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risk factors for AKI and chronic kidney disease
older age → stiffening of blood vessels
diabetes ½ → high blood sugars cause dmg to small blood vessels, causing kidney damage making and inability to filter
hypertension → kidneys aren’t able to function effectively
hyperlipidemia → causes stenosis, damages vasculature, unable to filter
s/s of respiratory failure
hypoxemic respiratory failure: not enough oxygenation
PaO2: less than 60 and PaCO2: normal/low
dyspnea (sob), tachypnea
irritability, confusion
cyanosis
tachycardia, arrythmia
somnolence (drowsiness, excessive sleepiness)
hypercapnic respiratory failure: increase in carbon dioxide, typically more mental-related changes
PaCO2: greater than 50
headache
change of behavior
coma
asterixis (eyeballs shake back and forth), tremors
papilledema/blurred vision
warm extremities
smoke inhalation injury: assessments and tx
inhalation injury assessment → singed hairs, burns, swollen neck, black soot, itchy/difficult vision, runny nose, abd. pain, nausea, stidor, chest tightness
treatment → bronchoscopy (scope in airway to evaluate burns in enclosed spaces where injury is likely), intubate
pancreatitis s/s
acute:
deliberating pain located in epigastric/L. upper abd. region, abrupt onset
pain after eating (especially fatty meals or drinking alcohol), can radiate to back (observe for guarding)
swelling in the abdomen
nausea, vomiting, dehydration
fever, chills, tachycardia (due to extreme pain), diaphoresis, tachypnea
jaundice
hyperglycemia (insulin is not being released properly)
steatorrhea (pale, loose, foul-smelling, floating, bulky stool)
internal bleeding
cullen’s and grey-tuners’s sign: c-shaped ecchymosis around or w/in pancreas; ecchymosis occuring on the side/flank, rare and dangerous condition in hemorrhagic pancreatitis
psuedocysts: necrotic/calcified tissue walls off fluid in pancreas, can rupture → peritonitis
chronic:
also epigastric pain that radiates to the back
will be fearful of eating → recurrent challenges affect quality of life and contributed to increased disability, absences from social/work events, cognitive disruptions, anxiety, and depression
osteoporosis
malabsorption
nausea and vomiting
weight loss
steatorrhea
pseudocysts
pancreatitis lab work
amylase and lipase levels → increased as it leaks out into the bloodstream when not used for digestion
triglyceride levels → increased, used to see if contributing to gallstone creation
blood glucose → see severity of disease
complete blood count, metabolic panel
abdominal ultrasound or CT → if gallstones are suspected
c-reactive protien → produced by liver in response to inflammation
endoscopic retrograde cholangiopancreatography → uses endoscope and x-rays to diagnose
discharge education for pancreatitis patients
strategies for maintaining low-fat, nutritent-dense diet
rich in vegetables, fruits, and whole grains
cessation of alcohol and smoking, high-cholesterol
maintaing adequate hydration
monitoring if fatty stools improved → prescribed pancrelipase (pancreatic enzyme supplement)
monitoring blood sugar levels depending on severity
priority tx for DKA/HHS patients
DKA:
provide NS fluid to improve hypovolemia → switch to IV fluids and electrolyte replacement
DO NOT bolus back-to-back as it can cause fluid overload and cerebral edema → neuro changes or seizures
give insulin gtt to improve glucose levels
Q15mins BS check, 1:1 w/ patient
monitor potassium level → starting insulin will make potassium go back into the cell so potassium levels should NOT be low when starting
provide insulin drip and potassium replacement levels as it is not indicated to not give insulin due to glucose levels
strict I and Os → DO NOT WANT PATIENT EATING, will need to be NPO then on clear liquids as they will spike glucose levels and impacts insulin drip rate
monitoring potassium levels (for both hyper and hypo)
provide patient education on checking blood glucose levels
Q4hrs when sick
if fasting or NPO, check w/ MD on insulin dosing
HHS:
provide IV fluid replacement (replace first 12hrs with specific amount then 24hrs later do different amount due to electrolyte levels and kg)
frequent blood pressure checks due to hypovolemia
insulin gtt
monitoring potassium levels during → monitor arrythmias
strict intake and output
discharge education on managing blood sugar at home
signs of cushing’s triad
recognizing common ECGs and tx
discharge goals for TBI patients
clinical presentations for the stages of shock
s/s of DIC and treatments
cirrhosis treatment
s/s of increasing ICP
treatments for septic shock
s/s of MI and treatments
common MI medications and their functions