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What are some causes of altered bowel elimination
insufficient dietary intake
inadequate fluid intake
meds (chronic laxative use)
lack of exercise
ignoring urge to defecate
infections/inflammation
neoplasms
change in routine
emotional factors
How does bowel elimination change in childhood
development of control over sphincters and mm for urination and bowel elimination occur in early childhood
How does bowel elimination change in pregnancy
increased pressure and bladder and intestines can cause urinary and bowel frequency
how does bowel elimination change in elderly
physiological changes include decreased renal blood flow, loss of nephrons, atrophy of smooth mm in colon and reduced mucous secretions all contribution to constipation or incontinence
signs of impaired bowel elimination
decreased frequency
difficulty passing stools
decreased stool volume
retention of feces in rectum
abdo pain
bloating
changes in bowel habits
Nursing interventions to optimize bowel elimination
promoting adequate fibre intake
encouraging fluid intake
promoting activity
health education on bowel habits
addressing emotional factors
collaborative interventions to optimize bowel elimination
adjust meds
conduct further ax to reveal underlying medical conditions
individualized care plans
subjective GI assessment
Dx hx
surgical hx
medication hx
abdo pain
N/V
food intolerance
changes in appetite or weight
dysphagia
last BM
constipation or diarrhea
Objective GI ax
inspection
auscultation
palpation
percussion
normal inspection findings in GI ax
symmetry of shape and color
flat or rounded contour
no visible lesions
intact skin
normal auscultation findings in GI ax
presence of normal bowel sounds
normal palpation findings in GI ax
absence of pain or tenderness
absence of masses
voluntary guarding
GI assessment in infants
breast or formula fed
infants have protuberant abdo contour
ax the umbilical cord
respirations observed in abdo
GI assessment in children
protuberant contour until age 4
often can't provide more subjective info than "my stomach hurts"
what is hematemesis
vomiting of red blood
what does hematemesis usually indicate and what is it due to
indicates upper GI bleed
typically d/t peptic ulcers, vascular lesion
Coffee ground emesis
Committing of dark brown, granular material
what is coffee ground emesis usually a result of
upper GI bleed that has slowed or stopped with conversion of red hemoglobin to brown hematin by gastric acid
what is hematochezia
passage of gross blood from the rectum
what does hematochezia usually indicate
lower GI bleed but can result from vigorous upper bleed
what is melena
black tarry stool
what is melena usually due to
typically upper GI bleed, but possibly from small bowel or R colon
Common etiologies of upper GI bleeds
Duodenal ulcer
Gastric or duodenal erosions
Varices
Gastric ulcer
Mallory-Weiss tear
Erosive esophagitis
Angioma
Arteriovenous malformations
GI stromal tumours
common etiologies of lower GI bleeds
Anal fissures
Angiodysplasia
Colitis
Colonic carcinoma
Colonic polyps
Diverticular disease
IBD: ulcerative colitis / Crohn's
Internal hemorrhoids
Common etiologies of GI bleeds in the elderly
Hemorrhoids and colorectal cancer most common causes of minor bleeding
Peptic ulcer, diverticular disease, and angiodysplasia most common causes of major bleeding
Important history in GI bleeding
Ascertain quantity and frequency of blood passage
Patients with hematemesis should be asked whether blood was passed with initial vomiting or only after several nonbloody emesis
Ask questions to distinguish b/w hematemis and hemoptysis cause pts get them confused
PMH to collect for GI blleds
inquire about previous GI blleding
known IBD
liver disease
meds
drugs and alcohol use
Meds that can contribute to GI bleeding
NSAIDS
anticoags
antiplatelets
bismuth
iron
common manifestations of GI bleeds
abdo pain
dysphagia
weight loss
easy bleeding or bruising
S/S of anemia (weakness, fatigue, dizziness(
Nausea / retching
change in bowel habits
Orthostatic changes seen w/ GI bleeding
>10bpm change in pulse
drop of ≥10mmHg in BP
both commonly develop after acute loss (≥2L) of blood
external stigmata of GI bleeding
petechiae
ecchymosis
chronic liver disease signs
portan HTN signs
chronic liver disease external signs
spider angiomas
ascites
palmar erythema
external signs of portal HTN
splenomegaly
dilated abdominal wall vessels
GI bleed H2T ax: CNS signs
decreased mentation
ALOC
lightheadedness
fainting
dizziness
GI bleed H2T ax: HEENT signs
pale eyes
pale mucosa and lips
GI bleed H2T ax: respiratory signs
decreased O2
SOB
GI bleed H2T ax: CV signs
chest pain
tachycardia
hypoTN
GI bleed H2T ax: GI signs
abdo pain
anal fissures
hemorrhoids
masses
hematemesis
melena
GI bleed H2T ax: hematologic signs
anemia
GI bleed H2T ax: integumentary signs
pallor
Red flags that suggest shock r/t GI bleeds
syncope
hypoTN
pallor
diaphoresis
tachycardia
oliguria
how are GI bleeds dx
CBC
coag profile
NGT
upper endoscopy (upper bleed)
colonoscopy (lower bleed(
angiography
Priority interventions for GI bleeds
maintain patent airway
fluid resuscitation / blood transfusion if indicated
medication
hemostasis
surgery
IV fluid resus protocol for GI bleeds
obtain IV access immediately (large bore in AC)
IV NS 500mL - 1L until signs improve
max 2L
pts who don't improve should get transfusion w packed RBCs until intravascular volume is restored
Medications used for upper GI bleeds
proton pump inhibitors
Meds used for variceal bleeds and dose
Octreotide (somatostatin)
50mcg bolus followed by continuous infusion of 50mcg/hr