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primary care
normal check up
examine all systems
refer to specialists as needed
liver
cleans blood
digests food
stores energy
secretes bile
gallbladder
stores bile
pancreas
digestion of fats, carbs, proteins
produces insulin and glucagon
stomach
storage/mixing of food
partial digestion
small intestine
primary site for digestion and absorption of food (chyme)
large intestine
chyme converted to feces
reabsorption of water
spleen
storage and regulation of RBC
produces antibodies and lymphocytes
adrenal glands
secrete epinephrine, norepineprhine, cortisol, estrogen, aldosterone, and androgen
kidnyes
filter waste, ions, and drugs in blood
ureters/bladder
transport urine to bladder/storage of urine
palpitation
physical assessment
abdominal musculature
rectus abdominus: stabilization/lumbar flexion; most superficial layer
internal oblique: stabilization/ipsilateral rotation
external oblique: stabilization/contralateral rotation
transverse abdominus: protect organs/spinal stabilization/forced expiration; deepest layer
quadrants of abdomen
midline extends from center of sternum through pubic bone
horizontal line extends through umbilicus
right upper, right lower, left upper, left lower
acute appendicitis
mild to severe pain in lower right quadrant w nausea, vomiting, low grade fever
inflammation of appendix does not mean burst but still medical emergency
mononucleosis
viral disease (epstein-barr virus) severe fatigue, sore throat, headache, swelling of spleen
athlete out for 4-6 wks bc of spleen
blow to solar plexus
paralysis diaphragm, stopping respiration, getting wind knocked out of you
type 1 diabetes
pancreas produces little to no insulin
difficult to manage —> need to keep blood sugar regulated w sugar and glucagon
type 2 diabetes
developed resistance to insulin due to prolonged high levels
specific to type 2: numbness/tingling of hand and/or feet (lose sensation w age), freq infections, slow healing wounds
general diabetes symptoms
increased thirst, freq urination, and unexplained weight loss
assessment
auscultation 1st: use stethoscope; need to do before touching bc touching creates artificial sounds (normal: gurgling, rumbling, clicking; abnormal: loud, reduced, absent)
palpitation: stand on left side, 4 regions start away from pain, feel for pain/rigidity/masses
percussion: dull- over solid organs; tympanic- over air filled structures
assessments
looking at pee color and stool consistency (watery is fatty diet or stomach issues)
lungs flow
nose/mouth > throat > trachea > bronchi > bronchioles
pneumothorax
collapsed lung
pleural cavity becomes filled w air
hemothorax
punctured lung
presence of blood w/in pleural cavity
asthma
spasm of smooth muscle in bronchioles, causing narrowing of airway
exercise induced bronchospasm (asthma)
difficulty getting air OUT
pneumonia
infection of lung and bronchioles cause by virus bacteria or fungus
vocal cord dysfunction
difficulty getting air IN (inhalers would not work)
assessing and reporting respiration
rate: breaths per min (BPM), count # of breaths in 60s
rhythm: regularity
depth: shallow, normal, deep
effort: labored vs quiet breathing (accessory musculature)
color: pallor (pale) and cyanosis (blue lips- cold or not getting enough o2)
posture: hands on knees, holding ch
respiration abnormalities
panic attack: increase rate, normal rhythm, shallow
hyperventilation: involves shoulders and rib flares
tachypnea
rapid breathing: > 24 breaths/min
hyperpnea
tachypnea w very large breaths
bradypnea
slow breathing <12 breaths/min
dyspnea
difficulty breathing: at rest, w/ activity, changes w position, temp?
can be w/ asthma when lying down
hypopnea
shallow, slow breaths
orthopnea
shortness of breath when lying down
cough
characteristics: dry, barking, productive
timing: worse at night (asthma/acid reflux), after exercise
phlegm colors
white: allergies/viral infection
green: bacterial infection (pneumonia)
red: mean lung injury or malignancy
brown: lung disease (smoking)
lung ausculatation
posterior
r/l upper lobes b/w C7 (moves)-T3
r/l lower lobes b/w t4-t10
pre participation physical evaluations
purpose of PPE: facilitate and encourage safe participation not to exclude athletes (recommended by AAFP)
identify any potential or correctable conditions that may impair athlete’s ability to full perform
student-athletes required to have comprehensive PPE upon entry into middle of highschool or transfer
annual updates on comprehensive health history, problem focused areas, vital signs
PPE required for student entrance into intercollegiate athletics
history
PPE started as a way to screen soliders
NCAA recommends PPE but details up to institution
PPE could be only contact adolescents have w medical provider in a year
goals of PPE
determine general physical and psychological health (new)
eval for conditions that may be life threatening or disabling (scoliosis, heart disease, sickle cell)
eval for conditions that predispose injury or illness
provide opportunity for dicussion of health and lifestyle issues
serve as entry point into healthcare system
key concepts of PPE
medical history effective at identifying 75% of problems affecting sport participation
PPE serves as sole source of med eval for 30-88% of adolescents annually
0.3-1.3% denied participation (heart-related and c-spine instability)
3.2-13.9% requiring further eval
PPE vs well-child
ppe: things that predispose to injury
well-child exam: normal check up of overall health
family and personal health history PPE
med history detects majority of gen and musculoskeletal conditions
barriers: health literacy and limited english so difficult to interpret ?s
scared to tell truth
physical exam of PPE
height, weight
vitals
ENT
auscultation
abdominal
genitalia
skin
female athletes
other PPE sections
mental health portion
physical fitness testing
medications
special medical tests PPE
optional
dental, vision, cardiac, neurologic, gen med (ferritin, urinalysis, sickle cell, diabetes)
sudden cardiac death
PPE prevents
males higher than females, black higher than white, basketball highest incidence
etiology: most common finding in athletes w SCD was structurally normal heart w no signs of disease
athletic PPE
both AHA and ESC recommend screening all athletes w a comprehensive history and physical
controversy exists abt whether or not to perform ECGs (electrical actvity in heart) on all athletes
hypertrophic cardiomyopathy
thickened cardiac muscle causes decreased cardiac output
heart murmur
abnormal, periodic sound due to changes in anatomy of heart valves
commotio cordis
traumatic blow to chest during heart repolarization—> stops heart
15-30msec before twave
heart cycle
r atria
r ventricles
lungs (oxygenate)
l atria
l ventricles
body
continuous cycle w each beat
sinus node (SA)
loc in r atria and generates electrical stimulus
atrioventricular node (AV)
impulses slowed down for very short pd
bundle of his
bundle of his pathway leading to ventricles
divides into right and left pathways called bundle branches
purkinje fibers
stimulate l and r ventricles
electrocardiogram
graphic record of direction and magnitude of electrical activity generated by depolarization and repolarization of atria and ventricles
can suggest or detect cardiomyopathies, ion channelopathies, myocarditis, and ventricular pre-excitation
other causes of SCD in young athletes (ex: anomalous coronary arteries, premature coronary artherosclerosis and aortopathies) not readily detected
electrical activity of heart
p wave: depolarization of atrial myocardium
QRS complex: depolarization of ventricular myocardium
t wave: repolarization of ventricular myocardium
cardiac auscultation
A- aortic valve (semilunar); r sternal border, 2nd intercostal space
P- pulmonary valve (semilunar); l sternal border, 2nd intercostal space
E- erbs point; l sternal border; 3rd intercostal space
To- tricuspid valve (AV); l sternal border, 4th intercostal space
Man- mitral valve (AV); midclavicular line, 5th intercostal space