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GERD
Regurgitation of stomach contents into esophagus
Predisposing conditions for GERD
Hiatal hernia
Incompetent lower esophageal sphincter (LES, often caused by meds that cause relaxation such as nitroglycerin)
Impaired esophageal motility
Decreased gastric emptying
Signs and symptoms of GERD
Pyrosis (heart burn)
Wheezing, coughing, dyspnea (aspirating)
Sore throat
Choking
Regurgitation
History and physical assessment questions for GERD
What makes it better/worse? What’s their diet like? Are they in any medications? Etc
When do you do an endoscopy in a patient with GERD
When the patient experiences:
Dysphasia
Odynophagia
Bleeding
Vomiting
Weight loss
When is a barium swallow often done for patients with GERD
When it gets worse at night
What are esophageal manmetry studies and pH monitoring both looking for in patients with GERD?
The presence of acid
EGD pre procedure
Education and consent
NPO after MN
No dentures
Throat sprayed with xylocaine during procedure and patient is sedated
EGD post procedure
NPO until gag reflex returns (2-4 hours)
Sore throat
Monitor for aspiration
Vital signs
Bleeding (decreased BP and increased HR)
Pain (bloating from air used for procedure)
Barium swallow: Pre procedure
NPO at least 8 hours before
Assess swallowing ability
Barium swallow: post procedure
Monitor elimination and encourage fluids (BM to pass contrast material, white stool)
Monitor for signs and symptoms of obstruction
May need cathartics/laxatives until stool is no longer white
Management of GERD
Diet (avoid aggravating foods)
Meds
Weight loss
Smoking
Small frequent meals
Sit up after meals and avoid eating before bed
GERD medication: step up approach
starting with the weakest/otc option and moving up when it stops working
Antacids
H2 receptor blockers
Proton pump inhibitor
GERD medication: step down approach
Starting with the strongest/ prescription medication and working your way down
Proton pump inhibitor
H2 receptor blockers
Antacids
When is surgical intervention used for GERD
Rarely, when life and med changes don’t work
Antacids
Tums, maalox, mylanta
Antacids on an empty stomach
Reduce acid for a short time
Antacids taken at onset of distress after eating
Reduce acid for longer
How long should you wait to take other meds for after taking an antacid
1 hour
H2 receptor blockers
Ranitidine (Zantac), famotidine (Pepcid)
H2 receptor blockers MOA
Decrease HCL secretion
When should you take H2 receptor blockers
With meals
Proton pump inhibitors
Omeprazole (loser), rabeprazole (pariet), esomeprazole (nexium)
PPI MOA
Reduces gastric acid secretion
What PPI should you take one hour before a meal?
Esomeprazole
What are some effects of long term PPI use
Osteoporosis
Infection risk (No stomach acid to protect against infection)
Complications of untreated GERD
Esophagitis (inflammation can lead to tissue damage and scar tissue)
Dysphagia
Barrett’s esophagus (change in cells lining esophagus, cells may turn into cancer)
Bronchospasm
Pneumonia
GERD patient teaching
Diet
When to take meds
Positioning after eating (Sit up)
Small, frequent meals
Avoid irritating foods
Life style changes (decaf coffee, stop smoking…)
Hiatal hernia
Protrusion of stomach into esophagus through an opening in the diaphragm
Types of hiatal hernia
Sliding-most common
Para-esophageal- “rolling”
What is the most common cause of GERD in older people
Hiatal hernias
What happens to the diaphragm that allows a hiatal hernia to form
It loses elasticity
Why might a person with a hernia feel full faster/easier
Because a portion of their stomach is up near their esophagus
Signs and symptoms of a hiatal hernia
Similar to GERD
Heartburn after meals
Dysphagia
Pain when bending or straining
Hiatal hernia diagnosis
Barium swallow
Endoscopy
Hiatal hernia treatment
Same as GERD
Gastritis
Inflammation of stomach mucosa, breakdown in normal gastric mucosal barrier
Acute gastritis
Lasts hours to days, develops quickly. May be onset after an irritating meal or alcohol
Chronic gastritis
Lasts weeks to years
Causes of gastritis
Medications (NSAIDs, digitalis, corticosteroids)
Diet (alcohol, spicy, irritating foods)
H-Pylori
Autoimmune component
Infections
Signs and symptoms of acute gastritis
Anorexia
Nausea and vomiting
Epigastric tenderness
Feeling full
Hemorrhage
Signs and symptoms of chronic gastritis
Same as acute
Vitamin B12 deficiency (important for growth/maturation of RBCs, can lead to anemia, may need monthly injection)
Gastritis diagnosis
History and physical (drug/alcohol use)
Endoscopy with biopsy
H- pylori testing
CBC-anemia
Stool sample-occult blood
Nursing care of acute gastritis
Eliminate cause
Supportive care
If vomiting occurs: bed rest, NPO, IV fluids, antiemetics
CF when symptoms subside
Drug therapy-reduce irritation of gastric mucosa
Nursing care of chronic gastritis
Evaluate, eliminate cause (stop ETOH, H-pylori treatment)
Non irritating diet (6 small meals a day)
No smoking
Vitamin B12 (may need injections)
What vitamin is important regarding gastritis
Vitamin B12
Peptic ulcer disease
Erosion of mucosal wall
Acute peptic ulcer disease
Superficial erosion, minimal inflammation
Chronic peptic ulcer disease
Long duration, erosion of muscular wall
Types of peptic ulcer disease
Gastric
Duodenal
Stress related (often result of trauma such as a burn)
Most common cause of PUD
H-pylori
Causes of PUD
H pylori
Meds (NSAIDs, corticosteroids )
Stress (burns, sepsis, trauma)
Diet (caffeine, spicy foods)
ETOH
Smoking
PUD signs and symptoms
Common to have no symptoms
Pain
Weight loss
Bleeding (hematemesis (vomiting blood)/gastric or melena/dudodenal)
Acute stress related ulcers
PUD gastric signs and symptoms
Burning, gaseous pain
Occurs 1-2 hours after a meal
Key difference between duodenal and gastric PUD
Duodenal pain occurs 2-4 hours after a meal (at night) and gastric pain occurs 1-2 hours after a meal
Duodenal PUD signs and symptoms
Burning, cramp like pain
Pain at night
Pain 2-4 hours after a meal
Dark, bloody stool
PUD diagnostic tests
Endoscopy
H-pylori testing
Barium studies/swallow
Labs:
CBC (bleeding/inflammation)
Urinalysis (can detect h pylori)
Liver enzymes may be elevated
Amylase (pancreas)
Stools (looking for h pylori and blood)
H pylori testing
Non invasive: serum blood tests (IgG, indicates H pylori has been present at some point), urea breath tests (increase in CO2 value if H pylori is present), stool test (not as accurate as breath test)
Invasive: biopsy of stomach (endoscopic procedure)
Conservative therapy Care of PUD
Rest
Bland diet
No smoking
Meds
Reduce stress
Care for acute exacerbation of PUD
IV fluids
NPO
NG to suction if complications occur
PUD medications
H2 receptor blockers
PPI
Antibiotics
Antacids
Anticholinergic
Cytoprotective agents
H pylori treatment
Triple therapy for 7-14 days, first line of therapy (teach pt to stay in full course of meds even if symptoms subside)
PPI (Prilosec/omeprazole)
Amoxicillin
Biaxin (clarithromycin)
Complications of PUD (emergencies)
Hemorrhage
Perforation (sudden, severe abdominal pain, rigid abdomen, shallow, rapid rests, peritonitis)
Gastric outlet obstruction (abdominal discomfort, projectile vomiting, loud, visible peristalsis)
Nursing care of PUD
History and physical-detect and prevent
Rest
Diet changes (may be NPO during acute exacerbation—> NG, fluid replacement and oral care)
Drug therapy
Stop smoking
Long term follow up and care
Physical and emotional rest