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Layers of skin
Epidermis (B KLM)
keratinocytes, melanocytes, langerhans cells, basal cells
Dermis
collagen, reticulum, fibroblasts, macrophages, lymphatic glands, blood vessels, nerve fibers
Skin receptors and function- Meissner’s corpuscles
detect light touch and texture (MeLT)
Skin receptors and function- Merkel Disks
Detect light tough, texture, and pressure
Skin receptors and function- pacinian corpuscles
Detect deep pressure and vibration
Skin receptors and function- Ruffini endings
Detect warmth, stretch deformation within joints
Skin receptors and function- Free nerve endings
Detect pain, temperature, touch, pressure, tickle and itch
Skin receptors and function- krause end bulbs
detect cold temperature
Pressure Ulcers- General
Pressure wounds are located over bony pressure areas and are staged related to the depth of the wound bed
Occur due to immobility
A pressure injury CAN NOT BE BACKSTAGED
Pressure Ulcers- Stages
Deep tissue injury- intact skin, purple maroon appearance
Stage 1- intact skin with non-blancable redness
Stage 2- partial thickness wound, superficial in nature with pink/ red wound bed (shallow)
Stage 3- full thickness wound, subcutaneous fat tissue visible but no bone, tendon, or muscle exposed (deep)
slough/ eschar present, undermining and tunneling may occur
Stage 4- full thickness with exposed bone, tendon, or muscle
slough/ eschar present, undermining and tunneling often occur
Unstageable- wound bed covered with slough/ eschar (unable to identify the depth)
Pressure injury- interventions
Positioning and pressure redistribution devices
Bed- every 2 hours
W/C- every 15 min
Frail patients- every 30 min
Elevate heels and keep the head of the bed <30 degrees unless necessary
avoid WB on bony prominence/ wounds
Air-filled cushions reduce pressure and friction while sitting
educate patients/ caregivers on pressure relief strategies
Arterial wounds/ insufficiency
Arterial insufficiency- Refers to a lack of adequate blood flow to a region of the body
Arterial wounds:
Location: Toes, lateral malleolus, heels, anterior shins
Drainage: Minimal Arterial insufficiency- Refers to a lack of adequate blood flow to a region of the body
Arterial wounds:
Location: Toes, lateral malleolus, heels, anterior shins
Drainage: Minimal to slight exudate (usually dry)
Wound margins: Discrete punched out
Wound appearance: pale
Edema: none
Odor: none
Pain: likely (ischemic) Arterial insufficiency- Refers to a lack of adequate blood flow to a region of the body
Venous wounds/ insufficiency
Venous insufficiency- Refers to inadequate drainage of venous blood from a body part, usually resulting in edema and/or skin abnormalities and ulcerations
Venous wounds:
Location: Near medial/ lateral malleolus, above malleoli, below knee
Drainage: moderate to heavy exudate
Wound margins: irregular borders
Wound appearance: hyperpigmentation (hemosiderin staining)
Edema: likely
Odor: strong
Pain: none
Interventions- Arterial disorders
Best: supervised interval walking (ischemic pain)
Positioning: legs down (dependent)
Education: smoking cessation; diabetes/ lipid/ BP control
Contraindication: Compression if ABI is abnormal; cold; vigorous massage
Interventions- Venous Disorders (CVI/ Venous Ulcers)
Best: Compression therapy (walk with compression garments)
Positioning: Elevation
Education: Avoid prolonged sitting/ standing; encourage movement
Contraindication: Heat on edematous limbs; prolonged dependent positioning; high-impact exercise with active ulcer
Key points to remember- Different types of wounds
Diabetic ulcers- generally located on the WB surface of the foot
Venous insufficiency- frequently are proximal to the medial malleolus. they are edematous
Arterial ulcers- generally located on lateral malleolus, distal toes, or areas of trauma
Pressure ulcers- result of unrelieved external pressure on an area
Burn- Epidermal
Vascularity: intact
Color: Erythematous, pink/ red
Surface appearance/ pain: delayed pain; tenderness
Swelling/ healing/ scarring: minimal edema, heals spontaneously
Burns- Superficial partial thickness
Vascularity: Blanching with brisk capillary refill
Color: Bright pink/ red
Surface appearance/ pain: very painful
Swelling/ healing/ scarring: moderate edema; heals spontaneously, minimal scarring
Burns- Deep partial thickness
Vascularity: Blanching with slow capillary refill
Color: Mixed red and waxy white
Surface appearance/ pain: Sensitive to pressure, but insensitive to light touch/ pinprick
Swelling/ healing/ scarring: marked edema, slow healing, scarring
Burns- Full-thickness
Vascularity: no blanching
Color: white (ischemic)
Surface appearance/ pain: Anesthetic; body hairs pull out easily
Swelling/ healing/ scarring: Area depressed, requires skin grafting; scarring +
Burns- Subdermal
Vascularity: none
Color: Charred
Surface appearance/ pain: Anesthetic, muscle, and nerve damage present
Swelling/ healing/ scarring: Tissue loss, heals with skin graft or flap, scarring +
Rule of 9’s- Adult
Head (A): 4.5
Arm (A): 4.5
Leg (A): 9
Torso (A): 18
Perineum: 1
Rule of 9’s- Child
Head (A): 8.5
Arm (A): 4.5
Leg (A): 6.5
Torso (A): 18
Perineum: 1
Scars
Normal: Flat and similar to skin color
Hypertrophic scar: a healed wound with thick fibrous tissue that remains within the original wound border
Keloid: condition which excessive scar tissue grows outside of the original margins of the wound
Types of wound drainage
Transudate- clear, thin, and watery
Serous- clear/ amber, thin and watery
Serosanguineous- Clear/ tinge of red/ brown, thin and watery
indicates a wound is healing
Sanguineous- Bloody, bright red fluid, indicates inflamed wound
Pus- yellow/ brown, moderate to very thick
Infected pus- hues of yellow, blue, and green, thick, and usually indicates infection, drainage may be foul
may not indicate infection as WBC macrophage necrotic tissue
Maceration
wound is too moist, edges and periwound will become macerated
identified as white, friable, overly hydrated, and sometimes wrinkled skin
Cause: inappropriate wound care, uncontrolled wound drainage, perspiration, or incontinence
Desiccation
If a wound lacks moisture, the wound and periwound will become desiccated
It is identified as cracked, with dry or flaky edges, and the tissue within the wound bed may be hard or crusty
Cuase: inappropriate wound care, inadequate moisture, infection, dehydration
Debridement- Selective
Removal of only nonviable tissues from a wound
Sharp debridement- use of scalpel, scissors, forceps (precise removal)
Enzymatic debridement- use of a topical application of enzymes (collagenase)
Autolytic debridement- Use of the body’s own mechanisms to remove nonviable tissue
Biologic (maggot therapy)- Sterile larvae digest necrotic tissue
Blue SEA
Debridement- Nonselective
Removal of both non-viable and viable tissues from a wound
> 50% dead tissue
Wet to dry dressing- application of a moistened gauze over area of necrotic tissue to be completely dried and removed
Wound irrigation- moves necrotic tissue from wound bed using pressurized fluid
Hydrotherapy: using a whirlpool with agitation directed toward a wound requiring debridement
Dressings
Based on exudate and if the wound is infected
Heavy exudate: calcium alginates, hydrofibers
Moderate exudate: foams
Minimal exudate: hydrogel dressing, hydrocolloid
Very mild exudate: transparent films
Infected wounds: hydrofiber, hydrogels, calcium alginates, and gauze
NO HYDROCOLLOIDS IN INFECTED WOUNDS