cardiac rehab exam 1 study guide

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Last updated 7:34 PM on 10/12/26
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106 Terms

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Cardiovascular Disease

Host of diseases that affect the heart and/or blood vessels.

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Forms of Cardiovascular Disease

Coronary artery disease (CAD), cerebral vascular disease (stroke), peripheral artery disease (PAD), heart failure, hypertension.

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<p><strong>Coronary Arteries</strong></p>

Coronary Arteries

Supply oxygen to the heart muscle.

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Coronary Artery Disease (CAD)

Plaque develops in the coronary arteries and creates blockages, leading to a heart attack.

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Cerebral Vascular Disease (stroke)

Significant plaque blockages restrict blood flow to the brain, leading to a stroke.

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Peripheral Artery Disease (PAD)

Plaque blockages in the legs blood vessels leading to discomfort. Pain goes away when walking begins.

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Heart Failure

Heart muscle is in a weakened state and is unable to pump enough blood to meet body’s needs for blood + oxygen.

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Hypertension

Where the force of blood against the artery walls is consistently too high, leading to heart disease or stroke.

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Atherosclerosis

The accumulation of fat and fibrous tissue in medium sized blood vessels.

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Atherosclerosis begins with…

Repeated/prolonged exposure to smoking, high LDL, high BP, obesity, diabetes, and sedentary lifestyle.

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% of blockage → beginning to feel symptoms

75%

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Myocardial Oxygen Consumption

Amount of oxygen that is consumed/used by the heart muscle.

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Myocardial Oxygen Demand

Amount of oxygen that is required/needed by the heart muscle.

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Myocardial Oxygen Supply

Amount of oxygen that is being delivered to the heart muscle.

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Physiological determinants of myocardial oxygen demand and myocardial oxygen supply

MVO2 demand = MVO2 supply

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Ischemia

An inadequate delivery of blood and oxygen to the heart muscle. MVO2 demand > MVO2 supply

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Angina

Discomfort associated with ischemia. Includes lightheadedness, left arm pain, chest pain, neck, jaw, upper back (women), upper abdominal pain.

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Myocardial Infarction

Heart attack; occurs when blood flow to a part of the heart is blocked due to blood clot in coronary artery. Prolonged period of ischemia of at least 30-60 minutes results in the death of heart muscle.

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Research based benefits of cardiac rehab:

25% reduction in mortality, being able to return to work/leisure activities sooner, and higher exercise capacity.

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Phase 1: Inpatient Cardiac Rehab

Events: Myocardial infarction, PCI, CABG, chronic heart failure, heart transplant.

Goals: Offset effects of bedrest, evaluate ability to return to daily activities, prepare for discharge/entry to outpatient CR.

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Phase 2-3: Early Outpatient CR

3 weeks after discharge

Goals: detect changes in disease status, return to recreational activities, implement safe/ effective program, educate on risk factors.

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Phase 4: Maintenance Cardiac Rehab

Supervised but not monitored, self-reliant on equipment, in program indefinitely.

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Functions of the cardiovascular system:

Transport blood through arteries, veins and capillaries to deliver oxygen and nutrients to every cell while removing CO2 and waste products.

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4 Chambers of the Heart

Right atrium, right ventricle, left atrium, left ventricle.

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Sequence of blood flow through the heart:

Vena cava → Right Atrium → Right Ventricle → Lungs → Left Atrium → Left Ventricle → Aorta

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Function of heart valves:

Ensure one-way blood flow through the heart and prevent backflow.

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Impulse pathway

SA Node → AV node → AV bundle → bundle branches → Purkinje fibers

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Autonomic Nervous System and HR

ANS acts on the SA node.

SNS → accelerates HR

PNS → slows HR

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Endocrine system and HR

Sympathetic → releases norepinephrine

Parasympathetic → releases acetylcholine

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Diastole

Phase where the heart muscle relaxes and allows the chambers to fill with blood.

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Systole

Phase where the heart muscle contracts and pumps blood from the chambers into the arteries.

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Stroke Volume

The volume of blood pumped from the ventricles per beat.

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Ejection Fraction

How much blood the left ventricle pumps out with each contraction.

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Cardiac Output

The volume of blood pumped by each ventricle of the heart over a given period of time (measured in liters/min).

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<p><strong>Left Main Coronary Artery</strong></p>

Left Main Coronary Artery

Sends blood to the left side of the heart; including the left ventricle + left atrium.

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<p><strong>Left Anterior Descending Artery</strong></p>

Left Anterior Descending Artery

Branches off the left coronary artery; sends blood to the front left side of heart + septum.

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<p><strong>Circumflex Artery</strong></p>

Circumflex Artery

Branches off left coronary artery and circles the heart; sends blood to the outer + back side of the heart.

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<p><strong>Right Main Coronary Artery</strong></p>

Right Main Coronary Artery

Sends blood to the right ventricle, right atrium, SA node, and AV node.

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<p><strong>Right Acute Marginal Artery</strong></p>

Right Acute Marginal Artery

Supplies blood to the right ventricle.

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<p><strong>Posterior Descending Artery</strong></p>

Posterior Descending Artery

Supplies the ventricles and interventricular septum.

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HR + Acute Exercise

Increased HR, increased cardiac output, increased SBP.

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Stroke Volume + Acute Exercise

Increased stroke volume, venous return.

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Cardiac Output + Acute Exercise

Increased cardiac output (volume of blood increases).

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Blood Pressure + Acute Exercise

Increases systolic blood pressure while diastolic blood pressure remains stable.

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<p><strong>P wave</strong></p>

P wave

Activation/depolarization of the atrium.

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<p><strong>PR interval</strong></p>

PR interval

Reflects conduction through the AV node.

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<p><strong>QRS duration</strong></p>

QRS duration

Activation/depolarization of the two ventricles.

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<p><strong>ST segment</strong></p>

ST segment

Interval between ventricular depolarization and repolarization.

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<p><strong>T wave</strong></p>

T wave

Follows QRS; represents the relaxation/repolarization of the ventricles.

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<p><strong>QT interval</strong></p>

QT interval

Represents time taken for ventricular depolarization and repolarization.

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<p><strong>Normal Sinus Rhythm</strong></p>

Normal Sinus Rhythm

60-100 bpm.

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Sinus Bradycardia

Less than 60 bpm.

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Sinus Trachycardia

More than 100 bpm.

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<p><strong>Sinus Arrythmia</strong></p>

Sinus Arrythmia

1:1 ratio of P waves to QRS complexes, variation in heart rate (distances in QRS complexes). Based on ANS changes with beating.

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<p><strong>PAC (premature atrial complexes)</strong></p>

PAC (premature atrial complexes)

Early beat, P wave may appear different, normal width QRS complex.

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<p><strong>PVC (premature ventricular complexes)</strong></p><p></p>

PVC (premature ventricular complexes)


Early beat, premature beat not preceded by P wave, large, upside-down QRS complex.

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<p><strong>Ventricular Bigeminy</strong></p><p></p>

Ventricular Bigeminy


PVC every other beat.

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<p><strong>Ventricular trigeminy</strong></p><p></p>

Ventricular trigeminy


PVC every 3rd beat.

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<p><strong>Ventricular Couplet</strong></p>

Ventricular Couplet

2 PVC every row.

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<p><strong>Ventricular Triplet</strong></p><p></p>

Ventricular Triplet


3 PVC every row.

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<p><strong>Unifocal PVCs</strong></p><p></p>

Unifocal PVCs


Multiple PVCs coming from the same ecotpic focus, identical.

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<p>Multifocal PVCs</p><p></p>

Multifocal PVCs


Coming from more than 1 ectopic focus, not identical.

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<p><strong>Sinus Tachycardia in ECG</strong></p><p></p>

Sinus Tachycardia in ECG


Gradual onset, normal P waves, normal QRS.

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<p><strong>Supraventricular Tachycardia</strong></p><p></p>

Supraventricular Tachycardia


Sudden onset, HR 150-250, P waves abnormal/buried in T wave, normal width QRS.

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<p><strong>Atrial Flutter</strong></p><p></p>

Atrial Flutter


“Saw-tooth” pattern, flutter waves override P waves and T waves, normal width QRS complex.

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<p><strong>Atrial Fibrillation</strong></p><p></p>

Atrial Fibrillation


Irregular rhythm, no organized P waves, normal QRS.

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Implications for atrial fibrillation:

Drop in cardiac output/symptoms, higher risk of stroke, medications.

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<p><strong>Ventricular Tachycardia</strong></p><p></p>

Ventricular Tachycardia


>3 consecutive ventricular beats, HR: 100-200, wide QRS, dissociation between P waves and QRS complexes. (serious + urgent)


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<p><strong>Ventricular Fibrillation</strong></p><p></p>

Ventricular Fibrillation


No organized ventricular activity, lethal rhythm.

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<p><strong>Components of a pacemaker:</strong></p><p></p>

Components of a pacemaker:


Pulse generator and placing wires (leads).

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Pacemakers treat:

Severe bradycardia, SA block (idioventricular), AV block (2nd + 3rd degree).

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<p><strong>Pacemakers changes on ECG:</strong></p><p></p>

Pacemakers changes on ECG:


Sharp vertical spikes, wide QRS complexes, abnormal/flat P wave, T waves in opposite direction.

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SA Node Blocks

Junctional rhythm and Idioventricular rhythm.

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<p><strong>Junctional Rhythm</strong></p><p></p>

Junctional Rhythm


40-60 bpm, impulses blocked + unable to leave SA node → inverted P waves, normal width QRS.

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<p><strong>Idioventricular Rhythm</strong></p><p></p>

Idioventricular Rhythm


15-40 bpm, impulses coming from ventricles + can’t travel ro SA + AV node due to blockage → no P waves, wide QRS.

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AV node blocks

1st degree, 2nd degree (mobitz 1 + 2), 3rd degree.

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<p><strong>1st Degree AV Block</strong></p><p></p>

1st Degree AV Block


Delay in conduction, fixed prolonged PR interval > 0.20 sec.

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<p><strong>2nd degree AV block - Mobitz 1 (wenckebach)</strong></p><p></p>

2nd degree AV block - Mobitz 1 (wenckebach)


Intermittent blocked conduction — progressive lengthening of PR interval → dropped QRS complex.

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<p><strong>2nd Degree AV Block - Mobitz 2</strong></p><p></p>

2nd Degree AV Block - Mobitz 2


Intermittent blocked conduction — fixed PR interval with occasional dropped QRS complex.

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<p><strong>3rd Degree AV Block</strong></p><p></p>

3rd Degree AV Block


Complete blocked conduction — atria and ventricles are beating independently of one another.

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<p><strong>Bundle Branch Blocks</strong></p><p></p>

Bundle Branch Blocks


One of the bundles is blocked, sequential depolarization, wide QRS.

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<p><strong>Bundle Branch Blocks (right) leads:</strong></p><p></p>

Bundle Branch Blocks (right) leads:


V1-V2.

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<p><strong>Bundle Branch Blocks (left) leads:</strong></p><p></p>

Bundle Branch Blocks (left) leads:


V5-V6.

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Primary Prevention of Heart Disease:

Quit smoking, heart-healthy diet, regular physical activity, weight control, manage cholesterol + BP.

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Secondary Prevention of Heart Disease:

Prevents recurring cardiovascular events → cardiac rehab, anti-platelet therapy, BP control, ACE inhibitors/beta-blockers.

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Smoking

Positive cardiovascular risk factor if individual is a current smoker or has quit within the past 6 months.

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Smoking – Pharmacological Interventions

Nicotine replacement therapy, varenicline, bupropion.

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Blood Pressure Risk for CVD

Hypertension >130/80 mmHg.

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AHA/ACC lifestyle recommendations for lowering BP:

Quit smoking/drinking, limit caffeine, manage diabetes, weight, cholesterol, diet, become physically active.

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LDL Cholesterol + Atherosclerosis

Primary driver → initiates plaque formation.

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HDL cholesterol + Atherosclerosis

Linked to lower atherosclerosis.

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Blood Glucose as a risk factor:

>250 mg/dL

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Type 1 Diabetes

Autoimmune condition where the body stops making insulin.

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Type 2 Diabetes

Autoimmune condition caused by insulin resistance and the body not using insulin effectively.

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ACSM criteria for physical inactivity

Not participating in at least 30 mins of moderate-intensity physical activity on at least 3 days per week for at least 3 months.

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MET-min weekly

1 MET = 3.5 x weight (kg) / 200

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Recommendation for physical inactivity (min/week) for general population:

150 min → moderate aerobic activity

75 min → vigorous aerobic activity

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Recommendation for physical activity (min/week) for heart disease patients:

240-360 min of moderate activty a week.

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ACSM criteria for obesity - BMI

BMI greater than 30 kg/m2.

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ACSM criteria for obesity - waist girth

Greater than 38 inches in women, 40 inches in men.