Neurologic Dysfunction

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Last updated 7:27 PM on 4/11/26
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21 Terms

1
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Altered level of Consciousness (LOC): Causes, presentation

  • Level of responsiveness and consciousness is the most important indicator of the patient’s condition → based on patients baseline

  • LOC is a continuum from normal alertness and full cognition (consciousness) to coma

  • Altered LOC is not the disorder but the result of a pathology

    • Causes: neuro (head injury, stroke), toxic (drugs, ETOH), or metabolic (hepatic/kidney injury, DKA)

    • Coma: unconsciousness, unarousable unresponsiveness

    • Akinetic mutism: unresponsiveness to the environment, makes no movement or sound but sometimes opens eyes

    • Persistent vegetative state: devoid of cognitive function but has sleep–wake cycles

    • Locked-in syndrome: inability to move or respond except for eye movements due to a lesion affecting the pons

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What are the components that are assessed with Altered Level of Consciousness?

  • Eye opening/response

  • Verbal response

  • Motor response (posturing)

  • Alertness

  • Respiratory status

  • Symmetry

  • Reflexes

  • Becomes restless, anxious initially if decreased state of consciousness

<ul><li><p>Eye opening/response</p></li><li><p>Verbal response</p></li><li><p>Motor response (posturing)</p></li><li><p>Alertness</p></li><li><p>Respiratory status</p></li><li><p>Symmetry</p></li><li><p>Reflexes</p></li><li><p>Becomes restless, anxious initially if decreased state of consciousness</p></li></ul><p></p>
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What are the two abnormal posture response to stimuli?

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Planning and Goals for the patient with Altered Level of Consciousness

  • Goals may include:

    • Maintenance of clear airway

    • Protection from injury

    • Attainment of fluid volume balance

    • Maintenance of skin integrity

    • Absence of corneal irritation

    • Effective thermoregulation

    • Accute perception of environmental stimuli

    • Maintenance of intact family or support system

    • Absence of complications

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What are the cerebral response to intercranial pressure (ICP)?

  • Cerebral perfusion pressure (CPP) is closely linked to ICP

  • CPP = MAP – ICP

  • Normal CCP is 70 to 100

  • A CCP of less than < 50 results in permanent neurologic damage

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Increased Intracranial Pressure

  • Monro–Kellie hypothesis: because of limited space in the skull, an increase in any one of components of the skull (brain tissue, blood, CSF) will cause a change in the volume of the others

  • Compensation to maintain a normal ICP of 10 to 20 mm Hg is normally accomplished by shifting or displacing CSF

  • With disease or injury, ICP may increase

    • Increased ICP decreases cerebral perfusion and causes ischemia, cell death, and (further) edema

  • Brain tissues may shift through the dura and result in herniation

  • Autoregulation: refers to the brain’s ability to change the diameter of blood vessels to maintain cerebral blood flow

    • CO2 is decreased → vasoconstriction

    • CO2 is increased → vasodilatation

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What are the early manifestations of increased ICP?

  • Changes in LOC

  • Any change in condition

    • Restlessness, confusion, increasing drowsiness, increased respiratory effort, purposeless movements

  • Pupillary changes and impaired ocular movements

  • Weakness in one extremity or one side

  • Headache: constant, increasing in intensity, or aggravated by movement or straining

<ul><li><p>Changes in LOC</p></li><li><p>Any change in condition</p><ul><li><p>Restlessness, confusion, increasing drowsiness, increased respiratory effort, purposeless movements</p></li></ul></li><li><p>Pupillary changes and impaired ocular movements</p></li><li><p>Weakness in one extremity or one side</p></li><li><p>Headache: constant, increasing in intensity, or aggravated by movement or straining</p></li></ul><p></p>
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What are the late manifestations of increased ICP?

  • Respiratory and vasomotor changes

  • VS: Increase in systolic blood pressure, widening of pulse pressure, and slowing of the heart rate; pulse may fluctuate apidly from tachycardia to bradycardia; temperature increase

    • Cushing triad: bradypnea, bradycardia, hypertension

  • Projectile vomiting

  • Further deterioration of LOC: stupor to coma

  • Hemiplegia, decortication, decerebration, or flaccidity

  • Respiratory pattern alterations including Cheyne–Stokes breathing and arrest

  • Loss of brainstem reflexes: pupil, gag, corneal, and swallowing

<ul><li><p>Respiratory and vasomotor changes</p></li><li><p>VS: Increase in systolic blood pressure, widening of pulse pressure, and slowing of the heart rate; pulse may fluctuate apidly from tachycardia to bradycardia; temperature increase</p><ul><li><p> <mark data-color="purple" style="background-color: purple; color: inherit;">Cushing triad: bradypnea, bradycardia, hypertension</mark></p></li></ul></li><li><p>Projectile vomiting</p></li><li><p>Further deterioration of LOC: stupor to coma</p></li><li><p>Hemiplegia, decortication, decerebration, or flaccidity</p></li><li><p>Respiratory pattern alterations including Cheyne–Stokes breathing and arrest</p></li><li><p>Loss of brainstem reflexes: pupil, gag, corneal, and swallowing</p></li></ul><p></p>
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What is assessment of patient with increased intracranial pressure?

  • Obtain history of events leading to illness

  • Evaluate mental status, LOC

  • Assessment of selected cranial nerves

  • Assess cerebellar function, reflexes, motor and sensory function

  • Glasgow Coma Scale, pupil checks

  • Frequent vital signs

  • Assessment of intracranial pressure

<ul><li><p>Obtain history of events leading to illness</p></li><li><p>Evaluate mental status, LOC</p></li><li><p>Assessment of selected cranial nerves</p></li><li><p>Assess cerebellar function, reflexes, motor and sensory function</p></li><li><p>Glasgow Coma Scale, pupil checks</p></li><li><p>Frequent vital signs</p></li><li><p>Assessment of intracranial pressure</p></li></ul><p></p>
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What are the nursing interventions for the patient with increased ICP?

  • Frequent monitoring of respiratory status and lung sounds and measures to maintain a patent airway

  • Position with head in neutral position and elevation of HOB 30 to 45 degrees to promote venous drainage unless contraindicated

  • Avoid hip flexion, Valsalva maneuver, abdominal distention, or other stimuli that may increase ICP

  • Maintain a calm, quiet atmosphere and protect patient from stress

  • Monitor fluid status carefully; every hour I&O during acute phase

  • Use strict aseptic technique for management of ICP monitoring system

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Types of intracranial surgery

  • CraniOtomy: Opening of the skull

    • Purposes: remove tumor, relieve elevated ICP, evacuate a blood clot, control hemorrhage

    • Refer to Table 61-3

  • CraniECtomy: excision of portion of skill

  • Cranioplasty: repair of cranial defect using a plastic or metal plate

  • Burr holes: circular openings for exploration or diagnosis to provide access to ventricles or for shunting procedures, aspirate a hematoma or abscess, or make a bone flap

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What is the preoperative care: medical management with intracranial surgery?

  • Preoperative diagnostic tests: CT scan, MRI, angiography, or transcranial Doppler flow studies

  • Medications are usually given to reduce risk of seizures

  • Corticosteroids, fluid restriction, hyperosmotic agent (mannitol), and diuretics may be used to reduce cerebral edema

    • Mannitol is used to decrease ICP(diuretic)

  • Antibiotics may be given to reduce potential infection

  • Diazepam may be used to alleviate anxiety

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What is the postoperative care for intracranial surgery?

  • Postoperative care is aimed at

    • Detecting and reducing cerebral edema

    • Relieving pain

    • Preventing seizures

    • Monitoring ICP and neurologic status

  • The patient may be intubated and have arterial and central venous lines

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What is the assessment of the pqtient undergoing intracranial surgery?

  • Careful, frequent monitoring of respiratory function, including ABGs

  • Monitor VS and LOC frequently; note any potential signs of increasing ICP

  • Assess dressing and for evidence of bleeding or CSF drainage

  • Monitor for potential seizures

    • Time seizure to know how long it lasted and possible damage

    • If seizures occur, carefully record and report

  • Monitor for signs and symptoms of complications

  • Monitor fluid status and laboratory data

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What are the nursing interventions done for patients undergoing intracranial surgery?

  • Maintaining cerebral perfusion

    • Monitor respiratory status; even slight hypoxia or hypercapnia can affect cerebral perfusion

    • Assess VS and neurologic status every 15 minutes to every hour

    • Strategies to reduce cerebral edema; cerebral edema peaks 24 to 36 hours

    • Strategies to control factors that increase ICP

    • Avoid extreme head rotation

    • HOB may be flat or elevated 30 degrees according to needs related to the surgery and surgeon preference

  • Regulating temperature

    • Cover patient appropriately

    • Treat high temperature elevations vigorously; apply ice bags, use hypothermia blanket, administer prescribed acetaminophen

  • Improving gas exchange

    • Turn and reposition every 2 hours

    • Encourage deep breathing and incentive spirometry

    • Suction or encourage coughing cautiously as needed (suctioning and coughing increases ICP)

    • Humidification of oxygen may help loosen secretions

  • Sensory deprivation

    • Periorbital edema may impair vision, announce presence to avoid startling the patient; cool compresses over eyes and elevation of HOB may be used to reduce edema if not contraindicated

  • Enhancing self-image

    • Encourage verbalization

    • Encourage social interaction and social support

    • Attention to grooming

    • Cover head with turban and, later, a wig

  • Monitor I&O, weight, blood glucose, serum and urine electrolyte levels, and osmolality and urine specific gravity

  • Preventing infections

    • Assess incision for signs of hematoma or infection

    • Assess for potential CSF leak

    • Instruct patient to avoid coughing, sneezing, or nose blowing, which may increase the risk of CSF leakage

    • Use strict aseptic technique

  • Patient education for self-care

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Seizures

  • Abnormal episodes of motor, sensory, autonomic, or psychic activity (or a combination of these) resulting from a sudden, abnormal, uncontrolled electrical discharge from cerebral neurons

    • Can be d/t light sounds or stress

  • Classification of seizures

    • Focal: originates in one hemisphere

    • Generalized: occur and engage bilaterally

    • Unknown: epilepsy spasms

    • “Provoked” related to acute, reversible condition

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What are the specific causes of seizures?

  • Cerebrovascular disease

  • Hypoxemia

  • Fever (childhood)

  • Head injury

  • Hypertension

  • Central nervous system infections

  • Metabolic and toxic conditions

  • Brain tumor

  • Drug and alcohol withdrawal

  • Allergies

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What is the plan of care for a patient experiencing a seizure?

  • Observation and documentation of patient signs and symptoms before, during, and after seizure

  • Nursing actions during seizure for patient safety and protection

  • After seizure care to prevent complications

  • Refer to Chart 61-4

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Headache

  • Also known as cephalalgia

  • One of the most common physical complaints

  • Primary headache has no known organic cause and includes migraine, tension headache, and cluster headache

  • Secondary headache is a symptom with an organic cause such as a brain tumor or aneurysm

  • Headache may cause significant discomfort for the person and can interfere with activities and lifestyle

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Assessment of headache

  • A detailed description of the headache is obtained

  • Include medication history and use

  • The types of headaches manifest differently in different persons and symptoms in one individual may also change over time

  • Although most headaches do not indicate serious disease, persistent headaches require investigation

  • Persons undergoing a headache evaluation require a detailed history and physical assessment with neurologic exam to rule out various physical and psychological causes

  • Diagnostic testing may be used to evaluate underlying cause if there are abnormalities on the neurologic exam

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What is the nursing management of headache:pain and education?

  • Provide individualized care and treatment

  • Prophylactic medications may be used for recurrent migraines

  • Migraines and cluster headaches require abortive medications instituted as soon as possible with onset

  • Hydration, low stimulating environment, cold compress

  • Provide medications as prescribed

  • Provide comfort measures

    • Quiet, dark room

    • Massage

    • Local heat for tension