14. Equine Nutrition 2: Nutrition Issues and Disorders

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Last updated 12:13 AM on 4/11/26
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44 Terms

1
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bermuda gras

what type of forage is associated with ileal impactions which can lead to colic

2
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tall fescue (endophyte)

which type of forage produces ergot alkaloids which can lead to agalactica, prolonged gestation, and thickened placentas in mares

3
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alsike clover

which forage type is associated with liver failure and photosensitization which ultimately displays as neurologic signs and wasting

4
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red clover

which forage type produces a slaframine toxin which can lead to hypersalivation aka "slobbers"

5
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diarrhea, colic, and laminitis

high grain diets are associated with delivery of too much starch to LI (30-50%) which is rapidly fermented and leads to.....

<p>high grain diets are associated with delivery of too much starch to LI (30-50%) which is rapidly fermented and leads to.....</p>
6
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sugars, fructans, and starch

high NSC forage has what contents

7
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high NSC intake (excess starch/fructans)-->reach hindgut where they are rapidly fermented--> pH drops leading to acidosis--> death of gram-negative bacteria--> endotoxin release--> inflammation--> laminitis

what is the pathophysiology process of diet-induced lamitis

8
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rapid fermentation by gram positive (produce lactic acid) decreasing pH and killing off gram negative bacteria to release endotoxin

overall the process of diet-induced laminitis is associated with....

9
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small intestine

horses have limited ____ ____ starch digestion

10
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hindgut

excess carbs will shift fermentation to ____ which is central to laminitis disease via acidotic conditions

11
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1. a storage carb in cool-season grasses...high in spring grasses

2. not digested in SI

3. rapidly fermented in hindgut

characteristics of fructans in grasses

<p>characteristics of fructans in grasses</p>
12
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cool weather, drought, frost, late afternoon

when do fructan levels increase in grasses

<p>when do fructan levels increase in grasses</p>
13
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increase

when fructans are high--> (increase/decrease) laminitis risk

14
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1. intake is high

2. NSC > ~12%

3. horses are insulin dysregulated

pasture can contain enough NSC to induce laminitis especially when....

<p>pasture can contain enough NSC to induce laminitis especially when....</p>
15
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<10%

when it comes to ESC (sugar) and starch content for laminitis horses, what is ideal

<p>when it comes to ESC (sugar) and starch content for laminitis horses, what is ideal</p>
16
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WSC + starch

NSC= ____ + ____

<p>NSC= ____ + ____</p>
17
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ESC (sugars) + oligosaccharides, fructans, pectins, and some polysaccharides

WSC = _____

<p>WSC = _____</p>
18
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estimate of fructans in cool season grass

WSC-ESC = ____

<p>WSC-ESC = ____</p>
19
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1. restrict grazing in spring

2. feed mature, lower NSC (<12%) hay

3. soak hay for 30-60 mins to decrease NSC by 30%

4. maintain ideal BCS

5. monitor high risk horses

6. limit gain to max of 0.5% BW

7. feed at least 50% hay

how can we prevent pasture-associated laminitis

<p>how can we prevent pasture-associated laminitis</p>
20
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1. limited facilities

2. cost constraints

3. compliance issues

client factor challenges in managing laminitis

21
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1. dry lot

2. high quality hay and nutrient analysis

3. farrier care

4. medical management

management needs associated with preventing laminitis

22
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bermuda grass

which forage is associated with ileal impactions

23
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1. high concentrate diets

2. limited pasture access

3. stall confinement

what are some predisposing factors of cribbing (aerophagia)

<p>what are some predisposing factors of cribbing (aerophagia)</p>
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1. increase forage

2. environmental enrichment

3. physical deterrents like collars and fencing

management for cribbing (aerophagia)

<p>management for cribbing (aerophagia)</p>
25
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1. performance horses---->60-90%

2. foals---->25-50%

gastric ulcers are most common in what kinds of horses

<p>gastric ulcers are most common in what kinds of horses</p>
26
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non-glandular stratified squamous mucosa

gastric ulcers occur most commonly in what part of the stomach

<p>gastric ulcers occur most commonly in what part of the stomach</p>
27
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gastric ulcers

high grain diets, intermittent feeding, stall confinement, and intense exercise are all risk factors for what

28
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1. continuous forage access to stimulate saliva

2. alfalfa for buffering capacity up to 5 hrs due to protein and calcium content

3. reduce stress

4. aloe vera juice...1x/d for prevention, 2x/d for tx.

5. corn oil to increase pH

prevention methods of gastric ulcers

29
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1. omeprazole-gastrogard which is Rx

2. omeprazole-ulcergard which is OTC and 1/4 dose of gastrogard

what are the primary methods to tx. gastric ulcers

<p>what are the primary methods to tx. gastric ulcers</p>
30
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fructans

which carb in grass can cause excessive LI fermentation and laminitis

31
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form of allergic respiratory distress due to dust/mold which causes chronic cough, nasal discharge, and respiratory difficulty

what is heaves/recurrent airway obstruction

32
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1. avoid dusty hay and storing hay above horses

2. feed cubes

3. feed soaked hay....but remember nutrient losses

4. maximize pasture turnout

5. use hay gain system to steam-sterilize hay

management practices of heaves/recurrent airway obstruction

<p>management practices of heaves/recurrent airway obstruction</p>
33
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1. ~30% reduction in sugar content

2. >50% reduction in potassium

what nutrients are leached when you soak hay

34
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when food is stuck in esophagus (usually behind base of heard or in thoracic inlet) and causes hypersalivation, retching, not eating, coughing, and nasal discharge

what is choke/esophageal obstruction

<p>what is choke/esophageal obstruction</p>
35
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1. rapid eating

2. dry feed...pellets or beet pulp

3. poor dentetion

risk factors associated with choke/esophageal obstruction

36
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1. soak feed

2. slow feeder

3. provide dental care

how can we prevent choke/esophageal obstruction

<p>how can we prevent choke/esophageal obstruction</p>
37
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1. poor forage quality

2. long term deficiency

what are some causes of vitamin E deficiency in horses

38
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1. equine degenerative myeloencephalopathy: hereditary or vit E deficiency associated with intracellular damage via oxidation leading to ataxia, proprioceptive deficits, and weakness typically in hindlimbs

2. equine lower motor neuron disease: neurodegenerative disorder associated with progressive weakness, fasciculations, mm. wasting, and WL

what are the primary conditions associated with vitamin E deficiency in horses and describe them

39
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older horses deficient in vit E for 18 months or longer...peaks about 16YO

what horses are most at risk for equine lower motor neuron disease

40
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vitamin E and selenium

when tx. vitamin E deficiency in horses ___ and ___ are often combined together

<p>when tx. vitamin E deficiency in horses ___ and ___ are often combined together</p>
41
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fumonisin toxicity due to mycotoxin

what causes leukoencephalomalacia aka moldy corn disease

<p>what causes leukoencephalomalacia aka moldy corn disease</p>
42
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liquefactive necrosis of cerebral white matter due to fumonisins disrupting sphingolipid metabolism and ultimately blood vessels

what is the pathology of leukoencephalomalacia aka moldy corn disease

<p>what is the pathology of leukoencephalomalacia aka moldy corn disease</p>
43
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1. progressive ataxia, depression, anorexia, delirium, aimless wandering, recumbency, coma, and death

2. death 12hrs-twk after C/s onset

clinical signs associated with leukoencephalomalacia aka moldy corn disease

<p>clinical signs associated with leukoencephalomalacia aka moldy corn disease</p>
44
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send moldy corn to toxicology lab for conformation

how can we dx. leukoencephalomalacia aka moldy corn disease

<p>how can we dx. leukoencephalomalacia aka moldy corn disease</p>