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129 Terms

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Elective surgery

Planned in advance (e.g., cosmetic surgery, cataract removal).

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Urgent Surgery

Needs to be performed soon but is not an emergency (e.g., gallbladder removal due to cholecystitis).

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Emergency Surgery

Performed immediately to save life or prevent serious complications (e.g., appendectomy for ruptured appendix).

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Minor Surgery

Low risk, minimal complications (e.g., skin biopsy, removal of warts).

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Diagnostic

Confirms or determines a condition (e.g., biopsy).

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Curative

Removes the cause of a condition (e.g., tumor removal).

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Palliative

Relieves symptoms without curing (e.g., colostomy for bowel obstruction).

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Reconstructive/Restorative

Restores function or appearance (e.g., plastic surgery after burns).

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Transplant

Replaces malfunctioning organs (e.g., kidney transplant).

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Cosmetic

Enhances appearance (e.g., rhinoplasty).

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Preoperative Phase

Begins when the decision for surgery is made and ends when the patient is transferred to the operating room. It includes assessments, patient education, and preparation.

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Intraoperative Phase

Starts when the patient enters the operating room and ends when they are transferred to the post-anesthesia care unit (PACU).

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Postoperative Phase

  • Begins after surgery and continues until full recovery. It is divided into:

    • Immediate postoperative phase: Monitoring in PACU.

    • Intermediate postoperative phase: Hospital recovery.

    • Extended postoperative phase: Ongoing rehabilitation and home care.

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Identify essential aspects of the pre-operative assessment.

  • Medical History: Chronic conditions (e.g., diabetes, hypertension), allergies, past surgeries.

  • Physical Examination: Vital signs, respiratory and cardiac status, hydration level.

  • Laboratory Tests: CBC, electrolytes, coagulation studies.

  • Medication Review: Blood thinners, insulin, steroids.

  • Psychosocial Assessment: Anxiety, coping mechanisms, support system.

  • Informed Consent: Ensuring the patient understands the procedure, risks, and benefits.

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Preoperative Phase

  • Educate the patient on the procedure and expectations.

  • Ensure informed consent is obtained.

  • Complete physical and psychological assessments.

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Intraoperative:

  • Maintain sterility and assist surgical team.

  • Monitor patient’s vital signs and anesthesia effects.

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Postoperative:

  • Monitor for complications (bleeding, infection).

  • Manage pain and encourage early ambulation.

  • Educate on wound care and follow-up.

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Describe essential preoperative teaching including; pain control, leg exercises, coughing and deep breathing exercises.

  • Pain Control: Explain pain management options (analgesics, PCA pump).

  • Leg Exercises: Prevent deep vein thrombosis (ankle pumps, leg raises).

  • Coughing & Deep Breathing: Prevent respiratory complications like pneumonia (incentive spirometry, splinting the incision with a pillow).

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Describe essential aspects of preparing a person for surgery, including skin preparation

  • Fasting (NPO Status): Usually 6-8 hours before surgery.

  • Bowel Preparation: If required (e.g., for colorectal surgery).

  • Skin Preparation:

    • Cleansing with antiseptic solution (chlorhexidine or betadine).

    • Shaving (if required) using clippers to reduce infection risk.

  • Preoperative Medications: Sedatives, antibiotics, or anti-nausea drugs.

  • Marking of site to prevent wrong site surgery

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Review the purpose of pre-operative medications and nursing considerations

  • Sedatives (e.g., benzodiazepines like midazolam): Reduce anxiety and induce relaxation.

  • Analgesics (e.g., opioids like morphine): Manage preoperative pain.

  • Anticholinergics (e.g., atropine): Reduce secretions to prevent aspiration.

  • Antiemetics (e.g., ondansetron, metoclopramide): Prevent nausea and vomiting.

  • Proton Pump Inhibitors (e.g., pantoprazole) or H2 Blockers (e.g., ranitidine): Reduce stomach acid and prevent aspiration pneumonia.

  • Antibiotics (e.g., cefazolin): Given prophylactically to reduce the risk of surgical site infections.

  • Nursing Considerations:

    • Verify the correct medication, dosage, and timing.

    • Assess for allergies and contraindications.

    • Educate the patient on medication effects.

    • Monitor vital signs for any adverse reactions.

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Anaesthetic Nurse

The anaesthetic nurse assists the anaesthetist in preparing and administering anaesthesia, ensuring patient safety during induction, maintenance, and recovery.

  • Participate in patient identification & other processes outlined in ‘Surgical Safety Checklist’.

  • Advocate for patient.

  • Collaborate with & assist anaesthetist.

  • Anticipate & provide equipment & supplies.

  • Prepares & checks anaesthetic machine, equipment, IV & arterial lines as required.

  • Assist in maintaining patient airway.

  • Applies monitoring devices.

  • Understands, prepares & assists in any anaesthetic emergencies.

  • Monitors airway devices, ETT cuff measurements, invasive pressures & all other equipment relating to patient observations.

  • Measures & documents blood, fluid & other loses in patient's fluid balance chart.

  • Assists with patient transfer before & after surgery.

  • Collaborates with post-anaesthesia care unit staff

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Circulating Nurse (Scout Nurse)

The circulating nurse works outside the sterile field, coordinating activities and ensuring a smooth surgical process.

  • Participates in processes outlined in surgical safety checklist.

  • Advocates for patient.

  • Assists to prepare room ensuring that all supplies & equipment are available.

  • Anticipates needs of surgical team before & during surgery.

  • Monitors draping procedure & intraoperative environment for breaches in aseptic technique & initiates corrective action.

  • Prepares, records, labels & sends blood pathology

  • Ensures correct handling & labelling of surgically removed human tissue & explanted items.

  • Performs & documents surgical count with instrument nurse.

  • Documents intraoperative nursing care & medications used by operative team & patient outcomes.

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Instrument nurse (Scrub nurse)

The scrub nurse works within the sterile field, directly assisting the surgeon by handling surgical instruments.

  • Participates in process outlined in surgical safety checklist

  • Advocates for patient

  • Prepares instrument table & organises sterile equipment in a functional manner for surgery

  • Anticipates needs of surgical team prior to & during surgery

  • Works directly with surgical team

  • Adheres to, & maintains, surgical aseptic technique throughout procedure

  • Monitors breaches in surgical aseptic technique & initiate corrective action

  • Assists with skin prep & draping of surgical site

  • Passes equipment to surgeon & assistants by anticipating their needs

  • Performs surgical count with circulating nurse

  • Ensures correct handling & labelling of surgically removed human tissue & explanted items

  • Documents intraoperative nursing care & patient outcomes.

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Safety and risk management requirements

  • Maintain a sterile field and aseptic technique.

  • Prevent surgical site infections through proper hand hygiene and sterile draping.

  • Ensure correct patient positioning to prevent pressure injuries.

  • Perform surgical time-outs to confirm patient identity, procedure, and site.

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Specific pre-, intra- and post-operative communication for patients

  • Preoperative: Educate the patient, answer questions, obtain informed consent.

  • Intraoperative: Communicate with the surgical team, document findings, and monitor the patient’s condition.

  • Postoperative: Provide handover to PACU staff, ensure smooth transition to recovery.

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Differentiate between each type of anaesthesia and their purposes

  • General Anaesthesia: Induces unconsciousness and complete muscle relaxation (e.g., for major surgeries like open-heart surgery).

  • Regional Anaesthesia: Blocks sensation in a specific area while the patient remains conscious (e.g., epidural for childbirth).

  • Local Anaesthesia: Numbs a small area for minor procedures (e.g., lidocaine injection for suturing).

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Identify potential post-anaesthesia complications and develop management strategies

  • Respiratory Issues: Airway obstruction, hypoxia.

  • Cardiovascular Instability: Hypotension, arrhythmias.

  • Nausea and Vomiting: Common with general anaesthesia.

  • Delayed Awakening: Due to prolonged drug effects.

  • Management Strategies:

    • Monitor vital signs and oxygenation.

    • Provide antiemetics and IV fluids.

    • Ensure airway patency and assist with ventilation if needed.

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Examine equipment used in airway management including endotracheal tubes (ETT), laryngeal mask airway (LMA), oropharyngeal and nasopharyngeal airway adjuncts.

  • Endotracheal Tube (ETT): Inserted into the trachea for mechanical ventilation.

  • Laryngeal Mask Airway (LMA): Sits over the larynx to maintain an open airway.

  • Oropharyngeal Airway (OPA): Prevents airway obstruction by keeping the tongue from blocking the throat.

  • Nasopharyngeal Airway (NPA): Inserted through the nostril to maintain airway patency.

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Discuss common respiratory surgical/procedural interventions for airway disruption.

  • Tracheostomy: Creating an opening in the trachea for long-term ventilation.

    • Acute airway obstruction is a medical emergency-insertion of a tracheostomy or similar airway may be required

    • Tracheostomy—a surgically created stoma (opening) in trachea, performed to establish an airway:

      • Bypassing an upper airway obstruction.

      • Facilitating secretion removal.

      • Allowing for long-term mechanical ventilation.

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Immediate post-anaesthetic phase assessment A-E

  • A(irway): Patency & adequacy

  • B(reathing): Adequacy of ventilation/oxygenation

    • Arterial oxygen saturation

    • Adequacy of ventilation (CO2 increase- no adequate breath)

      • Respiratory rate, rhythm, depth

      • Use of accessory muscles (neck, diaphragm, positioning, abdominal muscles-rib)

      • Air entry & breath sounds

  • C(irculation): Adequacy of circulation

    • Heart rate & rhythm

    • Peripheral pulses amplitude & equality

    • Blood pressure

    • Capillary filling time (capp. refill) & skin warmth

    • Fluid status

      • Intake & output (estimated blood loss from surgery) (urine output anything less then 30ml concerning)

      • IV infusions

      • Signs of dehydration/fluid overload

  • D(isability): Neurological function

    • Level of consciousness (AVPU +/- GCS) (check with baseline)

    • Presence of protective reflexes (e.g. gag, cough, swallow)

    • Duration of emergence form unconsciousness

    • Ability to move extremities/limb power

    • Blood glucose

    • Pain

  • E(xposure):

    • Condition of operative site

      • Status of dressing

      • Drainage (type, amount, colour)

    • Drains

      • Patency

      • Type

      • Suction on/off

    • Body temperature

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Postoperative care- lifespan and other considerations

  • Children:

    • Infants & young children- may not state their pain levels-can use other signs-crying, fussiness, increased heart rate & BP, agitation (FLACC-face, legs, arms, cry, consolability)

    • Good pain control help facilitate healing process & recovery

    • Some PACU’s have parents in area when child is waking & stable-calms both child & can reduce parent anxiety

  • Older adults

    • Have less efficient reserves –can take longer to recover –more fatigue & weakness post op (less elasticity)

    • Patients with dementia-increase in confusion & agitation- safety risk-calm & reassuring attitude important

    • Follow up phone calls if outpatient surgery useful for check on patient condition & if patient understood post op instructions/care

  • Bariatric patients- Increased risk for:

    • Airway obstruction-fat deposits in neck

    • Atelectasis –increased pressure on diaphragm from excess abdominal tissue

    • Difficulty clearing anaesthetic medications from system-pain control more complex (as it sits in the fat cells)

    • Thromboembolism

    • Pressure areas

  • Other issues to consider:

    • Diabetes Mellitus –healing impaired

    • Impaired vision or hearing (back on pt so they are more orientated)

    • Peripheral vascular disease-healing impaired

    • Previous cerebrovascular accident (CVA/Stroke)

    • Smoking, alcohol, drugs (resistance to normal doses)

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This week’s focus is on the immediate postoperative phase in the PACU, and considers:

  • Close monitoring of patient is critical for safety- Unobstructed view of all patients

  • Situational awareness important- need for recognizing deterioration & acting quickly

  • Patient unable to protect themselves from environmental stimuli- reliant on nurse depending on level of consciousness =1:1 care for unconscious patients

  • Use of side rails

  • High risk of sudden deterioration

  • Observations 5-15 minutely

  • Continuous SpO2 +/- ECG (continuously monitoring)

  • Clinical reasoning (CR Cycle) helps identify & consider cues to predict & then intervene when needed

  • Knowing how to communicate issues accurately & timely is important

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Pneumonia

  • Inflammation of alveoli

    • Causes: infection, toxins, irritants causing inflammation. Immobility & impaired ventilation results in lung collapse with growth of lung pathogens

    • Signs: increased temperature, cough –productive-with blood, purulent sputum, dyspneoa, chest pain

    • Preventative management: Deep breathing & coughing, early moving in bed & ambulation

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Atelectasis (lung collapse)

  • Alveoli collapse & not ventilated!

    • Causes: mucus plugs causing blockage in bronchial airways, decreased lung expansion, analgesics, immobility

    • Signs: Dyspneoa, tachypnoea, tachycardia; diaphoresis, anxiety; pleural pain, decreased chest wall movement; dull or absent breath sounds; decreased oxygen saturation (SpO2)

    • Preventative management: Deep breathing & coughing, early moving in bed & ambulation

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Pulmonary Embolism (PE)

  • Blood clot that has moved to lungs blocking a pulmonary artery, thus obstructing blood flow to a portion of lung

    • Causes: Stasis of venous blood from immobility, venous injury from fractures or during surgery, use of oral contraceptives high in oestrogen, pre-existing coagulation or circulatory disorder

    • Signs: Sudden chest pain, shortness of breath, cyanosis, shock (tachycardia, low blood pressure)

    • Preventative management: Turning, ambulation, anti-embolic stockings, sequential compression device (SCD)-promotes venous blood flow

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Pneumothorax

occurs when air leaks into the space between the lung and the chest wall. This air accumulation puts pressure on the lung, causing it to partially or fully collapse

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Hemothorax

is a condition where blood collects in the pleural space, the area between the lung and the chest wall

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Chest drains are inserted following:

  • cardiothoracic surgery;

  • after chest trauma;

  • following a spontaneous pneumothorax;

  • or from any conditions resulting in accumulation of content in pleural space.

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Chest drainage systems have 3 main components:

  1. A collection chamber or bottle (single or multi-chamber) which collects fluid drained from chest

  2. A water seal (one way valve) to prevent which prevents air from re-entering chest on inspiration. Chest tube end sits 2cm below sterile water line.

  3.  A low suction control source &/or vent to allow air to escape from system

<ol type="1"><li><p><span>A collection chamber or bottle (single or multi-chamber) which collects fluid drained from chest</span></p></li><li><p><span>A water seal (one way valve) to prevent which prevents air from re-entering chest on inspiration. Chest tube end sits 2cm below sterile water line.</span></p></li><li><p><span>&nbsp;A low suction control source &amp;/or vent to allow air to escape from system</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/4d936674-bdf6-400b-9a15-2d292fabbc99.png" data-width="100%" data-align="center"></li></ol><p></p>
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Non-invasive ventilation (NIV)- NIV

  • Non-invasive: Delivered via face or nasal masks or nasal cannulae (HFNC)

  • Positive pressure ventilation: air “forced” into patient’s airway, as opposed to natural breathing whereby air enters under negative pressure (diaphragm drops, drop in intrathoracic pressure)

  • Can only be delivered to patient who is breathing spontaneously & able to protect airway e.g. able to clear secretions. (NIV pt is awake)

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NIV- Complications:

  • Hypotension (increased intrathoracic pressure)

  • Pulmonary barotrauma

  • Gastric distension (increased risk of aspiration)

  • Air leaks

  • Mask/tube discomfort

  • Pressure injuries (face & occiput)

  • Eye irritation

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NIV- Nursing care:

  • Close observation (1:1 or 1:2 ratio typically)

  • High dependency area (PACU, CCU, HDU or ICU setting)

  • Hourly observation (at minimum)

  • May require NGT (aspiration risk)

  • Communication & reassurance

  • Planned breaks (oral & eye care, meals, pressure area care)

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Airway Management:

  • Ensure airway patency (e.g., suctioning secretions, positioning).

  • Use airway adjuncts like oral or nasal airways if needed.

  • Be prepared for reintubation in severe cases.

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Prevention and Early Detection of Complications NIV:

  • Frequent respiratory assessments (rate, depth, effort, breath sounds).

  • Monitor for signs of respiratory distress (cyanosis, tachypnea, use of accessory muscles).

  • Encourage early ambulation and repositioning to reduce the risk of atelectasis and PE.

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NIV Pain Management:

  • Adequate pain control (opioids, NSAIDs, regional anesthesia) to allow effective breathing.

  • Encourage splinting of the incision while coughing or deep breathing to reduce discomfort.

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High Flow Nasal Cannula/Prongs (HFNC/HFNP)- NIV

  • Supplemental oxygen delivered under a high flow

  • Settings:

    • Flow: Litres of gas per minute delivered (gas = air & O2)

      • 20 – 60L gas/min

    • FiO2: Between 21% - 100% FiO2

  • Benefits:

    • Generates PEEP in lower airways

      • Cannot control PEEP like in CPAP (can't measure it)

    • Decreases physiological dead space (improving ventilation)

    • Reduces respiratory effort

    • Therapy better tolerated by patients

  • Uses: Bridge between CPAP & Low Flow oxygen (step down/up)

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Continuous Positive Airway Pressure (CPAP)- NIV

  • Gas delivered under constant positive pressure during inspiration & expiration

  • “Splints open” airways

  • Recruits collapsed alveoli

  • Two main settings:

    • Fraction of inspired oxygen (FiO2) = Between 21% - 100% oxygen

    • PEEP = Positive End Expiratory Pressure. Pressure remaining in lungs at end expiration. Typically set between 5 – 10cmH2O

  • Used for: post operative atelectasis, pulmonary oedema, Type 2 Respiratory Failure (hypercapnia) & COPD

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Chronic Obstructive Pulmonary Disease (COPD)

  • COPD revision

    • A COPD exacerbation is characterized by dyspnea &/or cough & sputum that worsens over ≤14 days; it may be accompanied by tachypnea &/or tachycardia & is often associated with increased local & systemic inflammation caused by airway infection, pulmonary embolism, pollution, or other airway insult.

  • Management goals:

    • Reverse airflow limitation using short-acting inhaled bronchodilators & oral systemic glucocorticoids e.g. prednisone

    • Treat infection, which is implicated in most exacerbations-some patients require antibiotics

    • Exclude other causes for which additional therapy is needed (eg, pulmonary embolism)

    • Ensure appropriate oxygenation-target of 88 to 92 percent pulse oxygen saturation= preventing worsening of hypercapnia

    • Avoid intubation & mechanical ventilation: NIV is preferred. Hiflow nasal cannula can be used which can provide a low level of continuous positive airway pressure (CPAP)

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Pharmacological Approaches for pain

  • Opioids (e.g., morphine, fentanyl) for moderate to severe pain.

  • Non-opioid analgesics (e.g., paracetamol, NSAIDs like ibuprofen) to reduce inflammation and opioid use.

  • Local anesthetics (e.g., nerve blocks, epidurals) for site-specific pain control.

  • Adjuvant medications (e.g., anticonvulsants like gabapentin, antidepressants like amitriptyline) for neuropathic pain.

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Non-Pharmacological Approaches for pain

  • Cognitive-behavioral therapy (CBT)

  • Cold/heat application

  • Relaxation techniques, such as breathing exercises

  • Patient education about pain expectations and control

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Management of pain

  • Regular pain assessments using validated scales (e.g., Numeric Rating Scale, Wong-Baker Faces Scale)

  • Adjusting plans based on patient-specific factors (age, comorbidities, type of surgery)

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Potential Complications of Post-operative Pain Management

  • Opioid-Related Complications:

    • Respiratory depression

    • Nausea and vomiting

    • Constipation

    • Opioid tolerance and dependency

    • Sedation and confusion (especially in elderly patients)

  • NSAID-Related Complications:

    • Gastrointestinal bleeding

    • Renal impairment

    • Cardiovascular risks (e.g., hypertension, heart failure exacerbation)

  • Local Anesthetic Complications:

    • Local anesthetic systemic toxicity (LAST)

    • Nerve damage

    • Infection at injection sites

  • Inadequate Pain Control:

    • Delayed mobilization, increasing the risk of:

      • Deep vein thrombosis (DVT)

      • Pulmonary embolism (PE)

      • Pneumonia

      • Poor wound healing

    • Chronic post-surgical pain development

  • Psychological Impact:

    • Increased anxiety and fear

    • Depression

    • Post-traumatic stress symptoms

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Schedule 8

  • otherwise known as Controlled Drugs, are substances & preparations for therapeutic use which have high potential for abuse & addiction.

  • All drugs that are schedule 8 must be approved by health department at time of writing a prescription for that drug; prescriptions must be written by hand & not printed.

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Schedule 4 Restricted (S4R)

These schedule 4 medicines are liable to abuse & may cause dependence. These medicines may be targeted for unauthorised use or diversion into illicit activities. These medications require more stringent storage & recording policies to allow more rigid monitoring & increase accountability of all health care providers.

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Schedule 8 care

  • S8’s kept in a lockable hardwood or metal cupboard. Cupboard must be securely fixed to a wall or floor.

  • Red Keys & Special inventory book (HA14) –record stock amounts, checks-date/time recorded, full name of patient, dose administered, amount discarded, amount from stock & balance remaining, name of prescriber, & signatures of staff –both also sign on med chart/IV chart

  • Never open cupboard without 2nd nurse present

  • Two nurses must be present for whole process eg. Both nurses check pt ID band & MAR from drug room to bedside

  • Two RN’s nurses count total at end of every shift. Discrepancies reported & investigated-procedure/policy

  • Errors need to be ruled in ink, dated & initialled & entry still legible

  • If any portion to be discarded, 2nd nurse witness & sign

  • Protect yourself & your colleagues-follow steps

  • Some S4R/S8 cupboards have video cameras & swipe card entry to identify you when you open the first door

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Epidural and spinal care – management and care

  • Infusion of opiates into epidural or intrathecal (subarachnoid) space

  • Epidural = thin plastic catheter inserted into epidural space & taped onto patients back –can connect to an infusion or PCEA style set up (locked as opioid in it)

  • Spinal anaesthetic= single injection of medication into CSF- motor block (unable to move legs); works within 5-10mins, very little medication absorbed into systemic circulation

  • Benefits= significantly less post op complications compared to systemic analgesia

  • Safety issues: staff training & management; anaesthetist inserting is responsible for initial connecting & management; patient must have IV access for emergency treatment e.g. hypotension, or LA toxicity; monitored by APS in hospital-notify asap if patient starting on anticoagulant medications; only specific epidural lines, tubing & labels used (see samples); may have opioid &/or LA in infusion bag; nurse checks insertion site regularly –site intact with occlusive but window dressing-monitor for infection signs, & epidural abscess or swelling; check catheter not kinked. Most epidural catheters removed 3rd or 4th day-regional catheters 7-10 days. Falls risks –muscle weakness &/or orthostatic hypotension from LA.

  • Complications: accidental disconnection- do not reattach –call APS asap; epidurals associated with risks of respiratory depression (assessed 1 hourly/24 hours post insertion), hypotension, pruritis, motor blockade, & urinary retention. If sensory block is at T3 or above this could affect respiratory centers in brain- infusion must be stopped & APS contacted asap (see hospital guidelines for more information & care).

<ul><li><p><span><strong>Infusion of opiates into epidural or intrathecal (subarachnoid) space</strong></span></p></li><li><p><span><strong>Epidural</strong> = thin plastic catheter inserted into epidural space &amp; taped onto patients back –can connect to an infusion or PCEA style set up (locked as opioid in it)</span></p></li><li><p><span><strong>Spinal anaesthetic</strong>= single injection of medication into CSF- motor block (unable to move legs); works within 5-10mins, very little medication absorbed into systemic circulation</span></p></li><li><p><span><strong>Benefits</strong>= significantly less post op complications compared to systemic analgesia</span></p></li><li><p><span><strong>Safety issues</strong>: staff training &amp; management; anaesthetist inserting is responsible for initial connecting &amp; management; patient must have IV access for emergency treatment e.g. hypotension, or LA toxicity; monitored by APS in hospital-notify <strong><u>asap</u></strong> if patient starting on anticoagulant medications; only specific epidural lines, tubing &amp; labels used (see samples); may have opioid &amp;/or LA in infusion bag; nurse checks insertion site regularly –site intact with occlusive but window dressing-monitor for infection signs, &amp; epidural abscess or swelling; check catheter not kinked. Most epidural catheters removed 3rd or 4th day-regional catheters 7-10 days. Falls risks –muscle weakness &amp;/or orthostatic hypotension from LA.</span></p></li><li><p><span><strong>Complications</strong>: accidental disconnection- do not reattach –call APS asap; epidurals associated with risks of respiratory depression (assessed 1 hourly/24 hours post insertion), hypotension, pruritis, motor blockade, &amp; urinary retention. If sensory block is at T3 or above this could affect respiratory centers in brain- infusion must be stopped &amp; APS contacted asap (see hospital guidelines for more information &amp; care).</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/fe430ca3-14e5-4595-b6f0-91be285763ba.png" data-width="100%" data-align="center"></li></ul><p></p>
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PCA care and management

  • PCA’s enables patient to self administer pain relief-pushes button & pre-set dose delivered with lock out interval of 5-15mins= less peaks & troughs for pain management (pt must press it for themselves, not family or health workers)

  • IV route most common, but subcut & epidural routes other options

  • Pre-determined dose by infusion pump set by nurse as per Dr’s prescription

  • Used in acute pain from surgical incisions, labour/delivery, traumatic injury, or chronic pain

  • Prescriber orders- analgesic dose, route, dose & lock out intervals-nurse responsible for education to patient, set up, & monitoring

  • Two RN’s to double check settings, & any changes or updates

  • Documentation –ongoing for pain levels, dosages & responses & usage

  • Safety aspects built in- prevents overdosage (if set up correctly), abuse & theft (locked with red keys ‘DD keys’).

  • Complications: respiratory depression & hypotension.

  • Patient safety =vital signs (resps & spo2), pain, sedation score, infusion rate, nausea & vomiting, constipation, IV site care, & patient understanding of PCA.

  • Can be set up to have a continuous infusion in background

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Describe the Nursing management and assessment of post-operative emergence delirium. 

  • Acute confusion which occurs during recovery from general anaesthetic

  • Usually short-term & may resolve within minutes

  • Common in paediatric populations & older patients

  • Initial management of emergence delirium &/or agitation includes:

    • Reassurance & reorientation.

    • Treatment of acute pain.

    • Treatment of other discomfort (bladder distention, hypothermia).

    • Laboratory testing to determine if hypoxemia, hypercarbia, hypoglycemia, or electrolyte imbalances are present.

    • Consideration of effects of certain residual anaesthetic agents

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Explore Post-Operative Cognitive Dysfunction. 

  • New cognitive deficits that appear immediately postoperatively

  • Not well understood thus far & requires further research

  • Suspected to have an inflammatory process resulting in cognitive dysfunction

  • Clinical observational studies show that POCD arises more frequently after extensive surgery under general anaesthesia, after secondary surgery, & when there are postoperative complications.

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Discuss potential contributors and complications associated with delayed emergence

  • Lack of consciousness 20-30 minutes after last administration of anaesthetic-related medications

  • Contributing factors:

    • Hepatic or renal insufficiency

    • Age & weight

    • Body temperature

    • Medication length of action

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Postoperative factors influencing urinary and renal function

  • Pathological conditions & types of renal failure-

  • Surgical procedures

  • Patient assessment questions

  • Bladder scanning and residual urine

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Postoperative renal and urinary complications

  • Altered urine production

  • Urinary retention post op

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Altered urine production

  • Normal function= 5-6 times a day, 1500ml or less output Abnormal= Revise terms & causes-prereading see table 51.3 in text

    • Oliguria (less than 30mls an hr), Anuria (lack of urine production), Polyuria (Litres more of urine than normal- due to diabetes, new medications, pituitary tumour, increased fluids), Frequency/Nocturia (urination in night), Urgency, Enuresis (involuntary), Retention

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Urinary retention post op

  • Patients must void within 8 hours post op –report to medical staff

  • Anaesthetics depress bladder tone- resolves around 6-8 hours post op; note surgery type affecting urinary function if delays

  • Strict Fluid balance –input/output post op important-usually 2 days

  • Interventions: IDC may be required if retention persists if techniques aren’t successful- such as positioning (male-standing/female-sitting), running water, ensuring fluid intake, ambulation

  • Goal should be to seek help in the team prior to MET criteria

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Infants & children factors affecting voiding

  • UTI’s common –can affect kidneys, bladder or both-if frequent- needs Dr review;

  • Teaching perineal hygiene important-Girls- wipe front to back, cotton underwear

  • Taught to go asap when sensation to void is felt

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Older adults factors affecting voiding

  • Older women-UTI’s- can lead to incontinence, confusion, & falls; Menopause -decreased oestrogen=decreased perineal tone & bladder support –stress incontinence, leakage on exertion, & UTI’s

  • Older men-enlarged prostate-incomplete emptying of bladder=urinary retention & overflow dribbling of urine (take longer to empty their bladder & stop start, cultural change, functional incontinent)

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Other issues to consider affecting voiding

  • Sex reassignment surgery=changes to urethra- longer term issues –stress incontinence, overactive bladder, & reduced blood flow

  • Note genital mutilation or obstetric fistulas (recurrent UTI’s, leakage) in some populations

  • Overactive bladder –sudden urge to void-from nerve damage from diabetes, stroke, injury, or surgery

  • Functional incontinence= from impaired mobility –joint & bone pain-surgery & neuromuscular issues –difficult to get to bathroom

  • Cognitive impairments- Dementia

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Pathological conditions & types of renal failure-

  • Pre-renal – haemorrhage, dehydration, burns, shock, low intravascular volume

  • Intra Renal – nephrotoxic agents (gentamicin), transfusion reactions, diseases of glomeruli, renal neoplasms, systemic disease (DM), infections, hereditary (polycystic kidney disease)

  • Post-renal – obstruction (calculi -stones, clots, tumours),

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Surgical procedures

  • Post cystoscopy –inflammation

  • Bleeding post op-risk of clots

  • Spinal anaesthetics –decrease awareness to void

  • Abdo surgery- structures adjacent to urinary system-swelling & affecting voiding e.g. uterus

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Prevention of urinary tract infections

  • More common in women; nosocomial occurs in healthcare & mostly related to IDC insertion; Most UTIs caused by bacteria common to intestinal environment (e.g. Escherichia coli)- This GI bacteria can move into urethra following urethral trauma, irritation, & manipulation. See readings for preventative measures

  • Urinary sheaths- used for managing incontinence (less risk for UTI than IDC)

  • Suprapubic catheter (other options)- Dr inserts surgically through abdominal wall into bladder with LA & with closed drainage system; temporary or long term; maintenance of drainage tube –can become blocked easily; dressing care at site of insertion

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Key points and nursing care

  • Strict aseptic technique

  • If resistance felt on insertion- STOP & seek help

  • Document post insertion-sticker in notes

  • Encourage fluids

  • Perineal care

  • Long term IDC

  • Strict FBC

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Care of clients with bladder irrigations

  • Bladder Irrigation; continuous or intermittent; closed or open

  • Indications- to prevent blood clots & mucous occluding catheter eg. post genitourinary surgery; to instil medication

  • Medical order

  • Strict Aseptic technique

  • Warm irrigating fluids

  • Strict FBC

  • Monitor & assess drainage- observations 30 minutely

  • Bladder scans pre/prior to installation of fluid

  • Review sample hospital policies

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Renal failure: Peritoneal Dialysis

  • Dialysis = fluids & molecules pass through a semi-permeable membrane with osmosis

  • Peritoneal dialysis (PD)- dialysis solution is instilled into abdominal cavity through a catheter, allowed to rest there while fluid & molecules exchange & then removed through catheter.

  • In PD peritoneal membrane acts as semipermeable membrane.

  • Peritoneal dialysis facilitates removal of waste products & excess fluid from bloodstream

  • process uses a specialised solution called dialysate, which is administered into peritoneal cavity

  • peritoneal cavity is then drained via gravity into a wastage bag for disposal

  • Performed at regular intervals until function returns or forever

Complications:

  • Exit-site infection

  • Peritonitis

  • Hernias-abdo wall from volume pressure of fluid

  • Lower back problems

  • Bleeding

  • Pulmonary complications-fluid shifts

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Renal failure: Haemodialysis

  • Person’s blood flows through vascular catheters, passes by dialysis solution in an external machine & then returns to person.

  • It is used to correct fluid & electrolyte imbalances & to remove waste products in kidney failure. It can also be used to treat medication overdoses.

  • In HD an artificial membrane (usually made of cellulose-based or synthetic materials) is used as semipermeable membrane & is in contact with patient's blood.

Haemodialysis Types of access:

  • Arteriovenous fistulas & grafts (never take a blood pressure or blood tests or insert IV in this arm; has a ‘thrill’ vibration on palpation, & ‘bruit’ sound)

  • Temporary vascular access-Dialysis catheter (VasCath)

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Cardiac physiology

  • What is Cardiac Output (CO)?

    • Amount of blood heart pumps through circulatory system in a minute.

  • What is Stroke Volume?

    • amount of blood put out by left ventricle of heart in one contraction.

  • CO = SV x HR

<ul><li><p><span><strong>What is Cardiac Output (CO)?</strong></span></p><ul><li><p><span>Amount of blood heart pumps through circulatory system in a minute.</span></p></li></ul></li><li><p><span><strong>What is Stroke Volume?</strong></span></p><ul><li><p><span>amount of blood put out by left ventricle of heart in one contraction.</span></p></li></ul></li><li><p><span><strong>CO = SV x HR</strong></span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/f4409a02-599f-4996-a9e0-3a75c670ebbd.png" data-width="100%" data-align="center"></li></ul><p></p>
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Heart rate

  • Influenced by autonomic nervous system (e.g. ‘Fight or flight’); blood pressure; hormones (thyroid); & medications.

  • Heart rates >150bpm cause inadequate filling time (diastole) thus a HR increases CO; if SV stays same.

  • Chronotropic effects (chrono= time) are those that change heart rate (SA node). Positive chronotropes >HR, whilst negative chronotropes <HR.

<ul><li><p><span>Influenced by autonomic nervous system (e.g. ‘Fight or flight’); blood pressure; hormones (thyroid); &amp; medications.</span></p></li><li><p><span>Heart rates &gt;150bpm cause inadequate filling time (diastole) thus a HR increases CO; if SV stays same.</span></p></li><li><p><span>Chronotropic effects (chrono= time) are those that change heart rate (SA node). Positive chronotropes &gt;HR, whilst negative chronotropes &lt;HR.</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/0b2402f7-aaa0-4b51-a0bc-f562cc544e72.png" data-width="100%" data-align="center"></li></ul><p></p>
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Stroke volume is determined by three factors

  • Preload: filling pressure of heart at end of diastole (volume circulating into atria/ventricles . Low preload-blood transfusions. High preload- furosemide)

  • Contractility: Inotropic state of heart-strength of contraction. Influenced by autonomic nervous system & medications. Positive inotropic drugs >contractility whilst negative inotropic drugs (force of heart)

  • Afterload: Pressure against which heart must work to eject blood during systole (resistance of ventricle must overcome to pump blood out into aorta & pulmonary artery)

<ul><li><p><span><strong>Preload</strong>: filling pressure of heart at end of diastole (volume circulating into atria/ventricles . Low preload-blood transfusions. High preload- furosemide)</span></p></li><li><p><span><strong>Contractility:</strong> Inotropic state of heart-strength of contraction. Influenced by autonomic nervous system &amp; medications. Positive inotropic drugs &gt;contractility whilst negative inotropic drugs (force of heart)</span></p></li><li><p><span><strong>Afterload:</strong> Pressure against which heart must work to eject blood during systole (resistance of ventricle must overcome to pump blood out into aorta &amp; pulmonary artery)</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/018b578b-0369-4a06-a974-76dde3389c06.png" data-width="100%" data-align="center"></li></ul><p></p>
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Hypovolaemia

  • Inadequate circulating blood volume

  • Causes: Fluid deficit, haemorrhage

  • Signs: Tachycardia, decreased urine output, decreased blood pressure

  • Preventative management: Early detection of signs; fluid &/or blood replacement

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Hypovolaemic shock

  • Inadequate tissue perfusion resulting from markedly reduced circulating blood volume

  • Causes: Severe hypovolaemia from fluid deficit or haemorrhage

  • Signs: Rapid weak pulse, dyspnoea, tachypnoea; restlessness & anxiety; urine output less than 30 mL/hr; decreased blood pressure; cool, clammy skin, thirst, pallor

  • Preventative management: Maintain blood volume through adequate fluid replacement, prevent haemorrhage; early detection of signs

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Haemorrhage

  • Internal or External bleeding

  • Causes: Disruption of sutures, insecure sutures, insecure ligation of blood vessels

  • Signs: Overt bleeding (dressings saturated with bright blood; bright, free-flowing blood in drains or chest tubes), increased pain, increasing abdominal girth, swelling or bruising around incision

  • Preventative management: Early detection of signs

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Thrombophlebitis

  • Inflammation of veins, usually of legs & associated with a blood clot

  • Causes: Slowed venous blood flow due to immobility or prolonged sitting; trauma to vein, resulting in inflammation & increased blood coagulability

  • Signs: Aching, cramping pain; affected area is swollen, red & hot to touch; vein feels hard; discomfort in calf when foot is dorsiflexed or when person walks (Homans’ sign)

  • Preventative management: Early ambulation, leg exercises, anti-embolic stockings, SCDs, adequate fluid intake

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Thrombus

  • Blood clot attached to wall of vein or artery (most commonly leg veins)

  • Causes: As for thrombophlebitis for venous thrombi; disruption or inflammation of arterial wall for arterial thrombi

  • Signs: Venous: same as thrombophlebitis

  • Arterial: pain & pallor of affected extremity; decreased or absent peripheral pulses

  • Preventative management: Venous: same as thrombophlebitis Arterial: maintain prescribed position; early detection of detection of signs

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Embolus

  • Foreign body or clot that has moved from its site of formation to another area of body (e.g. lungs, heart or brain)

  • Causes: Venous or arterial thrombus; broken intravenous catheter, fat or amniotic fluid

  • Signs: In venous system, usually becomes a pulmonary embolus (see pulmonary embolism); signs of arterial emboli may depend on location

  • Preventative management: Turning, ambulation, leg exercises, SCDs; careful maintenance of IV catheters

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Cardiac tamponade

  • is a clinical syndrome caused by accumulation of fluid in pericardial space, resulting in reduced ventricular filling & subsequent hemodynamic compromise (squashes heart)

  • Condition is a medical emergency, complication of which include profound shock, multi organ failure & death.

  • Signs:

    • Tachypnea

    • Tachycardia

    • Elevated jugular venous pressure (JVP) & CVP

    • Confusion

    • Decreased urine output

    • Hypotension

    • Peripheral cyanosis

  • Management:

    • Urgent medical review

    • Drainage/evacuation of fluid from pericardial space

<ul><li><p><span>is a clinical syndrome caused by accumulation of fluid in pericardial space, resulting in reduced ventricular filling &amp; subsequent hemodynamic compromise (squashes heart)</span></p></li><li><p><span>Condition is a medical emergency, complication of which include profound shock, multi organ failure &amp; death.</span></p></li><li><p><span><strong>Signs:</strong></span></p><ul><li><p><span>Tachypnea</span></p></li><li><p><span>Tachycardia</span></p></li><li><p><span>Elevated jugular venous pressure (JVP) &amp; CVP</span></p></li><li><p><span>Confusion</span></p></li><li><p><span>Decreased urine output</span></p></li><li><p><span>Hypotension</span></p></li><li><p><span>Peripheral cyanosis</span></p></li></ul></li><li><p><span><strong>Management:</strong></span></p><ul><li><p><span>Urgent medical review</span></p></li><li><p><span>Drainage/evacuation of fluid from pericardial space</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/d701d3cf-f615-4f95-b3dc-fc00a12861db.png" data-width="100%" data-align="center"></li></ul></li></ul><p></p>
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Why measure CVP:

  • CVP is used to assess adequacy of blood volume & is an indicator of right ventricular preload.

    • CVP also reflects limit to venous return & informs about right ventricular function.

    • CVP measurements may be helpful to support & guide fluid management

    • Measured through a central line & measures right atrial pressure

    • There are no valves between right atria & superior vena cava so pressure measured at tip of catheter is a measure of right atrial pressure

    • Right atrial pressure is an indirect measurement of right ventricular preload

    • Helps to assess cardiac function & venous return to heart

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Measuring CVP:

  • Fluid filled tubing connected to a transducer is attached to distal lumen of central line.

  • Measure from phlebostatic axis (4th intercostal space mid axillary line).

  • Temporarily cease fluids being administered through lumen for measurement.

  • Ensure reading occurs at end expiration.

  • Recommence fluids.

  • Document – normal CVP reading can be between 0-8cm H20, abnormal e.g. cardiac tamponade could be 12cm H20

<ul><li><p><span>Fluid filled tubing connected to a transducer is attached to distal lumen of central line.</span></p></li><li><p><span>Measure from phlebostatic axis (4th intercostal space mid axillary line).</span></p></li><li><p><span>Temporarily cease fluids being administered through lumen for measurement.</span></p></li><li><p><span>Ensure reading occurs at end expiration.</span></p></li><li><p><span>Recommence fluids.</span></p></li><li><p><span>Document – normal CVP reading can be between 0-8cm H20, abnormal e.g. cardiac tamponade could be 12cm H20</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/fae8c00e-669d-47b3-9365-a70fd38e1204.png" data-width="100%" data-align="center"></li></ul><p></p>
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Jugular Venous Pressure (JVP):

  • Non-invasive evaluation of central venous pressure (CVP) can be achieved by assessing Jugular Venous Pressure (JVP)

  • Elevated CVP will present clinically as a pulsation of internal jugular vein when a patient is inclined at 45 degrees; however, it can be noted in an upright patient in severe cases.

  • Elevated JVP readings can indicate heart failure & fluid overload needing diuresis

<ul><li><p><span>Non-invasive evaluation of central venous pressure (CVP) can be achieved by assessing Jugular Venous Pressure (JVP)</span></p></li><li><p><span>Elevated CVP will present clinically as a pulsation of internal jugular vein when a patient is inclined at 45 degrees; however, it can be noted in an upright patient in severe cases.</span></p></li><li><p><span>Elevated JVP readings can indicate heart failure &amp; fluid overload needing diuresis</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/90a74174-74a3-4fe7-a3c2-24bff9c1c61c.png" data-width="100%" data-align="center"></li></ul><p></p>
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<p><span><strong>Discuss complex perfusion dysfunction and nursing management for patients with heart failure.</strong></span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/130dec3c-f289-4582-b0eb-eaca8e572e0f.png" data-width="100%" data-align="center"><p></p>

Discuss complex perfusion dysfunction and nursing management for patients with heart failure.

  • Cardiomyopathy (CMP)

  • Dilated CMP

  • Hypertrophic CMP

  • Restrictive CMP

  • Heart Failure

  • Left side heart failure

  • Right-sided heart failure

  • Management options and nursing care:

    • Treatment of underlying cause

    • Nitrates

    • Beta adrenergic blockers antiarrhythmics

    • ACE inhibitiors

    • Diuretics

    • Ventricular assist device

    • Implantable cardioverter/defibrillator

    • Surgical – Heart Transplantation

<ul><li><p><span>Cardiomyopathy (CMP)</span></p></li><li><p><span>Dilated CMP</span></p></li><li><p><span>Hypertrophic CMP</span></p></li><li><p><span>Restrictive CMP</span></p></li><li><p><span>Heart Failure</span></p></li><li><p><span>Left side heart failure</span></p></li><li><p><span>Right-sided heart failure</span></p></li><li><p><span>Management options and nursing care:</span></p><ul><li><p><span>Treatment of underlying cause</span></p></li><li><p><span>Nitrates</span></p></li><li><p><span>Beta adrenergic blockers antiarrhythmics</span></p></li><li><p><span>ACE inhibitiors</span></p></li><li><p><span>Diuretics</span></p></li><li><p><span>Ventricular assist device</span></p></li><li><p><span>Implantable cardioverter/defibrillator</span></p></li><li><p><span>Surgical – Heart Transplantation</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/99ca5f2f-5be9-416a-9411-df0631f457ea.png" data-width="100%" data-align="center"></li></ul></li></ul><p></p>
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Management of heart failure:

  • Main treatment goals:

    • Treat underlying cause & contributing factors

    • Maximise CO

    • Provide treatment to alleviate symptoms

    • Improve ventricular function

    • Improve quality of life

    • Preserve target organ function

    • Improve mortality & morbidity.

    • Oxygen therapy

    • Relieve dyspnoea & fatigue

    • Physical & emotional rest

    • Conserve energy & decrease oxygen needs

    • Structured exercise program

    • Biventricular pacing/cardiac

    • IABP & VADs as bridge or destination therapy for stage IV HF

  • Nutritional therapy:

    • Low-sodium diet

    • Recommend Dietary Approaches to Stop Hypertension (DASH) diet

    • Sodium is usually restricted to 2 g/day

    • If fluid restriction is required, < 2 L/day.

    • Daily weights

  • Medications:

    • Diuretics

    • RAAS inhibitors

      • ACE inhibitors

      • Angiotensin II receptor blockers

      • Aldosterone antagonists

    • β-adrenergic blockers

    • Vasodilators

      • Nitrates

    • Positive inotropic agents

      • Digitalis (be alert for digitalis toxicity)

<ul><li><p><span>Main treatment goals:</span></p><ul><li><p><span>Treat underlying cause &amp; contributing factors</span></p></li><li><p><span>Maximise CO</span></p></li><li><p><span>Provide treatment to alleviate symptoms</span></p></li><li><p><span>Improve ventricular function</span></p></li><li><p><span>Improve quality of life</span></p></li><li><p><span>Preserve target organ function</span></p></li><li><p><span>Improve mortality &amp; morbidity.</span></p></li><li><p><span>Oxygen therapy</span></p></li><li><p><span>Relieve dyspnoea &amp; fatigue</span></p></li><li><p><span>Physical &amp; emotional rest</span></p></li><li><p><span>Conserve energy &amp; decrease oxygen needs</span></p></li><li><p><span>Structured exercise program</span></p></li><li><p><span>Biventricular pacing/cardiac</span></p></li><li><p><span>IABP &amp; VADs as bridge or destination therapy for stage IV HF</span></p></li></ul></li><li><p><span>Nutritional therapy:</span></p><ul><li><p><span>Low-sodium diet</span></p></li><li><p><span>Recommend Dietary Approaches to Stop Hypertension (DASH) diet</span></p></li><li><p><span>Sodium is usually restricted to 2 g/day</span></p></li><li><p><span>If fluid restriction is required, &lt; 2 L/day.</span></p></li><li><p><span>Daily weights</span></p></li></ul></li><li><p><span>Medications:</span></p><ul><li><p><span>Diuretics</span></p></li><li><p><span>RAAS inhibitors</span></p><ul><li><p><span>ACE inhibitors</span></p></li><li><p><span>Angiotensin II receptor blockers</span></p></li><li><p><span>Aldosterone antagonists</span></p></li></ul></li><li><p><span>β-adrenergic blockers</span></p></li><li><p><span>Vasodilators</span></p><ul><li><p><span>Nitrates</span></p></li></ul></li><li><p><span>Positive inotropic agents</span></p><ul><li><p><span>Digitalis (be alert for digitalis toxicity)</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/28894849-3ce0-4628-8e27-cc3dafd5583b.png" data-width="100%" data-align="center"></li></ul></li></ul></li></ul><p></p>
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Tetralogy of Fallot

complex congenital heart defect that consists of four anatomical abnormalities:

  1. Ventricular septal defect (VSD): A hole between the right and left ventricles.

  2. Pulmonary stenosis: Narrowing of the pulmonary valve and artery, reducing blood flow to the lungs.

  3. Overriding aorta: The aorta is positioned above the VSD, receiving blood from both ventricles.

  4. Right ventricular hypertrophy: Thickening of the right ventricle muscle due to increased workload.

    Clinical Features

    • Cyanosis: Bluish skin, lips, and nails due to low oxygen levels.

    • "Tet spells": Sudden episodes of deep blue skin, nails, and lips after crying, feeding, or agitation (caused by a sudden drop in oxygen).

    • Poor feeding and growth (failure to thrive).

    • Clubbing: Enlargement of fingers and toes.

    • Murmurs: Heart murmurs heard during auscultation.

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Blood transfusion indications:

  • Blood loss (trauma, surgery, haemorrhage)

  • Severe anaemia

  • Replacement of fluid & protein

  • Restoration of oncotic pressure

  • Replacement essential clotting factors

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Blood grouping and matching:

  • Recipient to donor –blood is a living tissue –considered ‘transplantation’-risk of fatal transfusion reaction-strict process required

  • Blood groups= A, B, O, & AB, & rhesus (Rh) factor +/- ve (85% of pop’n has antibodies on their RBC= Rh +ve)

  • Blood of one group incompatible with another group & mismatches (group & Rh) causes haemolytic reactions. Type O –we can give to all (no antigens)

  • Autologous blood can be given long before patient goes for surgery & given back to them during/after

<ul><li><p><span>Recipient to donor –blood is a living tissue –considered ‘transplantation’-risk of fatal transfusion reaction-strict process required</span></p></li><li><p><span>Blood groups= A, B, O, &amp; AB, &amp; rhesus (Rh) factor +/- ve (85% of pop’n has antibodies on their RBC= Rh +ve)</span></p></li><li><p><span>Blood of one group incompatible with another group &amp; mismatches (group &amp; Rh) causes haemolytic reactions. Type O –we can give to all (no antigens)</span></p></li><li><p><span>Autologous blood can be given long before patient goes for surgery &amp; given back to them during/after</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/69f07c5e-8075-42c1-8106-b57411da0e3d.png" data-width="100%" data-align="center"></li></ul><p></p>
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Before requesting issue of a blood product or collecting the blood product, clinical staff must check the following:

  • Prescription has been satisfactorily completed, & a valid, informed consent has been obtained & indication for transfusion has been documented in patient’s health-care record

  • Patient has been assessed, including baseline vital signs, to determine whether it is appropriate to undertake transfusion at planned time

  • IV access is appropriate 18–20G or larger & patent, & all necessary equipment is available & in working order (e.g. infusion pumps, Blood IV set or blood warmers)

  • Appropriately trained & competent staff are available for duration of transfusion, including two staff to perform blood product & patient identity checks at patient’s side-blood is not spiked/started until all checks completed

  • Patient is available to proceed with transfusion; for example, patient is not scheduled for a procedure.

  • Once a unit of blood is removed from blood fridge, transfusion must:

    • Commence within 30 minutes.

    • Be completed within 4 hours.

  • Normal saline is used to prime line- isotonic & doesn’t affect RBC (no lysis or clumping)

  • Transfusion reactions usually occur in first 15mins- start infusion slowly & remain with patient for 15- 30mins to observe- vital signs= 15/60mins

  • Check & follow hospital policies

<ul><li><p><span>Prescription has been satisfactorily completed, &amp; a valid, informed consent has been obtained &amp; indication for transfusion has been documented in patient’s health-care record</span></p></li><li><p><span>Patient has been assessed, including baseline vital signs, to determine whether it is appropriate to undertake transfusion at planned time</span></p></li><li><p><span>IV access is appropriate 18–20G or larger &amp; patent, &amp; all necessary equipment is available &amp; in working order (e.g. infusion pumps, Blood IV set or blood warmers)</span></p></li><li><p><span>Appropriately trained &amp; competent staff are available for duration of transfusion, including two staff to perform blood product &amp; patient identity checks at patient’s side-blood is not spiked/started until all checks completed</span></p></li><li><p><span>Patient is available to proceed with transfusion; for example, patient is not scheduled for a procedure.</span></p></li><li><p><span>Once a unit of blood is removed from blood fridge, transfusion must:</span></p><ul><li><p><span>Commence within 30 minutes.</span></p></li><li><p><span>Be completed within 4 hours.</span></p></li></ul></li><li><p><span>Normal saline is used to prime line- isotonic &amp; doesn’t affect RBC (no lysis or clumping)</span></p></li><li><p><span>Transfusion reactions usually occur in first 15mins- start infusion slowly &amp; remain with patient for 15- 30mins to observe- vital signs= 15/60mins</span></p></li><li><p><span>Check &amp; follow hospital policies</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/b488c6b9-358c-4805-b21e-c3344f7065ef.png" data-width="100%" data-align="center"></li></ul><p></p>
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Reactions - refer to hosp clinical practice guidelines

Clinical Signs

Nursing Intervention

  • Haemolytic Reaction:

    • Febrile, chills, headache, SOB, Chest pain, Tachycardia, hypotension

(incompatibility) Stop transfusion & ring for RMO! Monitor obs & FBC, urinalysis. Check blood pack & paperwork for discrepancies. Send remaining blood & lines to lab. Infuse NaCl with new lines. CALL MET if meets criteria. Document! (incident report, Progress notes)

  • Allergic Reaction:

    • Itching, urticarial rash, wheezing (mild)

    • Severe- dyspnoea, chest pain, circulatory collapse, cardiac arrest.

  • STOP TRANSFUSION

  • Keep IV line open with normal saline

  • Check blood pack label & patient identification labels are correct i.e. correct unit has been given to correct patient.

  • NOTIFY RMO

  • Severe- Administer CPR if needed, administer medications or oxygen as ordered.

Febrile Reaction: Febrile, chills, warm, flushed skin, headache, anxiety, muscle pain.

(incompatibility) Stop transfusion & ring for RMO! Give antipyretics as ordered. Keep  vein open with normal saline.

Fluid overload/circulatory overload: Dyspnoea, chest pain, anxiety, diaphoresis, blood tinged sputum, fine crackles on auscultation of the chest

Stop transfusion

Notify medical staff-frequent obs, check medication chart for frusemide to be given between units, FBC, nursing care as per fluid overload

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Discuss nursing management of post operative patients undergoing gastrointestinal surgery

  • Nursing management:

    • Post op movement/activity/mobilisation –often improves return of gastrointestinal function- from ‘gas cramps’-abdominal distension & loss of bowel sounds –slowed GI function from surgery or handling of bowel in surgery

    • Fluids- IV post op- patients often dehydrated- NBM, fluid loss during surgery, renal retention of sodium & water from stress. Strict FBC important post op- IV continues until fluids by mouth to maintain FBC 

    • FBC includes all intake (e.g. IV, blood, volume expanders, oral, & enteral fluids) & output (drainage, stoma, vomitus, NGT output, bladder irrigations & urine). Note drainage changes- e.g. new bleeding, bile- may detect abnormality- e.g. breakdown of anastomosis

    • PONV- note factors = anaesthetic, opioids, hypotension, abdo surgery, female more likely, non-smoker, history of migraines or previous PONV (post-operative nausea & vomiting)

    • Antiemetics- 5-HT3 receptor antagonist (Serotonin) is first line of choice e.g. ondansetron. Dopamine antagonists- metoclopramide. H1 antihistamines can be used- promethazine, hyoscine can be used in vestibular disturbances such as motion sickness

    • Oral fluids used cautiously post op-to reduce risk of lung injury (aspiration)-ice chips used first- then sips of h20-avoid straw (more air into stomach), then clear fluids, then normal diet in steps. Chewing gum may help- but with caution, crackers

    • Diet –high protein= (tissue repair & healing), high carbohydrate (energy & vitamins C & D for tissue formation)-may need dietitian support for supplementary drinks or feeding

    • NGT – used to decompress stomach post GI surgery (prevents abdo distension), & may prevent PONV- remains insitu until peristalsis returns (bowel sounds & tolerating diet)-note if on straight drainage vs stopper

    • Bowel care- sluggish post op due to: fasting, dehydration, medications, bowel manipulation. If bowel sounds, & flatus returns –positive sign of peristalsis – oral intake could resume (if medical staff approve). May take 2-3 days before normal bowel function returns for patient. Note pain with incisions- patient may be reluctant to bear down with bowel motion with pain- management important-splinting wound, bowel care=stool softeners, laxatives (glycerin suppositories), microlax enemas if needed

    • Some diagnostic procedures (colonoscopy, sigmoidoscopy) require bowel cleansing & fasting e.g. golytely, glycoprep, Movicol, or picolax prior to procedure-some of these also used for faecal impaction…

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Post op nausea & vomiting (PONV

  • Negative effects can include: wound dehiscence, oesophageal rupture, aspiration, dehydration, increased intracranial pressure (when you Valsalva blood goes into your brain), & pneumothorax.

  • Causes: Pain, abdominal distension, ingesting food or fluids before return of peristalsis, certain medications, anxiety

  • Signs: Complaints of feeling sick to the stomach, retching or gagging

  • Preventative management: IV fluids until peristalsis returns (bowel sound); then clear fluids, full fluids & regular diet; anti-emetic drugs if ordered; analgesics for pain

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Constipation

  • Infrequent or no stool passage for abnormal length of time (e.g. within 48 hours after solid diet started)

  • Causes: Lack of dietary roughage, analgesics (decreased intestinal motility), immobility

  • Signs: Absence of stool elimination, abdominal distension & discomfort

  • Preventative management: Adequate fluid intake, high fibre diet, early ambulation

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Abdominal distension (gas)

  • Retention of gases within intestines. Note some abdominal procedures e.g. laparoscopy instills CO2 into abdo to expand & to visualize structures (insufflation)-can also irritate phrenic nerve – referred pain in shoulder & chest

  • Causes: Slowed motility of intestines due to handling of bowel during surgery & effects of anaesthetic

  • Signs: Obvious abdominal distension, abdominal discomfort (gas pains), absence of bowel sounds

  • Preventative management: Early ambulation; avoid using a straw, provide ice chips or water at room temperature

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Paralytic ileus

  • Intestinal obstruction characterized by lack of peristaltic activity

  • Causes: Handling bowel during surgery, anaesthetic, electrolyte imbalance, wound infection

  • Signs: Abdominal pain & distension; constipation; absent bowel sounds; vomiting-note could be bile looking or if severe- faecal, can be noted on Xray

  • Preventative management: Treat underlying causes, assess bowel sounds frequently & carefully –auscultation, assess for signs of abdominal distension above, NGT to decompress abdo distension

<ul><li><p><span>Intestinal obstruction characterized by lack of peristaltic activity</span></p></li><li><p><span><strong>Causes:</strong> Handling bowel during surgery, anaesthetic, electrolyte imbalance, wound infection</span></p></li><li><p><span><strong>Signs:</strong> Abdominal pain &amp; distension; constipation; absent bowel sounds; vomiting-note could be bile looking or if severe- faecal, can be noted on Xray</span></p></li><li><p><span><strong>Preventative management:</strong> Treat underlying causes, assess bowel sounds frequently &amp; carefully –auscultation, assess for signs of abdominal distension above, NGT to decompress abdo distension</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/e3ff2066-87f1-4334-af23-c05398fcc1c3.png" data-width="100%" data-align="center"></li></ul><p></p>
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<p><span><strong>Abdominal Surgery</strong></span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/631cf41d-855d-40c5-b7b4-7356d21c6488.png" data-width="100%" data-align="center"><p></p>

Abdominal Surgery

  • Note pattern of scars

  • Adhesions-scar tissue

  • Specific clinical pathways/care plans for abdo surgery in hospital-preop/postop/discharge care

<ul><li><p><span>Note pattern of scars</span></p></li><li><p><span>Adhesions-scar tissue</span></p></li><li><p><span>Specific clinical pathways/care plans for abdo surgery in hospital-preop/postop/discharge care</span></p><img src="https://knowt-user-attachments.s3.amazonaws.com/383f3075-f2f4-4612-8f98-93db02d7e315.png" data-width="100%" data-align="center"></li></ul><p></p>