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what is the primary purpose of a motor speech disorder (MSD) evaluation?
determine if the patient has dysarthria
what are the components of the speech mechanism?
tongue
movement of mandible
lungs
vocal folds
velum
diaphragm
an MSD evaluation should differentiate:
dysarthria from apraxia
MSD evaluations describeโฆ
features of speech and integrity of structure and function of the speech mechanism
an MSD evaluation should specify:
severity
MSD evaluation establishes
diagnosis possibilities and a diagnois
a MSD evaluation determines
goals for treatment
determining whether speech characteristics are normal or abnormal requires:
evaluation of each speech subsystem
differential diagnosis is used to:
narrow diagnostic possibilites and arrive at a specific diagnosis
what examination helps determine structural adequacy for speech?
oral peripheral examination
the oral peripheral exam assesses structures for:
speech and nonspeech activities
to determine structural integrity of the speech mechanism tasks are given to assess:
nonverbal oral movement control and sequencing
when assessing nonverbal oral movement control and movement you are looking for disorders likeโฆ
apraxia
functional integrity is primarily assessed using:
neuromotor speech examination
the neuromotor speech exam tasks for:
assessing speech planning or programming capacity
the functional integrity of the speech mechanism determines:
adequacy of musculature for speech movements
what speech systems are evaluated for deficits in a neuromotor speech exam?
articulatory
phonatory
laryngeal
resonance
prosody
when considering diagnosis, the clinician should first determine whether the disorder is:
neurologic
organic
chronic
acute
if a neurologic disorder is present, the clinician should determine:
whether it is a motor speech disorder (or other related disorder)
diagnosis is based on:
interpretation of findings
a diagnosis may sometimes:
not always be stated with certainty
true or false: people with Parkinsonโs can also have a stroke
true
why should severity always be estimated?
to establish prognosis, therapy goals, and ensure patient complaints match
severity ratings help establish:
a baseline
if a patient is saying their voice gets strained after a longer period of time what should the speech therapist do during assessment?
have them talk more to assess speech and their concerns
treatment planning should identify:
characteristics amendable to treatment
treatment goals should consider:
functional impact of dysarthria/apraxia on patientโs functional activities
what is a goal for dysarthria or apraxia?
for the patient to be more intelligible (will always be a goal surrounding communication)
what are the three essential components to evaluation?
history, identification of salient speech features, and identification of confirmatory signs
case history helps reveal:
time course of disorder and patient observations of the problem
oral responses during case history taking provide:
a sample of typical speech conditions for the client
what are the six salient features that influence speech production?
strength of the muscle
speed at which they can move the muscle
range of movement
steadiness
time
accuracy
muscles typically have ______ strength
reserve
weakness is most apparent in:
LMN lesions
weakness can affect:
laryngeal, velopharyngeal, and articulatory musculature
weakness of the laryngeal, velopharyngeal, and articulatory musculature causes weak:
vocal cords, velum, tongue muscles, etc
weakness can be exhibited in all:
5 components of the speech mechanism
true or false: we use all of our muscle strength when we talk
false
phasic movements
quick, unsustained, discrete movements
phasic movements are required for:
speech
phasic movements are mediated through:
direct activation UMN pathway to alpha motor neurons
true or false: excessive speed is uncommon in MSDs
true
excessive speech speed is most commonly associated with:
hypokinetic dysarthria
excessive speech rate is usually due to:
decreased range of motion
slow speech rate is most common in:
spa*tic dysarthria
slow speech rate has the greatest effect on:
prosody
flaccid dysarthria causes:
muscle paresis and muscle paralysis
spa*stic dysarthria causes:
tight muscles that make it harder to move
in hypokinetic dysarthria, range of motion is typically:
decreased
abnormal range has a major impact on:
prosody (restrictive or excessive)
range of speech affects all:
speech mechanisms, not just one
true or false: problems usually include decreased range of motion, not increased
true
hyperkinesia is the result of:
neurologic disease affecting steadiness
tremor is an example of impaired:
steadiness
tremors commonly affect:
phonation, and possibly prosdoy
tremor can be heard during:
vowel production
tremor may be seen on:
an oral mechanism exam
examples of steadiness include:
dystonia, dyskinesia, chorea, athetosis
random, unpredictable movements can affect:
any component of speech
hyperkinetic
decreased accuracy and altered prosody
tone can be:
excessive or reduced
tone is known to:
fluctuate
abnormal tone can affect:
any of the speech mechanisms
what type of tone does flaccid dysarthria have?
reduced tone
spastic and hypokinetic dysarthria have what kind of tone?
consistently increased tone
hyperkinetic has what kind of tone?
variable tone
myasthenia gravis becomes more ______ over time.
flaccid
those with hyperkinetic dysarthria have _____ tone AT FIRST, but as they move it changes.
normal
excessive force and tone results in:
overshooting targets
decrased force and tone results in:
undershooting targets
accuracy in speech can be consistent or inconsistent depending on consistency of:
strength, speed, range, steadiness, and tone
accuracy problems are generally perceived more easily in:
articulation and prosody (but can occur within any of the components)
in ataxic dysarthria, inaccuracy is primarily due to:
impaired timing and coordination
in hyperkinetic dysarthria inaccuracy results from:
involuntary movements
true or false: confirmatory signs can be related to the speech mechanism or not
true
confirmatory signs that:
assist with confirmation of the diagnosis
confirmatory signs are NOT:
diagnosis of MSDs (do not need to be present)
examples of non-speech signs within the speech system include:
atrophy
reduced tone
fasciculations
emotional lability
reduced reflexes and/or presence of pathologic reflexes
an example of reduced tone includes:
dropping of face
an example of fasciculations includes:
flaccid dysarthria
emotional lability is the:
unpredictable outburst of emotion
non-speech signs outside of the speech system include:
gait disturbances
abnormal muscle reflexes (Babinski Reflex)
loss of automatic or volitional movement (limb apraxia)
difficulty initiating limb movements
abnormalities of strength, speed, accuracy, tone, steadiness, and range of movement of muscles
what information should be collected during history?
basic background data (including any premorbid speech problems)
introduction and goal setting
basic demographics
time course of complaints
association of deficits
patients awareness of symptoms/perception of deficit
degree of disability or handicap caused by the problem
what kind of things have been tried to manage the problems?
awareness of medical diagnosis and prognosis
during examination of the face, the clinician should assess for:
symmetry
dentures/edentulous
espressionless/masklike
abnormal involuntary movements
mild facial symmetries relatively normal
forehead wrinkling
eyebrow raising
pucker and retraction of lips
during examination of the jaw, the clinician should assess for:
lightly closed or slightly open
involuntary movements
opened or closed against resistance
during examination of the tongue, the clinician should assess for:
full or symmetric at rest
abnormal movements at rest? excessively moist or dry?
deviation to one side during protrusion
able to push out cheek on the other side (with tongue)
speed, range, and regularity of tongue during lateralization
during examination of the palate, the clinician should assess for:
using tongue depressor, is palate symmetrical? does it hang low?
during phonation of /ah/ (long a) is palate symmetrical? sustained elevation for 6-8 seconds? repeated elevation - differences?
evidence of nasal airflow on mirror during vowel prolongation
change with nares occluded
can they protrude tongue and puff cheeks simultaneously
during examination of the larynx, the clinician should assess for:
sharp cough (not loud, but sharp) and glottal coup on command?
presence of inhalation stridor on rapid and/or deep inhalation
during examination of respiration (during quiet breathing), the clinician should assess for:
posture control
shortness of breath (16 to 18 breathing cycles/min; 2-3 sec per cycle)
chest wall and shoulders during breathing
hiccups
hiccups are what kind of movement?
hyperkinetic
inhalation order
vocal folds are vibrating as the air is going through
edentulous
no teeth
what does water glass manometer for determining:
the ability to generate and sustain respiratory driving pressure sufficient for speech
for the water glass manometer you must:
maintain a stream of bubbles for 5 seconds with a straw at a depth of 5 cm
typical vital capacity at age 10 is approximately:
2 liters
why should the absence of normal reflexes be interpreted cautiously?
they may or may not be present in the general population (interpret with caution)
the gag reflex is elicited by stroking what structures:
back of tongue, faucial arches, or posterior pharyngeal wall on both sides
a normal gag reflex response includes:
elevation of the palate, retraction of tongue, and contraction of posterior and lateral pharyngeal walls
an elicited gag reflex is considered clinically significant when:
it is asymmetrical
if the gag reflex is asymmetircal, the clincian should:
ask the patient if it feels different on each side