exam 2: psychopathology chapter 5

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Last updated 9:17 PM on 9/24/22
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50 Terms

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fear
central nervous system's physiological and emotional response to a serious threat to one's well-being
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anxiety
central nervous systems physiological and emotional response to a vague sense of threat or danger
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generalized anxiety disorder (GAD)
disorders marked by persistent and excessive feelings of anxiety and worry about numerous events and activities
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most common disorders in US
-19% of us adults experience one of six DSM-5-TR anxiety
disorders
-around 31% develop one of the disorders at some point in their lives
-about 42% of these individuals seek treatment
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GAD diagnosis
1. 6 months or more, person experiences disproportionate uncontrollable, and ongoing anxiety and worry about multiple matters
2. symptoms include at least 3 of: edginesss, fatigue, poor concentration, irritability, muscle tension, sleep problems
3. significant distress or impairment
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sociocultural perspective (GAD)
-GAD is most likely to develop in people faced with dangerous ongoing social conditions or highly threatened environments (supported by research)
-societal stress: poverty, race and ethnicity, widespread illness
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separation anxiety disorder
-individuals feel extreme anxiety, and often panic, whenever they are separated from key people in their lives
-most common among young children
-DSM-5-TR determined it can develop in adults
-new categorization is tan anxiety disorder is controversial
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separation anxiety disorder diagnosis
-developing inappropriate and excessive fear or anxiety concerning separation from those to whom the individual is attached as evidenced by 3 of: fear, anxiety, or avoidance is persistent at least 4 weeks in children and adolescents and 6 months or more in adults and the disturbance causes clinically significant distress or impairment
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Freud on GAD
-all children experience some degree of anxiety and use ego mechanisms to control this
-GAD occurs with high anxiety levels or inadequate defense mechanisms
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todays psychodynamic theorists (GAD)
-disagree with some Freudian GAD explanations, but agree GAD Cana be traced to early parent-child relationships
-research testing psychodynamic explanations (GAD and defense mechanisms, repressed memories, harsh punishment, overprotectiveness)
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psychodynamic therapies (GAD)
-free association
-therapist interpretations of transference, resistance, and dreams to reduce fear of id impulses and control
specific treatment: Freudians focus less on fear and more on control of the id
-object-relations therapists attempt to help patients identify and settle early relationship problems
-short term psychodynamic therapy is more effective than longer treatments
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humanistic perspective (GAD)
-GAD arises when people stop looking at themselves honestly and acceptingly
-Carl Rogers: lack of unconditional positive regard in childhood leads to conditions of worth
-threatening self judgements break through an cause anxiety setting the stage for GAD to develop
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humanistic treatment approach (GAD)
client-centered therapy: used to show unconditional positive regard for clients and empathize with them
-despite optimistic case reports, controlled studies shave failed to offer strong support
-only limited support for Roger's explanation of GAD and other forms of abnormal behavior
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cognitive behavioral perspective (GAD)
-problematic behaviors and dysfunctional thinking often cause psychological disorders
-treatment focus involves the nature of behavior and throughs
-early approach: maladaptive or basic irrational assumptions, silent assumptions
-newer explanations: metacognitive theory, intolerance of uncertainty theory, avoidance theory
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cognitive behavioral therapies (GAD)
-changing maladaptive assumptions (Ellis rational-emotive therapy)
-breaking down worrying: mindfulness-based cognitive behavioral therapy
-acceptance and commitment therapy- mindfulness meditation
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biological perspective (GAD)
-caused chiefly by biological factors, supported by family pedigree studies and brain researchers, and challenged by competing explanations of shared environment
-fear reactions are tied to brain circuits
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biological explanations (GAD)
-everyday fear reactions are tied to brain circuits- fear circuit hyperactivity may be tied to the development of GA
-benzodiazepines provide anxiety relief-: benzodiazepine receptors ordinarily receive neurotransmitter gamma-aminobutyric acid
-GABA carries an inhibitory message that ends the firing of neuron receptors- low GAB could help produce excessive brain circuit communication and contribute to GAD development
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drug therapies (GAD)
-early 1950's: barbiturates (sedative-hypnotics)
-late 50's: benzodiazepines (significant problems)
-more recently: antidepressants that increase serotonin and norepinephrine neurotransmitter activity, antipsychotics
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phobias vs. fear
-more intense and persistent fear
-greater desire to avoid a feared object or situation
-create distress that interferes with functioning
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specific phobias
-yearly symptoms exist in 9% of US population
-13% experience symptoms during lifetime
-women 2 to 1 for men
-32% at most seek treatment
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specific phobia diagnosis
-marked, persistent, and disproportionate fear of a particular object or situation; usually lasting 6+ months
-exposure to an object produces immediate fear
-avoidance of the feared situation
-significant distress or impairment
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agoraphobia diagnosis
-pronounce, disproportionate, or repeated fear about being in the act least two delineated situations
-avoidance of agoraphobic situations
-symptoms usually continue for at least 6 months
-significant distress or impairment
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agoraphobia
-yearly symptoms exist in around 1% of US population
-1.3% experience symptoms during lifetime, gender differences
-around 46% seek treatment
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causes for specific phobias
-cognitive-behavioral theories receive most research support
-focus primarily on the behavioral dimension
-first fear of certain objects, situation, or events are learned through conditioning
-once fears are acquired, individuals avoid dreaded object or situation and permit fears to become entrenched
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phobia research
-early laboratory studies of classical conditioning fear (little Albert)
-modeling: bandora
fear reactions are not always conditioned: disorders no ordinarily acquired through classical conditioning or modeling
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behavioral-evolutionary explanation (PH)
-some specific phobias are much more common than others
-species biological predisposition to develop certain fears (preparedness)
-explains why some phobia are more common than other
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phobias treatments
-actual contact with the feared object or situation is key to greater success in all forms of exposure treatment
-systematic desensitization: relaxation training, fear hierarchy, in vivo or overt desensitization, virtual reality
-flooding, modeling
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agoraphobia treatment
-variety of exposure therapy approaches (support groups, home-based self-care programs)
-successful in around 70% of agoraphobic clients (as many as half relapse)
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social anxiety disorder
-pronounced repeated anxiety about the social situation in which an individual could be exposed to scrutiny by others (6 months or longer)
-fear of being negatively evaluated by or offensive to others
-exposure to the social situation almost always produces anxiety
-avoidance of feared situations
-significant distress or impairment
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cause of social anxiety disorder
cognitive behavioral perspective: a group of social realm dysfunctional beliefs and expectations help; the anticipation of social disasters and dread social situations
-avoidance and safety behaviors performed to reduce or prevent disasters
-tied to genetic predispositions, trait tendencies, biological abnormalities, trauma, overprotective parents
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treatment for social anxiety disorder
overwhelming social fears
-- medications: benzodiazepine or antidepressents
--cognitive behavioral therapy: exposure and systematic therapy discussions
lack of social skills
--social skills and assertiveness training
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social media jitterss
computer and mobile device use can produce more common forms of anxiety including generalized and social
-instagram, snapchat, TikTok, Facebook
-fomo, nomophobia
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panic attacks
-periodic short bouts of panic that occur suddenly, reach a peak within minutes and gradually pass
-fears at least 4 of the following: heart palpitations, tingling in hands or feet, shortness of breath, sweating, hot or cold flashes, trembling, chest pain, choking sensation, faintness, dizziness, feeling of unreality
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panic disorder
-unforeseen panic attacks occur repeatedly
-one or more of the attacks preceded either: at least a month of continual concern about future attacks
or a month of dysfunction behavior changes associated with attacks
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biological perspective of panic disorder
initial theory: panic attacks caused by abnormal norepinephrine activity in locus ceruleus
more recent: brain circuits and amygdala are the more complex root of the problem, may be inherited predisposition to abnormalities
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biological factors contributing to panic disorder
-hyperactive panic circuit: amygdala, hippocampus, ventromedial nucleus of hypothalamus, central gray matter, locus coerulues
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drug therapies for panic disorder
-various antidepressants bring some improvement to more than 2/3 of patients
-drugs function in norepinephrine receptors in the panic brain circuit
-improvements require maintenance of drug therapy
-some benzodiazepines (Xanax) have proved helpful
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cognitive behavioral perspective on panic disorders
-biological factors are only part of the cause of panic attacks
-bodily sensations are misinterpreted as signs of medical catastrophe and controlled by avoidance and safety behaviors
-anxiety sensitivity may exist

-biological challenge test: procedure used to produce panic and assess panic disorder
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cognitive behavioral therapy for panic disorder
-seeks to correct people's misinterpretations of their bodily sensations
-educate about nature of panic attacks
-teach applications of more accurate interpretations
-teach skills for coping with anxiety, including biological challenge procedures
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obsessions
persistent thoughts, ideas, impulses, or images that seem to invade a person's consciousness
-features: though feel both intrusive and foreign, attempts to ignore or resist them trigger anxiety, awareness the thoughts are excessive
-basic themes: dirt/contamination, violence and aggression, orderliness, religion, sexuality
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compulsions
repetitive and rigid behaviors or mental acts that people feel they must perform to prevent or reduce anxiety
-features: various forms of voluntary behaviors or mental acts, feeling mandatory/unstoppable, recognition the behaviors are unreasonable, performing behaviors that reduce anxiety for a short time, behaviors often develop into rituals
-themes: cleaning compulsions, checking compulsions, order or balance, touching, verbalizing and/or counting compulsions
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OCD
-occurrence of repeated obsessions, compulsions, or both
-obsessions or compulsions take up considerable time
-significant distress or impairment
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psychodynamic perspective for OCD
theory: battle between id and ego defense mechanisms lessens anxiety in overt thoughts and actions
-freud: oct related to anal stages of development
-not all psychodynamic theorists agree
treatment: classical techniques of free association and therapist interpretation; have little research support
-short term psychodynamic therapies are more direct and action-oriented
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cognitive behavior perspective of OCD
disorder grows from human tendencies to have unwanted, intrusive, unpleasant though
-to avoid negative outcomes individuals attempt to neutralize their thoughts with actions
-reduction in anxiety after neutralizing strategies
-exceptionally high standards of conduct; thought-action fusion
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cognitive behavior therapy for OCD
focus on cognitive processes that help to produce and maintain obsessive thoughts and compulsive acts
-use exposure and response prevention exercises: set example, videoconferencing in recent years, between 50%-70% see improvement with therapy
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biological perspective of OCD
early research: genetic focus- family pedigree and twin studies, heritability estimates 26%- 33%
recent research: brain circuit hyperactive in people with OCD
-abnormal serotonin activity, abnormal brain structure and functioning, some research evidence suggests these two lines may be connected
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biological treatment of OCD
-serotonin-enhancing antidepressants: clomipramine and similar drugs, improvement within 50%-80%-- relapse occurs if medication is stopped
-research suggests that combination therapy may be most effective
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religious OCD
-fear of not having enough faith, fear of hell, fear of being unclean or contaminated according to rules of one's religion, fear of committing immoral behavior
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OCD related disorders
-hoarding disorder
-trichotillomania (pulling hair)
-excoriation (skin-picking)
-body dysmorphic disorder
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epidemiology of OCD
OCD- lasts an average of 8.9 years
-mean time consumed by symptoms
-obsessions 5.9 hours
-compulsions 4.6 hours