Week 10 Peds GI

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53 Terms

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Pediatric differences

  • frequent feedings

  • frequent bowel movements

  • faster peristalsis

  • breast fed stools and frequency

  • formula fed stools and frequency


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How to assess for dehydration in children?

  • assess skin turgor

  • fontanels

  • lack of tearing

  • dry mucus membranes

  • thirst

  • skin temperature

  • activity level


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S/S GI disorders in infants/children

  • vomting/regurgitation

  • irritability/fussiness

  • abdominal pain/distention

  • Failure to thrive

  • weight lost

  • stool changes


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Subjective assessment Peds GI

  • family hx

  • diet

  • gaining/losing weight

  • through hx feeding pattern

  • allergies (lactose intolerant, celiac dx)

  • spitting up

  • elimination pattern

  • I/Os

  • encorpresis/constipation


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Objective assessment Peds GI

  • inspect distention, symmetry, bulges or masses, umbilicus

  • auscultate hyper/hypo bowel sounds

  • percuss tympany vs dullness

  • palpate light vs deep, rebound tenderness, McBurney’s point


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Disorders of the GI system include

  • structural defects

  • disorders of motility

  • inflammatory disorders

  • disorders of malabsorption

  • hepatic disorders

  • injuries to the GI system


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Structural defects

  • cleft lip and cleft palate

  • esophageal atresia and tracheoesophageal fistula

  • pyloric stenosis

  • intussusception

  • abdominal wall defects

  • anorectal malformations

  • umbilical hernia


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Cleft lip and cleft palate is a

congenital anomaly that occurs as a result of failure of soft tissue or bony structure to fuse during weeks 6-12 gestation

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Characteristics Cleft lip and Cleft palate

  • may appear by itself or together

  • cleft lip can be unilateral or bilateral

  • most common craniofacial deformities in the US

  • multifactorial causes


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Complications with CL/CP

  • feeding problems (ability to suck, swallow, and breath without distress)

  • speech development

  • otologic

  • dental and orthodontic

  • developmental delays


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Cleft lip management

  • special feeding techniques until surgery

  • Haberman feeder

  • ESSR method


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What is the Haberman feeder technique used for infant with cleft lip?

  • hold infant in upright position

  • stimulate sucking by rubbing nipple to allow child to get food into back of throat without strong sucking

  • allow time to swallow and burp frequently


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What is the ESSR method for cleft lip?

  • enlarge nipple

  • stimulate sucking

  • swallow rest


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Cleft lip repair

  • surgery usually done at 1-3 months

  • z shape suture line


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Post op cleft lip repair

  • logan bar over child’s mouth to reduce tension on suture line

  • clean sutures, use saline to prevent crusting, antibiotic ointment on suture line

  • elbow restraints (release every 2 hours)

  • breck feeder technique

  • position on back or side lying


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Cleft palate is repaired in stages between

6 months and 2 years of age

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Repairing cleft palate protects against

  • altered dentition and speech dysfunction

  • frequent ear infection (otitis media) d/t open nasopharynx

  • need to be able to use cup to drink


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Cleft palate repair post op

  • maintain airway, O2 sat monitor

  • no bottles, straws, pacifiers, spoons for at least 7-10 days

  • elbow restraints and mittens

  • clears to soft diet

  • all feedings followed by rinsing mouth with water to clean suture line

  • pain med

  • no brushing teeth 1-2 weeks


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Esophageal Atresia (EA) & Tracheoesophageal Fistula (TEF)

  • associated with maternal polyhydramnios

  • may occur alone or together (usually tgt)


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Esophageal Atresia (EA) is a

congenital disruption of upper and lower portions of esophagus

surgical emergency

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Tracheoesophageal Fistula (TEF) is a

abnormal communication b/t trachea and esophagus

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S/S Esophageal Atresia (EA) and Tracheoesophageal Fistula (TEF)

immediate newborn period

  • respiratory distress

  • difficulty feeding

  • excessive drooling

  • choking, coughing

  • cyanosis


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Pre-op EA and TEF

  • maintain airway

  • NG tube to suction

  • prevent aspiration (HOB increase, NPO)

  • IV fluids, IV abx

  • G tube may be placed to drain stomach content/allow air to escape

  • surgical repair single or staged surgery depending on age, size, length of gap


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Post-op EA and TEF

  • initally NPO

  • TPN

  • g tube feeds

  • feeds start with clears then advance over several days

  • complications: tears, stricture, reflux, infection

  • family teaching


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Pyloric stenosis characteristics

  • etiology unknown but often affects first born males

  • present at birth

  • food causes irritation, progressive edema and further narrowing


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Pylroic stenosis is diagnosed by

ultrasound

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Pyloric stenosis occurs

between stomach and duodenum

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Pyloric stenosis is

hypertrophy of the circular muscles of the pylorus hat causes narrowing of the pyloric canal between the stomach and duodenum

<p>hypertrophy of the circular muscles of the pylorus hat causes narrowing of the pyloric canal between the stomach and duodenum</p>
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Physical assessment of pyloric stenosis

  • vomiting progresses from mild regurgitation to projectile vomiting

  • vomiting occurs shortly after meals

  • vomiting is forceful (2-4feet)

  • constant hunger, fussiness, crying

  • decrease in size and number of stools

  • failure to gain or lose weight

  • dehydration & electrolyte imbalance

  • olive shaped mass sometimes palpable on abdominal exam


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How is pyloric stenosis treated?

surgery= pyloromyotomy

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Pre-op Pyloric stenosis surgery

  • NPO

  • IVFs for fluids/electrolyte management

  • NG tube


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Post-op Pyloric stenosis surgery

  • maintain IVF, analgesics, NG tube

  • start feeds


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Gastroschisis & Omphalocele are

Abdominal wall defects

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Gastroschisis & Omphalocele

  • a congenital defect of the ventral abdominal wall, characterized by herniation of abdominal visceral outside the abdominal wall


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gastroschisis occurs

to the side of umbilicus

<p>to the side of umbilicus</p>
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omphalocele occurs

through the umbilical cord

<p>through the umbilical cord</p>
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Nursing management for Gastroschisis & Omphalocele

  • prevent infection by covering sac with sterile gauze soaked in NS, then sterile plastic

  • monitor VS closely; assess temperature

  • NPO, maintain IVFs, TPN

  • parental support


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Anal stenosis & Anal atresia are

Anorectal malformations

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Anal stenosis is

thickened & constricted anal wall (produce ribbon-like stools

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Anal atresia (imperforated anus)

  • failure to pass meconium

  • exam reveals absent anal opening


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What would you see in anal stenosis & anal atresia?

stool in urine or vagina due to fistulas

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Treatment for Anorectal malformations depends on

where it is located and if it is low/high defect

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Treatment for low defect Anorectal malformations

anoplasty and serial digital dilations are effective

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Treatment for high defect anorectal malformations

2 step procedure that may involve a temporary colostomy

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Umbilical hernia is

hernia protrusion of an organ or part of an organ through the muscle wall of the cavity that normally contains it

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Umbilical hernia results from

imperfect closure of the umbilical muscle ring and often associated with separation of the abdominal muscles

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Clinical manifestations Umbilical Hernia

  • herniated umbilicus protrudes with coughing, crying, or straining

  • hernia can be reduced by pushing contents back into fibrous ring

  • most defects spontaneously resolve by 3-4 years old, as muscular ring closes

  • surgery indicated in cases of strangulation, increased protrusion after 2 years of age or no improvement in large defect after 4 years


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