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atresia
body passice is closed or missing
ex: esophageal atresia โ incomplete formation of esophagus.
dysphagia
difficulty swallowing โ from obstruction in esophagus like esophageal atresia/stenosis/diverticula or neurological disorders
can also be caused by surgeries and procedures and meds that relax muscles
clinical manifestations
sensation of something stuck in throat
coughing
pocketing food in mouth
delayed swallowing
diganostic
physical exam
hx
x rays
egd
treatment
speech therapy
prevent aspiration
hepatomegaly
enlargement of liver โ caused by hepatits, CHF, cancer and cirrohsis
s/s โ abdominal pain, fatigue, jaundice, obesity
fulminant
severe medical conditions that develop suddenly and progess rapidly often fatal
chyme
mixture of food that has been chemically digested and churned in stomach
cholelithiasis
gallstones, stones in the gallbladder
cholecystits
inflmmation or infection in the biliary system caused by calculi
cholecystectomy
removal of gallbladder or calculi
ascites
fluid accumlated in the peritoneal cavity
jaundice
yellowing of the skin and sclera caused by excessive bilirubin
albumin
vital protein produced by liver that maintain fluid blanace, prevent blood vessel leakage, and transports hormones, drugs, and enzymes
ammonia
toxic waste product of protein digestion that healthy liver converts to urea
encephalopathy
any disease or dysfunction of the brain that alters structure and function
paracentesis
low risk prodcedure to remove ascites from abdomen to relieve pressure
bilirubin
yellowish pigment produced when rbc break down
varices
abnormal dilated swollen veins in lower esophagus or stomach caused by high bp in liver due to cirrhosis
bristol stool chart
types
separate hard lumps hard to pass
sausage shaped but lumpy
like a susage with cracks on surface
laike a susage smooth and soft
soft blobs with clear cut passsed easily
fluffy pieces with ragged edges mushy stool
watery no solid
melena
black tarry stool indicated upper gi bleed
hematochezia
bright red stool, lower gi bleed
steatorrhea (yellow color)
fatty greasy, foul smell, floating stool indicate malabsoprtion or celiac disease
oocult blood
blood in stool not visible to eye
acholic stool
pale, clay colored indicates lack of bile or gall bladder problems
bilious stool
greenish bile stained stool
acute hepatitis sympotmatic phase
prodromal phase - 2 wks after exposure to virus, symptoms of nausea, vomiting, malaise, anorexia, low grade fever, headache
icteric phase - 1-2 wks after prodomal last 6 wks
jaundice, dark color urine - bilirubinuria, clay colored stools, hepatomegaly, ruq pain
recovery phase - resolution of jaundice 6-8 wks after exposure, liver enlarged for up to 3 months
peritonitis
inflammation fo peritoneum, result from chemical irriation or organism invasion
assessment finding of abdomen from peritonitis
rigid boardlike abdomen
abdominal msucle spasm
tenderness
pain when flxing hips, pressure
three main nursing problems w GI
imbalanced nutrition
abnormal bm
risk for bleeding
barium swallow
fluoroscopy that uses x ray and chalky contract liquid to examine throat and esophagus
to diagnose
swallowing difficulties
reflux
blockages
upper gi series
x ra test using fluoroscopy and barium contract to image esophagus, stomach, and dodenum
manometry
measures pressure muscle contraction strength and relaxation of esohpagus and sphincters when swallowing
breath test
measure methane and hydrogen gases
acute vs chronic gastritis
acute
mild, transient irritation
can be severe ulceration with hemmorhage
presence of PMNs in mucosa
nausea epigatric pain
chronic
develops gradually
last for months or years
atrophy or metaplasia seen
dull epigastric pain
sensation of fullness after minimal intake
asymptomatic
pathogensis of hp in gastritis
hp embeds in mucosal layer activating toxins and enzymes that cause inflammation.
symptoms of duodenal vs gstric ulcers
duodenal
its epigastric pain is relieved with food but returns 2-3 hrs after eating
gastric
pain worsened with food
pancreatits
inflammation of pancreas
caused by
cholelithiasis
alochol abuse
biliary dysfunction
hepatotoxic drugs
metabolic disorders
trauma
endocrine disorders
tumors
leads to
renal failure
malnutrition
cancer
diabetes
respiratory distress
s/s
upper adom pain
nausea vomiting
mild jaundice
fever
indigestion
loss of weight
steatorrhea
flatulence
diagnostic
physical
serum amulase
lipase level
serum calcium
CBC
liver enzyme panel
x ray
ct
bowel obstruction
blockage of intestinal contents
caused by
gi tract dysfuntion
foreign bodies
tumors
adhesions
hernia
volvulus
stricture
fecal impact
intussusception
appendicitis patho
obstruction in appendix caused by lymphoid hyperplasia leading to distention, bacterial overgrowth, ischemia, inflammation.
the inflammation causes edema causing small obstruction as fluid builds microorganisms proliferate it compresses blood vessels in the area.
IBD (inflammatory bowel disease)
chronic inflammation in gi tract usually intestine. seen in women
crohn disease
a chronic inflammatory bowel disease (IBD) causing persistent inflammation of the digestive tract, most commonly the small intestine.
clinical manifestations
abdominal pain,
severe diarrhea,
fatigue,
weght loss,
and malnutrition
crohn disease patho
chronic, transmural inflammatory bowel disease driven by a dysregulated immune response to intestinal bacteria in genetically susceptible individuals
ulcerative colitis patho
a dysfunctional immune response (often Th2-mediated) against commensal bacteria, leading to a breached intestinal epithelial barrier, increased mucosal permeability, and chronic inflammation with crypt abscesses and surface ulcers
ulcerative colitis clinical manifest
chronic diarrhea often with blood/mucus, severe fecal urgency, abdominal cramping, and rectal pain.
colorectal cancer s/s
persistent change in bowel habits (diarrhea/constipation), rectal bleeding, unexplained anemia, and abdominal pain