1/42
Looks like no tags are added yet.
Name | Mastery | Learn | Test | Matching | Spaced | Call with Kai |
|---|
No analytics yet
Send a link to your students to track their progress
when can we rule in acute LBP w/ radiating pain
1. symptoms reproduced/aggravated with mid range and worsen w/ end spinal ROM, lower limb tension/SLR, and/or slump test
2. signs of nerve root involvement
when can we rule out acute LBP with radiating symptoms?
lower limb tension (SLR) and slump test don't reproduce LBP or leg pain
when can we rule in acute LBP with referred/related LE pain?
1. low back and LE pain can be centralized and diminished w/ positioning, manual procedures, and/or repeated movement
common signs of acute LBP with referred/related LE pain
lateral trunk shift, decreased lumbar lordosis, limited lumbar extension ROM, movement coordination impairments
when can we rule out acute LBP with referred LE pain
baseline assessment of pain location and level is not changed with prolonged position change, manual therapy, or repeated movement
when can we rule in chronic LBP with referred LE pain
symptoms reproduced/aggravated with sustained end range tension/SLR/slump test
when can we rule out chronic LBP with referred LE pain
lower limb tension/slump test do not reproduce LBP or leg pain
extension classification typical MOI
bending, lifting, twisting
extension typical pt presentation
age late 20s-early 50s
acute LBP, often with radiating features
shift behavior, antalgic posture, muscular spasm, trouble staying in upright postures
why does full flexion tend to cause injury
support must shift from muscles to disks/ligaments when flexed
shear force>tissue tolerance
disc failure is characterized by ___ failure and ___ protrusion of the disc
annular; posterior
what may exacerbate mal-effects of fully flexed spine
vibration
put these in order of lowest to highest intra-discal pressure: sitting, supine, standing
supine
what happens when we bend forward to disc pressure? what about when we bend forward with a weight?
increase; further increase
what are some reasons why patients might centralize with extension
extension causes nucleus to migrate anteriorly
extension moves compression from disc to facet joints
extension allows discs to rehydrate --> dec compression
blocked extension principle characteristics
flexion worsens symptoms, cannot test extension hypothesis, not usually a candidate for mobiilzation
unstable/volatile status characteristics
symptoms worsen rapidly with flexion movement/position, may achieve rapid improvement with extension
stable/mechanical status characteristics
gradual improvement with sustained/repeated extension, worsen with sustained/repeated flexion
what aspect of clinical status, if answered by the patient, requires further explanation
remain the same (improve and worsen are self explanatory)
what constitutes a status quo designation?
neither centralization, nor peripheralization occur
transient increase/decrease in pain occurs during movement
what might we consider with status quo patients
mobility deficits, active rest
examples of worsened symptoms
inc neuro signs, parasthesia inc/peripheralizes, pain inc/peripheralize, deformity inc, AROM dec
examples of improved symptoms
dec neuro signs, parasthesia dec/centralizes, pain dec/centralizes, deformity dec, AROM inc
what constitutes peripheralization
pain/parasthesia moves distally, new parasthesia produced, symptoms present/produced increase in intensity and remain increased for 30+ seconds after movement stops
what constitutes centralization
pain/parasthesia moves centrally towards spine, parasthesia goes away, symptom diminishes/abolished during movement and stays that way for 30+ seconds after movement stops
acute extension classification
unstable pattern, peripheralization w/ flexion, centralization w/ extension
chronic extension classification
stable pattern, pain increases but status quo w/ flexion, pain decreases but status quo w/ extension, history suggesting extension preference is present
co-existing factors in pt history
diagnosed disc or degenerative pathology, LE symptoms, positive neurological symptoms
co-existing factors in pt exam
status improvement during movement testing (flexion, extension, pelvic translocation, repeated/sustained movement)
further movement testing procedures for extension classificaton
repeated/sustained extension in standing/quadruped/prone to confirm that extension movement and/or posture improves status
extension classification pt education
limit sitting, maintain lordosis when sitting (discuss lumbar supports, sitting surfaces), how to get out of bed while maintaining extension (prone on elbows)
80% of lateral shifts are ___
away from herniation
other terms for lateral shift
lumbar list, trunk list, sciatic scoliosis
pathomechanics associated with lateral shift
disc herniation, muscle spasm (protective), segmental instability
typical history elements associated with lateral shift
flexion MOI, acute onset, visual deformity that worsens w/ WB, sitting and walking worsen symptoms, radicular signs/symptoms
typical exam findings with lateral shift
visible deformity
+ side bend test
signs of nerve root compression
asymmetrical side bending
asymmetrical pelvic translocation
if status improves with pelvic translocation in lateral shift pt, what should be attempted next
extension in pelvic translocation
T/F: NWB positions usually increase lateral shift
F, reduce
prone progression for lateral shift
prone lying with shift correction
prone lying with shift over-correction
prone extension exercises
if prone progression does not improve status, what should be considered next?
traction (especially if pt has radicular sx)
after correction of lateral shift, patient must avoid ___
all flexion
progression of treatment for lateral shift patients
NWB correction, WB correction, extension progression, motor control
manual therapy techniques to enable extension
prone P-A, pelvic leg lift, pelvic prone/SB/ext, lumbar extension (closing), side lying pelvic correction (lazy man's roll), long axis distraction