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Hemorrhage
- External or internal loss of a large amount of blood in a short period
- Do not become distracted by ugly but non-life threatening bleeding
Bleeding Control
- MOI of the bleed, don't get hurt
- BSI gloves at minimum (eye protection, face shield, gown)
- Signs and symptoms of shock
Arterial Bleeding
- Bright red
- Spurting, pulsating
Venous Bleeding
- Darker red
- Flowing blood
Capillary Bleeding
- Medium red
- Oozing
Severity of Bleeding
- Volume loss and rate of bleeding (>15% leads to shock)
- Patient's state of health
- Age and size (Adult: 70ml/kg, Peds: 80ml/kg)
Determining Blood Loss
Assess mental status of patient (pulse, skin vitals, respiration, BP)
Control of External Hemorrhage
- Direct pressure (with gloved hand or sterile material)
- Elevate above the heart (preferably with pressure)
- If suspected fracture, don't elevate until splinted
Tourniquets
- Devices used to stop bleeding in an emergency
- Can be left on for 8-10 hours
Bleeding from Nose or Mouth
- Check airway patency - suction needed?
- If nose bleed: do not tip head back - aspiration/throw up blood
- Be in lateral recumbent, sit up/lean over to drain
Bleeding from Ear
- May be due to basilar skull fracture (CSF present)
- Use loose dressing, do not put pressure or block drainage - avoids brain bleed
- May have CSF
Bull's Eye Test
Drop of blood on gauze and see if yellow ring forms, if so basilar skull fracture - CSF travels faster than blood
Internal Bleeding
- MOI determines suspicion - think anatomy damages
- Observe for pain, tenderness, bruising, distended abdomen (enlarging belly), bleeding from GI or genitourinary, shock
- Initiate transport quickly if suspected
Signs of Shock
- Decreased blood pressure
- Rapid, weak pulse
- Mottled to gray, clammy, cold skin
- Change in mental status
Decreasing Clotting and Increasing Bleeding
- Coagulopathy
- Movement or removal of dressing
- Decrease in body temperature
Coagulopathy
Impaired clotting leading to continued bleeding
Body Temperature and Bleeding
- Normal body temp is 98.6
- Drop 1 degree below and clotting slows
- If patient is wet, increase losing heat due to evaporation
Ambulance on Scene for Cold Patient
- Close doors when patient is in trunk
- Turn on heat, if your not sweating patient is cold and bleeding
Non-Steroidal Anti-Inflammatory Drugs
- Provide mild pain relief
- Aspirin
- Ibuprofen
- Naproxen
Anti-Coagulant
Breaks up formation
Anti-Platelet
Stops clot formation
Most Common Anti-Platelet
Plavix (Clopidogrel)
Most Common Anti-Coagulants
- Coumadin (Warfarin)
- Heparin (Heparin)
- Eliquis (Apixaban)
- Xarelto (Rivaroxaban)
Hypovolemic Shock Hemorrhagic
- Shock resulting from external or internal bleeding
- Plasma/whole blood loss (RBC, platelets, plasma)
Hypovolemic Shock Non-hemorrhagic (Fluid Deplition)
- Vomiting, diarrhea, dehydration, burns
- Plasma lost but other components intact
- Losing fluids causes fluid to come out of vessels but leaves RBC/WBC
Cardiogenic Shock
- Shock caused by inadequate function of the heart, or pump failure
- Loss of cardiac contractility, reduced cardiac output
- Heart attack, disease, trauma, age, dysrhythmias
- Too fast: chambers can't fill; Too slow: can't maintain pressure
Disruptive Shock
- Septic shock, Neurogenic shock, Anaphylactic shock
- Loss of peripheral vascular resistance
- Blood volume/heart function remains
- Blood vessels dilate and become leaky
Neurogenic Shock
- Hypoperfusion due to nerve paralysis
- Spinal cord injuries resulting in the dilation of blood vessels
- Increases the volume of the circulatory system beyond the point where it can be filled (warm skin rather than cold)
Anaphylactic Shock
- Severe shock caused by an allergic reaction
- Decrease in venous return, poor distribution of blood
Septic Shock
- Shock caused by severe infection, usually a bacterial infection
- Decrease in venous return, poor distribution of blood
Phases of Shock
-Compensated
-Decompensated
-Irreversible
Compensated Shock
- Early stage of shock
- Body's mechanisms attempting to maintain perfusion (BP)
- Normotension despite other vital sign changes
- Stop bleeding, O2, give fluids, keep warm
Compensated Shock Signs
- Normotension despite other vital sign changes
- Increased cardiac output (increased heart rate/stroke volume
- Shift blood to vital areas (away from skin/muscles)
- EMT: Stop bleeding, O2, give fluids, keep warm
Sympathetic Nervous Stimulation in Shock
- Body goes into hyperdrive cranking out adrenaline hormones
- Epinephrine and Norepinephrine
Alpha Receptors
- EPI: Smooth muscle contraction
- NE: Smooth muscle contraction
- Vasoconstriction, inability to defecate/urinate
Beta Receptors
- NE: Heart and lungs
- Increase cardiac output
Antidiuretic Hormone
- Released during compensated shock
- Inhibits urine output
- Thirsty but not peeing
Glucagon
- Released during compensated shock
- Converts glycogen to glucose
- Need insulin to use
Decompensated Shock
- Compensatory mechanism no longer effective
- Ischemia (higher need for O2 than what body is bringing) of organs leading to decrease function
- Unresponsiveness
- Cadriovascular collapse
- No more glycogen, fluid too low, BP falls
Fall in Blood Pressure
Increase clotting
Irreversible Shock
- Organ failure
- Final stage of shock
- Wide spread formation of microemboli blocking capillaries throughout body
SAMPLE of Shock: History
- Trauma: immediate or past few days
- Medical: allergic reaction, fever, infection, vomiting/diarrhea
- Increased thirst
- Decrease urine output (apple juice color)
- Medication: Anti-Hypertensives or Diuretics
SAMPLE of Shock: Physical Exam
- Increase pulse but weak or loss in peripheral
- Altered mental status
- Cool, pale, moist, sweaty skin
- LAST CHANGE: decreasing BP - indicator of decompensation
Treating Shock
- High flow O2
- Assess ABC
- Maintain body heat
- Elevate legs to help with venous return, never above shoulders
- Do not give food or drink
- Request AEMT/Medic and notify hospital