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This set of flashcards covers key vocabulary related to motor speech disorders, dysphagia physiology and assessment, apraxia types, cranial-motor anatomy, and common therapeutic techniques discussed in the lecture notes.
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Chorea
Smooth, random, dance-like involuntary movements; typical of Huntington’s disease.
Ataxia
Drunken-like, uncoordinated speech and body movement caused by cerebellar damage.
Dystonia
Sustained, twisting muscle contractions that produce abnormal postures or movements.
Myoclonus
Sudden, brief, jerky muscle contractions often seen in epilepsy or metabolic disorders.
Tremor
Rhythmic, involuntary oscillation of a body part; common in Parkinson’s disease and essential tremor.
Bradykinesia
Pathologically slow initiation and execution of voluntary movement.
Rigidity
Uniform resistance to passive movement, giving a ‘lead-pipe’ or ‘cogwheel’ feel.
Spasticity
Velocity-dependent increase in muscle tone with exaggerated stretch reflexes.
Hyperkinesia
Excessive, involuntary movements due to over-activity of motor pathways.
Akinesia
Marked difficulty initiating voluntary movement, as in advanced Parkinson’s disease.
Upper Motor Neurons (UMNs)
Cortical and subcortical neurons that initiate voluntary movement and modulate tone; bilateral damage causes spasticity.
Lower Motor Neurons (LMNs)
Brainstem or spinal neurons that directly innervate muscle; lesions cause weakness, atrophy, fasciculations.
Spastic Dysarthria
Motor speech disorder from bilateral UMN damage; strained voice, slow rate, hyper-reflexes.
Flaccid Dysarthria
Speech disorder from LMN damage; hypernasality, breathy voice, fasciculations, rapid fatigue.
Ataxic Dysarthria
Cerebellar-based speech disorder with irregular articulatory breakdowns and excess-equal stress.
Hypokinetic Dysarthria
Basal ganglia disorder due to dopamine depletion; monopitch, reduced loudness, short rushes of speech.
Hyperkinetic Dysarthria
Basal ganglia disorder with excess dopamine; involuntary voice breaks, variable rate, distorted vowels.
Unilateral UMN Dysarthria
Mild dysarthria from one-sided UMN damage causing weakness and spasticity on the contralateral side.
Basal Ganglia
Subcortical nuclei regulating movement initiation, amplitude, and inhibition.
Cerebellum
Brain structure that coordinates timing, force, and sequence of movements for smooth motor control.
Area 4 – Primary Motor Cortex
Precentral gyrus controlling voluntary skeletal muscle; lesions cause weakness or paralysis.
Area 44 – Broca’s Area
Left inferior frontal gyrus responsible for speech production; damage produces Broca’s aphasia.
Areas 3, 1, 2 – Somatosensory Cortex
Post-central gyrus processing tactile input; damage yields numbness or impaired sensation.
Area 6 – Supplementary Motor Cortex
Anterior to motor cortex; plans and sequences complex movements.
Sound Production Treatment (SPT)
Evidence-based apraxia therapy using modeling, repetition and cueing to improve consonant accuracy.
Minimal Contrasts
Therapy technique practicing word pairs differing by a single phoneme to refine articulation.
Supraglottic Swallow
Breath-hold, swallow, cough sequence used to protect airway when closure is delayed or weak.
Super-Supraglottic Swallow
Hard breath-hold plus swallow & cough providing extra laryngeal closure for severe airway risk.
Effortful Swallow
Intentional, forceful swallow to increase tongue base retraction and pharyngeal pressure.
Mendelsohn Maneuver
Voluntarily sustaining laryngeal elevation during swallow to prolong UES opening.
Chin Tuck
Postural adjustment of lowering the chin to widen vallecular space and reduce aspiration risk.
Head Turn (to weaker side)
Rotating head toward damaged pharyngeal side to direct bolus down the stronger channel.
Penetration–Aspiration Scale (PAS)
Eight-point rating of material entering airway, ranging from 1 (no entry) to 8 (silent aspiration).
Videofluoroscopic Swallow Study (VFSS/MBSS)
Dynamic X-ray that visualizes bolus flow through all swallow phases using barium contrast.
Fiberoptic Endoscopic Evaluation of Swallowing (FEES)
Trans-nasal endoscopy providing direct laryngeal view before and after the swallow without radiation.
Cervical Auscultation
Listening to swallow sounds at the neck to infer pharyngeal movement and airway closure.
Surface Electromyography (sEMG)
Non-invasive electrodes measuring muscle activity during oral and pharyngeal swallow stages.
3-Ounce Water Swallow Test
Bedside screen where patient drinks 90 ml of water continuously; coughing or wet voice signals risk.
Yale Swallow Protocol
Standardized screening combining oral-motor exam, cognition check, and 3-ounce water swallow.
Heads-Up Effortful Swallow
Swallow performed with neck extension to assist clearance of vallecular residue; may worsen pyriform residue.
Lee Silverman Voice Treatment (LSVT LOUD)
High-effort program increasing vocal loudness and respiratory drive for Parkinson’s hypokinetic speech.
Apraxia of Speech (AOS)
Motor planning disorder causing groping, inconsistent sound errors, and disrupted prosody.
Childhood Apraxia of Speech (CAS)
Pediatric motor-planning deficit with inconsistent errors and increasing difficulty as utterances lengthen.
Inconsistent Phonological Disorder (IPD)
Phonological disorder with variable phoneme selection errors but preserved motor planning and imitation.
Ideational Apraxia
Inability to conceptualize multi-step tasks leading to misuse or sequence errors with tools/objects.
Ideomotor Apraxia
Breakdown in translating a motor idea into action; gestures poor on command but better with imitation.
Buccofacial (Oral) Apraxia
Impaired voluntary non-speech oral movements such as blowing or tongue protrusion.
Gait Apraxia
Difficulty initiating or planning walking despite normal strength, producing a ‘magnetic’ shuffle.
Myasthenia Gravis
Autoimmune LMN junction disorder causing fatigable weakness; can produce flaccid dysarthria.
Amyotrophic Lateral Sclerosis (ALS)
Neurodegenerative disease causing mixed spastic-flaccid dysarthria and progressive weakness.
Guillain-Barré Syndrome
Acute demyelinating polyneuropathy producing LMN flaccid dysarthria and dysphagia.
Orofacial Dyskinesia
Involuntary lip, tongue or jaw movements often related to excess dopamine states.
Masako Maneuver
Tongue-hold swallow strengthening pharyngeal constrictors by protruding tongue between teeth while swallowing.
Upper Esophageal Sphincter (UES)
Cricopharyngeal opening between pharynx and esophagus that relaxes during the swallow.
Lower Esophageal Sphincter (LES)
Distal esophageal valve that closes to prevent gastric reflux after bolus entry into stomach.
Oral Preparatory Phase
Voluntary stage where food is chewed, mixed with saliva, and formed into a cohesive bolus.
Oral (Propulsive) Phase
Tongue propels bolus posteriorly toward the oropharynx to trigger swallowing reflex.
Pharyngeal Phase
Reflexive stage with velopharyngeal closure, laryngeal elevation, and UES opening to move bolus.
Esophageal Phase
Involuntary peristaltic transport of bolus from UES through esophagus to stomach.
Thin Liquid
Fluid consistency like water; highest aspiration risk.
Nectar-Thick Liquid
Mildly thick fluid similar to apricot nectar; flows slowly.
Honey-Thick Liquid
Moderately thick fluid that pours like honey.
Pudding-Thick Liquid
Extremely thick, spoonable consistency resembling yogurt; does not pour.
Soft/Mechanical Soft Solid
Easily chewed foods such as cooked vegetables or tender meats.
Pureed Solid
Smooth, cohesive food requiring no chewing, e.g., applesauce.
Eight-Step Continuum
Hierarchical cueing program that gradually fades clinician support to improve word production in AOS.
Fasciculations
Visible, involuntary muscle twitches indicative of LMN damage.
Jaw Jerk Reflex
Pathologic hyperactive stretch reflex of the masseter seen in bilateral UMN lesions.
Spasmodic Dysphonia
Hyperkinetic laryngeal disorder causing spasms during speech, treated as a hyperkinetic dysarthria.
Cricopharyngeal Muscle
Principal muscle of the UES that relaxes during the pharyngeal swallow to allow bolus passage.
Diadochokinetic Rate
Speed and regularity of rapid alternating speech movements; slowed in CAS and ataxia.