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Latent phase
onset of labor to 4cm dilatation
Active phase
4cm to complete
dilatation
Second stage:
From full dilatation
to birth of baby
First stage
Time span from beginning of regular contractions to complete cervical dilatation
Latent phase: NULLIPARA
ave
upper Normal
8.6 hours
20 hours
Latent phase: MULTIPARA
Ave
upper Normal
5.3
14
Active phase: Nullipara
Ave
Upper Normal
4.9 — 1.2 cm/hr
12
Active phase: multipara
Ave
Upper Normal
2.5 — 1.5 cm/hr
6 h
second stage
Full dilation to birth of baby
second stage: NULLIPARA
ave
upper normal
1
2 h without epidural, 3h with epidural
second stage: MULTIPARA
ave
upper normal
0.5
under 2 h without epidural, under 2h with epidural
placenta stage
Nullipara
Multipara
30 min
30 min
inertia
DYSFUNCTIONAL LABOR
DYSFUNCTIONAL LABOR (inertia)
Force of labor is less then usual
DYSFUNCTIONAL LABOR (inertia)
Sluggish contractions
DYSFUNCTIONAL LABOR (inertia)
Can occur at any point in labor but
classified as primary (occurring at the
onset of labor)
primary
occurring at the onset of labor
DYSFUNCTIONAL LABOR (inertia)
Causes (7)
o Primagavida
o CPD
o ROP or extension of fetal head than flexion
o Failure of uterine muscle to contract properly
o Overdistension of uterus (multiple pregnancy,
polyhydramnios, excessively oversized fetus)
o A mother exhausted from labor
o Inappropriate use of analgesia
DYSFUNCTIONAL LABOR
(Inertia)
Cause: Primagavida
DYSFUNCTIONAL LABOR
(Inertia)
Cause: CPD
DYSFUNCTIONAL LABOR
(Inertia)
Cause: ROP or extension of fetal head than flexion
DYSFUNCTIONAL LABOR
(Inertia)
cause: Failure of uterine muscle to contract properly
DYSFUNCTIONAL LABOR
(Inertia)
Cause:Overdistension of uterus (multiple pregnancy,
polyhydramnios, excessively oversized fetus)
DYSFUNCTIONAL LABOR
(Inertia)
Cause: A mother exhausted from labor
DYSFUNCTIONAL LABOR
(Inertia)
Cause: Inappropriate use of analgesia
Uterine contractions
basic force that moves the fetus through
the birth canal
adenosine triphosphate
major electrolytes
protein
epinephrine
norepinephrine
progesterone
prostaglandin
INEFFECTIVE UTERINE FORCE interplay of ____ and the influence of ____ and (5)
(calcium, sodium and potassium);
major
electrolytes 3
INEFFECTIVE UTERINE FORCE
▪ Interplay of adenosine triphosphate and the influence of major
electrolytes (calcium, sodium and potassium); and proteins
(action and myosin), epinephrine and norepinephrine,
progesterone and prostaglandin
action
myosin
INEFFECTIVE UTERINE FORCE proteins (2)
Dysfunctional labor occurs
▪ When they have less strength than usual or are rapid but
ineffective __ occurs
INEFFECTIVE UTERINE FORCE
When they have less strength than usual or are rapid but
ineffective====Dysfunctional labor occurs
Hypertonic Uterine Contractions
Occurs most often in Primigravidas
Hypertonic Uterine Contractions
▪ Marked by an increase in resting tone to more than 15mmHg
Hypertonic Uterine Contractions
Contractions are ineffectual, erratic, uncoordinated, and
involve only a portion of the uterus
Hypertonic Uterine Contractions
Increase in frequency of contractions, but intensity is decreased
Hypertonic Uterine Contractions
do not bring about dilation and effacement of the cervix
Hypertonic Uterine Contractions
Most commonly seen in latent phase of labor
primigravidas
Hypertonic Uterine Contractions
occurs often in:
15mmhg
Hypertonic Uterine Contractions
resting tone increases in
Frequency in contractions
intensity
Hypertonic Uterine Contractions
increase in___
decrease in ___
latent phase of labor
Hypertonic Uterine Contractions
most commonly seen in
dilation and effacement of the cervix
Hypertonic Uterine Contractions
do not bring about (2)
Muscle fibers of the myometrium do not repolarize
(relax after contraction)
▪ More painful than usual
▪ Myometrium becomes tender from constant lack of
relaxation
▪ Results to anoxia of uterine cells
Hypertonic Uterine Contractions CAUSES (4)
Hypertonic Uterine Contractions
cause
Muscle fibers of the myometrium do not repolarize
(relax after contraction)
Hypertonic Uterine Contractions
More painful than usual
Hypertonic Uterine Contractions
Myometrium becomes tender from constant lack of
relaxation
Hypertonic Uterine Contractions
Results to anoxia of uterine cells
anoxia of uterine cells
hypertonic uterine contractions results to
repolarize
(relax after contraction)
PAINFUL contractions in resting tone uterine muscle
anoxia
Dilatation and effacement of the cervix does not
occur
Prolonged latent phase
fetal distress occur early
anxious and discouraged
hypertonic uterine contractions
SIGNS AND SYMPTOMS(2)
stay at 2 - 3 cm
do not dilate as should
HUC
prolonged latent phase (2)
uterine resting tone
placental perfusion
HUC
Fetal distress occurs early= high, decrease in -
Lack of relaxation between contractions
may not allow optimal uterine artery
filling
HUC
Danger
fetal anoxia
Lack of relaxation between contractions
may not allow optimal uterine artery
filling can lead to
comfort measure (warm shower, mouth care, imagery, music, back rub)
mild sedation
bedrest
hydration
tocolytics to reduce high uterine tone
cs (as needed)
HUC
treatment (6)
tocolytics
___ to reduce high uterine tone
o Decelerations in the FHR
o Abnormally long first stage of labor
o Lack of progress with pushing (second-stage arrest)
HUC
TREATMENT
▪ Cesarean Section Delivery if:(3)
(second-stage arrest
Lack of progress with pushing
HYPOTONIC UTERINE CONTRACTIONS
Number of contractions is unusually infrequent
HYPOTONIC UTERINE CONTRACTIONS
Resting tone remains less than 10 mmHg
HYPOTONIC UTERINE CONTRACTIONS
Strength of contractions does not rise above
25mmHg
not more than 2-3 in 10 min
less than 10mmHg
does not rise above 22mmHg
HYPOTONIC UTERINE CONTRACTIONS
num of contraction
resting tone
strength
the active phase of labor
o After administration of anesthesia
o Bladder or bowel distention
hypo UC
▪ Apt to occur during: (3)
length of labor
bladder or bowel distention increases
overstretching of the uterus
bowel or bladder distention preventing descent
Etiology and Pathophysiology (2)
o large baby
o multiple babies
o Polyhydramnios
o multiple parity
hypo UC
Etiology and Pathophysiology
▪ Overstretching of the uterus (4)
analgesia
Etiology and Pathophysiology
Bowel or bladder distention preventing descent excessive use of
▪ Weak contractions – become mild
▪ Infrequent (every 10 – 15 minutes +) and brief
▪ Can be easily indented with fingertip pressure at peak of
contraction
▪ Prolonged ACTIVE Phase
▪ Psychological trauma - frustrated
hypo Signs and Symptoms (5)
o Exhaustion of the mother
o Risk for postpartal hemorrhage
hypo UC
s and sx prolonged active phase (2)
ambulation
nipple stimulation
enema
amniotomy
Hypo UC
Treatment (4)
Ambulation
getting up and walking will increase contractions
Nipple Stimulation
causes release of endogenous Pitocin which can
stimulate contractions
enema
warmth of ___ may stimulate contractions
endogenous Pitocin
Nipple Stimulation
o causes release of _______ which can
stimulate contractions
Amniotomy
o artificial rupture of the membranes
✓Contractions are more similar to those of
spontaneous labor
✓Usually no risk of rupture of the uterus
✓Does not require as close surveillance
Amniotomy
o Advantages of doing this before Pitocin (3)
AROM-Cot
amniotomy finger cot for the artificial rupture of membranes
o Delivery must occur
o Increase danger of prolapse of umbilical cord
o Compression and molding of the fetal head
(caput)
Amniotomy
▪ Disadvantages (3)
caput
molding of the fetal head
▪ # 1-Check the fetal heart tones
▪ Assess color, odor, amount
▪ Provide with perineal care
▪ Monitor contractions
▪ Check temperature every 2 hours
Amniotomy - Nursing Care (5)
Pitocin
for augmentation of labor
Pitocin
Use only if CPD is not present
CPD
PITOCIN is used only when ___ is not present
Pitocin
hang as a secondary infusion, never as
primary
20 units /1000 cc fluid
Pitocin give
▪ Use only if CPD is not present
▪ Give 20 units /1000 cc fluid
▪ hang as a secondary infusion, never as
primary
Pitocin – for augmentation of labor (3)
Achieve contractions every 2 - 3 minutes of good
intensity with relaxation between
Pitocin - GOAL:
pitocin
Achieve contractions every 2 - 3 minutes of good
intensity with relaxation between
o Assess contractions--are they increasing but not
tetanic
o Assess dilation and effacement
o Monitor vital signs and FHT’s
Pitocin Nursing care (3)
Prolonged Labor
A labor lasting more than 18-24 hours
o Cervical dilation
✓Primigravida 1.2 cm / hr
✓Multigravida 1.5 cm / hr
▪ Descent
o 1 cm. / hr in primigravida
o 2 cm./ hr. in multigravida
labor NORMAL
cx and descend (Primi and Multi)
Rapid delivery
Delivery/delivery outside normal setting
Rapid delivery
Everything is OUT OF CONTROL
▪ Do NOT leave the mother alone
▪ Try to make the place clean
▪ Try to get the mother in control (Panting)
▪ Apply gentle pressure to the fetal head
nursing care for rapid delivery (4)
BANDL’S RING
PATHOLOGIC RETRACTION RINGS
BANDL’S RING
Hard band that forms across the uterus at the junction of the
upper and lower uterine segments and interferes with fetal
descent
BANDL’S RING
Usually appears during the 2nd stage of labor
BANDL’S RING
Palpated as horizontal indentation across the abdomen