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1st Generation Drugs: Dolasetron, Ondansetron, Granisetron
Serotonin 5-HT3 Receptor Antagonists
Potent antiemetic
Antiemetic action is restricted to emesis caused by vagal stimulation and chemotherapy
2nd Generation Drugs: Palonosetron
Serotonin 5-HT3 Receptor Antagonists
For CINV, Radiation therapy and Post-op. N&V
Aprepitant
NK-1 Receptor Antagonists
Selective antagonist of the binding of Substance P to the neurokinin-1 (NK1) receptor
IV: Fosaprepitant
NK-1 Receptor Antagonists
Used in combination with 5HT3-receptor antagonists and corticosteroids for the prevention of acute and delayed nausea and vomiting from highly emetogenic chemotherapeutic regimens
Dexamethasone
Corticosteroids
Mechanism is unclear
Prevention of acute and delayed CINV
Recommended in combination with aprepitant before highly emetogenic chemotherapy
Domperidone
Dopamine Antagonists
Used in the preventive and curative treatment of nausea and vomiting and dyspepsia with motility disorder
Metoclopramide
Dopamine Antagonists
Same as domperidone
Phenothiazine Derivatives: Promethazine, Prochlorperazine
Tetrahydrocannabinol (THC)
Cannabinoids
A psychoactive constituent from marijuana
Used to control nausea and vomiting during chemotherapy in the 1970's
Antiemetic mechanism is unknown
Nabilone
Cannabinoids
A synthetic cannabinoid
Is more active than prochlorperazine in preventing chemotherapy-induced emesis, including cisplatin-containing regimens
Levonantradol
Cannabinoids
Synthetic cannabinoid
Antihistamines
Block the histamine receptors in the vomiting center and thus reduce activity along these pathways
Includes: Buclizine, Cyclizine, Dimenhydrinate, Diphenhydramine, Meclizine
Reduce N&V associated with motion sickness
Scopolamine
Anticholinergics
Available as transdermal patch for motion sickness
Benzodiazepines
Suppress vestibular responses (GABA modulation) and reduce cerebral cortex input to vomiting center
Good for anticipatory nausea and vomiting before cancer chemotherapy