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obstructive pulmonary disease
asthma
COPD
cystic fibrosis
COPD
preventable, treatable, but often progressive disease characterized by persistent airflow limitation
associated with enhanced chronic inflammatory response in airways and lungs
primarily caused by cigarette smoking and other noxious particles and gases
previously considered a combination of chronic bronchitis and emphysema
COPD pathophysiology
long-term disease that causes ongoing inflammation in the airways, alveoli, and blood vessels in lungs
makes it hard ti breathe out air fully
lungs lose natural elasticity
airways become blocked due to too much mucus, edema of lining, and tightening of muscles around airways (bronchospasm)
clinical manifestations of COPD
chronic intermittent cough (often first symptom)
increased anteroposterior diameter of chest due to hyperinflation (barrel chest)
breath sounds
decreased
wheezing, rales, or ronchi
prolonged expiration
signs of advanced disease include
pursed lip breathing
neck vein distention/peripheral edema (may indicate pulmonary HTN)
cachexia, fatigue, anorexia, weight loss
diagnostic studies for COPD
history and physical exam
spirometry
cxr (not diagnostic)
serum a1-antitrypsin levels
ABGs
6 minute walk test
COPD assessment test (CAT) or clinical COPD questionnaire (CCQ)
clinical course of COPD
slowly worsens over time
irreversible airflow limitation
exacerbations
sudden worsening of symptoms (triggers like infection, air pollution)
may require hospitalization
often increase in frequency as disease progresses
disease stages (based on severity)
mild → moderate → severe → very severe
based on lung function tests
COPD related nursing problems
activity intolerance
ineffective breathing pattern
ineffective airway clearance
impaired gas exchange
anxiety
poor nutritional status
COPD related nursing implications
smoking cessation = single most effective and cost effective intervention to reduce risk for COPD and disease progression
teach influenza and pneumonia vaccine adherence
teach early detection of respiratory infections
inhaler therapy = mainstay for COPD self-management/adherence is critical
long acting beta agonist (mainstay of treatment)
long acting muscarinic antagonist
inhaled corticosteroids
O2 administration - target O2 sat 88-92%
teach tripod position and pursed lip breathing
review cough techniques
teach energy conservation and relaxation exercises
will need extra calories for increased WOB
provide psychosocial support
management of acute exacerbation
worsening of dyspnea, cough or sputum beyond typical variations that require a change in treatment plan
oxygen is mainstay of hospital treatment when hypoxemia is present
keep O2 sat >90%
oxygen should never be withheld
noninvasive ventilation (positive pressure ventilation) when hypercapnic
bronchodilators
chest physiotherapy
possibly antibiotics