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Body image disturbance
overestimate actual size
unrealistically low ideal size
social comparison
sensitivity to fullness
Basic eating disorder (ED) classifications
Anorexia = restricting subtype, binge-eating/purging subtype
Bulimia = purging subtype, non-purging subtype
Binge eating
usually large amounts of food *
Lack of control *
most likely at home, at night, after unstructured activity, in a negative mood
Purging
attempts to compensate for binge eating and prevent weight gain
self-induced vomiting
laxative misuse
diuretics
excessive exercise
enemas
chewing/spitting out food
Anorexia nervosa (AN)
Consuming less calories/food than required to maintain a normal body weight, leading to significantly low body weight in the context of age, sex, developmental trajectory, and physical health
intense fear of gaining weight or persistent behavior that interferes with weight gain
disturbance in the way in which one’s body weight or shape is experienced, undue influence (when someone unfairly pressures or manipulates another) of body weight/shape on self-evaluation, or persistent lack of recognition of the seriousness of the current low body weight
Restricting type = no binges/ purges
Binge-eating/purging type = as defined
Bulimia Nervosa (BN)
recurrent binge eating episodes
recurrent, inappropriate compensatory behavior
at least 1x/wk. for 3 mos.
undue influence of body weight/shape on self-evaluation
Binge eating disorder (BED)
binge eating at least 1x/wk. for 3 mos.
requires 3 or more:
eating rapidly
uncomfortably full
not physically hungry
eating alone (embarrassment)
disgusted, depressed, or guilty
no compensatory behavior
distress
Risk factors
Family
maternal body dissatisfaction, internalization of thin-ideal, dietary restraint, bulimic symptoms
parents of higher weight
Sociocultural
internalization of thin-idea
Personal
dieting
childhood obesity
body image dissatisfaction
low self-esteem
depression/anxiety/OCD
Peer influence
teasing
peers value thin ideal
peers diet or purge
Overvalued beliefs
perfectionism
asceticism (self-disciplined)
Personality
control
impulsivity
Complications: Anorexia
weight loss
amenorrhea (loss of menstrual cycle)
dehydration and electrolyte imbalance
bradycardia and arrhythmias (irregular heartbeats)
postural hypotension (drop in blood pressure)
This can result in hypothermia, dental problems, osteopenia (low bone density), delayed gastric emptying, irritability, infertility, morality, suicide
Complications: Bulimia
electrolyte imbalance
gastrointestinal distress
delayed gastric emptying
menstrual irregularities
postural hypotension (drop in blood pressure)
This can result in esophageal tears/ruptures, arrhythmias, dental problems, parotid swelling (the salivary gland located between the jaw and ear), weight fluctuations, metabolic alkalosis (acid imbalance), chronic renal failure
Treatments for AN and BN
Nutrition support
nasogastric tube
nutritional supplements
Medication
antidepressants
neuroleptics and antipsychotics
appetite stimulants
psychotherapy
psychodynamic
family systems
interpersonal
cognitive-behavioral - focuses on nutrition interventions (meal planning, weekly goals, hydration), psychoeducation (food pyramid, truth about purging), distraction/alternative behaviors, cognitive restructuring, body image interventions (no weighing/checking, in vivo body exposure), in vivo food exposure, exposure with response prevention, relapse prevention
Personality Disorders (PD)
Personality = individual’s beliefs, traits, actions
Personality traits = how an individual acts and and respond to situations — build your personality
Personality disorder = an enduring pattern of inner experience and behavior that deviates markedly from the expectations of the individual’s culture, is pervasive and inflexible, has an onset in adolescence or early adulthood, is stable over time, and leads to distress or impairment—cognition, emotions, interpersonal functioning, and/or impulse control
Problems in diagnosing PDS
moderate inter-rather reliability
poor test-retest reliability
overlap between PDS
heterogeneity (dissimilar) within each PD
high comorbidity—2 or more medical conditions in the same person at the same time
gender distribution and gender bias
categorical vs. dimensional PDS
Categorical view
PDs qualitatively distinct from normal—not just more extreme versions of normal personality traits, they’re fundamentally different in kind, not just degree
Dimensional View
PDs are extremes on personality dimension(s)
5-factor model: extraversion (outgoing), agreeableness (cooperative/empathetic), conscientiousness (organization/self discipline), emotional stability, openness to experience
Personality Disorder clusters
Cluster A—odd or eccentric
Paranoid, schizoid, schizotypal
Cluster B—dramatic, emotional, or erratic
Antisocial, borderline, histrionic, narcissistic
Cluster C—anxious or fearful
Avoidant, dependent, obsessive-compulsive (WE DON’T GET TO CLUSTER C)
Cluster A
Paranoid PD — more common in men
Pervasive, unjustified, mistrust and suspicion of others; hostility; jealously
limited close relationships; cold and distant affect
excessive trust in own ideas and abilities; critical of weakness and fault in others
Etiology
possibly genetic
cognitive: early learning that the world is dangerous and others are devious
Treatment for Paranoid PD
few seek help, develop trust, cognitive therapy—thoughts about others, lack good outcome studies
Schizoid PD — more common in men
Pervasive pattern of detachment from social relationships
perceptions of extrasensory abilities or magical control over others
attention focus
Etiology
social isolation preference resembles autism
cognitive: I am self-sufficient; others are intrusive
Treatment for Schizoid PD
few seek help, cognitive therapy—value of interpersonal relationships, building empathy and social skills, lack good outcome studies
Schizotypal PD — slightly more common in men
Odd and unusual behavior, thoughts, and appearance
magical thinking, ideas of reference (hidden meaning), illusions (things not present)
individuals believe unrelated events pertain to them in important ways; bodily illusions
demonstrate difficulty keeping attention focused
conversation is typically digressive and vague, even sprinkled with loose associations
Etiology
a phenotype of schizophrenia genotype?
Treatment
social skills training, antipsychotic medications, address comorbid depression, lack good outcome studies
Cluster B
Antisocial PD — more common in low SES and highly comorbid with substance use disorders
noncompliance with social norms, violate rights of others, lack of empathy/remorse
lie repeatedly, reckless, and impulsive
little regard for other individuals, and can be cruel, sadistic, aggressive, and violent
Psychopathy vs ASPD
Psychopathy-broader term
not all those with psychopathy have ASPD and vice-versa
there is an overlap and lack of overlap among antisocial personality disorder, psychopathy, and criminality
Etiology
family factors
lack of affection, severe parental rejection, inconsistent (or no) discipline, family history of criminal/violent behavior
Genetic contributions
family, twin, and adoption studies support
MZ (identical) > DZ (fraternal) twins for ASPD concordance
Gene-environment interaction
neurobiological influences
brain damage—little support
imbalance between behavioral inhibition system (BIS) and reward system in brain
Treatment
few seek help, poor prognosis, incarceration often the only variable alternative, prevention emphasis
Borderline PD — more common in women
pattern of unstable moods and relationships fear of abandonment, impulsivity, poor self-image
self harm and suicidal gestures
70% attempt suicide; 10% die by suicide
Etiology
Runs in families (genetics), early trauma abuse
Linehan’s biosocial theory = invalidating environment—family members respond erratically to child’s thoughts and feelings
emotional dysregulation → great demands on the fam → invalidation by parents through punishing/ignoring the demands → emotional outbursts by child to which parents attend
Treatment
Antidepressant medications, Dialectical behavior therapy (identify/regulate emotions, problem solving, exposure to traumatic memories, best available treatment—good outcome studies)
Histrionic PD — more common in women
Overly dramatic, attention-seeking, self-centered, sexually provocative, emotionally shallow, impulsive
Etiology
Psychoanalytical seductiveness, sext-typed variant of antisocial PD?
Treatment
Long-term consequences of attention seeking
address problematic interpersonal behaviors
lack outcome studies
Our response to fear
Two pathways where the automatic nervous system and the endocrine system produce arousal and fear reactions
1. sympathetic nervous system pathway (SNS) = acts on blood vessels, organs, and glands in ways to prepare the body for action, especially in life threatening situations
provides resources for the fight/flight response
mobilizes the body to attack, defend, or flee when encountering a potential threat
works with the peripheral nervous system
2. hypothalamic-pituitary-adrenal (HPA) pathway = hormonal (slow, long lasting stress response), it’s apart of the endocrine system and releases cortisol
parasympathetic nervous system (PNS) = returns the body to a resting state by counteracting the actions of the sympathetic nervous system
performs restorative functions — “rest & digest”
allows the body to regenerate when it is safe to do so
works with the SNS to prepare the body for challenges
Trauma in the DSM-5 and PTSD
trauma = exposure to actual or threatened death, serious injury, or sexual violence in one (or more) of the following ways:
direct exposure
witnessing the event in person
learning that a relative or close friend experienced the event
experiencing repeated or extreme exposure to aversive details of the traumatic event(s) (e.g., first responders collecting human remains; police officers repeatedly exposed to details of child abuse)
note: this does not apply to exposure through electronic media, television, movies, or pictures, unless this exposure is work related
PTSD disorder
at least one traumatic event
at least one re-experiencing symptom
at least one avoidance symptom
at least two negative alterations in cognitions and mood symptoms
at least two arousal and reactivity symptoms
symptoms last at least a month
symptoms cause impairment
4 PTSD major symptom clusters
re-experiencing: intrusive thoughts, nightmares, flashbacks—feeling like you’re back in the situation, prolonged physiological responses when reminded of the trauma
avoidance: avoiding thoughts of feelings related to the event, avoiding people places, or things related to the event
negative alterations in cognition and mood: inability to remember important parts of the event, negative beliefs about oneself, other people, or the world, distorted cognitions that lead to blame of self or others related to event, persistent negative emotional state, loss of interest in activities, feelings of detachment from others, inability to experience positive emotions
arousal and reactivity: problems with concentration, reckless or self-destructive behavior, irritability, insomnia (difficulty falling or staying asleep), hypervigilance, exaggerated startle response
Developing acute and post traumatic stress disorders
Biological factors
brain—body stress routes
brain’s stress circuit
inherited predisposition
Childhood experiences — can increase risk for later PTSD
chronic neglect or abuse
poverty
parental conflict
catastrophe
family members with psychological disorders
Cognitive factors and coping styles
preexisting memory impairments, intolerance of uncertainty, inflexible coping styles, and negative worldview versus resiliency and manageable stress exposure in childhood
Social supports systems
weak family and social support systems
Severity and nature of the trauma
more severe or prolonged trauma
more direct exposure to trauma
intentionally inflicted trauma
mutilation, severe physical injury, or sexual assault
Treatment for acute and post traumatic stress disorders
antidepressant drug therapy
cognitive-behavioral therapy
cognitive processing therapy
mindfulness-based techniques
exposure techniques; prolonged exposure
eye movement desensitization and reprocessing (EMDR)
couple or family therapy
group therapy
Dissociative disorders
group of disorders triggered by traumatic events
when changes in memory lack a clear physical cause, they are called dissociative disorders
one part of the person’s memory typically seems to be dissociated, or separated, from the rest
Types of dissociative disorders
Dissociative amnesia = inability to recall important info, usually of an upsetting nature, about one’s life
memory loss is much more extensive than normal forgetting and is not caused by physical factors
often the amnesia episode is directly triggered by a specific upsetting event or trauma
leads to significant distress or impairment
Types of dissociative amnesia
localized: most common type; loss of all memory of events occurring within a limited period
selective: loss of memory for some, but not all, events occurring within a period
generalized: loss of memory beginning with an event, but extending back in time; may lose sense of identity; may fail to recognize family and friends
continuous: forgetting continues into the future; quite rare in cases of dissociative amnesia
Dissociative fugue = extreme version of dissociative amnesia
people not only forget their personal identities and details of their past, but also flee to an entirely different location
may be brief or more severe
Dissociative identity disorder (multiple personality disorder)
person experiences a disruption to his or hers identity, as reflected by at least two separate personality states ir experiences of possession
person repeatedly experiences memory gaps regarding daily events, key personal info, or traumatic events, beyond ordinary forgetting
leads to significant distress or impairment
symptoms are not caused by a substance or medical condition
each has a unique set of memories, behaviors, thoughts, and emotions
subpersonalities often display dramatically different characteristics
sudden movement from one subpersonality to another (switching) is usually triggered by stress
Subpersonalities interact
3 kinds of relationships:
Mutually amnesic relationships= subpersonalities have no awareness of one another
Mutually cognizant patterns = each subpersonality is aware of the rest
One-way amnesic relationships = most common pattern; some personalities are aware of others, but the awareness is not mutual. Those who are aware (“co-conscious subpersonalities”) are “quiet observers”
average number subpersonalities is now thought to be 15 for women and 8 for men; often appear in groups of 2 or 3
Epidemiology
women are diagnosed 3x more than men
men # alters = 13
childhood onset
‘abuse history
chronic
Etiology
psychodynamic model = dissociation: extreme repression
behavioral model = negative reinforcement maintains dissociation
Treatment for DID
Unlike those with dissociative amnesia or fugue, people with DID do not typically recover without treatment
therapists usually try to help clients:
recognize fully the nature of their disorder
recover the gaps in their memory
integrate their subpersonalities into one functional personality