Focus on bringing unconscious struggles into consciousness (free association, dream analysis)
Insight: increase patient’s understanding of their own psychological processes
Encouragement of personal growth through self-understanding → congruence
Safe and comfortable setting, empathy, reflective listening
unconditional positive regard
Incorporates techniques from both cognitive therapy and behavioral therapy to correct faulty thinking and change maladaptive behaviors
treats mood disorders like depression
disorders typically diagnosed in childhood, yet often continue throughout one’s lifespan
ADHD, Schizophrenia, autism, etc
Little to no eye contact
Failure to develop a normal theory of mind
No language abilities or delayed acquisition of language to normal skills
Difficulty maintaining conversations with others because of their social skills deficits
High levels of repetitive, routine behaviour
Extremely limited and focused preoccupations
Unusually increased or decreased sensitivity to stimuli
Strong evidence for genetic connection
Environmental factors interacting with genetic factors associated with ASD during sensitive periods of brain development
Parental age
The inability to maintain sustained attention or on-task behaviour for an age-appropriate length of time (problems completing and organizing work, following instructions)
Engaging in structural activities is challenging (waiting in line), behaviour is noisy, active, and appear to take action without thinking
Genetics play a significant role in the development of ADHD
Environmental factors: lead contamination, low birth weight, and prenatal exposure to tobacco, alcohol, and other drugs
Underactive frontal lobes or smaller amygdala, basal ganglia, and hippocampus
excess in behaviour
Delusions: false personal beliefs based on incorrect inferences about reality
Hallucinations: false sensory experiences
Disorganized Speech: loosening of associations; speech pattern in which thoughts are disorganized or meaningless
Disorganized Behaviour: disorganized or abnormal motor behaviour, e.g., catatonia
deficits in functioning, harder to treat
Isolation, withdrawal
Apathy
Blunted emotion
Slowed, monotonous speech
Pharmacological: antipsychotics are very effective
Side effects: tardive dyskinesia (results in uncontrollable sudden erratic motor behaviors
Second-gen meds: clozapine (lower risk of tardive dyskinesia)
No effect on negative symptoms
lithium, used for treatment
Patients tend to refuse treatment as it will dampen their ability to emote
characterized by lengthy periods of depressed mood, loss of pleasure in normal activities, disturbances in sleep and appetite, difficulty concentrating, feelings of hopelessness, and possible thoughts of suicide
presence of depressive mood and anhedonia
unrealistic fear of open spaces, being outside the home alone, or being in a crowd
often claustrophobic
Autonomic system arousal
Worry/anxiety/tenseness
Restlessness
Excessive startle response
Cognitive Factors
attention to and perception of threat
Ambiguous stimuli
interpretation of bodily sensations (panic disorder)
Genetics; inhibited temperamental style
Fear circuitry dysfunction (amygdala, prefrontal cortex)
Problems with the hypothalamic-pituitary-adrenal (HPA) axis → the system responsible for the release of cortisol into the bloodstream during periods of stress
Anxiolytics: xanax → increase GABA
Cognitive-Behavioural Therapy
Exposure Therapy (phobias)
Strong genetic vulnerability, childhood trauma
Classical (anxiety paired to some event, originally linked to one thing grows to many other things) and operant conditioning (behaviours linked with relief)
Genetic component
Structural and functional differences in the brain → frontal lobe dysfunction, enlarged ventricles
Environmental stress → socioeconomic status, prenatal environment
Drug use (THC)