1/38
Week 3 material
Name | Mastery | Learn | Test | Matching | Spaced | Call with Kai | Chat |
|---|
No analytics yet
Send a link to your students to track their progress
metabolic acidosis
hyperkalemia associated
hypokalemia causes
b2 ag (terbutaline, albuterol), diuretics, amphotericin, gastro losses, low intake, hypomag
hypokal treatment
pills, aldosterone antagonists, iv, magnesium
hyperkal causes
intake, decreased elim, unresponsive to aldosterone, metabolic acidosis, hemolyzed sample
Hyperkal symptoms
Muscle weakness, ecg/arrythmia
hyperkal treatment
1)If arrythmia then cacl/cagluc periph 2)insulin or insulin with dextrose <250 3)albuterol 4)bicarb if acidotic 5)dialysis/resin exchange
hyperkal alternate treatment
loop diuretics with normal renal fxn, lokelma+SPS
hypomagnesium causes (Gi absorb + renal elim)
GI loss, renal disorder, magnesium citrate laxative overuse, diuretics, ampho B, tacrolimus, cyclosporine
hypomag treatment
oral tablets then bolus
hypermag treatment
Ca for arrythmia, bolus, NS preferred unless acidosis,loops, hemodialysis if renal failure. Reduce intake.
Calcium is more bound to albumin when
metabolic alkalosis occurs
hypocal causes
hypopth, vit d deficit, renal fail, hypomag, loops, calcitonin
hypocal symptoms
cramps, seizure, arrythmia, dermatitis, ecg issues
hypocal treatment
Oral cal, vit d, bolus
hypercal symptoms
constipation, n/v, kidney stones, coma, ecg changes (short qt)
hypercal treatment- asymptomatic>12
Bolus(unless hf), IV Loop, IV Calcitonin, IV Glucocorticoids, IV Bisphosphonate
Hypercal treatment-symptomatic ok kidneys
Bolus(unless hf), loop, calcitonin, glucocort
Hypercal treatment-symptomatic bad kidneys
Hemodialysis, calcitonin, glucocort
Hypophos causes
Low Gi absorb (sucralfate, cacarb, sevelamer), Increased excretion (acetazolamide) Redistribution (Insulin, alcohol, ketoacidosis)
Hyperphos causes
Low GFR, sodium phosphate enema, bisphos (-nate), rhabdomyolysis
Base excess
Indicates alkalosis, positive
Acidemia/alkalemia values
<7.35 >7.45
Lungs regulate which component
PaCO2 via ventilation
Kidneys regulate which component
HCO3 via cl excretion, less cl = more basic
Ideal BG values
pH 7.4, pCo2 40±5 HCO3 24±2
Metabolic acid compensatory
40-1.3(24-bicarb)
Metabolic alk compensatory
40-0.6(bicarb-24)
metabolic acid general causes
higher inorganic acid, high acid from renal failure, low bicarb
metabolic acid non-anionic gap causes
diarrhea, cacl, rapid ns
metabolic acid causes anionic gap (mudpiles)
methanol, urea, diabetic ketoacidosis, paraldehyde, isoniazid/ischemia, lactic acid, ethylene glycol, salicylates/starvation
mudpiles lactic acid drugs
metformin, linezolid, propofol, pento/phenobarb, ativan
Plasmalyte, LR, NS, D5W pH
7.4, 6.5, 5.5, 5
Metab acid treatment
Treat disorder, replace body fluid, give bicarb by mouth or bolus, or sodium acetate if good liver
metabolic alk causes (saline responsive)
vomiting, diuretics, too much bicarb, k deficit
metabolic alk causes saline resistant
excess mineralcorticoid, mg deficit, milk-alkali, blood transfusion, estrogen
metabolic alk treatment saline responsive
correct disorder, give NS bolus, 2nd acetazolamide with K, 3rd arginine hcl(good liver/kidney)
metabolic alk treatment saline resist
change mineralcort, give spironolactone, give K
resp acid causes
sedatives, stroke, asthma/copd, brainstem injury
resp alk causes
anxiety, pain, anemia, high altitude