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risk factors- perinatal mortality
inadequate/ poor quality colostrum
poor hygiene
lack of navel dressing
lack of supervision
poor weather
infectious disease
ewe condition
birthweight
iodine deficiency
birthweights
single 5.5 to 7
twins 5-6
triplets greater than 4
for meat breed x f1 ewe, reduce by 1kg for hill breed
hygiene
good lambing env
clean and dry
drainage
stocking rate
what to do if not breathing
doxapram hydrochloride, rub chest, stimulate nostril
colostrum
high in fat and antibodies
immunity and energy source
best source is the dam
alternative sources: ewes on same farm, another farm, cow (care with anti ovine factor), commercially bought in)
how to know if lamb has had colostrum
if not- vocalisation
steal
if not identified may become dull
beware suckling wool
palpate abdomen
aboout 30 min after lambing
colostrum requirements
50ml/kg on 2hrs
200ml/kg in 24 hrs
not either or option
restrain ewe and train lamb
bottle
stomach tube
pick up those needing supplementation
hungry/assisted birth/meconium stained
how to do navel dressing
10 percent iodine in alcohol base
strong iodine
first 15mins then 2-4hrs later
common conditions
dystocia
abortion associated pathogens
hypothermia
watery mouth
navel ill
watery mouth
e coli colonisation of gut of newborn
due to intensive husbandry- poor hygiene, contaminated fleece, no colostrum intake
endotoxaemia
twins, esp triplets aged 12-36 hour
dull, lethargic, depressed and reluctant to suck
profuse salivation
wet lower jaw
increasing abdominal distension although lamb has not been sucking
watery mouth- treatment
soapy water enemas
mild laxatives/purgatives
oral antibiotics in early phase
oral electrolyte therapy at 50mls/kg fourtimes daily
penicillin type drug injected intramuscularly
watery mouth- prevention
abundant clean and dry straw bedding
use of paraformaldehyde powder on straw
clean and disinfect of individual pens between lambing ewes
collect and dispose placenta
colostrum
oral antibiotic prep within first 15 min of birth as last resort
yogurt 30mls per lamb (only anecdotal)
hypothermia
linked to management
not enough energy to maintain core body temp
primary and secondary
37 or less
weak or comatose
primary hypothemrmia
glucose in blood sufficient but env losses too high
mismothering/wet
warm up (warming box 45 degrees) and feed once able to swallow
secondary
over 6hrs
brown fat all used and energy requirements supplied by colostrum
not enough energy to maintain temp
body temp drops
dull depressed lateral recumbency
cold mouth
comatose if not caught soon enough
if warmed up as before no glucose to support increased metabolism-hypogylaemic shock
give energy before warming!
treating a comatose hypothermic lamb
kettle
water
50ml syringe
19g 25mm needle
glucose 20 or 40 percent
oxytetracycline spray
boil water and mix 50:50 with 40 glucose solution in 50ml solution
inject into abdomen
normal gestation period
143-147 days
1st stage labour
3 to 6 hrs
increased activity- nest building
separate from flock
short abdominal contractions
build to cervical dialtion
mucus hanging from vulva
2nd stage labour
actual lambing
10-60mins
active and powerful straining
amnion should rupture, may not
normal presentation (nose and 2 feet)
3rd stage labour
expulsion of foetal membranes
2-3 hours after lambing
common presentations
head and 2 feet
both legs back (normal)
leg back (ewe on side with leg back uppermost,) repulse lamb,pull leg forward, lamb ewe
bilateral shoulder flexion- (head out only) repulse lamb and bring legs forward, epidural
breach (tail out)- repulse, bring hind limbs to vulva, epidural
twins at same time- push one back, push other out, repeat
head to side- repulse and pull head to fore, use snare to keep head up, lambs freq dead
other issues with ewes
metritis
ringwomb
vaginal prolapse
uterine prolapse
twin lamb disease
hypocalcaemia
abortion
mastitsis
metritis
infection of womb
sequelae of poor lambing practice
dull ewe
poor milk yield
swollen vulva with red brown discharge
antibiotic
ringwomb
failure of cervix to fully dilate
2 fingers able to get in
manual dilation
c section
vaginal prolapse risk factor
pre lambing issue
vagina and cervix
risk factors:
over conditioned
lack of movement
high fibre diet
triplet
lameness
hypocalcaemia
vaginal prolapse signs
visual observation of prolapse
straining like first stage labour
isolation
reluctance to feed
cervix may be open depending on stage of gestation
vaginal prolapse- treatment
first aid- clean and protect
epidural
ewe standing
lift bladder to allow urination
retain (truss/harness, plastic devices, suture)
lamb ewe
truss/harness
rope of manufactured harness
applies pressure to area around vulva
good for early prolapses
check regularly
plastic devices
mild early cases
spoon retains tisue in correct position
tied to wool on flank
buhner suture
veterinary intervention
epidural mandatory
correct standing
release at lambing
uterine prolapse
large single ewes predispose
due to pain or swelling of posterior reproductive tract
identified by visual signs
uterine prolapse treatment
vet emergency
epidural
antibiotic course
NSAID
feed
water
supplement lambs
evisceration through vaginal tear
risk factors as for prolapse
no treatment and euthanasia required
pregnancy toxaemia causes
energy demands exceed supply
triplets
poor roughage quality
inadequate concentrate supplementation
clinical signs of pregnancy toxaemia
does not come to feed trough
isolated
dull and depressed
blind
head pressing
fine muscle tremors of head
weakness and recumbency
death
pregnancy toxaemia treatment
palatable feeds to promote appetite
fresh water
IV glucose injection
glucocorticoid injection
prevention: feed to lamb number, monitor BCS up to lambing
hypocalcaemia- causes
in late gestation
older ewes
change in ration
stress
inapproprate mineral supplementation
clinical signs of hypocalcaemia
depressed
weak
unable to stand
rumenal stasis and bloat
reflux of ruminal contents with green fluid around nostrils and lower jaw
coma and death within 48-72hrs
hypocalcaemia treatment
40ml IV calcium over 30-60s
subcut slower
abortion
should be less than 2 percent of the flock
expulsion of a non viable foetus before term
zoonotic- hygiene, pregnant women at risk
isolate the animal and get a diagnosis
abortion prevention
vaccination
biosecurity
mastitsis signs
ACUTE(hot painful)
heat pain swelling
discolouration
clots in milk
sick ewe
not feeding lambs
CHRONIC
palpable masses
decreased yield
treatment of mastitsis
early treatment with antibiotics and NSAIDs
risk factors of mastitis
maintainig ewes BCS at 3
extra supplementation for thin ewes and old ewes or cull older
teat lesions may predispose to infection
consider culling ewes with poor udder conformation
check udder for abnormal masses
separate
ensure shelter during bad weather
provide extra nutrition for ewes with multiple lambs
test for maedi visna
put freshly weaned ewes on a low plane of nutrition for 2 weeks
joint ill/ navel ill
infection of the lamb due to ingress of bacteria through unhealed navel
meningitis
joint infection
liver abscess
local navel abscess
spinal abscess
etc
joint ill/ navel ill prevention and treatment
navel dressing
hygiene
colostrum intake
penicillin injection
depending on specific clinical syndrome may be unsuccessful