous drug eruption? tx?

exanthematous/morbiliform rash- generalized distribution of "bright-red" macules & papules that coalesce to form plaques- usually begins 2-14 days after medication initiation (abx, NSAIDs, allopurinol, thiazide diuretics)

tx: oral antihistamines

52
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what is the 2nd Mc type of cutaneous drug eruption that occurs w/i minutes to hrs after drug administration? tx?

urticarial or angioedema (type I)- triggers include abx, NSAIDs, opiates, radiocontrast media

tx: systemic corticosteroids, antihistamines

53
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what is the 3rd MC cutaneous drug eruption that is characterized by target lesions? tx?

erythema multiforme (type IV)- triggered by sulfonamines, PCNs, phenobarbital, dilantin

tx: symptomatic therapy

54
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in regards to urticaria, local pinpoint pressure that results in the skin to wheal in that area is called? local rubbing causing urticaria is called?

dermatographism

Darier's sign

55
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how is urticaria treated?

removal of offending agent, antihistamines (H1 blockers), corticosteroids, or H2 blockers as adjuvant therapy (cimetidine, ranitidine, famoidine)

56
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erythema multiforme is MC associated with

HSV, mycoplasma, s. pneumo, sulfa drugs, beta-lactams, phenytoin, phenobarbital, malignancies, autoimmune

57
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red purpuric papules and macules with a hypopigmented rim around the center and a erythematous halo around that span the palms and soles of the feet? same lesions but involving 1 or more mucous membranes (oral, genital, or ocular mucosa) and <10% BSA with no epidermal detachment.

erythema multiforme minor; major

<p>erythema multiforme minor; major</p>
58
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what is the treatment for erythema multiforme?

symptomatic- d/c offending drug, antihistamines, analgesics, steroid/lidocaine/diphenhydramine mouthwash for oral lesions, and systemic steroids if severe

59
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Steven-Johnson Syndrome and TEN are usually due to what offending agents?

MC d/t drug eruptions from sulfa, anticonvulsants, NSAIDs, allopurinol, abx, infxns like mycoplasma, HIV, HSV, malignancy, or idiopathtic

60
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SJS is defined as sloughing of ____% BSA where are TEN is defined as sloughing of ____% BSA

SJS <10%

TEN >30%

61
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Dx? fever, URI, widespread blisters, erythematous/pruritic papules with 1 or more mucous membranes involved with epidermal detachment (+ Nikolsky sign), <10% BSA; tx?

dx: SJS (if >30% = TEN)

tx: treat like severe burn- admit to burn unit, pain control, withdrawal of offending meds, fluid & electrolyte replacement, wound care

+ Nikolsky sign is when rubbing of skin exfoliates the outer layer

<p>dx: SJS (if &gt;30% = TEN)</p><p>tx: treat like severe burn- admit to burn unit, pain control, withdrawal of offending meds, fluid &amp; electrolyte replacement, wound care</p><p>+ Nikolsky sign is when rubbing of skin exfoliates the outer layer</p>
62
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what is the pathophysiology behind the 4 causes of acne vulgaris?

1. inc sebum production (d/t inc androgens)

2. clogged sebaceous glands

3. propionibacterium acne overgrowth (nml flora that overgrows in blocked pores and causes an inflammatory response)

4. inflammatory response

63
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what type of acne are blackheads? whiteheads? papules/pustules? cystic/nodular?

blackheads- open comedones (incomplete blockage)

whiteheads- closed comedones (complete blockage)

papules/pustules- inflammatory

cystic/nodular- severe and often heals with scarring

<p>blackheads- open comedones (incomplete blockage)</p><p>whiteheads- closed comedones (complete blockage)</p><p>papules/pustules- inflammatory</p><p>cystic/nodular- severe and often heals with scarring</p>
64
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what are tx options for mild, moderate, and severe acne vulgaris?

-mild: topical retinoids (for comedone and inflammatory acne), benzoyl peroxide (decreases p. acne growth), topical abx (clindamycin), OCPs (dec androgen production reducing sebum production)

-mod: " + oral abx (tetracyclines such as doxycycline and minocycline), spironolactone (dec androgen production)

-severe: isotretinoin (accutane) works on all 4 pathophys routes. SEs include psych, hepatitis, inc TG/chol, arthralgias, leukopenia, premature long bone closure, dry skin, and highly teratogenic (so have to have 2 - preg tests before starting and then monthly thereafter + 2 forms of birth control while on it)

65
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verruca are caused by _____ and commonly seen on the hands appearing as firm, hyperkeratotic papules b/t 1-10mm with red-brown punctations (thrombosed capillaries that are pathognomonic) with reg or irregular borders; tx?

HPV

tx: cryotherapy (liquid nitrogen), electrocautery, OTC salicylic acid, CO2, laser, intralesional bleomycin, intralesional candida albicans to trigger immune response to area

<p>HPV</p><p>tx: cryotherapy (liquid nitrogen), electrocautery, OTC salicylic acid, CO2, laser, intralesional bleomycin, intralesional candida albicans to trigger immune response to area</p>
66
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Dx? papulopustules on erythematous base around mouth sparing ther vermillion border which may become confluent into plaques with scales; tx?

dx: perioral dermatitis- MC in young women, may have h/o topical corticosteroid use

tx: metronidazole or erythromycin topically, tetracyclines orally (doxy, minocycline), and avoidance of topical steroids

67
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what does the atopic triad of conditions include?

asthma, atopic dermatitis/eczema, and allergic rhinitis - usually people with one of these have another if not all

68
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Dx? erythematous, ill-defined papules/blisters/plaques that later dry and crust over with scales most commonly in flexor creases; tx?

dx: atopic dermatitis (aka eczema)

tx: topical corticosteroids + antihistamines for itching w/ daily application of non-scented lotions/emollients and switching to gentle detergent, topical calcineurin inhibitors another option (tacrolimus, pimecrolimus alternative to steroids but can cause irritation and possible lymphoma/skin cancer risk)

<p>dx: atopic dermatitis (aka eczema)</p><p>tx: topical corticosteroids + antihistamines for itching w/ daily application of non-scented lotions/emollients and switching to gentle detergent, topical calcineurin inhibitors another option (tacrolimus, pimecrolimus alternative to steroids but can cause irritation and possible lymphoma/skin cancer risk)</p>
69
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Dx? sharply defined discoid/coin-shaped lesions of dried patches/plaques of skin commonly seen on dorsum of hands, feet and extensor surfaces; tx?

dx: nummular eczema

tx: same as atopic dermatitis (topical corticosteroids, antihistamines for itching, and hydrating skin techniques)

<p>dx: nummular eczema</p><p>tx: same as atopic dermatitis (topical corticosteroids, antihistamines for itching, and hydrating skin techniques)</p>
70
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poison ivy rash is a type of ________ dermatitis; tx?

contact dermatitis- irritant (others include detergent, chemicals, cleaners, acids, metals) tx is avoidance of irritants & topical corticosteroids

<p>contact dermatitis- irritant (others include detergent, chemicals, cleaners, acids, metals) tx is avoidance of irritants &amp; topical corticosteroids</p>
71
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Dx? an idiopathic cell-mediated immune response with inc incidence with HepC that manifests as purple, polygonal, planar, pruritic papules with fine scales and irregular borders most commonly on flexor surfaces of extremities and skin, mouth, scalp, genitals, nails and mucous membranes that usually resolves in 8-12 mos; tx?

dx: lichen planus

tx: 1st line topical corticosteroids + antihistamines for itching; 2nd line po steroids, UVB therapy, retinoids

<p>dx: lichen planus</p><p>tx: 1st line topical corticosteroids + antihistamines for itching; 2nd line po steroids, UVB therapy, retinoids</p>
72
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when new skin lesions appear at sites of trauma as seen in lichen planus and psoriasis, what is this called?

Koebner's phenomenon

<p>Koebner's phenomenon</p>
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fine white lines on the skin lesions or oral mucosa seen in lichen planus is aka

Wickham striae

<p>Wickham striae</p>
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Dx? a rash that starts as a solitary salmon-colored macule on the trunk that is 2-6 cm in diameter and progresses to smaller, very pruritic 1cm round/oval salmon-colored papules with white circular (collarette) scaling along cleavage lines in a Christmas tree pattern that is confined to trunk and proximal extremities (sparing face) and usually resolved in 6-12 weeks; tx?

dx: pityriasis rosea

tx: symptomatic only: antihistamines for itching, topical corticosteroids, oatmeal baths, UVB if severe and initiated early on

<p>dx: pityriasis rosea</p><p>tx: symptomatic only: antihistamines for itching, topical corticosteroids, oatmeal baths, UVB if severe and initiated early on</p>
75
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tinea versicolor is caused by an overgrowth of normal skin flora, a yeast called __________ that causes hypopigmented scaly patches to appear on trunk, face and extremities.

Malassezia furfur

<p>Malassezia furfur</p>
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how is tinea versicolor worked up and diagnosed (besides PE) and treated?

-KOH prep from skin scraping showing hyphae and spores

-Woods lamp showing yellow-green fluorescence over affected spots

tx: topical antifungals: selenium sulfide, sodium sulfacetamide, zinc pyruthione, and "azoles"

if failed topicals: systemic antifungal therapy with itraconazole or fluconazole

<p>-KOH prep from skin scraping showing hyphae and spores</p><p>-Woods lamp showing yellow-green fluorescence over affected spots</p><p>tx: topical antifungals: selenium sulfide, sodium sulfacetamide, zinc pyruthione, and "azoles"</p><p>if failed topicals: systemic antifungal therapy with itraconazole or fluconazole</p>
77
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what is the 1st line tx for tinea capitis?

capitis: po griseofulvin (or terbinafine, itraconazole or fluconazole 2nd line)

all other tinea/dermatophytosis infxns are treated with topical antifungals unless failed then po antifungals (EXCEPT for onychomycosis- nail fungus)

<p>capitis: po griseofulvin (or terbinafine, itraconazole or fluconazole 2nd line)</p><p>all other tinea/dermatophytosis infxns are treated with topical antifungals unless failed then po antifungals (EXCEPT for onychomycosis- nail fungus)</p>
78
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what are the 3 types of impetigo? which is the MC type?

1. nonbullous- MC - characteristics "honey colored crust" - vesicles and pustules - MC cause is s. aureus, 2nd MC GABHS (strep. pyogenes)

2. bullous- vesciles turn into bullae then rupture into thin crusts - accompanying fever and diarrhea - s. aureus MC cause but rare and if seen will be in newborn/infants

3. ecthyma- ulcerative pyoderma caused by GABHS that will scar with healing - also rare

<p>1. nonbullous- MC - characteristics "honey colored crust" - vesicles and pustules - MC cause is s. aureus, 2nd MC GABHS (strep. pyogenes)</p><p>2. bullous- vesciles turn into bullae then rupture into thin crusts - accompanying fever and diarrhea - s. aureus MC cause but rare and if seen will be in newborn/infants</p><p>3. ecthyma- ulcerative pyoderma caused by GABHS that will scar with healing - also rare</p>
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how is impetigo treated?

-topical mupirocin DOC TID x 10 days - can substitute for Bacitracin too

-if severe or systemic sxs (fever) give systemic antibiotics (cephalexin, dicloxacillin, clindamycin, erythromycin, azithromycin, clarithromycin)

<p>-topical mupirocin DOC TID x 10 days - can substitute for Bacitracin too</p><p>-if severe or systemic sxs (fever) give systemic antibiotics (cephalexin, dicloxacillin, clindamycin, erythromycin, azithromycin, clarithromycin)</p>
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Dx? intensely pruritic papules, vesicles, and linear burrows commonly found in the intertrigous zones (between folds) including web spaces between fingers/toes, and scalp with increased itching at night- in males on scrotum, glans, or penile shaft; tx?

dx: scabies (caused by sarcoptes scabiei) - possible to see eggs/mites on scraping/microscopy w/ mineral oil

tx: DOC Permethrin cream applied from neck down and left on for 8-14 hrs before showing with repeat tx in 1 week + washing all bedding after storing in a bag for at least 72h; Lindane also indicated but more SE of seizure, teratogenic, etc; for pregnant women and infants 6-10% sulfur in petroleum jelly; oral ivermectin if extensive

<p>dx: scabies (caused by sarcoptes scabiei) - possible to see eggs/mites on scraping/microscopy w/ mineral oil</p><p>tx: DOC Permethrin cream applied from neck down and left on for 8-14 hrs before showing with repeat tx in 1 week + washing all bedding after storing in a bag for at least 72h; Lindane also indicated but more SE of seizure, teratogenic, etc; for pregnant women and infants 6-10% sulfur in petroleum jelly; oral ivermectin if extensive</p>
81
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Dx? intense itching of the scalp, papular urticaria on scalp, white oval-shaped capsules at base of hair shafts; tx?

dx: lice (pediculosis)

tx: permethrin cream x 10 min unless infected elsewhere besides head then 8-10 hrs; Lindane cream 2nd line but neurotoxic; oral Ivermectin in refractory cases; wash all bedding/clothes & seal toys in plastic bag x 14 days

<p>dx: lice (pediculosis)</p><p>tx: permethrin cream x 10 min unless infected elsewhere besides head then 8-10 hrs; Lindane cream 2nd line but neurotoxic; oral Ivermectin in refractory cases; wash all bedding/clothes &amp; seal toys in plastic bag x 14 days</p>
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Dx? hair loss that occurs at temporal, mid-frontal, or vertex areas of scalp d/t dihydrotestosterone (DHT); tx?

dx: androgenetic alopecia

tx: minoxidil (rogaine) for recent onset and smaller area & oral finasteride (5-alpha reductase inhibitor aka androgen inhibitor)

I think they meant to put alopecia areata on the topic list instead of this which is autoimmune causing patchy hairloss seen in kids, MC in girls- treated w/ intralesional/topical corticosteroids or topical immunotherapy w/ allergen like squaric acid

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what are the burn BSA percentages of infants/children?

18% entire head, 9% entire arm x 2, 36% entire trunk, 14% entire leg x 2

(in adults, 9% head, 9% arm x 2, 36% trunk, 18% leg x 2, genitalia 1%)

<p>18% entire head, 9% entire arm x 2, 36% entire trunk, 14% entire leg x 2</p><p>(in adults, 9% head, 9% arm x 2, 36% trunk, 18% leg x 2, genitalia 1%)</p>
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in infants/children, <____% TBSA burn is considered a minor burn but >____% TBSA burn is considered a major burn

minor: <5%

major: >20%

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burns are best washed with? cool sterile compresses are used for? chemical burns are cleaned how?

-burns best washed with mild soap and water- disinfectant may inhibit healing

-cool sterile compresses to stop thermal burning but ice NOT recommended

-chemical burns cleaned/irrigated with running water for at least 20 min

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what topical antibiotic is used for 2nd-3rd degree burns? superficial burns?

2nd/3rd: silver sulfadiazine unless <2 mos old, burn on face, or sulfa allergy

superficial: aloe vera and Bacitracin ointment

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IV fluid resuscitation for children with moderate to severe burns is the same as adults using the Parkland formula which is:

LR 4mL/kg/%TBSA given IV first 24 hrs with 1/2 given first 8 hrs then other 1/2 given over the next 16 hrs

REMEMBER this is on top of the daily fluid requirement

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roseola infantum (sixth's disease) is caused by

human herpes virus 6 or 7

<p>human herpes virus 6 or 7</p>
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Dx? a 3 year old comes in with a h/o high fever that lasted 4 days and has broken but now has a rash that is rose/pink colored maculopapular and blanchable that started on the trunk/back and has spread to the face; tx?

dx: roseola infantum (sixth's disease) caused by human herpes virus 6 or 7 (this is the ONLY childhood exanthem that starts on trunk and spreads to face!)

tx: supportive, anti-inflammatory, anti-pyretic (to prevent febrile seizures)

<p>dx: roseola infantum (sixth's disease) caused by human herpes virus 6 or 7 (this is the ONLY childhood exanthem that starts on trunk and spreads to face!)</p><p>tx: supportive, anti-inflammatory, anti-pyretic (to prevent febrile seizures)</p>
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coxsackie virus type A vs type B causes what conditions?

A: hand, foot, mouth disease & herpangina

B: pericarditits/myocarditis & pleurodynia

A & B: aseptic meningitis, rashes, common cold sx

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Dx? mild fever, URI sx, dec appetite, vesicular lesions with erythematous halos in oral cavity (esp buccal mucosa and tongue), exanthem 1-2 days later of vesicular, macular, or maculopapular lesions on distal extremitis (often includes palms and soles); tx?

dx: hand, foot, mouth disease (caused by coxsackie A)

tx: supportive (antipyretics, topical lidocaine)

<p>dx: hand, foot, mouth disease (caused by coxsackie A)</p><p>tx: supportive (antipyretics, topical lidocaine)</p>
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Dx? sudden onset of high fevers, stomatitis (small vesicles on soft palate, uvula & tonsillar pillars that ulcerate before healing), sore throat, lasting 3-5 days; tx?

dx: herpangina (caused by coxsackie A)

tx: supportive (antipyretics, topical lidocaine)

<p>dx: herpangina (caused by coxsackie A)</p><p>tx: supportive (antipyretics, topical lidocaine)</p>
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viral pericarditis/myocarditis in infants and children are most commonly caused by

coxsackie B

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Dx? fever, severe pleuritic chest pain, paroxysmal spasms of chest/abdominal muscles including diaphragm, and HA; tx?

dx: pleurodynia (caused by coxsackie B)

tx: supportive (anti-pyretics, anti-inflammatories)

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Dx? coryza, fever, rash on face/cheeks that appears like slapped cheeks with circumoral pallor 2-4 days, lacy reticular rash on extremities (esp UE) sparing palms/soles that resolves in 2-3 wks, arthralgias, associated fetal loss in pregnancy; tx?

dx: erythema infectiosum (fifth disease/slapped cheek syndrome) caused by parvovirus B19 - do serologies to confirm dx

tx: supportive, anti-inflammatories, anti-pyretics

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in pts w/ sickle cell dz or G6PD deficiency, infection w/ parvovirus B19 may precipitate

aplastic crisis

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Dx/cause? low grade fever, myalgias, HA, parotid gland pain/swelling; tx? what lab work supports dx? prevention?

dx: mumps (paramyxovirus)

tx: supportive, anti-inflammatories

labs: serologies, inc amylase

prevention: MMR vaccine at 12-15 mos and again at 4-6 y/o

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what are some complications of mumps that can appear?

MC in older pts:

orchitis in males (unilateral), oophoritis, encephalitis, aseptic meningitis, MC cause of acute pancreatitis in children, deafness, arthritis, infertility

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Dx? high fever, cough, coryza, conjunctivitis (3 Cs), koplik spots (small red spots in buccal mucosa w/ pale blue/white center, followed 1-2 days later by maculopapular/morbiliform brick-red rash on face beginning at hairline then moving to extremities lasting ~7 days coalescing and darkening then fading from top to bottom; tx?

dx: rubeola (measles) caused by paramyxovirus (same as mumps)

tx: supportive, anti-inflammatories, vitamin A (reduces mortality)

complications: diarrhea, otitis media, PNA, conjunctivitis, encephalitis

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Dx? low grade fever, cough, anorexia, LAD (posterior cervical, posterior auricular), pink/lt red spotted maculopapular rash on face then to extremities that lasts 3 days, small red macules or petechiae on soft palate (Forchheimer spots), in young women +/- transient photosensitivity & joint pains; tx?

dx: rubella (german measles) caused by rubella virus

tx: supportive, anti-inflammatories

generally no complications