Burns

lost in a burn injury

sensory receptors

Free nerve endings (Nociceptors)

* Epidermis
* Pain, itch

Free nerve endings

* Dermis
* Pain

Merkel’s Disc

* Stratum spinosum
* Touch (Light touch)

Meissner’s Corpuscle

* Papillary dermis
* Touch (Fine, discriminative touch, and vibration)

Ruffini’s corpuscle

* Papillary dermis
* Warmth

Krause’s end bulb

* Papillary dermis
* Cold

Pacinian corpuscle

* Reticular dermis
* Pressure, vibration

Muscle spindle

proprioception/sense of position

GTO

tension

Alteration of vascular integrity → Formation of edema in interstitial spaces → Edema in adjacent tissues → Decreased ROM d/t swelling

below 111° F / 44° C

local tissue damage unless c prolonged exposure

between 44°-51° celsius

rate of cellular death doubles c each degree there’s a rise in temperature, sometimes, short exposure will lead to cellular death

over 124° F / 51°

brief exposure of tissue can damage tissue

thermal, chemical, electrical, or radioactive agents

tissue Injury or destruction results from

* Zone of Coagulation (Inner)
* Zone of Stasis (Intermediate)
* Zone of Hyperemia (Outer)

burn wounds consist of three zones

Zone of Coagulation (Inner)

* Cells are irreversibly injured, cell death occurs
* necrosis

Zone of Stasis (Intermediate)

* Cells are injured
* may die without specialized treatment
* usually within 24-48 hours

Zone of Hyperemia (Outer)

* minimal cell injury
* cells should recover

Jackson’s burn wound model

describes 3 zones of injury