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gastritis
inflammation of the stomach
-can be acute and chronic
-often patients with chronic gastritis have autoimmune disorders
erosive gastritis
Precursor to gastric ulcer
-inflammation eats through mucosal lining
-high risk for bleeding and infections
S/S of gastritis
acute: abdominal discomfort, headache, lassitude, N/V, hiccuping
chronic: epigastric discomfort, anorexia, heartburn after eating, belching, sour taste in mouth, N/V, intolerance of some foods
(may have vitamin deficiency to B12 due to erosion of mucosal lining-->where we absorb B12)
management of gastritis
acute:
-refrain from alcohol and food until symptoms subside
-if due to strong acid or alkali treatment to neutralize the agents avoid emetics, and lavage due to danger of perforation and damage to esophagus
chronic: promote rest, modify diet, avoid caffeine/alcohol/NSAIDs, and pharmacologic therapy, adequate fluid intake
Peptic ulcer
erosion of a mucous membrane forms an excavation in the stomach
-often feel relief after eating (coats ulcer)
-can happen anywhere in GI tract
-risk factors: excessive stomach acid, chronic use of NSAIDs/alcohol/smoking, and familial tendency
S/S: dull, gnawing pain or burning in the midepigastrium, heartburn/vomiting
treatment for peptic ulcers
-relieve pain: avoid aspirin, NSAIDs, alcohol; prescription meds
-reduce anxiety: help identify stressors
collaborative complications of ulcerations:
-bleeding
-infection
-perforation (risk for septic shock)
-pyloric obstruction (only if ulcer is at pylorus)
when should a patient take a PPI?
30 minutes before a meal
gastric cancer
WHAT: malignant tumor of the stomach
risk factors- chronic inflammation of stomach, H pylori infection, pernicious anemia, smoking, gastric ulcers, and genetics
S/S: pain relieved by anatacis, dyspepsia, early satiety, weight loss, abdominal pain, loss or decrease in appetite, bloating after meals, and N/V
TREATMENT: surgical removal of the tumor if possible, pain management, and palliative care
tumors of the small intestine
-64% malignant
-may be asymptomatic or present with pain, occult bleeding, weight loss, N/V, and intestinal obstruction
-assessment: includes CBC, bilirubin, caarcinoembryonic antigen
-diagnose by upper GI radiograph
-treat with surgery and chemotherapy
colorectal cancer
Risk Factors: genetics, food intake
S/S: change in bowel habits, blood in stool, obstruction, pain, and cramping
TREATMENT: depends on stage of disease
small intestine/colorectal cancer complications:
-GI bleeding
-perforation
-obstruction (need to be NPO)
-peritonitis, abscesses, risk for septic shock
IBS
chronic functional disorder characterized by recurrent abdominal pain associated with disordered bowel movements, which may include diarrhea, constipation, or both
-15% of adults suffer
triggers: chronic stress, sleep deprivation, surgery, infections, diverticulitis, and some foods
(finding the trigger is helpful to treat, getting enough hydration/sleep)
S/S: alteration in bowel patterns, pain, bloating, abdominal distention
malabsorption
impaired digestion or intestinal absorption of nutrients
conditions that can cause malabsorption:
-infectious disease
-luminal disorders
-post-op malabsorption
-mucosal disorders
hallmark signs of malabsorption
key sign: loose stool (diarrhea) that is bulky, foul-smelling, and often grayish (steatorrhea), weight loss, and vitamin deficiency
(often similar symptoms to IBS)
Celiac disease
malabsorption syndrome caused by an immune reaction to gluten (autoimmune response to consumption of products containing gluten)
-no treatment besides do not eat gluten
-promote fruits, veggies, and meats to eat
manifestations of celiac disease
diarrhea, constipation, abdominal pain/distention
Appendicitis
-most frequent cause of acute abdomen in the U.S
-appendix becomes inflamed and edematous as a result of becoming kinked or occluded (usually due to hard fecal mass)
-sepsis is a complication if it ruptures
-RLQ abdominal pain is key sign (that responds well to pressure)
-patients may be in knee to chest position to compress appendix
-immediate surgical intervention needed
never give pain meds to an UNDIAGNOSED abdominal/GI patient. Why?
because you cannot assess properly where the pain is etc.
-patient may not be able to feel appendix rupture
interventions for appendicitis
-NPO
-prepare for surgical intervention
-recognize when rupture has occurred (if patient feels all of a sudden better--> most likely just ruptured)
-fluid/electrolyte management
-provide comfort
Diverticular disease
condition in which bulging pouches (diverticula) in the GI tract push the mucosal lining through the surrounding muscle layer
-may occur anywhere in intestines
-disease increases with age and is associated with a low-fiber diet
-diagnosed with a colonsocopy
Diverticulosis
multiple abnormal outpouchings in the intestinal wall of the colon without inflammation
Diverticulitis
infection and inflammation of diverticula
interventions for diverticular disease:
-increase fluids
-soft foods
-bulk laxatives
intestinal obstruction
partial or complete blockage of the small or large intestine caused by a physical obstruction
-mechanical or functional/paralytic obstruction
goals for intestinal obstructions
-NG tube
-assess/measure output of NG tube
-assess for electrolyte/fluid balance
-monitor nutritional status
-assess for s/s improvement
IBD
inflammatory bowel disease
-Crohn's disease and ulcerative colitis
interventions for IBD:
-figure out relationship between food and diarrhea/constipation and emotional stressors
-provide ready access to bathroom or commode
-encourage rest when there is active peristalsis or movement when constipated
-administer anti-inflammatory agents/pain meds
-reduce anxiety
-goal is to maintain frequency, characteristics, and amount of stool
complications associated with IBD:
-electrolyte imbalance
-cardiac dysrhythmias
-GI bleeding with fluid loss
-perforation of the bowel
interventions for GI conditions:
-reduce anxiety
-pain (administer analgesics as prescribed)
-patient education (avoid triggers, positioning-->sitting upright or left-side lying)
-maintain function of NG tube (irrigate it, assess for abdominal distention, etc.)
dumping syndrome
Rapid emptying of gastric contents into small intestines. Client experience abd. pain, nausea, vomiting, explosive diarrhea, weakness, dizziness, palpitations & tachycardia
-parasympathetic NS is activated
-avoid drinking large amounts of fluid with meals
-lay down after eating (helps to keep the food where it needs to be instead of rushing into intestines too fast) --> combat gravity
the care of the patient with GI concerns:
-hx and symptoms
-dietary hx
-abdominal assessment
-fluid/electrolyte management
-weight loss inquiry (more than 10%)
-bowel patterns
-education (avoid triggers, smoking/caffeine/alcohol, limiting NSAIDs, and stress management)