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What is flexibility?
The normal extensibility of all soft tissues that allows the complete ROM of a joint
What does flexibility have a major influence on while exercising?
Mobility during dynamic motion
What can poor flexibility lead to?
Development of relative flexibility (process in which HMS seeks path of least resistance during functional movements)
What is the HMS also known as, which can be classified into what two things?? What systems does it comprise?
Kinetic chain - upper kinetic chain and lower kinetic chain
Muscular, skeletal, and nervous systems
What can muscle imbalances be caused by?
Postural distortions, repetitive movements, cumulative trauma, emotional distress, poor training technique, poor bodily control, or biased training patterns
What can muscle imbalance result in?
Altered reciprocal inhibition (ARI), synergistic dominance (neuromuscular phenomenon - synergists take over for weak or inhibited agonist - leads to ARI of antagonist), or osteokinematic or arthrokinematic dysfunction
What can altered joint motion be caused by?
Altered muscle length-tension relationships, force couple relationships, or poor joint surface motion, which results in poor movement efficiency
What is neuromuscular efficiency?
Ability of nervous system to recruit correct muscles, produce force, reduce force, and dynamically stabilize body’s structure in all three planes of motion
What must individuals have to allow for optimal neuromuscular efficiency?
Flexibility in all three planes of motion
What is the scientific rationale for flexibility training (what does it prevent)?
Concept of pattern overload (consistently repeating same pattern of motion or posture - places abnormal stress on body) and the cumulative injury cycle (progressive 6 step loop where trauma builds on itself to create permanent muscle imbalances - initiated by pattern overload)
What are the 6 steps of the cumulative injury cycle?
Tissue trauma: microscopic tears due to pattern overload)
Inflammation: body sends fluid and blood to protect + pain receptors
Muscle spasms: nervous system triggers protective tensions to keep area from moving
Adhesions: continuous spasm forms inelastic “knots” or scar tissue - alters normal extensibility
Altered neuromuscular control: nervous system changes how muscle fires and forces compensation
Muscle imbalance: compensation becomes permanent - alters normal joint mechanics and causes more tissue trauma
What are common types of flexibility exercise?
Self myofascial techniques and static, active, and dynamic stretching
What is self myofascial rolling believed to do?
Thought to produce both local mechanical and neurophysiological effect on myofascial tissue
What is static stretching?
Passively take muscle to point of tension and hold for >= 30 seconds
What is active stretching?
Use agonists and synergists to dynamically move joint into a ROM, hold for 1-2 seconds, and do sets and reps
What is dynamic stretching?
Using force production of muscle and body’s momentum to take joint through full available ROM
What is a contraindication? What is a precaution?
Situation where training technique is completely unsafe and must NOT be performed
Situation where technique can be performed, but must be done with utmost caution
When should you not perform flexibility training on a client?
Acute soft tissue injury, recent injury or surgery, contagious skin disease or open wound, arthritis during flare up, osteoporosis, deep vein thrombosis or blood clots, or cancer
What are considered controversial stretches and what should you do instead?
Inverted hurdler’s stretch → seated hamstring stretch
Plow pose → seated or lying hamstring stretch
Shoulder stand → doorway stretch
Straight leg toe touch → seated or standing hamstring stretch
Arching quad stretch → standing quad stretch
What are two prime examples of relative flexibility?
Squat with excess external foot rotation → limited ankle mobility prevents adequate dorsiflexion
Overhead shoulder press with hyperlordosis → weak core stabilizers
What are force couple relationships?
Synergistic action of multiple muscles working together to move a joint
What is reciprocal inhibition?
When agonist receives signal to contract, functional antagonist also receives inhibitory signal allowing it to lengthen
What is altered reciprocal inhibition (ARI)?
When over active agonist causes decreased neural drive to functional antagonist
What is synergistic dominance?
Neuromuscular phenomenon occurs when synergists take over function for weak or inhibited prime mover (agonist)
What is altered length-tension relationship?
When muscle’s resting length too long or short, reducing amount of force that can be produced
What is a muscle spindle?
Sensory receptors sensitive to change and ROC of length of muscle
What is a Golgi tendon organ (GTO)?
Specialized sensory receptor located at point where muscle fibers insert into tendons of skeletal muscle - sensitive to change and ROC of muscular tension
What is autogenic inhibition?
Process by which neural impulses that sense tension are greater than impulses that cause muscles to contract, providing inhibitory effect on the muscle spindles (prolonged increase in tension stimulates Golgi Tendon Organ (GTO), causing muscle to relax)
Which two flexibility techniques primarily utilize the principle of autogenic inhibition?
Self myofascial rolling (SMR) and static stretching (static stretching must be >= 30 seconds for GTO to override muscle spindles - spindles tell muscle to contract thinking it’s going to tear, while GTO hates excessive tension and tells muscle eventually to relax)
What is Davis’ law?
Soft tissue models along the line of stress
What are the mechanism of action and training variables for self myofascial rolling?
Mechanism of action: autogenic inhibition
Training variables: 1-3 sets, hold each tender area for 30 seconds
What are the mechanism of action and training variables for static stretching?
Mechanism of action: stretch tolerance and/or reciprocal inhibition (depending how stretch is performed)
Training variables: 1-3 sets, hold each stretch for 30 seconds
What are the mechanism of action and training variables for active stretching (formerly called active isolated stretching)?
Mechanism of action: reciprocal inhibition
Training variables: 1-3 sets, hold each stretch for 1-2 seconds, repeat 5-10 reps
What can you do to increase the effectiveness of stretching the hip flexor, adductor, and latissimus dorsi statically or actively?
Posteriorly rotate pelvis
What are the mechanism of action and training variables for dynamic stretching?
Mechanism of action: reciprocal inhibition
Training variables: 1-3 sets, 5-10 reps, 3-10 exercises