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lecture given 3/24/2026
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non-surgical periodontal therapy
not invasive (in a closed environment), local anesthesia may be required (comfort and hemostasis)
modalities: OHI, scaling, root planing, local antibiotics, systemic antibiotics, removal of iatrogenic factors, occlusal therapy
what are the 2 major forms of the inflammatory disease affecting the periodontium?
gingivitis and periodontitis
which is the primary etiological factor of periodontal disease?
bacterial plaque
what is the main outcome of periodontal disease?
inflammation/destruction of gingival tissues, destruction of periodontal attachment apparatus
dental biofilm induced gingivitis
reversible disease
clinical signs: gingival inflammation, plaque, calculus
primary goal: emphasis on plaque/calculus removal
other goals: eliminate/reduce etiologic factors to eliminate/reduce inflammation
appropriate supportive periodontal maintenance to prevent disease
therapeutic goal: establish gingival health
is oral hygiene only sufficient to treat gingivitis?
yes- experimental gingivitis study, depends on compliance, professional plaque control helps in disease prevention, local plaque retentive factors that pre-dispose to gingival inflammation
t/f the treatment plan for dental biofilm induced gingivitis on a reduced periodontium is the same as dental biofilm induced gingivitis
true- possibly additional root planing on exposed root surfaces if needed but rest is the same
what are the local contributing factors that may interfere with oral hygiene and the ability to control inflammation?
calculus, overhanging restorations, restoration margins, poorly designed contacts, overcontoured restorations, caries, poor temporization
scaling
removal of calculus from crown of teeth, supragingival/subgingival/intra-sulcular instrumentation to remove calculus/plaque/deposits/stains, short powerful pull stroke, use of hand/sonic/ultrasonic instruments
gingivitis- limited to tooth crown
periodontitis- performed on either coronal or radicular surfaces
what factors may affect treatment and therapeutic outcome of gingivitis?
systemic risk factors like smoking, diabetes, systemic diseases, stress, ect
what happens if gingivitis is not successfully treated?
continuation of inflammation, risk of progressing to periodontitis
what does it mean if gingivitis is successfully treated but gingival signs still persist?
look for underlying systemic conditions and non-dental biofilm induced gingival diseases
what are the 2 major therapeutic approaches for treating periodontitis?
anti-infective treatment- designed to halt the progression of periodontal attachment loss by removing etiologic factors and resolve inflammation
regenerative therapy- induced anti-infective treatment and is intended to restore structures destroyed by the disease
essential to both treatment approaches is the inclusion of periodontal maintenance procedures
can periodontitis be treated only with controlling supra gingival plaque/calculus and removal of iatrogenic factors?
it is less effective compared to supragingival plaque control and scaling/root planing when assessing all clinical parameters (BOP, pockets, attachment loss)
can periodontitis be treated without proper supra gingival plaque control?
meticulous oral hygiene on a daily basis is crutial to avoid bacterial colonization and progression of periodontal disease
what can be done to address gingivitis?
dental plaque index, oral hygiene instructions, scaling, polishing, eliminate local etiologic factors
what can be done to address periodontitis?
scaling and root planing, polishing, eliminate local etiologic factors, local or systemic antibiotics
what are the local contributing factors that may interfere with oral hygiene and predispose to periodontitis?
calculus, overhanging restorations, restoration margins, open contacts, overcontoured restorations, caries/resorption, ill fitting prosthetic appliance, root proximity, occlusal trauma, tooth mobility
root planing
subgingival instrumentation to remove calculus/plaque on root surface and contaminated cementum (bacterial plaque/toxins)
aiming to smooth root surface (diseased cementum removal), less plaque retentive
reconstruction of periodontal tissues’ morphology in a level compatible with gingival health
moderate pull stroke for final smoothing and root planing, long and overlapping
use of hand, sonic, ultrasonic instruments
gold standard of periodontal treatment and its clinical importance has been proven in many studies
only performed on root surfaces that have been denuded of periodontal attachment by periodontitis
may be a definitive treatment in some stages of periodontal disease, may be part of pre-surgical procedures in others, and an essential part of maintenance care
arduous and time consuming, among the most technically demanding procedures performed in dentistry
may be done by quadrants or full mouth
may need to be repeated during treatment phases and may require local anesthetic
what is the therapeutic goal when treating periodontitis?
to resolve inflammation and establish gingiva health, to eliminate/later microbial etiology and contributing risk factors, to arrest progression of disease, to maintain attachment levels/gain clinical attachment, to reduce/eliminate pockets, to maintain/improve bone levels, to stabilize occlusion, to prevent the recurrence of disease, to regenerate the periodontal attachment apparatus where indicated
to preseve the natural dentition in a state of health, comfort, and function with appropriate esthetics
what happens if periodontitis is not successfully treated in phase I?
additional therapy may be required- phase II
what are treatment considerations/limitations?
patient related: systemic health, age, compliance, therapeutic preferences, patient’s ability to control plaque
other factors: clinician’s ability to remove subgingival deposits, depth of pockets, local factors, restorative and prosthetic needs, severity of periodontitis
when performed with optimal access and skill, SRP will produce a…
decrease in gingival inflammation, reduction in periodontal probing depths, and a gain in periodontal attachment
what is the rational for root planing?
root smoothness, removal of diseased cementum, preparation for new attachment (long junctional epithelium)
what are the objectives for root planing?
securing biologically acceptable root surfaces, eliminating periodontal pathogens, resolving inflammation, decreasing probing depth, facilitating oral hygiene procedures, improving or maintaining attachment levels, preparing tissues for surgical procedures
repair
healing of a wound by tissue that does not fully restore the architecture or the function of the part
reattachment
to attach again, the reunion of connective tissue with a root surface on which viable periodontal tissue is present
true new attachment
the reunion of connective tissue with a root surface that has been deprived of its periodontal ligament
this reunion occurs by the formation of new cementum with inserting collagen fibers
long junctional epithelium new attachment
adhesion of the junctional epithelium to a root surface that has been deprived of its periodontal ligament or connective tissue attachment
regeneration
reproduction or reconstruction of a lost or injured part
ODU 11/12 explorer
double ended with mirror image working ends
small fine tip and rounded back (easier insertion into deep and narrow pockets)
universal application
used for detecting calculus
mechanical debridement of natural teeth
hand instruments (scalers, universal and area specific curettes)
power driven instruments- sonic air scalers, ultrasonics- magnetostrictive scalers, piezoelectric scalers
scalers
used for supragingival scaling and are designed to remove plaque and calculus from the tooth surface
curettes
instruments of choice for subgingival instrumentation where scalers cannot be used due to their large, rigid design and their dual-edge blades
can be broadly subdivided into universal and area specific
all share the same elements: rounded back, rounded toe, semi-circular cross section
universal curettes
designed for easy adaptation on all tooth surfaces
2 cutting edges, round toe
2R/RL- anterior
4R/4L- posterior
17/18 or 13/14 McCall
barnhart
area specific curettes/gracey curettes
are designed to work in specific areas of the dentition
a series of isntruments with offset blades were designed to reach the deepest and least accessible periodontal pockets easily without traumatic injury to the gingiva
one cutting edge, round toe
after five gracey curette
designed for instrumentation in deeper periodontal pockets
the terminal shank is elongated 3mm to provide better clearance around crowns and superior access to root contours and pockets 5mm or more in depth
blade thinned by 10% to ease gingival insertion and reduce tissue distention
mini five curettes
designed with the same elongated terminal shank and thinned blades as the after five gracey curettes
50% shorter blade for access to smaller roots, narrow pockets, furcations, and developmental grooves
vision curettes
modification of the gracey curettes
50% shorter blade, increased blade curvature, straighter terminal shank, longer terminal shank
includes visual features like blade ID mark, shank demarcation, and bands on handles (marks 5mm and 10mm)
the shorter blade provides better adaptation on narrow root surfaces and tight periodontal pockets and minimizes tissue displacement
what are the limitations of hand instruments?
technique sensitive- success of SRP procedures depends on the ability of the therapist and the patient to keep the oral cavity below the threshold for disease
anatomic limitations- prevent proper adaptation of the instrument to effectively remove root deposits, like deep narrow pockets, furcations, and concavitites
instrument limitations- size of the instrument does not allow adequate access at a particular site, like root grooves that are smaller than the working portion of the instrument or in areas of root proximity
power driven instrumentation
are usually used to provide a rapid calculus and stain removal with minimum discomfort to the patient or trauma to hard and soft tissues
may be used in both supra and subgingival areas
includes sonic scalers and ultrasonic scalers (magnetostrictive and piezoelectric)
sonic air scalers
functions in the audible range, 2,500 - 700 cps or Hz
attaches to a conventional handpiece
driven by compressed air
orbital or elliptical tip movement
all surfaces active
magnetostrictive ultrasonic scaler
20,000 - 42,000 cps or Hz
metal rod or stack of metal sheets
elliptical tip movement
all surfaces active
peizoelectric ultrasonic
functions above the audible range, 29,000 - 50,000 cps or Hz
ceramic transducer
linear tip movement
lateral surfaces are more active
what are the advantages of ultrasonics?
reduced clinician fatigue, less repetitive stress, increased access, less tissue distension, potential for antimicrobial delivery, benefits of lavage
what are the limitations of ultrasonics?
altered tactile sensitivity, fluid control/evacuation, effects of noise and vibration, contaminated aerosol production
what are contraindications for ultrasonics?
communicable disease that can be transmitted by aerosol, susceptibility to infection, respiratory risk, dysphagia, cardiac pacemaker (consult with cardiologist is needed to determine type of pacemaker)
what do you need to have caution with when using an ultrasonic?
titanium surfaces of implants- use proper tips
demineralized areas, exposed dentinal surfaces (where dentinal tubules can be uncovered and sentivity can be created)
margins of porcelain/amalgam restorations as they may be damaged or removed
hand versus power driven instruments
plaque removal can be equivalently accomplished by power-driven scalers or hand instrumets
when compared to sonic or ultrasonic instruments, manual instrumentation requires more time to achieve the same clinical result
equally effective in decreasing PD, BOP, and periodontal pathogens
root surface hand v power driven
roughness/smoothness- smoothest roots were produced by ultrasonics followed by curettes
alterations- ultrasonics used on medium power may do less damage to the root surface than hand or sonic scalers, as instrument contact time/tip to tooth angle/instrumentation pressure is increased the likelihood of root surface damage is also increased
endotoxin and cementum removal hand v power driven
endotoxin (lipopolysaccharide) is a surface substance which is superficially associated with the cementum and calculus
endotoxin can be removed by washing, brushing, lightly scaling, or polishing the contaminated root surface
extensive cementum removal is not necessary for periodontal healing
side effects of scaling and root planing
bleeding, gingiva tears/lacerations, necrosis of pocket gingival lining, pain, periodontal asbcess (residual calculus), dental hypersensitivity
mechanical debridement for implants
plastic/titanium/gold hand instruments
power driven instruments- ultrasonics with plastic tips, titanium brush
how effective is scaling and root planing when considering probing depths?
100 teeth extracted after SRP, 57% of teeth surfaces had remaining calculus
if PD was </= 4mm there was 90% calculus removal, 5-6mm there was 75% calculus removal, >6mm there was 65% calculus removal
how effective is scaling and root planing when considering root surfaces?
if the probing depth was 0-3mm, 85% of calculus was removed from interproximal surfaces and 80% was removed from buccal surfaces
if the probing depth was >4mm, 60% of calculus was removed from interproximal surfaces and 80% was removed from buccal surfaces
how effective is scaling and root planing when considering furcation involvement?
68% of molars had remaing calculus after SRP
prevalence of the width of furcation entrance smaler than hand instrument size: 58%
in single rooted teeth: 90% calculus removal / in multi rooted teeth: 70% calculus removal
furcation involvement in max molars: 40% calculus removal / fucation involvement in mand molars: 70% calculus removal
what are the microbiological effects of scaling and root planing?
anaerobes with no treatment, baseline 70.8% to 65.7% at 3 months
anaerobes with SRP, at basline 71.8% to 24.5% at 3 months
what is the full mouth disinfection protocol?
1: SRP full mouth- all teeth in 2 visits within 24 hrs, under local anesthesia
2: brushing the back of the tongue- for 1 minute, with 1% CHX gel
3: mouthwash- 2x for 1min, with 10mL of CHX at 0.2% and gargling the last 10 sec to reach the tonsils
4: subgingival irrigation of all pockets- 3x for 10 min, with 1% CHX gel, after each of the 2 sessions, and repeated at D8 using a 6 and 8mmg syringed labeled
5: mouthwash (at home)- with 10mL of CHX at 0.2%, 2x a day for 1 min, over 2 weeks
6: oral hygiene instructions- tooth brushing, interdental cleaning with brushes or other hygiene aid, brushing of the tongue
what does the research on full mouth disinfection say?
clinically, not superior to SRP per quadrant in terms of efficacy
differences are not significant after 4 mo
differences are similar independently of probing depth and furcation involvement
lasers in perio
gingivectomy, frenectomy, melanin pigmentation removal, removal of metal tattoos of gingiva, osseous recontouring`
subgingival debridement and curettage, removal of granulation tissue (flap surgery), maintence of implants, treatment of peri-implantitis
perioscope
periodontal endoscope
specifically designed to explore and visualize the periodontal pocket in patients with periodontitis
allows for subgingival visualization of the root surface at magnifications of 24x to 48x
a 0.99mm fiber optic bundle is delivered to the gingival margin coupled into an instrument called an explorer
minimally invasive non-surgical treatment
minimize need for surgery
clinical health
no clinical signs of inflammation
physiologic or reduced periodontium
gingivitis classification
clinical signs of inflammation
no CAL or reduced periodontium
periodontitis classification
clinical signs of inflammation
CAL
what are the realistic, ideal, and long term goals of periodontal treatment
realistic- resolution of inflammation, PD reduction, CAL gain/maintenance, mobility reduction
ideal- periodontal tissue regeneration
long term- treatment outcome maintenace, no disease progression