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cognition
brains ability to process, retain, use information
reasoning, judgment, perception, attnetion, comprehension, memory
cognitive disorders
disruption or impairment in higher level brain functions
neurocognitive disorders (NCDs)
disruption or impairment of higher level brain
signs and symptoms often mimic other mental illness
difficult to obtain direct evidence for definitive diagnosis
delirium - causes and risks
most frequent in older adults
medications
substance use or withdrawl
fluid and electrolyte imbalances
metabolic imbalances
brain tumor or injury
terminally ill
delirium - interventions
general- assess and reassess, eliminate underlying cause, provide safe environment, coordinate interdisciplinary treatment
biological- stop suspected meds, monitor changes, maintain fluid and hydration, promote sleep and nutrition, prevent aspriation, admin meds as prescribed
psychological- provide support, present reality, encourage expression of fears, reduce stimuli, limit choices
sociological- involve family if possible
delirium - evaluation
correction of underlying physchological alteration
prevention of injury
resolution of confusion and associated behaviors
return to prior level or functioning
dementia
disease process marked by progressive cognitive impariment with no change in the level of consciousness
impaired memory and judgement, aphasia, apraxia, agnosia
disturbance in executive functioning
emotional and behavioral changes
physical and functional decline
LOC is unchanged
“sundowning”
usually irreversible
aphasia
deterioration of language function
apraxia
imparied ability to execute motor functions
agnosia
inability to recognize or name objects
disturbance in executive function
inability to plan initiate, sequence, monitor, and stop complex behavior
dementia - common causes and risks
advanced age
prior head trauma
lifestyle factors (sedentary)
genetics
metabolic syndrome
substance use disorder
infections (HIV)
disorders affecting neuro: alzheimers, parkinsons, huntingtons, prion, lewy, ect
delirium
impairment in consciousness and rapid change in cognition over a short period
impaired memory and judgement
rambling incoherent speech
altered LOC
vivid dreams and nightmares
unstable vitals signs
usually caused by medical condition and considered an emergency
reversible if cause is treated
etiology of alzheimer’s disease
loss of neurons and brain volume
cell death
genetic factors
inflammation
oxidative stress, free radicals
stages of alzeimer’s disease dementia
preclinical- changes in brain, mild memory loss, no impact on ADLs
mild (early)- short term memory lapses, forget familiar words, impaired concentration
moderate (middle)- confusion, memory loss, wandering, help with ADLs, behavioral
changes
severe (late)- loses awareness of environment, loses ability to communitcate, help with
ADLs, incontinent
etiology of vascular dementia
characterizzed by a marked disruption in cerebral flow with destruction of brain cells
risk factors: advanced age, TIA, stroke, diabetes, HTN, hypercholestemia
signs and symptoms of vascular dementia
confusion, dizziness, wandering, slurred speech, muscle weakness, problem following instructions
nursing interventions for dementia
establish baseline
provide safety
therapeutic relationship
maintain independence
avoid meds with anticholinergic effects
promote quality of life
educate on home care and community resources
simple consistent routines
psychological nursing interventions for dementia
symptom → intervention
delusion - reduce trigger
confusion - eliminate stimuli
mood changes - address self worth
anxiety - reduce stress
cognitive decline - memory enhancement
social and behavioral nursing interventions for dementia
symptom → intervention
social isolation - social activites
apathy - engagement
agitation - redirection
wandering - motion and sound alarms
pharmacological interventions for dementia
donepezil - mild to severe stages of alzheimer’s
riveastigmine- mild to moderate stages
galantamine- mild to moderate stages
memantine- moderate to severe stages
suvorexant- mild to moderate stages
dementia - evaluation
individual remains safe and secure
cognitive decline is slowed
anxiety is maintained at low level
individual remains as independent as long as possible
protective factors for older adults - dementia
marriage
education
income
effective coping
positive outlook
healthy lifestyle
physical activity
psychosomatic
connection between mind and body
somatization
unconscious expression of psychological distress into physical manifestations, symptoms suggest medical illness but cannot be explained by underlying pathology
somatic symptoms disorders (brief description)
characterized by a preoccupation with worrying about their physical manifestations to the point where it assumes a central role
primary gain - somatic symptom disorder
relief of the unconscious psychological distress
secondary goal - somatic symptom disorder
unintended gain, rewards obtained from the physical symptoms that might otherwise not be received
general characteristics of somatic symptom disorders
chromic or recurrent
angry at medical community
comorbidity of anxiety and/or depression
more commen in women
difficulty in accepting a psychological diagnosis
somatic symptom disorder (in depth)
multiple vauge physical symptoms
involves more than one body symptoms
interferes with daily functioning
seeks medical from multiple doctors
often rejects psychological diagnosis as the cause
pain disorder
pain is primary physical symptom, generally unrelieved by analgesics
greatly affected by psychological factors
illness anxiety disorder
belief in/fear of having or developing a serious disease despite negative findings and clinician reassurances
very high anxiety, obsessive thoughts and fears about illness
seeks care from multiple providers or avoids seeking health care
functional neurological symptom disorder
characterized by transferring mental conflict into a physical symptom for which there is no organic cause
symptoms: usually sudden deficits in voluntary motor or sensory functions (blindness, paralysis, seizures)
may cause anxiety but often exhibits indifference
usually short, rarely chronic
malingering
intentional production of false or grossly exaggerated physcial or psychological symptoms
no real physical symptoms or exaggerated minor symptoms
able to stop the symptoms as soon as secondary gain is received
factitious disorder
previously munchausen syndrome
characterized by somatization in which the person intentionally causes an illness or exaggerates symptoms for the purpose of assuming sick person role
willfully controls the phsycial symptoms
nursing interventions for somatic symptom disorders
establish daily routine, adequate nutrition and sleep
recognize relationship between stress and physical symptoms
keep a journal
limit time spent on physical symptoms
teach coping strategies
pharm - ssris
cognitive behavioral therapy
behavioral therapy
groups
evaluation for somatic symptom disorders
recognizes the interaction of mind and body and the effects of stress
identifies conflicts or problems in their situation and relationships
copes well
assumes appropiate roles in work, family, community
substance use disorder can use
alcohol
caffeine
cannabis
hallucinogens
inhalants
opioids
sedatives
stimulants
tobacco
non substance use disorder (behavioral/process addictions)
gambling
shopping
social media
internet gaming
substance use disorder risk factors
genetics
adolescent population
chronic stress
history of trauma
lowered self esteem and tolerance for pain
few relationships and life successes
risk taking tendencies
substance use disorder protective factors
positive family support, social relationships, self esteem
caregiver involvement in child and adolescent
availability of community resources and programs
employment
substance use disorder sociocultural theories
alaskans and native americans have higer percentage
asians have lower percentage
peer pressure
older adults with history of alcohol use
substance use disorder - questions to ask
type of substance
pattern and frequency
amount used
age at onset
periods of abstinence
previous withdrawal manifestations
date of last substance use
blackouts, stress, sleep problems, weight loss/gain
CIWA-Ar
10 item scale to assess and manage alcohol withdrawal, sum indicates severity of withdrawal
mild < 15
moderate = 16-20
severe > 20
treating substance use disorder
tolerance
withdrawal
abstinence syndrome
opioids
intended effect- euphoria/pain relief
intoxication- slurred speech, impaired memory, decreased LOC
withdrawal manifestations- sweating, tremors, irrtability, weakness, N/V, muscle aches, fever
antidotes- naloxone, flumazenil
abstinence maintenance- methadone, clonidine, buprenorphine
substance use disorder - alcohol
0.08% is legally intoxicated
fetal alcohol syndrome
intended effects- relaxation, decreased social anxiety, stress reduction
excess- slurred speech, memory impairment, decreased motor skills
chronic use- cardiovascular damage, liver damage, gastritis, gastrointestinal bleeding
withdrawal manifestations- cramping, vomiting, tremors, restlessness, anxiety, increased blood pressure
meds to treat withdrawal- chlordiazepoxide, diazepam, lorazepam, oxazepam
abstinence maintenance- disulfiram, naltrexone, acamprosate
more CNS depressants
sedatives- benzodiazepines
hypnotics- barbiturates
anxiolytics- club drugs
cannabis- marijuana, hashish
hallucinogens- LSD
caffeine
tabacco
treatment for nicotine addiction
bupropion- decrease craving
nicotine replacement therapy
varenicline- release of dopamine
wernicke-korsakoff syndrome
Wernicke’s encephalopathy
acute and reversible
altered gait, vestibular dysfunction, confusion
treatment is large dose thiamine IV 2-3 daily for 1-2 weeks
Korsakoff’s syndrome (if wernicke’s is untreated)
chronic condition, thiamine for 3-12 months, recovery is 20%
substance use disorder - nursing care
objective, nonjudgmental
1:1 observation during withdrawal, low stimulations
seizure precautions
educate client and family about codependent behaviors
encourage self responsibility
develop emergency plan
substance use disorder - family therapy and client education
families learn use of specific substances
family coping, problem solving, indications of relapse, support groups
teach CBT
attend 12 step program
individual therapy, group therapy, pharmacological therapy
alexithymia
difficulty identifying and expressing feelings
anorexia nervosa
life threatening eating disorder characterized by clients restriction of nutrional intake necessary to maintain a minimally normal body weight, intense fear of gaining weight
binge eating
consuming a large amount of in a discrete period of usually 2 hours or less
body image disturbance
occurs when there is an extreme discrupancy between one’s body image and the perceptions of others perceptions of others and extreme dissatisfaction with one’s body image
bulimia nervosa
recurrent episodes of binge eating followed by inappropiate compensatory behaviors to avoid weight gain
enmeshment
lack of clear role boundaries
orthorexia nervosa
obsession with proper or healthful eating
pica
persistent ingestion of nonfood substances
purging
compensatory behaviors designed to eliminate food by means of self induced vomiting or misuse of laxitives, enemas, diuretics
rumination
repeated regurgitation of food that is then rechewed, reswalled, or spat out
eating disorders risk factors
family genetics
biological
interpersonal relationships
psychological influences
environmental factors
eating disorders - prodromal manifestations
weight changes
abnormal eating habits
ritualized mealtime behaviors
lying about food intake
preoccupation with weight and body image
compulsive and/or exercising
eating disorders - expected findings
mental status- overgeneralizations, all or nothing thinking, catastrophizing, emotional reasoning
vital signs- low/high blood pressure, decreased pulse and body temp, potential hypertension
weight- body weight is less than 85% than expected normal weight, overweight/obese
musculoskeletal- muscle weakness, decreased energy, loss of bone density
gastrointestinal- constipation, diarrhea, abdominal pain, self induced vomiting, gastric rupture, excessive use of laxitives
reproductive status- menstrual irregularities, amenorrhea
psychosocial- low self esteem, impulsivity, depressed mood, irritability, insommnia
integumentary- fine downy hair, yellow skin, cool extremities, poor skin tugor
head/neck/throat- enlargement of parotid gland, dental erosion and caries
cardiovascular- heart failure, cardiomyopathy, peripheral edema
fluid/electrolyte- acidosis, alkalosis, dehydration
anorexia nervosa (in depth)
persistent energy intake restriction
fear of gaining weight or becoming overweight
disturbance in self perceived weight or shape
types: restricting or binging/purging
bulimia nervosa (in depth)
recurrently eat large quantities of food
can be followed by inappropriate compensatory behaviors to rid the body of excessive calories
occurs on average of once per week for 3 months
binge eating is in a discrete period of time (less that 2 hours)
amount of food definitely larger than what most individuals would eat in a similar period of time
clients have a sense of lack of contorl over eating
types: purging or non purging
binge eating disorder
recurrently eat large quantities of food over short period of time
no use of compensatory behaviors
clients experience distress following the episode
an excessive food consumption must be accompanied by a sense of lack of control
at least once per week for 3 months
additional eating disorders
pica
rummination disorder
avoidant/restrictive food intake disorder
eating disorder - criteria for acute care treatment
weight loss of 20% of ideal body weight OR less than 10% body fat
unsuccessful weight gain
abnormal vital signs
ECG changes
electrolyte distrubances
meeting psychiatric criteria
eating disorders - nursing care
safe, structured, supportive environment
monitor weight, vital signs, I&O
moitor electolytes
teach effective coping with feelings
administer SSRIs as indicated
encourage care plan participation
increase/decrease calorie intake (depends on type)
therapy and support group
eating disorders - patient centered care
meds- SSRI (fluoxetine), other meds to treat comorbid disorders
interprofessional care- dietitian, CBT, family therapy
client education- develop maintenance plan, follow up treatment plan, support groups, individual therapy
eating disorders- complications
refeeding syndrome (life threatening)
cardiac dysrhythmias
severe bradycardia
hypotension
ADHD
inattentiveness, overactivity, and impulsiveness
autism spectrum disorder (ASD)
pervasive and usually severe impairment of reciprocal social interaction skills, communication deviance, and restricted stereotypical behavioral patterns
coprolalia
use of socially unacceptable words, frequently obscene
palilalia
repeating one’s own sounds or words
stereotypes motor behavior
hand flapping, body twisting, head banging
therapeutic play
used to understand the child’s thoughts and feelings and to promote communication
tic
sudden, rapid, recurrent, nonrhythmic, sterotyped motor movement or vocalization
tourette’s disorder
multiple motor tics and one or more vocal tics, which occur many times a day for more than 1 year
callous and unemotional traits
little empathy for others, does not feel bad or guilty or shows remorse for their behavior, has shallow or superficial emotions
conduct disorder
persistent behavior that violates societal norms, rules, laws, and the rights of others
distruptive behavior
problems with the person’s ability to regulate their own emotions or behaviors
externalizing behavior
problems with outward behaviors
intermittent explosive disorder (IED)
repeated episodes of impulsive, aggressive, violent behavior, and angry verbal outburst, usually lasting less than 30 minutes
internalizing behavior
problems with self regulation of emotions
oppositional defiant disorder (ODD)
enduring pattern of uncooperative, defiant, disobedient, and hostile behavior toward authority figures without major antisocial violantions
factors impeding diagnosis - childhood and adolescence
language skills
cognitive development
emotional development
wide variation of “normal” behaviors
difficult to determine
children and adolescent disorders - risk factors
gentic links, chromosomal abnormalities
biochemical
social and environmental
cultural and ethnic
resiliency
witnessing/experiencing traumatic events
oppositional defiant disorder
recurrent pattern of:
negativity
disobedience
hostility
defiant behaviors
stubbornness
limit testing
unwillingness to compromise
can develop into conduct disorder later in life
disruptive mood dysregulation disorder
severe and recurrent temper outburst that do not corrrelate with situation
observable by others
temper outbursts are present three or more times per week
at least within two settings (home, school)
intermittent explosive disorder
recurrent episodic violent and aggressive behavior with the possibility of hurting people, property, or animals
diagnosed as early as 6 yrs old
males more affected
verbal or physical aggression
overreaction to normal events followed by feelings of shame and regret
prevents the clients ability to have healthy relationships/employment
conduct disorder
persistent pattern of behavior that violates the rights of others or rules and norms of society
categories: aggression to people and animals, destruction of property, deceitfulness or theft, serious violations of rules
childhood onset: before age of 10, males more prevalent
adolescent onset: after age 10, gender equal
contributing factors: parental neglect, abuse, family history, lack of supervison, rejection, neglect
can turn into antisocial disorder later in life
conduct disorder manifestations
lack of remorse or care
bullies/threatens/intimidates/lies
low self esteem
temper outbursts
reckless behavior
shop lifts
destroys property
physcial cruelty to others
attention deficit hyperactive disorder (ADHD)
involves the inability of a person to control behaviors requiring sustained attention
characteristics: inattention, hyperactivity, impulsivity
behaviors must be present prior to age 12 and more than one setting
types: predominatly inattentive, predominantly hyperactive-impulsive, combined: client exhibits both inattentive and hyperactive impulsive behaviors
* risk for injury
autism spectrum disorder
thought to be genetic origin
affects individual’s ability to communicate and interact with others
cognitive and language development are delayed
inability to maintain eye contact, repetitive actions, strict routines
present in early childhood
more common in boys
wide variation in functioning
intellectual developmental disorders
onset of deficits and impairments during the developmental period of infancy or childhood
deficits with mental abilites: reasoning, abstract thinking, academic learning, learning from prior experiences
imparied ability to maintain personal independence and social responsibilty
deficits range from mild to severe
specific learning disorder
persistent difficulty in acquiring reading, writing, or mathematical skills
performance in one or more academic areas is significantly lower than the expected range
benefit from an Individualized Education Program (IEP)
communication disorders
persistent problems related to language and speech skills
speech dysfluencies, such as stuttering
difficulty with conversational skills