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Sample site: Periphery of lesion at alopecic–hair junction, scrape until minimal capillary oozing for burrowers.
Prep: Mineral oil on slide → specimen → coverslip → microscopy.
Record: Species, presenting sign, lesion description, specimen/site, elements seen, diagnosis, management.
Sample site: Periphery of lesion at _____–hair junction, scrape until minimal _____ oozing for burrowers.
Prep: _______ oil on slide → _____ → coverslip → microscopy.
Record: ______, presenting sign, lesion ______, specimen/site, elements seen, diagnosis, management.
Otodectes cynotis (ear mites)
Notoedres cati (notoedric mange / feline scabies)
Sarcoptes scabiei (sarcoptic mange / scabies
Demodex (demodectic mange)
Cheyletiella (walking dandruff)
ONSDC
Mites and mange
Ctenocephalides spp. (fleas)
Argas persicus (fowl tick / poultry tick)
Rhipicephalus sanguineus (brown dog tick)
Dermanyssus gallinae (poultry red mite)
Ancylostoma caninum larvae
CARDA
Other ectoparasites
Common ear mite of dogs and cats; highly contagious by direct contact, especially among housemates. Causes ear scratching, head shaking, and dark waxy/ceruminous discharge. Feeds mainly on desquamated epithelial cells, cerumen, and aural exudates in the ear canal; outside the ear, it may feed on blood, serum, and lymph. Slide treatment note: Otodex TX.
Otodectes cynotis (ear mites): Common ear mite of dogs and cats; highly contagious by direct contact, especially among housemates. Causes ear scratching, head shaking, and dark waxy/ceruminous discharge. Feeds mainly on desquamated epithelial cells, cerumen, and aural exudates in the ear canal; outside the ear, it may feed on blood, serum, and lymph. Slide treatment note: Otodex TX.
Otodectes cynotis (ear mites): Common ear mite of dogs and cats; highly contagious by direct contact, especially among housemates. Causes ear scratching, head shaking, and dark waxy/ceruminous discharge. Feeds mainly on desquamated epithelial cells, cerumen, and aural exudates in the ear canal; outside the ear, it may feed on blood, serum, and lymph. Slide treatment note: Otodex TX.
Notoedres cati (notoedric mange / feline scabies
Primarily a feline burrowing mite; may occasionally affect dogs. Produces intense pruritus, crusting, scaling, and alopecia, often beginning on the head, ears, and face. Slide treatment note: ivermectin.
Notoedres cati (notoedric mange / feline scabies): Primarily a feline burrowing mite; may occasionally affect dogs. Produces intense pruritus, crusting, scaling, and alopecia, often beginning on the head, ears, and face. Slide treatment note: ivermectin.
Notoedres cati (notoedric mange / feline scabies): Primarily a ______ burrowing mite; may occasionally affect dogs. Produces intense _____, _____, scaling, and _____, often beginning on the ____, ____, and face.
Slide treatment note: _______.
Sarcoptes scabiei (sarcoptic mange / scabies)
Highly contagious burrowing mite; can temporarily affect humans. Mites burrow into the skin, lay eggs, and feed on epidermal debris, blood, and skin material, causing intense pruritus, alopecia, crusts, and excoriations.
Sarcoptes scabiei (sarcoptic mange / scabies): Highly contagious burrowing mite; can temporarily affect humans. Mites burrow into the skin, lay eggs, and feed on epidermal debris, blood, and skin material, causing intense pruritus, alopecia, crusts, and excoriations.
Sarcoptes scabiei (sarcoptic mange / scabies): Highly ______ burrowing mite; can temporarily affect _____. Mites burrow into the ___, lay eggs, and feed on ______ debris, _____, and skin material, causing intense pruritus, alopecia, crusts, and ______.
Demodex (demodectic mange)
Follicular mite living mainly in hair follicles and sebaceous glands. Usually associated with underlying immune compromise; feeds on follicular/sebaceous secretions including sebum, keratin, and host cells. Slide note: “male” and “female” are life-cycle/sex stages of the mite, not different species.
Demodex (demodectic mange): Follicular mite living mainly in hair follicles and sebaceous glands. Usually associated with underlying immune compromise; feeds on follicular/sebaceous secretions including sebum, keratin, and host cells. Slide note: “male” and “female” are life-cycle/sex stages of the mite, not different species.
Demodex (demodectic mange): ______ mite living mainly in ____ follicles and _____ glands. Usually associated with underlying ______ compromise; feeds on follicular/sebaceous secretions including _____, _____, and host cells.
Slide note: “____” and “______” are life-cycle/sex stages of the mite, not different species.
Sarcoptes burrows in the skin, is highly contagious, spreads by direct contact or contaminated items, and may temporarily affect humans as scabies. Demodex lives in hair follicles/sebaceous glands and is usually linked with immune compromise.
_______ burrows in the skin, is highly contagious, spreads by direct contact or contaminated items, and may temporarily affect humans as scabies. ______ lives in hair follicles/sebaceous glands and is usually linked with immune compromise.
Cheyletiella (walking dandruff)
Surface mite causing cheyletiellosis; dandruff-like scaling may appear to move because of mite movement on the skin surface.
Cheyletiella (walking dandruff): Surface mite causing cheyletiellosis; dandruff-like scaling may appear to move because of mite movement on the skin surface.
Cheyletiella (walking dandruff): _______ mite causing cheyletiellosis; _____-like scaling may appear to move because of mite movement on the skin surface.
Feature | Sarcoptic mange | Demodectic mange |
Agent | Sarcoptes | Demodex |
Contagious? | Yes; highly contagious | Usually no; linked with immune issues |
Mite location | Burrows in skin | Hair follicles / sebaceous glands |
Treatment focus | Antiparasitic medication | Antiparasitic medication + manage underlying immune problem |
Feature | Sarcoptic mange | Demodectic mange |
Agent | Sarcoptes | Demodex |
Contagious? | Yes; highly contagious | Usually no; linked with ______ issues |
Mite location | Burrows in ____ | _____ follicles / _____ glands |
Treatment focus | _____ medication | Antiparasitic medication + manage underlying _______ problem |
Ctenocephalides spp. (fleas)
External parasites that feed on blood; may cause pruritus, irritation, flea allergy dermatitis, anemia in heavy infestations, and secondary skin lesions.
Argas persicus (fowl tick / poultry tick)
Small soft-bodied tick found mainly on domestic fowl such as chickens and ducks.
Rhipicephalus sanguineus (brown dog tick)
Important dog tick and vector; commonly associated with tick infestation on skin and ears.
Dermanyssus gallinae (poultry red mite)
Poultry mite that can cause irritation and blood loss.
Ancylostoma caninum larvae
Infective hookworm larvae may penetrate the skin and cause cutaneous larva migrans; lesions often appear on body parts that contact the ground. Slide note: larvae are migrating.
Macule
Patch
Papule
Plaque
Nodule
Cyst
Vesicle
Bulla
Pustule
Hematocyst
Petechiae
Purpura
Ecchymoses / bruises
MPPPN CVBPH PPE
Primary lesions
Macule
Small, flat, circumscribed color change; slide note: macule is less than 1 cm.
Patch
Larger flat discolored lesion; slide note: patch is a macule greater than 1 cm.
Papule
Small solid raised lesion; slide note: papule is elevated and less than 1 cm.
Plaque
Elevated, palpable, broad/flat-topped lesion larger than 10 mm or greater than 1 cm; may form by coalescing papules.
Nodule
Small solid bump / deeper solid lesion, usually involving dermis or subcutis.
Cyst
Sac-like cavity filled with solid, semisolid, liquid, air, or other material; slide note: a true cyst has a lining sac, while a false cyst lacks a lining and may be related to trauma or inflammation.
Vesicle
Small fluid-filled blister.
Bulla
Larger fluid-filled blister, usually greater than 1 cm.
Pustule
Elevated lesion filled with pus / purulent exudate.
Hematocyst
Blood-filled cyst-like or blister-like lesion; compare with bulla because both may look blister-like but contain different material.
Petechiae
Smallest hemorrhagic lesions; pinpoint non-blanching red to purple spots in skin or mucous membranes.
Purpura
Larger hemorrhagic lesions, about 4–10 mm, that may be multifocal or coalescing.
Ecchymoses/ bruises
Largest hemorrhagic lesions, usually greater than 1 cm.
Lesion | Surface / content | Key size / feature |
Macule | Flat discoloration | Less than 1 cm |
Patch | Flat discoloration | Greater than 1 cm |
Papule | Raised, solid | Less than 1 cm |
Plaque | Raised, broad | Greater than 1 cm / larger than 10 mm |
Vesicle | Fluid-filled | Small blister |
Bulla | Fluid-filled | Large blister, greater than 1 cm |
Lesion | Surface / content | Key size / feature |
______ | Flat discoloration | Less than 1 cm |
______ | Flat discoloration | Greater than 1 cm |
______ | Raised, solid | Less than 1 cm |
_____ | Raised, broad | Greater than 1 cm / larger than 10 mm |
_____ | Fluid-filled | Small blister |
_____ | Fluid-filled | Large blister, greater than 1 cm |
Comedo
Follicular plugging with keratin and sebum (“blackhead”); slide examples include Schnauzer comedo syndrome and blackheads.
Excoriation
Self-induced superficial skin abrasion, erosion, or ulceration from scratching, biting, rubbing, or chewing, often due to intense pruritus.
Fissure
Linear crack or split through the epidermis and sometimes dermis, often from loss of skin elasticity.
Scale
Visible flakes of stratum corneum / dried skin or epithelial tissue from abnormal keratinization.
Crust
Dried serum, blood, pus, inflammatory exudate, or dead epithelium on the skin surface.
Epidermal collarette
Circular rim of scale from ruptured vesicles or pustules; often seen as peeling edges around lesions.
Erosion
Superficial epidermal loss that heals without scarring; may be seen with excoriation from scratching, biting, or rubbing.
Ulceration
Full-thickness epidermal loss extending into dermis that may scar; excoriation may progress into ulceration when scratching/chewing is severe.
Scar
Permanent fibrous tissue / mark replacing normal dermis after injury.
Sclerosis
Pathologic hardening from increased fibrous connective tissue; slide example also included nuclear sclerosis, an aging opacity of the lens in dogs.
Induration
Firmness due to edema, inflammation, or fibrosis in dermis/subcutis.
Lichenification
Thickened skin with exaggerated lines from chronic rubbing/licking.
Hyperpigmentation
Increased melanin or darkening of skin after inflammation or endocrine disease.
Folliculitis
Inflammation of hair follicles; slide note: papule = elevated, pustule = pus, and folliculitis lesions may show papules, pustules, and crusts centered on hair follicles.
Perifolliculitis
Inflammation around the hair follicle.
Furunculitis / furunculosis
Deep infection/inflammation of a hair follicle with follicular rupture, abscess formation, pus, and necrotic tissue. (boils or caruncles)
Intertrigo
Maceration and inflammation of skin folds due to moisture, friction, accumulated skin secretions, and secondary infection.
Keratosis
Hyperkeratosis
Orthokeratosis / orthokeratotic hyperkeratosis
Parakeratosis / parakeratotic hyperkeratosis
Acanthosis nigricans / hyperpigmentation
Epitheliogenesis imperfecta
Hypertrichosis
Ichthyosis
Solar dermatosis
Callus
Paronychia
Seborrhea
Spongiosis
Erythema
Urticaria / hives
Wheal & flare reaction
Alopecia
Lupus erythematosus
KHOPA EHISC PSSEU WAL
Pathologic disorders of skin
Keratosis
General term for abnormal keratin formation or thickening/keratin accumulation in the epidermis or stratum corneum.
Hyperkeratosis
Specific increase in thickness of the stratum corneum due to increased keratin.
Orthokeratosis / orthokeratotic hyperkeratosis
Excess keratin with mature keratinization; cornified cells lack retained nuclei.
Parakeratosis / parakeratotic hyperkeratosis
Hyperkeratosis with retained nuclei in the stratum corneum / cornified layer.
Acanthosis nigricans / hyperpigmentation
Hyperpigmented, thickened skin often in flexures; not a specific disease but a reaction pattern to certain conditions.
Epitheliogenesis imperfecta
Congenital absence/defect of skin or mucosa leading to ulcerated areas at birth.
Hypertrichosis
Excessive hair growth beyond normal for breed/region.
Ichthyosis
Inherited disorder of cornification causing generalized scaling
Solar dermatosis
UV-induced skin injury causing erythema, scaling, and potential neoplasia.
callus
thickening of skin due to friction
intertrigo
skin fold dermatitis
folliculitis
inflammation of hair follicle
perifolliculitis
inflammation of structures surrounding hair follicle
furunculosis (penetrating folliculitis)
due to rupture of hair follicle
keratosis
excessive formation of keratin
paronychia
inflammation of skin surrounding the nails
seborrhea
increased greasiness, scaling and crusting of skin
spongiosis
intercellular edema
wheal and flare reaction
edema and redness of skin due to hypersensitivity/ allergic reactions