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Pruritis which worsens at night, which is especially interdigital
Scabies until proven otherwise
Combination of products to avoid when treating a fungal infection
Antifungals with steroids
Differential diagnoses of Tinea Cruris
Candidiasis,
Seborrheic dermatitis,
Intertrigo,
Psoriasis,
Erythrasma
Complications of Tinea pedis
Portal of entry for bacterial cellulitis
Treatment of macerated Tinea pedalis
20min aluminum acetate soaks, topical turbinafine
Treatment of dry and scaly Tinea pedis
Local Terbinafine, urea 10% lotion
Diagnosis and treatment of Tinea capitis
Confirm with fungal testing (systemic is necessary)
Oral tervinatine
Exception of Tinea capitis treatment
Microsporum responds better to oral Griseofulvin
Presentation of Tinea unguium
Thickened, discolored, brittle, distorted nails separated from the bed
Important diagnostic/treatment note of Tinea unguium:
Systemic treatment rq, so confirm fungal infection
DDx of Tinea unguium:
Trauma, psoriasis, plenty of other nail dystrophies
First line treatment of cutaneous candidiasis
Topical Nystatin or -azole
What tinea infections require systemic therapy?
tinea capitis, tinea unguium
Types of Pediculosis
Capitis, corporis, and pubisW
Where are lice/nits found in pediculosis corporis
clothing seams
Treatment of Pediculosis
Replace/clean contaminated laundry
Permethrin rinse, repeated at day 9
DDx of Pediculosis
Seborrheic Dermatitis
Intense general pruritis which is often worse at night, with 3mm burrows
Scabies signs &Sx
Common areas of scabies itching/burrows
Interdigital, wrists, palms, axillae, areolae, pubis, and scrotum
Important history to take that may indicate scabies
Whether other residents have had itching
What is the more-contagious subtype of scabies?
Crusted scabies
What is an important note once scabies Dx has been reached?
It’s highly contagious (especially crusted scabies)
What infestation skin disease is a major infection control concern?
Crusted scabies
Scabies treatment
Permethrin 5% cream, or oral ivermectin
Counseling for scabies treatment
Pruritis may persist for a time after treatment ends
Types of tinea
Capitis, Corporis, Cruris, Pedis, Unguium
Pruritis, sharp borders, advancing scaly edge, central clearing, sometimes accompanied by maceration/fissuring
Tinea
What discerns tinea cruris from candidiasis?
Scrotum is spared from Tinea cruris
KOH that reveals hyphae
Tineae
Where to take a KOH scaping from on a possible fungal infection?
Advancing border
Treatment of Tinea corporis, cruris, and pedis
Topical Terbinafine
DDx of tinea
Lupus erythematous,
syphilis,
granuloma annulare,
pityriasis rosea,
annular psoriasis
Bright red rash with satellite papules/pustules, often accompanied by maceration
Candidiasis
Common locations of candidiasis
Inframammary and abdominal folds, groin, axillae, diaper, fingers
KOH that reveals pseudohyphae
Candidiasis
Candidiasis treatment
Topical nystatin or -azole
Hypopigmented macules/patches with a fine scale. Asymptomatic, or mild pruritis
Tinea Versicolor
KOH that reveals short, “spaghetti and meatball” hyphae
Tinea Versicolor
Bright red, raised, sharply-demarcated plaque. Possible fever, chills, and malaise
Erysipelas
Superficial lymphatic infection
Erysipelas
What tests can support a clinical diagnosis of erysipelas?
CBC showing leukocytosis, blood cultures
Deep dermis/subcutaneous infection
Cellulitis
Tender, poorly-demarcated erythematous skin
Cellulitis
Tender, poorly-demarcated erythematous skin with associated lymphangitis and lyphadenopathy
Cellulitis
Cellulitis without drainage, fluctuance, or abcess
Non-purulent (strep)
Cellulitis with drainage, fluctuance, or an abcess
Purulent; Staph
Tender, poorly-demarcated erythematous skin which has fluctuance
Purulent cellulitis
Treatment for bright red skin that is raised and sharply demarcated or tender and poorly demarcated, but has no fluctuance
Oral cephalexin or dicloxacillin
IV cefazolin or nafcillin
Penicillin allergy: clindamycin
Oral treatment for suspected erysipelas or non-purulent cellulitis
Cephalexin or dicloxacillin
IV treatment for suspected erysipelas or non-purulent cellulitis
Cefazolin or nafcillin
Penicillin-allergic treatment for suspected erysipelas or non-purulent cellulitis
Clindamycin
counseling & guidelines for suspected erysipelas or non-purulent cellulitis
Reassess response in 1-2d.
If worsening, change antibiotic.
If purulent, change treatment
Maintain meds for 5d to 2 weeks
Treatment for purulent cellulitis
Drain abcess, then TMP-SMZ, doxy, clinda, or vanco
Rapidly-spreading redness with severe pain, can be accompanied by dusky/purple color, bullae, crepitus, or necrosis
Necrotizing Fasciitis
Treatment for suspected necrotizing fasciitis
URGENT surgical consult, and broad-spectrum IV antibiotics
Superficial blisters with purulent material, leaving “honey-colored” crusts
Impetigo
What is Pilonidial disease?
A hair-related acquired PROCESS of cyst formation
Treatment for Pilonidial disease
Drainage, or Surgical ref for chronic cases
Pathogens of folliculitis (3)
Staph (normal)
Pseudomonas (hot tub)
Malassezia (fungal)
Highly pruritic folliculitis
Malassezia (fungal)
Treatments of folliculitis
Top mupirocin or clinda
Dermatitis that presents with pruritis and vesicles
Allergic contact dermatitis
Dermatitis with burning and dryness
Irritant contact dermatitis
Morbilliform/maculopapular eruption with symmetric presentation
Likely cutaneous drug eruption, measles, contact dermatitis, seborrheic dermatitis, or tinea
How to discern between measles and drug eruption?
Correlate with recent drug exposure