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HA EXAM 2
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Ventilation
Moving air in and out of the lungs (breathing)
Diffusion (gas exchange)
O2 moves from alveoli into blood; CO2 moves from blood into alveoli
Perfusion
Blood flow that delivers O2 to the tissues
Big idea of oxygenation
Ventilation moves air, diffusion swaps gases, perfusion delivers blood. A problem in any one = low O2
Hypoxemia
Low O2 in the BLOOD (PaO2 < 80 mmHg, SpO2 < 95%)
Hypoxia
Low O2 at the TISSUES (result of hypoxemia or poor perfusion)
Hypercapnia (hypercarbia)
Too much CO2 in the blood (PaCO2 > 45)
Dyspnea
Feeling short of breath (subjective; what the client says)
Orthopnea
Short of breath lying flat; ask "How many pillows do you sleep on?"
Paroxysmal nocturnal dyspnea (PND)
Waking up at night gasping for air; think heart failure
Tachypnea
Adult RR > 20
Bradypnea
Adult RR < 12
Apnea
No breathing
Cyanosis
Bluish color; a LATE sign of hypoxia
Clubbing
Rounded fingertips, nail angle 180° or more; CHRONIC low O2 (COPD, CF, heart defects)
Atelectasis
Collapsed alveoli; common after surgery and with immobility
Surfactant
Substance that keeps alveoli from collapsing
Compliance
How easily the lungs stretch
Hemoptysis
Coughing up blood (think TB, cancer, PE)
Barrel chest
AP diameter = lateral diameter (1:1); seen in COPD/emphysema; EXPECTED finding in COPD
Normal adult chest shape
AP to lateral ratio about 1:2 (wider than deep)
Infant chest shape
Round chest, 1:1 ratio is NORMAL for infants
SpO2 vs SaO2
SpO2 = pulse oximeter reading; SaO2 = saturation measured in the ABG
Path of air (in order)
Nose/mouth → pharynx → larynx → trachea → carina → mainstem bronchi → bronchioles → alveoli
Job of the nose
Warms, filters, and humidifies air
Epiglottis
Flap that covers the airway so food doesn't go into the lungs
Carina
Where the trachea splits into right and left bronchi; about the level of the sternal angle (T4)
Alveoli
Tiny air sacs where gas exchange actually happens
Right lung lobes
3 lobes (upper, middle, lower)
Left lung lobes
2 lobes (makes room for the heart)
Right mainstem bronchus
Shorter, wider, straighter; aspirated objects and a too-deep ET tube usually go RIGHT
Pleura
Two thin layers (visceral on lung, parietal on chest wall) with fluid between so they glide
Pleural effusion
Fluid buildup in the pleural space
Pneumothorax
Air in the pleural space; lung collapses
Diaphragm
Main breathing muscle; contracts and moves DOWN so air rushes in
Inspiration vs expiration
Inspiration is ACTIVE; expiration is normally PASSIVE
Accessory muscles
Sternocleidomastoid, trapezius, intercostals; using them = respiratory distress
What controls breathing
Brainstem (medula and pons)
Normal drive to breathe
Rising CO2 level
Hypoxic drive (COPD)
Some chronic CO2 retainers breathe in response to low O2; give O2 carefully, titrate to SpO2 88–92%
Sternal angle (angle of Louis)
Bump on the sternum at the 2nd rib; used to count ribs and intercostal spaces
Apex of lungs
3–4 cm above the clavicles
Base of lungs
About 6th rib in front, 10th rib (T10) in back
Infants and young kids (respiratory)
Small narrow airways, nose breathers, belly breathers, higher RR; swelling or mucus blocks airways fast
Nasal flaring and retractions in kids
Signs of respiratory distress
Pregnancy and breathing
Uterus pushes diaphragm up; mild SOB late in pregnancy can be normal
Older adults (respiratory changes)
Weaker muscles and cough, stiffer chest, less elastic lungs, fewer cilia, kyphosis; higher risk of pneumonia, aspiration, atelectasis
Older adult first sign of hypoxia or infection
Confusion (may NOT have a fever)
Number one changeable lung risk
Smoking / vaping
Pack-years formula
Packs per day × years smoked (2 packs/day × 10 years = 20 pack-years)
Obesity and breathing
Limits diaphragm and chest expansion; linked to sleep apnea
Immobility and lungs
Secretions pool → atelectasis and pneumonia
Opioids, alcohol, sedatives
Slow the RR (respiratory depression); alcohol also raises aspiration risk
Anxiety and breathing
Causes fast breathing (hyperventilation)
Occupational lung exposures
Coal dust, asbestos, silica, chemicals, farm dust; always ask about jobs
High altitude
Less O2 in air; body makes more RBCs over time
Carbon monoxide and pulse ox
Pulse ox reads FALSELY NORMAL; need ABG with CO-oximetry; teach CO detectors
Radon
2nd leading cause of lung cancer; teach home radon testing
Crowded living spaces
Increase spread of TB and flu
Client in respiratory distress (interview)
Treat FIRST; ask short yes/no questions; finish history after stable
OLDCARTS
Onset, Location, Duration, Characteristics, Aggravating, Relieving, Treatment tried, Severity
Clear or white sputum
Normal or viral
Yellow or green sputum
Infection
Rust-colored sputum
Pneumococcal pneumonia
Pink frothy sputum
Pulmonary edema; EMERGENCY
Pleuritic chest pain
Chest pain that gets worse when breathing in
Night sweats + weight loss + cough
Think TB
ACE inhibitors (respiratory side effect)
Dry cough
Beta blockers (respiratory risk)
Can trigger bronchospasm in asthma
Order of physical assessment (lungs)
Inspect → palpate → percuss → auscultate
EARLIEST sign of low O2
Change in mental status (restlessness, anxiety, confusion) plus rising HR and RR
Normal adult RR
12–20 breaths/min
Normal SpO2
95–100% (88–92% may be the goal for some COPD clients)
Tripod position
Leaning forward on arms; sign of respiratory distress (also helps COPD breathe)
Speaking in full sentences
Good sign; can't speak full sentences = distress
Symmetric chest expansion
Thumbs at T10, client breathes in, thumbs should move apart equally
Crepitus (subcutaneous emphysema)
Crackling feeling under the skin = air leaking into tissue
Tactile fremitus
Vibration felt when client says "99"
Increased tactile fremitus
Consolidation (pneumonia); solid conducts better
Decreased tactile fremitus
Air or fluid (pneumothorax, effusion, emphysema)
Resonant percussion
Normal lung
Dull percussion
Fluid or solid (pneumonia, effusion)
Hyperresonant percussion
Too much air (emphysema, pneumothorax)
Normal capillary refill
Less than 3 seconds
Pulse ox: causes of bad readings
Cold fingers, poor circulation, movement, dark nail polish, acrylic nails; try earlobe or forehead
What pulse ox does NOT show
CO2; a client can have good SpO2 and still retain CO2
How to auscultate lungs
Diaphragm on bare skin, client sits up and breathes deep through mouth, side to side, top to bottom, one full breath per spot
Vesicular breath sounds
Soft, low, over most lung fields; inspiration longer than expiration
Bronchovesicular breath sounds
Medium; between scapulae and next to sternum (1st–2nd ICS); inspiration = expiration
Bronchial breath sounds
Loud, high, hollow over trachea; ABNORMAL if heard over lung fields (consolidation)
Fine crackles (rales)
Like hair rolled between fingers or Velcro, late inspiration; fluid in alveoli (pneumonia, HF, atelectasis); do NOT clear with cough
Coarse crackles
Louder, bubbly, gurgling; mucus in larger airways
Wheezes (sibilant)
High-pitched musical whistle, usually on expiration; narrowed airways (asthma, COPD, bronchospasm)
Rhonchi (sonorous wheeze)
Low-pitched snoring; thick secretions in large airways; often CLEARS with coughing
Stridor
Loud high-pitched crowing on INSPIRATION, heard without stethoscope; upper airway blockage; EMERGENCY
Causes of stridor
Croup, epiglottitis, laryngeal swelling, after extubation, anaphylaxis, foreign body
Pleural friction rub
Grating, leather rubbing; inflamed pleura (pleurisy, pneumonia, PE); painful; STOPS when client holds breath
Pleural rub vs pericardial rub
Pleural rub stops when holding breath; pericardial rub keeps going
Diminished or absent breath sounds
Pneumothorax, effusion, atelectasis, severe COPD, airway blockage
Memory trick for lung sounds
Wheeze = narrow airway; Crackles = fluid; Rhonchi = mucus (cough it out); Stridor = upper airway, call for help