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upper extremity arterial disease occurs much — frequently than lower extremity
causes of upper extremity arterial disease
mechanical obstruction (thoracic outlet syndrome, TOS.)
embolism
trauma
digital artery vasospasm (raynaud’s disease)
digital artery occlusion
brachial artery aka innominate artery
first major branch of the aortic arch
subclavian
the innominate artery divides into right common carotid & — arteries
rarely, right subclavian will originate directly from aorta
may arise distal to left subclavian and cause difficulty swallowing or laryngeal nerve palsy
left subclavian
arises directly from aortic arch as 3rd major branch
vertebral arteries
first major branch from both subclavian arteries
other subclavian artery branches
thyrocervical trunk
costocervical trunk
thoracic outlet
subclavian arteries exist chest through —
areas of potential compression of the subclavian arteries
over first rib between anterior and middle scalene muscles (scalene triangle)
costoclavicular space bound by clavicle and first rib
pectoralis minor space (infrequently involved)
compression of the subclavian arteries may cause
stenosis, aneurysmal dilation, laminar thrombosis, and dynamic compression with arm abduction
axillary artery
originates at the lateral margin of the first rib between anterior
lies deep to pectoralis major and minor muscles
brachial artery
axillary transitions to — at the level of the inferno lateral border of the teres major muscle
courses more superficial in medial arm
recurrent arteries that branch from the brachial artery
deep brachial, radial, and ulnar
they are important collaterals
anatomic variants of brachial artery
high takeoff of radial artery
accessory or duplicated —
origin of ulnar artery in upper arm (less common)
elbow
at — level, brachial artery divides into radial, ulnar, and interosseous arteries
radial artery
continues to wrist deep to flexor muscles
courses along radius
superficial and deep palmer arches
the radial artery branches into the —
ulnar artery
gives rise to interosseous artery
passes deep to forearm flexor muscles
courses toward wrist following ulna
metacarpal and digital
branches from palmar arches (superficial more than deep) give rise to — arteries
superficial palmar arch
the ulnar artery terminates as the —
45-60
using optimal — angle in longitudinal plane when recording PSV
anteroposterior (AP)
document diameter (— and lateral) when aneurysm is present
windows for the subclavian artery
sternal notch
may require lower frequency transducer with smaller footprint
supraclavicular
infraclavicular
followed as it courses under clavicle and over first rib
pectoralis major
axillary artery identified from anterior approach deep to — and minor muscles
teres major muscle
brachial artery begins as artery crosses —
courses more superficial in medial arm between biceps and triceps muscles
physiologic testing
is often included to help identify arterial insufficiency in the limb
qualities
multilevel pressures and/or waveforms provide — assessment of global perfusion
impediments to complete physiologic testing exam
wounds/dressings
intravenous catheters
orthopedic fixation devices
normal upper extremity waveform
normal
sharp systolic peak
brief period of diastolic flow
minimal continues forward flow in diastole
normal PSV in subclavian
varies from 80 to 120 cm/s
normal PSV in brachial, radial, and ulnar arteries
40-60 cm/s
abnormal waveform findings
elevated pSV
poststenotic turbulence
dampened distal waveforms with loss of end systolic flow reversal
waveform changes and brachial blood pressures can help determine stenosis significance
>50% stenosis
general guidelines suggest velocity ratio >2 is consistent with —
aneurysm
permanent localized dilation resulting in 50% increase in diameter of an artery compared to adjacent normal artery
TOS
aneurysms often occur in subclavian artery in association with —
other causes of aneurysms
atherosclerosis and trauma
associated more with axillary, brachial, radial, and ulnar arteries
not common
may present as a pulsatile mass
hypothenar hammer syndrome
arterial degreneration of ulnar artery as it passes deep to hook of hamate bone
associated with repeated use of palm of hand as a hammer
raynaud’s syndrome
intermittent digital ischemia from cold exposure or emotional stimuli
primary raynaud’s syndrome
abnormal digital artery vasospasm
results in pain and color changes (white, blue, red)
color order matters (worst-better)
anatomically, digital arteries appear normal
rarely results in tissue damage
secondary raynaud’s syndrome
underlying disease process responsible for symptoms
patients tend to develop occlusive lesions
associated with tissues necrosis
possible causes of secondary raynaud’s syndrome
autoimmune disorder (scleroderma)
mixed connective tissue disease
systemic lupus erythematosus
rheumatoid arthritis
drug induced vasospasm
cancer
proximal source
duplex ultrasound can be used to help determine if digital occlusive disease is caused secondarily from —
aneurysms
stenosis lesions
fibromuscular disease of forearm arteries
thoracic outlet syndrome
impingement of the neuromuscular bundle at the thoracic outlet
compressions
TOS — may be secondary to cervical ribs, abnormal fibrous bands, or hypertrophy of the scalene muscles
neurogenic
duplex ultrasound may be used to confirm — TOS; however, evidence is not supportive of this conclusion
20%
— of normal individuals can demonstrate subclavian artery compression with provocative maneuvers
younger patients
major arterial thoracic outlet syndrome occurs primarily in —
the subclavian artery
TOS results in compression and damage to —
high
ultrasound has — sensitivity and specificity for locating arterial trauma
proximal
significant atherosclerotic disease in the upper extremity usually involves the — subclavian artery
arterial occlusive disease
occurs most often in left subclavian
often an extension of atherosclerotic involvement of the aortic arch
rarely produces symptoms in upper extremity but may result in subclavian steal syndrome
reversal of flow in ipsilateral vertebral artery
takayasu’s arteritis
autoimmune disorder that affects the arteries of the aortic arch and visceral abdominal aorta
most common in women in their 20s and 30s
results in long segment occlusion or stenosis of affected arteries
acutely, associated with fever, malaise, arthralgias, and myalgias