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spinal shock
initial transient physiological depression below level of injury resulting in loss of sensory, autonomic, reflexes
clinical presentation that emerges as spinal shock resolves
hyperreflexia
neurogenic shock
hypotensive state due to loss of sympathetic outflow
result of neurogenic shock
vasodilation and decreased peripheral vascular resistance, hypotension, bradycardia, hypothermia, decreased CO
what can PTs do for shock
education, monitor pt, re-evaluate motor and sensory changes often
result of paralysis of phrenic nerve
impaired inspiratory/expiratory function, reduced vital capacity, reduced lung complicance, quicker fatigue
major cause of morbidity and mortality in SCI
respiratory compromise
PT interventions for respiratory compromise
assisted cough, ROM of chest wall, postural education, early and often mobilization, percussion
cardiovascular complications post SCI
loss of outflow and vascular tone, reduced ability to regulate BP, limited muscle contraction for venous return
common cardiovascular complications
OH and autonomic dysreflexia
what to do for OH
slow progression to upright position, ensure hydration, take medications, tilt table, ace wraps
autonomic dysreflexia physiology
noxious stimulus causes sympathetic response of vasoconstriction and hypertension
presentation of autonomic dysreflexia
increased BP and decreased HR (opposite of OH), headache, dizziness, goosebumps
who is at risk for autonomic dysreflexia
T6 and above
most common causes of autonomic dysreflexia
bowel and bladder
what to do for autonomic dysreflexia
sit pt up and take BP, find source of trigger
other cardiovascular complications that could occur in acute SCI
DVT and pulmonary embolus
why are pts with SCI at increased risk for pressure injuries
impaired sensation, immobilization, decreased bulk around bony prominences, shearing during transfers
what can PT do for pressure injuries
education, early and often mobilization, skin checks, pressure relief program
parts of pressure relief program
turning schedule and seated pressure reliefs
thermoregulation impairments
initial concern for hypothermia then hyperthermia following shock
bone complications
osteoporosis, increased risk of fracture, heterotropic ossification
cervical spinal precautions
no bending, rotation
spinal precautions
no bending, lifting, twisting, hip flexion >90
types of early equipment
adaptive equipment, abdominal binder, tilt table, positioning device
steps of neurological exam
review PMH → ask about goals, support → assess sensory, motor, functional mobility
points of education for pt and family
many changes, basic interventions, skin concerns, early and often mobility, rehab process
function of tenodesis
using wrist extension to flex fingers to create a functional grasp
useful ROM in LE
hip abduction and external rotation for bowel management and dressing, tightness in low back for trunk control
components of functional mobility assessment
need for equipment, need for assistance, transfers, bed mobility
common goals for SCI
focus on mobility, independence, prevention of secondary complications
risk factors of lower functional outcomes following SCI
other injuries, age, pain level, limited ROM, obesity, impaired cognition, psychological, other medical complications