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what are some assumptions of emotions
Emotions are all processes that involve the assessment of value- why strong emotions have strong value. Emotions are directed
help us seek pleasure and avoid discomfort
influence our behaviour because it narrows down the range of our behaviours
Emotions are adaptive, so when we have an absence of emotions, it can lead to deficits.
When animals have deficits in emotion-processing areas, they have major impairments in judgement
Emotions can be considered the intervening variable between stimulus and response- it modulates and modifies inputs into outputs
Emotions can be conscious, but aren’t always consciously identified. You can feel sad but dont know why you are sad
Emotions are comprised of subjective, physiological, expressive and behavioural changes. Emotions is not just a feeling, it has changes to our para/sympathetic NS and a chnage in our behaviour
what are some ways we can operationalize emotions
physiological
expressive
subjective
behavioural
cognitive
neural
what is the physiological way of operationalizing emotions
SCR- skin conductance response
ECG- heart rate monitor
and other autonomic measures
why can we use autonomic measures to quantify emotions
because emotions affect the the autonomic n.s
what is the expressive way of operationalizing emotions
EMG
we can code facial expressions and body language from videos
sort images : we can tell ppl to put images showing emotions in separate piles( sometimes we can tell them how many piles or let them freestyle it)
what is the subjective way of operationalizing emotions
self report
brain acitivity ( EEG and fmri)
behaviour( when people are happier they behave differently)
When is self report good and bad
good for happiness
bad when the emotion is considered socially prohibited like anger
what is the common sense view in emotions
Perception of bear leads us to feeling fear and the experience of fear causes physiological response
James Lange view of emotions
Perception of bear drives a change in your physiological response and we interpret these changes as being afraid
so the physiological response drives your psychology
what does the james lange view imply
that each emotion should have its own physiological response
each emotions is discrete and has its own behaviour and physiology
what are some of the evidence that support the Lange’s identity theory of emotion
The polygraph reaction patterns
in this experiment, we would induce fear in participants by shocking them
or induce anger in participants
and the conclusion of the study was that across a wide variety of physiological responses, there would be different physiological patterns for anger vs fear
The nummenma study
This experiment was about feelings and where the feelings are located in the body.
They asked people to imagine where in the body they would feel happy or sad etc.
But in this experiment, you are not inducing emotions, so the participants werent happy or sad
What is the cannon bard view
That the psychology and physiology responses are both driven by sensory processes.
The perception of the fear drives the fear and your physiology independently
what is the evidence that is against the James Lange view and supports minimally the cannon bard view
Better studies on spinal cord damage
If we separate the viscera ( organs of ANS) from the CNS in animals - you are interfering with the pathway from physiology to the sensation of fear
what Sherrington and Cannon saw was that the animals still expressed fear, so you can still induce emotion even with no physiological response
Same changes but different states- how can there be different emotions that have overlapping physiological changes ( this contradicts James Langue theory)
Viscera is relatively insensitive; we are not conscious of what is going in our body
The visceral changes are very slow, so they are too slow at driving psychological changes.
Artificial induction: you can stimulate the viscera and you would expect that to drive an emotional response but it doesn’t
What is the confederate experiment Shacter and singer
There are participants that think they are getting vitamin but it is actually epinephrine. It activates your sympathetic NS.
Now you put each of these participants in a room under different conditions.
What was schacter and singer’s theory on emotion
It is a two factory theory where we are interpreting our physiology ( arousal) using the events around us (the context)
What experiment supports the Two factory theory of emotion
The arousal on capilano suspension bridge
There was 2 conditions the experimental was the cap. bridge and the control was a 3m bridge .
They hired an attractive women as an interviewer to stand on the other side of the bridges
as men walked across the bridge, and she would interview them and gave them her number
They found 3 things
men who walked across the suspension bridge were more likely to call the women back
the stories they told the women was more romantic
the men were more likely to rate the interviewer on the suspension bridge more attractive vs. the men who walked across the control bridge
from the Capilano suspension bridge what can we conclude
Men who were walking across the cap.bridge had high arousal in the NS and when they met someone attractive, the men would interpret their physiology as being strongly attracted to the person
What is the
What is the only universal expression and how did we find this and proof
What is a duchenne vs non-duchenne smile
Duchenne smile is a true smile and it activates the orbicularis oculi muscles around the eyes and the zygomatic major muscle in our jaw
Non-duchenne smile is a fake smile and it only activates the zygomatic major muscle in our jaw
what are the 3 aspects of affect
Temperament
mood
emotion
What is temperament
it has been present since birth and it is a predisposition of how reactive you are
easy babies- have easy temperament, they eat and sleep better, cry less and are less emotionally reactive
difficult babies are the opposite
so easy babies as an adult are less emtionally reactive, introverted
and difficult babies as an adult are more reactive, extroverted
Mood vs emotion
Mood:
Low intensity and long lasting
it influences our day
not directed
emotions influence how we perceive ( bad mood shapes your overall experience- bad mood = negative perspective)
Emotion:
High intensity and very brief(flashes of anger)
directed
emotions influence how we act( we get angry and leave classroom)
Theories of sleep
-Circadian
-energy
-body restoration
-brain development
-adjust brain circuitry
-brain detox
Types of aggression
Predatory aggression
Affective aggression
What is predatory aggression? Where is it seen? What are the changes to the autonomic N.S?
In this type of aggression:
Animals are quiet
There is no emotional component
Animals are more likely to inflict harm and be violent
It is seen when one species kills another species for food, the attacks are usually aiming to kill
Very little change in the parasympathetic and sympathetic changes
How do we induce predatory aggression and what do we see
Stimulating
Lateral hypothalamus
MFB- median forebrain bundle
VTA
We are not inducing anger here, we are inducing motivated behaviours to kill for food
What is MFB again?
Bundle of axons near the hypothalamus that project from the VTA, so they are axons of dopamine neurons.
What is affective aggression? The point of it and the changes in our autonomic NS
In this type of aggression:
Animals/humans are very loud, animals will start hissing ( related to sham rage)
It is emotional
Animals/humans will not commit much violence or inflict harm
The point is to display a threat, not inflict harm
Very high levels of sympathetic nervous system activity
How do we induce affective aggression
Stimulate the
Ventromedial/Medial hypothalamus
Periaqueductal gray- also involved in defensive behaviours
What is the story between serotonin and aggression
It has been seen that when serotonin levels are VERY low, you are more likely to be aggressive
But the correlation between serotonin and aggression is very weak, so their relationship isnt linear, but it suggests that you need a minimum amount of serotonin and when you are below it, there is likely more aggressive behaviours
What is the evidence that shows serotonin levels relates to aggressive behaviours
If you do selective damage(lesion) to serotonin neurons using 5,7-DHT in rodents, the rats will become very aggressive. Rats are very calm.
If you use drugs that block serotonin synthesis, animals will become very aggressive
If you measure aggression levels in monkey colonies, the animals with the most aggression have the lowest serotonin levels
What does active stress mean
When things are done to you- someone pinches you, beats you, abuses you etc.
what does passive stress mean
When you are deprived of things you need for your well being-
maternal negligence, separation, deprivation of food or environmental stimuli
What are controllable stressors
A stressor an animal can influence or control
-when something bad is happening to you, you can do something about it
What are uncontrollable stressors
A stressor you have no control over
ex. Poverty or SES
what is stress immunization and learned helplessness
These are both stress responses that are determined by the type of stress you have been exposed to.
Stress immunization: is when you are more resilient to stress because you are exposed to controllable stressors
Learned helplessness: is when we are less resilient to stress
What happens when we are exposed to controllable stress
When we are exposed to controllable stress, we develop stress immunization and exposure to these stressors help us become more resilient to future stress, even if the future stressors are NOT CONTROLLABLE
What happens when we are exposed to uncontrollable stress
exposure to these stressors make us less resilient to stress and future stress even when the future stressors ARE CONTROLLABLE
How did we figure out stress immuniz. and learned helplessness
what are the 2 pathways of stress
Sympathetic response
and HPA axis
what is the sympathetic response to stress
It is our fast pathway because our neurons are targetting the adrenal medulla of our adrenal glands.
So our sympathetic neurons activate the adrenal medulla, which will release epinephrine and norepinephrine as hormones. This will get our heart racing and etc.
what is the HPA axis
This is our slower response because the signal travels in our bloodstream( endocrine pathway).
The hypothalamus has neuro-endocrine cells which look like neurons but release hormones. These cells release releasing hormones to the anterior pituitary gland which will release tropic hormones, These hormones travel via the bloodstream and target thr adrenal cortex of the adrenal glands.
The adrenal cortex releases cortisol and this is a longer lasting response
is the HPA axis always on
Yes it is part of our circadian rhytym but cortisol release is elevated under stress conditions
what does it mean when we say stress is an adaptive response to threat
The point of getting stressed out is to succesfully deal with a stressor and move on. Our brain thrives in acute stress, but once we are exposed to stress for a awhile and we are in a constant stressful state, it can have adverse affects, our brains weren’t meant for chronic stress
Why is acute stress beneficial
Mild levels of stress are beneficial bc it makes us perform better in aspects of reaction time, cognition, memory and accuracy
what are the costs and benefits of High stress levels
Benefits:
implicit memory/ skills
simple tasks, we are even better at them under high pressure
anything habitual and that is well-rehearsed
suppresses our immune system temporarily, so you will not be symptomatic.
Costs:
cognitive flexibility
working memory
executive function
Bilateral vmPFC damage: executive dysfunction
It is bilateral damage bc the ventromedial PFC is along the medial line
symptoms:
Most intellectual ability is preserved
Problems with prioritization
Emotional dysregulation ( start to see behaviours in inappropriate ways)
eg. acquired sociopathy in some dementia
Repetition of mistakes despite knowing that the mistake was suboptimal, and they will report they know the optimal strategy but refuse to do it
Loss of Motivation
Problems thinking ahead and sequencing steps for a task
Very poor cognitive flexibility, they are rigid in their thought and actions
Problems with attention and concentration
What is acquired sociopathy
caused by neurodegeneration in PFC but it is a change in personality
what happens for unilateral vmPFC damage to the right side
Same executive dysfunction as seen in bilateral damage
what happens for unilateral vmPFC damage to the left side
The deficits are subtle not as severe as damage to the right side
People have stable employment and relationships
Their personality is relatively unchanged
Their decision making ability is intact
Not alot of executive dysregulation