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problems getting or staying pregnant
infections
BCS
heat stress
postpartum disorders
dystocia
signs of calving
unsocial and restless
standing up/ laying down
isolation
reluctance to feed
water bag emerges
cow may rest for a short while
front feet appear
push
standing or laying
once head is through calf dives out
dystocia causes
breeding
over conditioned dam
malformation of the calf or dam
lengthened gestation
foetal maternal incompatibility- heifers are at risk
malposition
other conditions eg milk fever or twisted uterus
other calving/post calving issues
haemorrhage
prolapse.- dystocia, large calf, milk fever
milk fever (hypocalcaemia)
incorrect dry cow nutrition
breed
stress
LINKED TO
calving problems
RFM
uternine prolapse (weaken uterine contractions)
metritis, mastitis (weakens immunity)
poor appetite
displaced abomasums
ketosis
delayed return to oestrus
retained foetal membranes
failure to expel foetal membranes within 12-24hs after calving
potential infection acting as a week
risk factors include Abortion ▪ Stillbirth ▪ Twins ▪ Dystocia ▪ Metabolic disorders, especially milk fever ▪ Specific infections such as: o Brucellosis o Leptospirosis o Listeriosis o IBR
downer cow- calving paralysis
cow has nerve and muscle damage from trauma of calving
calf not presented properly
large calf
calving too long
downer cow diagnoses and treatment
history of hard calving
cow is usually alert eating drinking
normal temp
treatment
time
lifting
anti inflammatory agents
metritis/ endometritis
infection of uterus
usually observed 2 weeks post calving
hygiene at calving key
calving difficulties- consider antibiotics
cows with twins more at risk
displaced abomasum
left more likely than right
costly
during pregnancy, the uterus displaces the abomasum.
following calving, the empty uterus and poor rumen fill leave more space in the abdomen for the abomasum to float
milk fever or ketosis can make the abomasum not contract normally - filled with gas
ketosis
usually result of undersupply of energy in comparison to demand
poor dry matter intake
low energy density of rations
typically occurs 3-8 week post calving as energy demand for milk production increases
moderate milk drop and reduced feed intake
dung firm
pear drop smell on breath
some get nervous symptons- disorientation, excessive licking
ketosis diagnosis
blood test
high beta hydroxybutyrate levels
often blood test during herd routine at appropriate stage of lactation
too fat
problem with calving- fat around birth canal
risk of fatty liver
factors impacting negative energy balance
feed given
feed eaten
energy/ protein intake
dry matter intake
group management
breed
age
genetics
BCS
grass only feeding
low cost
limited to summer milk production where milk prices are lowest
Cows do not achieve sufficient DMI from grass alone to meet demand for lactation – leads to greater loss of body condition score
risk of hypomagnesaemia
spring time diarrhoea
nutrition and reproduction
feeding level can influence reproductive success
large decrease in BCS during early lactation have difficulty getting back in calf
excessive fat can be difficult getting back in calf
Glucose supporting rations aim to improve milk yield and can improve reproductive success
High dietary protein levels diets can lead to reduced fertility and can be assessed indirectly through blood urea-nitrogen levels.
lactaton period feeding systems- feeding for peak yield
amount of milk produced at peak yield helps set overall lactation yield
high peak yield= high lactation yield
give a high concentration allowance for first 6 weeks of lactation
but risk of rumen acidosis, prone to lower reproductive success
lactation period feeding systems- buffer feeding
feeding additional energy to high yielding grazing cows
often as maize silage plus brewers grain for 3hr indoor before afternoon milking
to increase and balance nurient intake in relation to lactation demand
usually provided when DMI is low
lead feeding
increasing concentrate feeding during last 3 weeks of pregnancy to adapt rumen flora to the switch to a high concentrate ratiion at the onset of lacation
to reduce requency of ketosis and acidosis associated with high levels of grain feeding
can increase risk of milk fever
DCAB feeding
decreasing ratio of cations to anions in the feed during last 3 weeks of pregnancy
helps minimise risk of milk fever
encourages mobilisation of calcium from bone
unpalatable
transition feeding
specific diet 3 weeks before and 3 weeks after calving
better rumen adaptation
monitoring rumen health
milk fat content more than 4 percent
VFA spectrum
fibre content
rumen acidosis
more than 60 lying and ruminating
faecal scoring
when to score lameness
after milking on way back to bed
concrete surface
not rushed
every 2 weeks/montly
what is lameness associated with
winter housing
poor floor hygiene indoors
long walking distances on hard tracks
inadequate foot care
sharp angular stones on the ground
assessing lameness
gait scoring
uneven weight bearing on feet
arched back when standing and walking
foot exam
mobility scores
0- walks with even weight bearing and rhythm on all 4 feet and flat back. long fluid strides
1- steps uneven or strides shortened. affected limb(s) not immediately identifiable
2- uneven weight bearing on limb immediately identifiable and or obviously shortened stride. usually arched back
3- unabale to walk as fast as brisk human pace. lame leg easy to identify. very lame.
infectious foot lesions
digital dermatitis
interdigital dermatitis
heel erosion/ slurry heel
non infectious foot lesions
sole ulcer
sole hemorrhage
white line disease