Movement analysis of gait

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Last updated 1:50 AM on 6/30/26
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37 Terms

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In normal gait, during the pre-swing phase the knee flexes to 40 degrees, this is achieved by:

  • activation of rectus femoris and sartorius during pre-swing

  • activation of iliopsoas during terminal stance

  • activation of semimembranosus during pre-swing

  • none of the above

none of the above - the knee flexes passively

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Phase 5: Pre-swing (aka toe off)

  • the second double stance interval in the gait cycle

  • ground contact by the blue leg causes the red leg to increase ankle plantar flexion, increase knee flexion, and decrease hip extension

  • hip: 10 deg hyperextension - adductors

  • knee: 40 deg flexion - no MM activity

  • ankle: 15 deg pf - no MM activity of PF - toe rocker)

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What is a possible cause of increased plantar flexion seen during stance phase?

  • excessive knee flexion

  • weak df

  • hip flexion contracture

  • weak hip abd

  • hip flexion contracture - DF only active in loading - tight hip flexors = torso flexes, hips stay back, knee extends, ankle pf

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Three functional tasks of normal gait

  • weight acceptance

  • single leg stance

  • limb advancement

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weight acceptance

  • initial contact (IC) and loading response (LR)

  • shock absorption, forward progression and stability

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single leg stance

  • midST, terminal stance

  • forward progress & stability

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limb advancement

  • PSw, ISw, MSw, TSw (all swing phases)

  • foot clearance and limb advancement

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gait deviations

aspects of gait that are not normal

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gait parameters

parts of gait we can measure

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Ranchos Los Amigos observation gait analysis

  • compare to norm

  • perform segmentally

  • ID major deviations during swing and stance phase (use RLA form)

  • comment on overall gait parameters (speed, step length, path deviation)

  • add variations to bring out deviations

  • hypothesize what impairments may be causing these deviations (atypical movements)

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Plantar flexion weakness - stance phase

  • extensor thrust in MSt to TST (usually chronic)

  • excessive df (crouched: knee flex) (acute) - knee buckling

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Extensor thrust

snapping knee back to genu recurvatum

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Quadriceps weakness - stance phase

  • hyperextension of the knee early in stance (IC or LR)

  • not very common - quads are first to come back after stroke

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hip extensor weakness - stance phase

  • excessive hip flexion at IC, LR, beginning of midstance

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hip abductor weakness - stance phase

  • trendelenburg/reverse trendelenburg

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dorsiflexion weakness - stance phase

  • foot flat contact or foot slap in LR (beginning of stance)

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anterior tib weakness - swing phase

  • poor clearance - steppage gait

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knee flexor weakness - swing phase

difficulty at initial swing

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knee extensor weakness - swing phase

doesn’t achieve full extension at TSwing

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hip flexor weakness - swing phase

poor swing and limb clearance

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absent or decreased HS in stance limb

impairments:

  • PROM restriction or spastic pf

  • weak df

  • sensory dysfunction

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excessive df in stance limb

  • weak pf

  • comes with increased knee flex

  • hamstring contracture

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excessive pf in stance limb

  • PROM restriction, spastic, weak pf

  • hip flex contracture

  • weak quads (early)

  • spastic quads

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knee hyperextension (thrust) in stance limb

  • PROM restriction, spastic, weak pf - midstance

  • quad weakness - loading

  • hip flex contracture

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knee wobble in stance limb

  • weak pf

  • quad weakness

  • sensory dysfunction

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Impaired motor control

  • lack of selective control

  • alterations in tone

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Lack of selective control

  • co-activation

  • emergence of primitive synergy patterns

    • mass flexion or extension patterns (stage 3 motor recovery)

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Alterations in tone

hypotonicity or hypertonicity

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you observe an extensor thrust (recurvatum) of the knee that occurs during midstance. A possible cause would be?

  • hamstring weakness

  • PF weakness

  • quadriceps weakness

  • all of the above

PF weakness - calf muscles don’t fire, so the tibia snaps back

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causes of gait deviations during WA and SLS

excessive knee hyperextension or extension thrust

  • recurvatum

  • pf spasticity

  • pf contracture (PROM restriction)

  • weak (poor motor control) of PF if occurring at MSt

  • weak quads if occurring early in cycle

  • hip flexion contracture

  • quadriceps hypertonicity

  • impaired proprioception at knee

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terminal stance places the __________ at any joint during the gait cycle

greatest muscle demand

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terminal stance - calf muscle activity _____ and allows _______

peaks, the heel to rise (stops the tibia from going forward)

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terminal stance - tibia and ankle become ______ and _______

a rigid lever, push knee into flexion

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terminal stance - where stance and _____ are linked

swing

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if terminal stance does not happen correctly, neither will ______

pre swing

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Forward trunk lean during WA or SLS - causes

  • intentional to reduce demand on quadriceps

  • necessary to progress over an excessively pf ankle

  • use of assistive devices

  • limited trunk ext ROM (spinal stenosis)

  • weak hip extensors

  • hip flexion contracture (restored PROM)

  • to substitute visual input for impaired proprioception

  • abdominal pain

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movement analysis of task

slide 19