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conduction system
made up of electrical cells
follows a specific pathway
SA-AV-Bundle of HIS- Bundle of branches- Purkinje Fibers
EKG
6chest leads V1-V6
6 limb leads
Unipolar AVR, AVL, AVF
Bipolar I(LA), II(LL), III(LL) leads
SA NODE
Receives blood from SA node artery which originates from the right coronary artery
has intermodal pathways
AV NODE
impulse delayed slightly
Ventricle filling
Supplied by the right coronary artery
AV NODE
impulse delayed slightly
Ventricle filling
Supplied by the right coronary artery
Bundle of HIS
Thin bundle of subendocardial threads (muscle fibers)
Receives blood from branches of the anterior and posterior descending coronary arteries
RIGHT Bundle Branch
This signal tells the RV when to contract, pushing blood to the lungs to get oxygen
LEFT Bundle Branch
signal makes LV contract, pumping oxygen rich blood out through aorta to the body
then divides into smaller branches called fascicles that spread electrical signal deep into the LV muscle
If the signal is blocked, LV get the signal late and the heartbeat can become uncoordinated
Purkinje Fibers
quickly spread electrical impulses to all parts of ventricular muscle
This makes R n L ventricles contract together-pushing
Their fast conduction speed ensures the ventricles contract for full and efficiently
Purkinje Fibers
quickly spread electrical impulses to all parts of ventricular muscle
This makes R n L ventricles contract together-pushing
Their fast conduction speed ensures the ventricles contract for full and efficiently
Intrinsic Rate
SA NODE 60-100
AV NODE 40-60
Purkinje fibers 20-40 (ventricular)
Interpreting an EKG
Many cardiac disorders:
check voltage calibration
heart rhythm and rate
Intervals (PR,ORS, QT)
Mean ORS axis
Abnormalities of P wave, QRS (hypertrophy, bundle branch block, infarction) ST segment n T wave
Voltage calibration
standard setting used to measure and display the strength (amplitude) of electrical signals
Normal 1mm vertical box on the EKG which represents 0.1mV (millivolts)
Tall QRS complexes, most common cause of this finding is an enlargement of thickened heart muscle called Hypertrophy
The rectangular upward deflection at the beginning of each line is the Voltage Calibration signal
Normal sinus rate with a 70 BPM

Heart Rhythm
P wave is followed by a QRS
P wave is upright in leads I, II, III
PR interval is 0.12 sec or greater (3small boxes)
Normal sinus rhythm 60-100
Bradycardia- less than 60 (slow)
Tachycardia- more than 100 (fast)
Intervals (PR,QRS, QT)
Normal ranges:
PR: 0.12- 0.20sec (3-5 boxes)
QRS: less than 0.10sec (less than 2.5 boxes)
QT: Males- less than 0.44s
Females- less than 0.46s
Abnormalities of P wave
aerial enlargement or loss of normal atrial activity
P wave is usually visualized best in lead II
When RA enlarges the initial P wave is larger than normal (taller than 2.5mm)

Abnormalities of QRS complex
ventricular hypertrophy: R n L ventricles cause the affected chamber to generate greater than normal electrical amplitude
Bundle branch blocks: may develop from ischemic or degenerative damage ex: fever
Most common:
R n L Bundle Branch Block
Ventricular Tachycardia
Pacemaker
ST segment and T wave abnormalities
common finding with wide range of causes, from benign conditions to life threatening ones like heart attack
They can signal myocardial ischemia (lack of blood flow to the heart), electrolyte imbalances (like high or low potassium), left ventricular hypertrophy, certain drugs, or other structural or neurological problems
Examples of ST segment and T wave abnormalities
Transient Myocardial Ischemia- temporary oxygen storage, no heart damage
Acute ST (STEM)- complete blockage, serious damage, emergency (DANGER!)
Acute non-ST (NSTEMI)- partial blockage, some damage, urgent treatment needed