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CAD assessment
ischemia to heart muscle leading to angina pectoris (chest pain) or sudden death
epigastric distress, pain radiating to jaw/L arm, impending doom, SOB, fatigue, weakness, diaphoresis, dizzy'/lightheaded
women = nausea, indigestion, palpations, numbness
CAD prevention
controlling cholesterol abnormalities
promoting smoking cessation
managing HTN
controlling diabetes
HTN limits
SBP > 140 and DBP > 90 based on 2+ separate assessments obtained 1-4 weeks apart
HTN assessment
none other than elevated BP
organ damage S/S: angina, SOB, altered speech/vision, epistaxis, headaches, vertigo, balance problems, fainting, nocturne, facial flushing
HTN treatments
lifestyle changes → wt loss, exercise, no caffeine/smoking, reduce fats, salts, and alc
pharmacological
diuretics: decrease blood volume
ACE inhibitors: decrease peripheral resistance
ARBs: decrease peripheral resistance
CCBs: vasodilation and slows rate of conduction
beta blockers: lowers HR and force of contractions
antihypertensive warnings
pt may report feeling worse on meds
potential for hypotension or F/E loss (diuretics)
reflex tachycardia compensation
suddenly stopping causes rebound HTN
elders susceptible to orthostatic hypotension and hyperkalemia
PAD assessment
intermittent claudication, pain relieved by rest in dependent position, numbness/birning in feet, decreased LE cap refill, diminished LE pulses, cold and cyanotic LE, pallor with elevation, dependent rubor, muscle atrophy, ulcers
PAD interventions
gradual exercise
promote vasodilation and avoid constriction
no restrictive garments
elevate only to heart level
PAD pharmacological agents
antiplatelets: prevents platelet aggregation, reduces blood viscosity
statins: promote dilation → avoid grapefruit!
analgesics: reduce pain
PVD assessment
pain worse when standing and subsides with elevation, leg cramps, edema of LE, thickening/browning of LE, shallow ulcers, varicose veins
PVD interventions
elevate legs
use compression socks
exercise
PVD pharmacological agents
diuretics: reduces edema
anticoagulants: DVT prevention
hemorrheologics: decreases blood viscosity to improve flow and reduce inflammation
DVT assessment
pain/tenderness in affected area, extreme SOB/chest pain for PE, redness, increased skin temp, elevated d-dimers
DVT interventions
monitor anticoagulant therapy and bleeding
elevate extremity to heart level
administer analgesics and anticoagulants
compression therapy to unaffected extremity
COPD assessment
dyspnea, chronić cough, recurrent wheeze, thin stature with decreased muscle mass, tripod position, barrel chest, rapid and shallow respirations with crackles
COPD pharmacological agents
bronchodilators: relaxes smooth muscles lining airway
B agonists and antocholijergics
corticosteroids: anti-inflammatory
mucolytic agents
antibiotics
airway enzyme inhibitor: suppresses enzyme causing airway inflammation
emphysema assessment
dyspnea early in disease, anxious and pursed lip breathing, hyperventilation, pink complexion, hunched, barrel chest, short and jerky sentences, very thin (increase WOB), hypoxemic
chronic bronchitis assessment
SOB, chest discomfort, dyspnea (later in disease), excessive sputum with recurrent cough, wheezing, dusky appearance, delayed cap refill, clubbing, hypoxia, increased use of accessory muscles, peripheral edema, obese
chronic bronchitis interventions
smoking cessation, controlled coughing, increased fluids, breathing treatments, COPD meds
asthma assessment
cough, dyspnea, wheezing
exacerbations: chest tightness, diaphoresis, tachycardia, hypoxemia, central cyanosis
asthma pharmacological treatment
quick relief
albuterol
ipratropium
IV systemic corticosteroids (prednisone)→ first choice for exacerbation
long term
inhaled corticosteroids → first choice
ICS + LABA
bronchodilators
TB assessment
night sweats, wt loss, fatigue, cough lasting 3+ weeks, coughing up blood/sputum
TB interventions
airborne precautions
TB skin test to all within contact
explain med regiment → 6-12 months with anti-TB agents; do NOT discontinue
osteoporosis assessment
low vitamin D and protein, high Ph intake, acute back pain, loss of or decreasing height, unstable gait/restricted movement, prolonged immobility, malabsorption disorder
osteoporosis intervention
diet rich in calcium and vitamin D (supplements)
exercise: weight bearing and aerobics
preventing injury and relieving pain
bisphosphonates: first line → decreases number of osteoclasts and inhibits bone reabsorption
OA
degenerative disease with no systemic involvement → localized inflammatory response; small joints
pain with activity that improves at rest
high ESR and CRP
OA assessment
joint stiffness, crepitus with movement, joint enlargement/effusion, limping gait
RA
inflammatory disease
pain at rest or in morning
affects large joints with systemic involvement
positive rheumatoid factor
RA assessment
early: fatigue/joint discomfort, inflammation, fever, paresthesia, anorexia
late: swan neck and boutonnières, muscle atrophy, anemia, skin lesions, enlarged lymph nodes, inflammatory complications (excess cortisol)
OA interventions
adaptive devices
heat with tenderness
balance activity with rest
NSAIDS, COX-2, corticosteroids, acetaminophen
RA interventions
control pain
splits (protect joints)
exercise program
methylprednisolone, ibuprofen, DMARD therapy
hemorrhage assessment
hypotension, weak/thready/rapid pulse, oliguria, restlessness, anxiety, disorientation, cold and clammy skin, deep and rapid respirations
hemorrhage interventions
prevention is key!
stop bleeding and replace blood volume
atelectasis assessment
decreased lung sounds, dyspnea, cyanosis, crackles, restlessness
atelectasis interventions
deep breathing with splints → promote full aeration of lungs
incentive spirometer
turning Q2 hrs and ambulation
maintain hydration
pneumonia assessment
dyspnea, fever, chills, cough with sputum, crackles, wheezes, chest pain