Vet tech prep power pages: surgical nursing review

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Last updated 1:21 PM on 5/30/26
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272 Terms

1
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The kidney is resoponsible for

regulating water and electrolyte balance in the body

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the kidney maintains

acid-base homeostasis

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the kidney aids in retaining

protein and glucose in the body

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the kidney excretes

waste and toxins

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the kidney plays a role in many

endocrine functions by secreting hormones

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renal refers to the

kidneys

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acid (kidney)

a substance that releases hydrogen ions in solution (pH <7).

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base (kidney)

a substance that accepts hydrogen ions in solution (pH >7).

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Nephron

functional unit of the kidney

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renin

hormone secreted by the kidney;

-mediates extracellular volume and arterial vasoconstriction (regulates blood pressure) via the renin-angiotensin system

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Aldosterone stimulates

active sodium reabsorption

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antidiuretic hormone (ADH) stimulates

passive water reabsorption

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the kidney is made up of

nephrons

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The nephron is the

functional unit of the kidney

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The nephron is made up of

the glomerulus

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the glomerulus is surrounded by

Bowman's capsule and its tubule

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The tubule is sectioned into the

proximal tubule, the medullary loop of henle, and the distal tubule which empties into the collecting duct.

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Filtration in the kidneys happens through the

glomerular capillaries.

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the filtration inside the glomerular capillaries is driven by

hydrostatic pressure as a direct result of arterial pressure.

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Glomerular Filtration Rate (GFR)

the rate at which filtration through the glomerular capillaries occurs

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The loop of henle is resposible for

concentrating the urine

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what is resorbed at the loop of henle

sodium and chloride

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The final urine concentration is dependent upon the

amount of antidiuretic hormone secreted by the pituitary gland

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The proximal tubule resorbs

all glucose and amino acids, and most bicarbonate, phosphate, and water.

25
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Renin increases the production of

Angiotensin II

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Renin is released in response to

intravascular volume drops (dehydration, blood loss)

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renin release causes then causes aldosterone release from

the adrenal cortex

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renin release also causes increased release of

ADH from the posterior pituitary causing increased thirst, and constriction of the efferent arteriole to maintain GFR. In fluid overload, the opposite occurs.

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Aldosterone causes

sodium and water resorption in the distal tubule and collecting duct

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in the distal tubule and collecting duct

sodium is exchanged for potassium and hydrogen

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what is released when sodium is low in the blood

aldosterone

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examples of sodium loss

Vomiting or loss of gastric acids

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sodium loss leads to

alkalosis

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Antidiuretic hormone makes the

distal tube and collecting duct more permeable to water, which increases urine concentration

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When antidiuretic hormone (ADH) is present

the urine is concentrated

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When antidiuretic hormone (ADH)is not present

37
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Nerve impulses from the hypothalamus stimulate

the pituitary to make ADH when osmotic blood pressure rises.

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Vitamin D (calcitriol) is produced by the

to help promote calcium absorption from the intestine

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the kidney also produces erythropoietin which

stimulates production of red blood cells.

40
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Kidney values on a blood panel

BUN and creatinine

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Amylase is excreted through

the kidneys

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if renal function is impaired, amylase

rises

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important factors on kidney function besides BUN/creatine is

UA and electrolyte values

44
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How many cranial nerves are there?

12

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cranial nerve diagram

knowt flashcard image
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CN 1

Olfactory nerve

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CN 1- olfactory

Mediates the sense of smell, observed when the pet sniffs around its environment

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CN 2

Optic

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CN 2- Optic

Carries visual signals from retina to occipital lobe of brain, observed as the pet tracks an object with its eyes. It also causes pupil constriction.

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The Menace response

is the waving of the hand at the dog's eye to see if it blinks (this nerve provides the vision; the blink is due to cranial nerve VII)

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CN III

oculomotor nerve

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CN III - Oculomotor Nerve

• Provides motor to most of the extraocular muscles (dorsal, ventral, and medial rectus) and for pupil constriction o Observing pupillary constriction in PLR

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CN IV (4)

trochlear nerve

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CN IV (4)- Trochlear Nerve

Provides motor function to the dorsal oblique extraocular muscle and rolls globe medially

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CN V (5)

Trigeminal Nerve - Maxillary, Mandibular, and Ophthalmic Branches

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CN V (5)- Trigeminal Nerve - Maxillary, Mandibular, and Ophthalmic Branches

• Provides motor to muscles of mastication (chewing muscles) and sensory to eyelids, cornea, tongue, nasal mucosa and mouth.

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CN VI (6)

abducens nerve

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CN VI (6)- Abducens Nerve

• Provides motor function to the lateral rectus extraocular muscle and retractor bulb

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CN VI (6)- Abducens Nerve is tested by

touching the globe and observing for retraction (also tests V for sensory) Responsible for physiologic nystagmus when turning head (also involves III, IV, and VIII)

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CN VII (7) facial nerve

• Provides motor to muscles of facial expression (eyelids,ears,lips) and sensory to medial pinna(ear flap). Also taste to rostral tongue

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CN VII (7)

facial nerve

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CN VII (7) facial nerve helps with

tear production (schirmer tear test)

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CN VII (7) facial nerve- Palpebral response

-motor for the blink reflex when touching medial canthus (also tests V for sensory)

64
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can observe for facial paralysis by

CN VII (7) facial nerve

symptoms: deviation of nose to one side or droopy lips

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CN VIII (8)

vestibulocochlear nerve

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CN VIII (8) -vestibulocochlear nerve

Sensory for hearing and head position

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A head tilt or nystagmus may suggest dysfunction of

CN VIII (8)- Vestibulocochlear Nerve (vestibular disease or inner ear disease)

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CN IX (9)

Glossopharyngeal nerve

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CN IX (9)- Glossopharyngeal Nerve

•Innervates the pharynx for swallowing (with X).

-Also innervates some salivary glands and provides taste innervation from caudal tongue

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examine the CN IX (9) - Glossopharyngeal Nerve by

eliciting a gag reflex and observing for dysphagia (difficulty swallowing)

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CN X (10)

vagus nerve

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CN X (10) - Vagus Nerve

•Innervates the larynx, esophagus, and pharynx. Also provides parasympathetic innervation to the heart and viscera

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CN X (10) - Vagus Nerve is tested by

a gag reflex along with CN IX (9)

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CN XI (11)

spinal accessory nerve

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CN XI (11) spinal accessory nerve

•Innervates cranial cervical (neck) muscles

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CN XII (12)

hypoglossal nerve

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CN XII (12) hypoglossal nerve

• Motor to the tongue (causes tongue movement)

78
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GDV

Gastric Dilatation Volvulus (Bloat)

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GDV is a condition most prevalent in

dogs

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GDV mortality rate

15-30%

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Is GDV an emergency?

yes patients are typically in critical condition with shock at presentation, requiring aggressive resuscitation and immediate supportive care and monitoring

82
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definitive GDV treatment includes

surgical decompression and derotation of the stomach followed by gastropexy to prevent recurrence.

83
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GDV is most common in

large breed, deep chested dogs. Great dane is most common breed.

84
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When antidiuretic hormone (ADH)is not present

the tubule is not very permeable to water so the urine is dilute

85
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GDV is when the

-stomach rotates counterclockwise when viewing from cranial to caudal in dorsal recumbency

-Also can be stated that stomach rotates clockwise when viewed caudal to cranial

86
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GDV can result in

-venous compression, congestion, and local compromise of perfusion to the stomach, resulting in necrosis

-tearing of short gastric vessels connecting the stomach and spleen

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GDV leads to hypovolemic shock because

Air accumulates in stomach, eventually impeding venous return to the heart via the vena cava and resulting in hypovolemic shock

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GDV can lead to

-global tissue ischemia and systemic inflammatory response

○ Inflammatory mediators and myocardial ischemia can lead to arrhythmias

89
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GDV happens more in

-large to giant breeds

-deep chested dogs

-Great Dane, German Shepherd,

Rottweiler, Irish Wolfhound, etc

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great danes have what percent likelihood of developing GDV

37%

91
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Other predisposing factors for GDV

○ Related to a dog that had a GDV

○ Anxious dogs

○ Very fast eaters

92
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Clinical signs of GDV

● Restless/nervous pacing

● Painful ● Non-productive retching/vomiting

● Abdominal distension

● Increased respiratory rate

● Signs consistent with shock/cardiovascular collapse

○ Tachycardia, weak pulses, pale mucous membranes, prolonged CRT

○ Depressed to comatose mentation

93
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We diagnose GDV by

-ECG- arrythmias common, VPC most likely

-blood gas- Metabolic acidosis (lactic acidosis) +/- respiratory compensation:, May have hypercapnia from gastric distention and impaired ventilation

-radiographs

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treatment of GDV includes first stabilizing the patient:

● Place two large-bore cephalic catheters (avoid saphenous since caudal venous return is poor)

● Shock dose crystalloid fluid therapy (80-90 ml/kg in fractions until resuscitation achieved) ○ Patient's large size often requires use of pressure bags for rapid administration of fluids

● Monitor blood pressure and ECG ● Lactate levels may provide some insight as to prognosis

95
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Treatment of GDV includes decompressing the stomach:

1. Orogastric tube

a. Pros: more effective emptying

b. Cons: requires heavy sedation, tube might not pass, possible esophageal trauma/rupture

2. Trocarization

a. Pros: more rapid intervention, does not require sedation

b. Cons: limited decompression, risk of lacerating gastric wall, puncturing spleen

c. Trocarize at point of maximum tympany, can't be sure if spleen is on the left or right

96
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Treatment of GDV inlcludes: Surgey:

● Goal is to fully decompress and reposition stomach, evaluate viability of stomach and resect any necrotic tissue. Also assess spleen, determine if splenectomy is indicated (rarely necessary)

. ● Perform gastropexy to prevent recurrence. ○ Incisional, circumcostal, belt-loop, incorporating, tube.

97
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survival rate of GDV

Reported survival 75-90% with surgery and post-operative care

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Negative prognosis indicators of GDV

○ Lactate > 6 mmol/L

○ Need for gastric resection/splenectomy

○ Long onset of signs to time of presentation (5 or 6 hours)

○ Recumbency at presentation

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If a patient has GDV and a gastropexy is performed, the recurrence of GDV is

less than 4%

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If a patient has GDV and a gastropexy is NOT performed, the recurrence of GDV is

recurrence is 50%