Advanced med-surg final essay topic questions

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Last updated 12:53 AM on 5/2/26
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15 Terms

1
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Describe how triage is performed during a disaster and how it differs from triage within the emergency department

triage in the ED--> prioritizes patients based on their level of illness/severity and the goal is the help patients with life-threatening conditions first

performed by: rapid assessment and color-coded based on condition (red, yellow, green) (may take several minutes per patient

resources: usually abundant (enough staff/beds etc.)

triage in MCI cases--> prioritizes patients based on the likelihood of survival and the goal is to do the greatest good for the greatest number of people

performed by: assessment of airway/circulation/mental status then color-coded based on findings (seconds to 1 minute per patient only for assessment)

resources: limited resources

2
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Differentiate between Addison's disease and Cushing's syndrome and associated plans

Addison's disease--> (Add the cortisol/salt/sugar)

S/S: low cortisol/aldosterone levels, low BP, hyponatremia/hyperkalemia, weight loss, hypoglycemia

INTERVENTIONS: primary goal is to replace hormones with hydrocortisone, and correct electrolyte imbalances, monitor vitals/BS/fluid status, and prevent addisonian crisis (teach about dosing during stress etc.)

Cushing's--> (cushioned with cortisol/salt/sugar)

S/S: high cortisol levels, round/moon face, HTN, hypernatremia, hypokalemia, weight gain, hyperglycemia

INTERVENTIONS: primary goal is to reduce cortisol levels through treating cause, meds, and/or surgery, and preventing hypertension/ correcting electrolyte imbalances

3
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Discuss the care plan for a patient suffering from DKA and HHS:

Assessment: vitals, neuro status, fluid status, kussmals respirations in DKA patients, blood glucose levels, BUN/creatinine, ABGS and presence of ketones

Interventions: fluid replacement (IV 0.9% NS), regular insulin IV, electrolyte management (treat hypokalemia) and determine underlying triggers/cause

4
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Describe differences for use of a lobectomy versus liver transplants in patients with liver failure:

Lobectomy:

-Surgical removal of a lobe of the liver, usually performed to treat localized liver disease, such as a tumor or severe localized injury

-Does not replace the entire liver—the remaining liver regenerates if healthy.

-for primary or metastatic liver tumors confined to one lobe, severe trauma or abscess localized to a single lobe, and certain cases of hepatic cysts or benign masses

-Requires adequate remaining liver function; not suitable in generalized liver failure (e.g., cirrhosis)

Liver Transplant:

-Surgical replacement of the entire diseased liver with a healthy donor liver

-End-stage liver disease / liver failure

-Completely replaces liver function

-Requires life-long immunosuppressive therapy to prevent rejection

5
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List 3 types of shock and at least one cause for each type

1) hypovolemic shock --> hemorrhage or severe dehydration

2) septic shock --> severe systemic infections (fungal, bacterial, or viral)

3) neurogenic shock--> spinal cord injuries or traumatic brain injury

6
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Discuss primary and secondary brain trauma

primary--> The immediate injury to brain tissue that occurs at the moment of trauma

examples: concussion, contusion, lacerations, and skull fractures

secondary --> Brain injury that occurs after the initial trauma, caused by physiologic and biochemical processes that worsen the primary injury

examples: increased ICP/cerebral edema

7
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What is Frank Starling's Law?

Starling's Law: The stroke volume of the heart increases in response to an increase in the volume of blood filling the heart, when all other factors remain constant

simplified = The more the heart fills with blood, the more it stretches, and the stronger it pumps — up to a point. So, stroke volume (blood pumped per beat) depends on how full the heart is before it contracts

8
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Discuss the components of frank starling's law and how they affect cardiac output

Key Components & Effects on Cardiac Output

Preload (how full the heart is before it beats)

Think: Stretch of the heart fibers like a rubber band.

Effect:

↑ Preload = ↑ stroke volume → ↑ CO

↓ Preload = ↓ stroke volume → ↓ CO

Contractility (how strong the heart can squeeze)

Think: The “muscle power” of the heart.

Effect:

↑ Contractility = heart squeezes harder → ↑ stroke volume → ↑ CO

↓ Contractility = weaker squeeze → ↓ stroke volume → ↓ CO

Afterload (resistance the heart has to pump against)

Think: Like pumping water through a narrow hose.

Effect:

↑ Afterload = heart works harder → stroke volume ↓ → CO ↓

↓ Afterload = easier to pump → stroke volume ↑ → CO ↑

9
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Acute phase of ARDs:

-Damage to alveolar–capillary membrane

-Fluid leaks into alveoli → pulmonary edema

-Decreased surfactant → alveoli collapse (atelectasis)

-Respiratory alkalosis early (from tachypnea)

S/S: tachypnea, crackles, restlessness, use of accessory muscles, hypoxemia, and increased labored breathing

PLAN OF CARE: Improve oxygenation, prevent further lung injury, high-flow oxygen, intubation and mechanical ventilation early if severe, use PEEP to keep alveoli open, and monitor ABGs

10
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Proliferative phase of ARDs:

-lungs begin repairing

-slight improvement in gas exchange

S/S: persistent hypoxemia, decreased lung compliance, hypercapnia, and tachypnea

PLAN OF CARE: Continue lung-protective ventilation,

avoid fluid overload, optimize nutrition, physical therapy, early mobility if stable, monitor for organ dysfunction, and prevention of complications (pressure injuries, DVTs)

11
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Late/fibrotic phase of ARDs:

-Lung tissue becomes scarred and fibrotic

-Decreased lung compliance (very stiff lungs)

-Permanent damage possible

S/S: severe hypoxemia, fatigue, very stiff lungs, worsening C02 retention, and in severe cases, multi-organ failure

PLAN OF CARE: Continue mechanical ventilation, may require high PEEP, monitor for right-sided HF, possible long-term oxygen needs, evaluate for tracheostomy if prolonged ventilation, and palliative support if poor prognosis

12
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Discuss the rationale for early enteral feeding in relation to prevention of GI microbial translocation

Microbial translocation = when bacteria or toxins from the GI tract cross the intestinal barrier and enter the bloodstream → can lead to sepsis, SIRS, and worsening organ failure

Early enteral feeding (providing nutrition through the gut within 24–48 hours) protects the intestinal lining and prevents this from happening

-when patient is NPO, intestinal lining can thin and bacteria can leak out

-enteral feedings stimulate peristalsis, preventing bacterial overgrowth

-maintains normal gut pH

13
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Level 1 Disaster

Small-scale event that can be managed completely by local resources

ex: house fires or minor flooding

-local EMS responds, local fire/police and local hospitals

14
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Level 2 disaster

Moderate disaster. Local resources are overwhelmed and state-level support is needed

ex: wildfires, moderate hurricane damage, flooding affecting multiple towns

-state national guard or state emergency operations may be needed

-state-level Red Cross

15
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Level 3 disaster

Large-scale or catastrophic disasters that overwhelm local and state resources → federal government steps in

ex: major hurricanes like Katrina, large earthquakes, pandemics, terrorism

-U.S public health gets involved, federal agencies such as CDC or national guard may be needed to get involved

-federal emergency management agency

-may even need international aid