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the epiphyseal plate is _____
the main site of longitudinal growth
what type of fracture?
you see a buckle that is incomplete (doesn’t go to other side, heals fast
torus fracture
What type of fracture?
you see that the periosteum is intact on one side (when you follow the bone cortex it is only messed up on one side)
greenstick fracture
what type of physeal fracture is this?
growth plate
the fracture is through the zone of hypertrophy (straight across)
growth disturbances are uncommon
treat with casting or splinting
Salter Harris Type 1
What type of physeal fracture is this?
metaphysis and growth plate
occurs above the growth plate
usually will not cause functional limitations
salter harris type 2
what is the most common type of physeal fracture?
salter harris type 2
What type of physeal fracture is this?
epiphysis and growth plate
chronic disability due to extension of fracture into the articular surface
treatment is surgical
salter harris type 3
what is the salter Harris scale?
What type of physeal fracture is this?
epiphysis, growth plate, and metaphysis
intra-articular fracture
goes from the articular surface through the growth plate
salter harris type 4
What type of physeal fracture is this?
compression or crush fracture
rare
growth is affected
injury occurs at the growth plate
salter harris type 5
What are some things that would constitute a high degree of suspicion for child abuse
note: there are a lot, so try to name a few, but most importantly just read them and try to understand them rather than memorize
babies with bruises (esp less then 12mo when they aren’t really walking)
back and torso bruising
inconsolable ccrying
burns
whip marks
unkempt or dirty appearance
nervous parent
quiet child with poor eye contact
child with an overly serious demeanor
parental putting down of child
inappropriate sexual behaviors in a young child
What are two things we commonly see with shaken baby?
subdural hematoma (a coup contrecoup injury) and retinal hemorrhages
mechanical instability of the hip
risk factors: female, first born, breeched, prenatal factors like not enough amniotic fluid, and swaddling
developmental dysplasia of the hip
What are the tests for DDH and what do we use to confirm?
barlow test: try to provoke hip dislocation
ortoloni test: move a dislocated hip back into acetabulum
galeazzi test: eval for current hip dislocation
use ultrasound to confirm
How do we treat DDH (developmental dysplasia of the hip)?
pavlik harness or spica cast
idiopathic avascular necrosis of the femoral head
male predominant
diagnosis made by radiography
Legg-Calve-Perthes disease
clinical presentation:
pain at hip, knee, groin, or anterior thigh
limp with or without pain and Trendelenburg gait
hip ROM decreased: esp abduction and internal rotation
Legg-Calve-Perthes disease
what are some ways we can treat Legg-Calve-Perthes disease?
minimal weight bearing
pain control
PT
bracing (Petrie cast)
if severe, surgery will be required
seen in obese, pubertal children during their growth spurt
more common in boys
you see: hip, thigh, groin, or knee pain, a limp, external rotation with flexion and decreased ROM
slipped capital femoral epiphysis
you see Salter harris type 1 fracture of the proximal femoral epiphysis and slippage through zone of hypertrophy
slipped capital femoral epiphysis
How do we treat slipped capital femoral epiphysis?
surgery
osteochondritis of the tibial tuberosity
pain and swelling at tibial tubercle
anterior knee pain made worse with kneeling, running, and jumping
Osgood-Schlatter disease
how do we treat Osgood-schlatter disease?
rest, ice, and ibuprofen
involves both the muscular and skeletal parts of the foot
treated with serial casting and possible surgery
you see plantar flexion of first ray, adduction of the forefoot/midfoot on the hindfoot, hindfoot points inward, and plantar flexion of the hindfoot
clubfoot
when you draw a line through heel you should cross through the second toe, but instead you cross lateral to the second toe
treatment involves stretching the foot (can be done by parent at home)
metatarsus adductus
pathogenesis: acute hematogenous spread, direct spread from adjacent infection, or direct inoculation
you see an initial site of infection, then an abscess forms
bacteriology: Staph aureus most common (E coli in neonates)
you see: fussiness, poor movement, point tenderness, etc.
for diagnosis need 2+: purulence of bone, positive culture, localized erythema, or positive imaging
acute osteomyelitis
How do we treat acute osteomyelitis?
IV therapy for minimum of 2-3 weeks
starts empiric until you get culture results
empiric: clindamycin, vancomycin, or linezolid
you see:
± fever
subtle onset
± limp
normal ESR (no inflammatory markers in blood)
a prior illness (usually viral or GI infection)
normal x-ray and ultrasound
transient synovitis
you see:
high fever
sudden onset
limp present
high ESR (inflammatory markers in blood)
no prior illness
abnormal x-ray and ultrasound
bacterial septic arthritis (medical emergency)
disorder of Ca and PO4 homeostasis
severe prolonged vit D deficiency
bones are softer and weaker so they bow
nutritional deficiency most common
Rickets
calcipenic versus phosphopenic rickets; what should we know?
calcipenic is more common; vitamin D deficiency or resistance
x-ray findings: bowing, rachitic rosary, and fraying at the ends of the bones
rickets